924 research outputs found
Gravitational radiation from string cosmology
No full textA spectrum of relic stochastic gravitational radiation, strongly tilted towards high frequencies, and characterized by two basic parameters is shown to emerge in a class of string theory models. We estimate the required sensitivity for detection of the predicted gravitational radiation and show that a region of our parameter space is within reach for some of the plannedgravitational-wave detectors
Observation of a fully reconstructed pair with long lifetimes in a high resolution hydrogen bubble chamber and the European Hybrid Spectrometer
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J / psi production in the hadronic decays of the Z
No full textJ/ψ mesons have been reconstructed from their decay to μ+μ- and e+e-, using the data collected by the DELPHI experiment during 1991 and 1992 at the LEP collider. From about 1 million hadronic Z decays 153 ± 17 J/ψ were found, 5.4 ± 2.3 ψ′ were obtained in the channel J/ψ (→μ+μ-)π+π- and 6.4 ± 2.7 χc in the channel J/ψ ( → μ+μ-)γ. As the dominant source of Jψ mesons is from bquarks, the following branching ratios: Br(b → J/ψ X) = (1.12 ± 0.12 (stat) ± 0.10 (syst.))%, Br(b → ψ′ X) = (0.48 ± 0.22 (stat.± 0.10 (syst.))%, Br(b → χc1 X) = (1.4 ± 0.6 (stat.)-0.2+0.4 (syst.))% were measured. From the proper time distribution of the J/ψ sample, the average lifetime of b-hadrons decaying into J/ψ was found to be: τB = 1.50-0.21+0.24 (stat.) ± 0.03 (syst.) ps. A search for completely reconstructed B meson decays to final states including a J/ψ gave a signal of 15 ± 5 events.0info:eu-repo/semantics/publishe
CDC42 switches IRSp53 from inhibition of actin growth to elongation by clustering of VASP
No full textFilopodia explore the environment, sensing soluble and mechanical cues during directional motility and tissue morphogenesis. How filopodia are initiated and spatially restricted to specific sites on the plasma membrane is still unclear. Here, we show that the membrane deforming and curvature sensing IRSp53 (Insulin Receptor Substrate of 53 kDa) protein slows down actin filament barbed end growth. This inhibition is relieved by CDC42 and counteracted by VASP, which also binds to IRSp53. The VASP: IRSp53 interaction is regulated by activated CDC42 and promotes high-density clustering of VASP, which is required for processive actin filament elongation. The interaction also mediates VASP recruitment to liposomes. In cells, IRSp53 and VASP accumulate at discrete foci at the leading edge, where filopodia are initiated. Genetic removal of IRSp53 impairs the formation of VASP foci, filopodia and chemotactic motility, while IRSp53 null mice display defective wound healing. Thus, IRSp53 dampens barbed end growth. CDC42 activation inhibits this activity and promotes IRSp53-dependent recruitment and clustering of VASP to drive actin assembly. These events result in spatial restriction of VASP filament elongation for initiation of filopodia during cell migration, invasion, and tissue repair
Study of W boson polarisations and measurement of TGCs in the reaction at energies between 189 and 208 GeV
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