2,007 research outputs found
Ein Prototyp eines Simulationssystem zur Lageroptimierung
No full textAuthor / eingereicht von Nikola Radic, BScMasterarbeit Universität Linz 2023Arbeit gesperr
Action of the mobius group ¨ M = hx, y : x 2 = y 6 = 1i on certain real quadratic fields
No full textLet C 0 = C ∪ {∞} be the extended complex plane and M = ­ x, y : x 2 = y 6 = 1® , where x(z) = −1 3z and y(z) = −1 3(z+1) are the linear fractional transformations from C 0 → C 0 . Let m be a squarefree positive integer. Then Q∗ ( √ n) = { a+ √ n c : a, c 6= 0, b = a 2−n c ∈ Z and (a, b, c) = 1} where n = k 2m, is a proper subset of Q( √ m) for all k ∈ N. For non-square n = 3h Qr i=1 p ki i , it was proved in an earlier paper by the same authors that the set Q 000 ( √ n) = { α t : α ∈ Q∗ ( √ n), t = 1, 3} is M-set ∀ h ≥ 0 whereas if h = 0 or 1, then Q∗∗∗√ n) = { a+ √ n c : a+ √ n c ∈ Q∗ ( √ n) and 3 | c} is an M-subset of Q 000 ( √ n) = Q∗ ( √ n) ∪ Q∗∗∗( √ 9n). In this paper we prove that if h ≥ 2, then Q 000 ( √ n) = (Q∗ ( pn 9 )\Q∗∗∗( pn 9 ))∪Q∗ ( √ n)∪Q∗∗∗( √ 9n) and also determine its proper M-subsets. In particular Q( √ m) \ Q = ∪Q 000 ( √ k 2m) for all k ∈ N
Essays in Honour of Thérèse Radic on her Eightieth Birthday: Special Issue
No full textThérèse Radic is widely recognized as Australia's foremost expert on Australian music. She is the author/editor of nine books, sixteen plays and some 200 articles, monographs, chapters and reviews on aspects of Australian music history. Her PhD of 1978 is still, after nearly forty years, the first port of call for anyone starting out to research Australian music history. Her generosity in sharing her extraordinary knowledge of Australian music history with other scholars, students and interested members of the general public is legendary. She has always been a feisty advocate for Australian music and for women in music, promoting these areas however, wherever and whenever possible. This volume celebrates her exceptional contribution to Australian music history, and the admiration and respect that she has inspired in generations of scholars. In 2013 Thérèse Radic was awarded the Don & Joan Squire Award for Voluntary Services to Musicology in Australia by the Musicological Society of Australia
Person
No full textThis record was harvested from a previous catalogue system and will be withdrawn in 2025. Information in this record may be superseded or incomplete. Visit this record in UMA's new catalogue at: https://archives.library.unimelb.edu.au/nodes/view/60032Therese Radic is a musicologist and author and was appointed an Australia Council committee member. The Council was established in 1975 by the Commonwealth Government as a statutory authority to formulate and carry out policies aimed at raising the standards of the arts in Australian, to encourage more Australians to become involved and inform oversease of Australian arts and culture
Unimpaired phase-sensitive amplification by vector four-wave mixing near the zero-dispersion frequency
No full textPhase-sensitive amplification (PSA), which is produced by degenerate four-wave mixing (FWM) in a randomly-birefringent fiber, has the potential to improve the performance of optical communication systems. Scalar FWM, which is driven by parallel pumps, is impaired by the generation of pump-pump and pump-signal harmonics, which limit the level, and modify the phase sensitivity, of the signal gain. In contrast, vector FWM, which is driven by perpendicular pumps, is not impaired by the generation of harmonics. Vector FWM produces PSA with the classical properties of a one-mode squeezing transformation. © 2007 Optical Society of America
Competition and risk-taking in investment banking
No full textHow does competition affect the investment banking business and the risks individual institutions are exposed to? Using a large sample of investment banks operating in seven developed economies over 1997-2014, we apply a panel VAR model to examine the relationships between competition and risk without assuming any a priori restrictions. Our main finding is that investment banks’ higher risk exposure, measured as a long-term capital-at-risk and return volatility, was facilitated by greater competitive pressures especially for full service investment banks but also for boutique investment banks. Overall, we find some evidence that more competition leads to more fragility before and during the recent financial crisis
Privatization: Implications Of A Shift From State To Private Ownership
No full textPrivatization—defined here as the transfer of ownership of state-owned organizations to private parties—has attracted the attention of scholars across multiple fields. Privatization programs have been based on the assumption, grounded in microeconomic theory, that a shift from public to private ownership will incentivize more efficient management of available resources. However, failure to deliver the expected outcomes in some cases and the more nuanced perspective on state-ownership offered by recent research in management seem to challenge this assumption, calling for revisiting this literature. Our comparative review of existing studies suggests that the mixed results of privatization programs could be partly explained by what was privatized, how it was privatized, and the regulatory regime under which it was privatized. By doing so, our review provides conceptual clarity and structure to a rich but fragmented body of literature, making seemingly divergent findings more legible, outlining theoretical gaps, and identifying avenues for future exploration
Environmental toxicity, redox signaling and lung inflammation:the role of glutathione
