319,908 research outputs found
Xinyuan-Liang/SC-shapes-the-maturation-of-cortical-morphology: v1.0.0
No full text<p>Relevant data for the article "Liang X, Sun L, Liao X, Lei T, Xia M, Duan D, Zeng Z, Xu Z, Men W, Wang Y, Tan S, Gao J, Qin S, Tao S, Dong Q, Zhao T, He Y, Structural connectome architecture shapes the maturation of cortical morphology from childhood to adolescence. bioRxiv, 2022, https://www.biorxiv.org/content/10.1101/2022.12.15.520527v2"
Contents include all source code and intermediate data during the main analysis. Demo data for plotting figures can be found in the "fig" folder. See the README file for details.</p>
Measurement of the ratio of prompt χ c to J / ψ production in pp collisions at √s = 7 TeV
Full text linkThe prompt production of charmonium χ c and J / ψ states is studied in proton-proton collisions at a centre-of-mass energy of √s = 7 TeV at the Large Hadron Collider. The χ c and J / ψ mesons are identified through their decays χ c → J / ψ γ and J / ψ → μ + μ - using 36 pb - 1 of data collected by the LHCb detector in 2010. The ratio of the prompt production cross-sections for χ c and J / ψ, σ (χ c → J / ψ γ) / σ (J / ψ), is determined as a function of the J / ψ transverse momentum in the range 2 < p T J / ψ < 15 GeV / c. The results are in excellent agreement with next-to-leading order non-relativistic expectations and show a significant discrepancy compared with the colour singlet model prediction at leading order, especially in the low p T J / ψ region
Onycholabis pedulangulus Liang and Imura 2003
No full textOnycholabis pedulangulus Liang and Imura, 2003:688 (China, Vietnam, Laos). Specimens Examined. Total 83 specimens; for 73 types, refer to Liang and Imura (2003); additional specimens: CHINA: Yunnan Province, 5 males and 5 females (IOZ, CAS), ‘‘ China, Yunnan Province, Tengchong, Wuhe Town, Longjiangqiao, riverside, 24853956.00N, 98840936.10E’’/‘‘ 1,215 m, 2003.10.28, Liang H. B., Shi X. C. coll. Institute of Zool., CAS and California Acad. Sciences’ ’. Description. See Liang and Imura (2003). Distribution. Known from Laos, Vietnam, and China. Remarks. Adults of this species have been found on river banks and on the walls of roadside drainage ditches.Published as part of Liang, Hongbin & Kavanaugh, David H., 2005, A Review of Genus Onycholabis Bates (Coleoptera: Carabidae: Platynini), with Description of a New Species from Western Yunnan, China, pp. 507-520 in The Coleopterists Bulletin 59 (4) on page 512, DOI: 10.1649/837.
Mellin-Transform-Based Performance Analysis of FFH -ary FSK Using Product Combining for Combatting Partial-Band Noise Jamming
No full text
Capacity and Error Performance of Reduced-Rank Transmitter Multiuser Preprocessing Based on Minimum Power Distortionless Response
No full textIn this contribution we first derive a transmitter multiuser preprocessing (TMP) scheme for a general multiuser multiple-input-multiple-output (MIMO) system based on the minimum power distortionless response (MPDR) criterion, which minimizes the power in the context of a given downlink mobile terminal (MT) under the distortionless condition. This optimization problem results in a solution, which has the same form as the minimum mean-square error (MMSE) multiuser detection (MUD). Hence, we then extend the well-known rank-reduction techniques in MMSE-MUD to the MPDR-TMP in order to mitigate the possible implementation problems of the MPDRTMP. In our study three classes of rank-reduction algorithms are considered, which are derived, respectively, based on the eigen-analysis methods of principal components (PC) and crossspectral metrics (CSMs) as well as on the Taylor polynomial approximation (TPA) approach, which does not depend on the eigen-analysis. In this contribution both the capacity and error performance of a downlink space-division multiple-access (SDMA) system is investigated, when either the full-rank or reduced-rank MPDR-TMP is invoked. From our study and simulation results, it can be shown that the MPDR-TMP scheme is highly efficient for achieving the capacity and for suppressing the multiuser interference (MUI). The reduced-rank techniques can be employed by the MPDR-TMP, so as to reduce its implementation complexity while achieving the near full-rank performance of the full-rank MPDR-TMP
Letter from Carl T. Hayden to C. H. Gensler, Havasupai Reservation
No full textLetter from Carl T. Hayden to C. H. Gensler, Havasupai Indian Reservation, regarding Hualapai and Cataract Canyons geography
Ahli kelab T-Rex UMP tawan puncak Gunung Liang
Full text linkRIADAH. Seramai 30 ahli Kelab Rekreasi (T-Rex) Universiti Malaysia Pahang (UMP) berjaya menawan puncak Gunung Liang yang terletak di Behrang, Perak baru-baru in
Altered Ca2+ handling and oxidative stress underlie mitochondrial damage and skeletal muscle dysfunction in aging and disease
No full textSkeletal muscle contraction relies on both high-fidelity calcium (Ca2+) signals and robust capacity for adenosine triphosphate (ATP) generation. Ca2+ release units (CRUs) are highly organized junctions between the terminal cisternae of the sarcoplasmic reticulum (SR) and the transverse tubule (T-tubule). CRUs provide the structural framework for rapid elevations in myoplasmic Ca2+ during excitation–contraction (EC) coupling, the process whereby depolarization of the T-tubule membrane triggers SR Ca2+ release through ryanodine receptor-1 (RyR1) channels. Under conditions of local or global depletion of SR Ca2+ stores, store-operated Ca2+ entry (SOCE) provides an additional source of Ca2+ that originates from the extracellular space. In addition to Ca2+, skeletal muscle also requires ATP to both produce force and to replenish SR Ca2+ stores. Mitochondria are the principal intracellular organelles responsible for ATP production via aerobic respiration. This review provides a broad overview of the literature supporting a role for impaired Ca2+ handling, dysfunctional Ca2+-dependent production of reactive oxygen/nitrogen species (ROS/RNS), and structural/functional alterations in CRUs and mitochondria in the loss of muscle mass, reduction in muscle contractility, and increase in muscle damage in sarcopenia and a wide range of muscle disorders including muscular dystrophy, rhabdomyolysis, central core disease, and disuse atrophy. Understanding the impact of these processes on normal muscle function will provide important insights into potential therapeutic targets designed to prevent or reverse muscle dysfunction during aging and disease
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