1,002,419 research outputs found
[Supplementary Offense Report by G. R. Rose and R. S. Stovall #1]
Supplementary offense report by G. R. Rose and R. S. Stovall. The report states that Buell Wesley Frazier drove Lee Harvey Oswald to work on the morning of President Kennedy's assassination. His sister, Mrs. Randle, saw Oswald put a long package into the back of the vehicle. Both individuals were taken in for questioning
Corrigendum to “Presence and function of kisspeptin/KISS1R system in swine ovarian follicles” (Theriogenology (2018) 115 (1–8), (S0093691X1830147X), (10.1016/j.theriogenology.2018.04.006))
The authors regret the following changes to the author group G. Basinia, F. Grassellia, S. Bussolatia, R. Ciccimarraa, M. Maranesib, A. Bufalarib, C. Dall'Agliob, F. Parilloc,#, M. Zeranib,c,*. a Dipartimento di Scienze Mediche Veterinarie, Università di Parma, 43126 Parma, Italy. b Dipartimento di Medicina Veterinaria, Università di Perugia, 06126 Perugia Italy. c Scuola di Bioscienze e Medicina Veterinaria, Università di Camerino, 62024 Matelica Italy. # Deceased. * Corresponding author: tel.: +39 0755857642; fax +39 0755857654. E-mail address: [email protected] (M. Zerani). And to the acknowledgements and figures
[Supplementary Offense Report by G. R. Rose and R. S. Stovall #2]
Handwritten copy of a supplementary offense report by G. R. Rose and R. S. Stovall. The report states that Buell Wesley Frazier drove Lee Harvey Oswald to work on the morning of President Kennedy's assassination. His sister, Mrs. Randle, saw Oswald put a long package into the back of the vehicle. Both individuals were taken in for questioning
Estetica
Organo dell'A.I.S.E. (Associazione Italiana degli Studiosi di Estetica). Comitato scientifico internazionale che vaglia gli articoli proposti, composto da: C. Angelino, O. Breidbach, M. Cacciari, M. Donà, F. Duque, G. Figal, M. Frank, A. Giannatiempo Quinzio, D. Harth, G. Marchianò, R. Milani, J.-L. Nancy, A, Pagnini, M. Pezzella, F. Rella, M. Saison, G. Vattimo, F. Volpi, M. Vozza, S. Zecchi
Holloway, F S G, NX70896
This record was harvested from a previous catalogue system and will be withdrawn in 2025. Information in this record may be superseded or incomplete. Visit this record in UMA's new catalogue at: https://archives.library.unimelb.edu.au/nodes/view/392953Surname: HOLLOWAY. Given Name(s) or Initials: F S G. Military Service Number or Last Known Location: NX70896. Missing, Wounded and Prisoner of War Enquiry Card Index Number: 35940.212224
Item: [2016.0049.25246] "Holloway, F S G, NX70896
Chidgey, F S G, 423061
This record was harvested from a previous catalogue system and will be withdrawn in 2025. Information in this record may be superseded or incomplete. Visit this record in UMA's new catalogue at: https://archives.library.unimelb.edu.au/nodes/view/376928Surname: CHIDGEY
Given Name(s) or Initials: F S G
Military Service Number or Last Known Location: 423061
Missing, Wounded and Prisoner of War Enquiry Card Index Number: 52764190687
Item: [2016.0049.09233] "Chidgey, F S G, 423061
Holm Oak (Quercus ilex L.) decline in Apulia, Southern Italy, a phytopathological overview.
Lucchese P.G., Chiaromonte E., Salamone D., Tarasco E., Pollastro S., Faretra F., Nigro F., 2025 – Holm Oak (Quercus ilex L.) decline in Apulia, Southern Italy, a phytopathological overview. 17th International Congress of Mediterranean Phytopathological Union (MPU). CIHEAM Bari, Italy, July 6-10 202
Evidence for the decay B0→J/ψω and measurement of the relative branching fractions of meson decays to J/ψη and J/ψη′
First evidence of the B 0 → J / ψ ω decay is found and the B s 0 → J / ψ η and B s 0 → J / ψ η ′ decays are studied using a dataset corresponding to an integrated luminosity of 1.0 fb -1 collected by the LHCb experiment in proton-proton collisions at a centre-of-mass energy of sqrt(s) = 7 TeV. The branching fractions of these decays are measured relative to that of the B 0 → J / ψ ρ 0 decay:frac(B (B 0 → J / ψ ω), B (B 0 → J / ψ ρ 0)) = 0.89 ± 0.19 (stat) - 0.13 + 0.07 (syst),frac(B (B s 0 → J / ψ η), B (B 0 → J / ψ ρ 0)) = 14.0 ± 1.2 (stat) - 1.5 + 1.1 (syst) - 1.0 + 1.1 (frac(f d, f s)),frac(B (B s 0 → J / ψ η ′), B (B 0 → J / ψ ρ 0)) = 12.7 ± 1.1 (stat) - 1.3 + 0.5 (syst) - 0.9 + 1.0 (frac(f d, f s)), where the last uncertainty is due to the knowledge of f d / f s, the ratio of b-quark hadronization factors that accounts for the different production rate of B 0 and B s 0 mesons. The ratio of the branching fractions of B s 0 → J / ψ η ′ and B s 0 → J / ψ η decays is measured to befrac(B (B s 0 → J / ψ η ′), B (B s 0 → J / ψ η)) = 0.90 ± 0.09 (stat) - 0.02 + 0.06 (syst)
Antioxidant ascorbic acid modulates nlrp3 inflammasome in lps-g treated oral stem cells through nfκb/caspase-1/il-1β pathway
Human gingival mesenchymal stem cells (hGMSCs) and endothelial committed hGMSCs (e-hGMSCs) have considerable potential to serve as an in vitro model to replicate the inflammation sustained by Porphyromonas gingivalis in periodontal and cardiovascular diseases. The present study aimed to investigate the effect of ascorbic acid (AA) on the inflammatory reverting action of lipopolysaccharide (LPS-G) on the cell metabolic activity, inflammation pathway and reactive oxygen species (ROS) generation in hGMSCs and e-hGMSCs. Cells were treated with LPS-G (5 μg mL−1 ) or AA (50 μg mL−1 ) and analyzed by 3-(4,5-Dimethylthiazol-2-yl)-2,5-Diphenyltetrazolium Bromide (MTT) assay, immunofluorescence and Western blot methods. The rate of cell metabolic activity was decreased significantly in LPS-G-treated groups, while groups co-treated with LPS-G and AA showed a logarithmic cell metabolic activity rate similar to untreated cells. AA treatment attenuated the inflammatory effect of LPS-G by reducing the expression of TLR4/MyD88/NFκB/NLRP3/Caspase-1/IL-1β, as demonstrated by Western blot analysis and immunofluorescence acquisition. LPS-G-induced cells displayed an increase in ROS production, while AA co-treated cells showed a protective effect. In summary, our work suggests that AA attenuated LPS-G-mediated inflammation and ROS generation in hGMSCs and e-hGMSCs via suppressing the NFκB/Caspase-1/IL-1β pathway. These findings indicate that AA may be considered as a potential factor involved in the modulation of the inflammatory pathway triggered by LPS-G in an vitro cellular model
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