59,571 research outputs found

    B. J. Gourley

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    "VX 66896 WOII BJ Gourley [A]rrived 18 Mile Camp 9 April 1942 [D]eparted Adelaide R 13 April 1943 Aust Fld Survey Sec AIF".VX 66896 Warrant Officer Class II B. J. Gourley. [A]rrived 18 Mile Camp, 9 April 1942. [D]eparted Adelaide River, 13 April 1943. Australian Field Survey Section, Australian Imperial Forces.Date:199

    Histories, Parker-Phillips Camp

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    The Daughters of Utah Pioneers, Phillips Camp biographies (circa 1940-1974) is a collection of biographical sketches of Utah pioneers submitted to the Phillips Camp, Daughters of Utah Pioneers, in Kaysville, Utah. The individual sketches give insight into the socioeconomic status of European, as well New World, converts to the Church of Jesus Christ of Latter-day Saints during the nineteenth century. They contain biographical and genealogical information, as well as descriptions of experiences crossing the Atlantic to America and traveling across the plains to Utah. Minute details of pioneering life in Davis County, Utah, and other frontier outposts of settlement are illuminated. Described also are individual occupations and survival techniques along with information on offices held in, and services to, the church and the community. Biographies include: George Blake Parker (1830-1920), 3 pages; Mary Lewis Parker (1825-1891), 3 pages; William Parker (1800-1822), 2 pages; Catharine Nichols Payne (1824)-1906), 5 pages; William L. Payne (1816-1892), 5 pages; Edward Phillips (1813-1896?), 10 pages; Hannah Simmonds Phillips (1824-1898), 9 pages; John Dee Phillips (1846-1887), 1 page; Mary Ann Press Dee Phillips (1773-1871), 3 pages; History of Phillips Camp, 2 page

    Evidence for the decay B0→J/ψω and measurement of the relative branching fractions of meson decays to J/ψη and J/ψη′

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    First evidence of the B 0 → J / ψ ω decay is found and the B s 0 → J / ψ η and B s 0 → J / ψ η ′ decays are studied using a dataset corresponding to an integrated luminosity of 1.0 fb -1 collected by the LHCb experiment in proton-proton collisions at a centre-of-mass energy of sqrt(s) = 7 TeV. The branching fractions of these decays are measured relative to that of the B 0 → J / ψ ρ 0 decay:frac(B (B 0 → J / ψ ω), B (B 0 → J / ψ ρ 0)) = 0.89 ± 0.19 (stat) - 0.13 + 0.07 (syst),frac(B (B s 0 → J / ψ η), B (B 0 → J / ψ ρ 0)) = 14.0 ± 1.2 (stat) - 1.5 + 1.1 (syst) - 1.0 + 1.1 (frac(f d, f s)),frac(B (B s 0 → J / ψ η ′), B (B 0 → J / ψ ρ 0)) = 12.7 ± 1.1 (stat) - 1.3 + 0.5 (syst) - 0.9 + 1.0 (frac(f d, f s)), where the last uncertainty is due to the knowledge of f d / f s, the ratio of b-quark hadronization factors that accounts for the different production rate of B 0 and B s 0 mesons. The ratio of the branching fractions of B s 0 → J / ψ η ′ and B s 0 → J / ψ η decays is measured to befrac(B (B s 0 → J / ψ η ′), B (B s 0 → J / ψ η)) = 0.90 ± 0.09 (stat) - 0.02 + 0.06 (syst)

    Letter from J. B. Caldwell to J. J. Brown

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    Letter from J. B. Caldwell to J. J. Brown, concerning donations to NFA camp fund

    Letter from J. B. Caldwell to T. J. Culler

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    Letter from J. B. Caldwell to T. J. Culler, concerning NFA camp fund donations

    Letter from J. B. Baird to S. B. Simmons

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    Letter from J. B. Baird to S. B. Simmons, concerning camp attendance of NFA members

    Letter from J. B. Caldwell to E. B. Coleman

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    Letter from J. B. Caldwell to E. B. Coleman, thanking him for camp fund donation

    Isoform-selective susceptibility of DISC1/phosphodiesterase-4 complexes to dissociation by elevated intracellular cAMP levels

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    Disrupted-in-schizophrenia 1 (DISC1) is a genetic susceptibility factor for schizophrenia and related severe psychiatric conditions. DISC1 is a multifunctional scaffold protein that is able to interact with several proteins, including the independently identified schizophrenia risk factor phosphodiesterase-4B (PDE4B). Here we report that the 100 kDa full-length DISC1 isoform (fl-DISC1) can bind members of each of the four gene, cAMP-specific PDE4 family. Elevation of intracellular cAMP levels, so as to activate protein kinase A, caused the release of PDE4D3 and PDE4C2 isoforms from fl-DISC1 while not affecting binding of PDE4B1 and PDE4A5 isoforms. Using a peptide array strategy, we show that PDE4D3 binds fl-DISC1 through two regions found in common with PDE4B isoforms, the interaction of which is supplemented because of the presence of additional PDE4B-specific binding sites. We propose that the additional binding sites found in PDE4B1 underpin its resistance to release during cAMP elevation. We identify, for the first time, a functional distinction between the 100 kDa long DISC1 isoform and the short 71 kDa isoform. Thus, changes in the expression pattern of DISC1 and PDE4 isoforms offers a means to reprogram their interaction and to determine whether the PDE4 sequestered by DISC1 is released after cAMP elevation. The PDE4B-specific binding sites encompass point mutations in mouse Disc1 that confer phenotypes related to schizophrenia and depression and that affect binding to PDE4B. Thus, genetic variation in DISC1 and PDE4 that influence either isoform expression or docking site functioning may directly affect psychopathology

    Observations of Bºs→ψ(2S)η and Bº(s)→ψ(2S)π+π- decays

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    First observations of the B0s →ψ(2S)η, B0 →ψ(2S)π + π − and B0s →ψ(2S)π + π − decays are made using a dataset corresponding to an integrated luminosity of 1.0 fb−1 collected by the LHCb experiment in proton–proton collisions at a centre-of-mass energy of √ s = 7 TeV. The ratios of the branching fractions of each of the ψ(2S) modes with respect to the corresponding J/ψ decays are B(B0s →ψ(2S)η) ÷ B(B0s →J/ψη) = 0.83± 0.14 (stat)±0.12 (syst) ±0.02 (B), ; B(B0→ψ(2S)π + π − ) ÷ B(B0→J/ψπ + π − ) = 0.56± 0.07 (stat)±0.05 (syst)± 0.01 (B), ; B(B0s →ψ(2S)π + π − ) ÷ B(B0s →J/ψπ + π − ) = 0.34± 0.04 (stat)±0.03 (syst)± 0.01 (B), where the third uncertainty corresponds to the uncertainties of the dilepton branching fractions of the J/ψ and ψ(2S) meson decays
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