No full textGlutathione (gamma-glutamyl-cysteinyl-glycine, GSH) is the most abundant intracellular antioxidant thiol and is central to redox defense during oxidative stress. GSH metabolism is tightly regulated and has been implicated in redox signaling and also in protection against environmental oxidant-mediated injury. Changes in the ratio of the reduced and disulfide form (GSH/GSSG) can affect signaling pathways that participate in a broad array of physiological responses from cell proliferation, autophagy and apoptosis to gene expression that involve H(2)O(2) as a second messenger. Oxidative stress due to oxidant/antioxidant imbalance and also due to environmental oxidants is an important component during inflammation and respiratory diseases such as chronic obstructive pulmonary disease, idiopathic pulmonary fibrosis, acute respiratory distress syndrome, and asthma. It is known to activate multiple stress kinase pathways and redox-sensitive transcription factors such as Nrf2, NF-kappaB and AP-1, which differentially regulate the genes for pro-inflammatory cytokines as well as the protective antioxidant genes. Understanding the regulatory mechanisms for the induction of antioxidants, such as GSH, versus pro-inflammatory mediators at sites of oxidant-directed injuries may allow for the development of novel therapies which will allow pharmacological manipulation of GSH synthesis during inflammation and oxidative injury. This article features the current knowledge about the role of GSH in redox signaling, GSH biosynthesis and particularly the regulation of transcription factor Nrf2 by GSH and downstream signaling during oxidative stress and inflammation in various pulmonary diseases. We also discussed the current therapeutic clinical trials using GSH and other thiol compounds, such as N-acetyl-l-cysteine, fudosteine, carbocysteine, erdosteine in environment-induced airways disease
NETosis 2: The Excitement Continues
No full textNETosis, a form of cell death that manifests by the release of decondensed chromatin to the extracellular space, provides valuable insights into mechanisms and consequences of cellular demise. Because extracellular chromatin can immobilize microbes, the extended nucleohistone network was called a neutrophil extracellular trap (NET), and the process of chromatin release was proposed to serve an innate immune defense function. Extracellular chromatin NETs were initially observed in studies of neutrophils and are most prominent in these types of granulocytes. Subsequent studies showed that other granulocytes and, in a limited way, other cells of the innate immune response may also release nuclear chromatin following certain kinds of stimulation. Variations of NETosis were noted with cells that remain temporarily motile after the release of chromatin. Numerous stimuli for NETosis were discovered, including bacterial breakdown products, inflammatory stimuli, particulate matter, certain crystals, immune complexes and activated thrombocytes. Fundamental explorations into the mechanisms of NETosis observed that neutrophil enzyme activity (PAD4, neutrophil elastase, proteinase 3 and myeloperoxidase) and signal transduction pathways contribute to the regulation of NETosis. Histones in NET chromatin become modified by peptidylarginine deiminase 4 (PAD4) and cleaved at specific sites by proteases, leading to extensive chromatin externalization. In addition, NETs serve for attachment of bactericidal enzymes including myeloperoxidase, leukocyte proteases, and the cathelicidin LL-37. NETs are decorated with proteases and may thus contribute to tissue destruction. However, the attachment of these enzymes to NET-associated supramolecular structures restricts systemic spread of the proteolytic activity. While the benefit of NETs in an infection appears obvious, NETs also participate as key protagonists in various pathologic states. Therefore, it is essential for NETs to be efficiently cleared; otherwise digestive enzymes may gain access to tissues where inflammation takes place. Persistent NET exposure at sites of inflammation may lead to a further complication: NET antigens may provoke acquired immune responses and, over time, could initiate autoimmune reactions, serve as antigen for nuclear autoantibodies and foster DNA immune complex-related inflammation. Neutrophil products and deiminated proteins comprise an important group of autoantigens in musculoskeletal disorders. Aberrant NET synthesis and/or clearance are often associated with inflammatory and autoimmune conditions. Recent evidence also implicates aberrant NET formation in the development of endothelial damage, atherosclerosis and thrombosis. Intravital microscopy provides evidence for conditions that induce NETosis in vivo. Furthermore, NETs can easily be detected in synovial fluid and tissue sections of patients with arthritis and gout. NETosis is thus of interest to researchers who investigate innate immune responses, host-pathogen interactions, chronic inflammatory disorders, cell and vascular biology, biochemistry, and autoimmunity. As we enter the second decade of research on NETosis, it is useful and timely to review the mechanisms and pathways of NET formation, their role in bacterial and fungal defense and their importance as inducers of autoimmune responses
Shannon Capacity Evaluation for 5G Communications Using the 3D Random Waypoint Mobility Model
No full textThis paper presents a mathematical analysis for estimating the Shannon capacity of a fifth-generation (5G) communication link established between a mobile user (MU) and a cellular base station. The analysis is carried out in a three-dimensional (3D) environment, adopting the random waypoint mobility model to statistically describe the displacement of the MUs, and including path-loss attenuation, directional antenna gains, and mid-scale fading. Closed-form expressions for the received signal power and for the channel capacity are derived for both line-of-sight (LoS) and non-LoS scenarios, assuming a noise-limited operating regime. The obtained analytical results, which are numerically validated by Monte Carlo simulations, are exploited to investigate the impact of the antenna gain and of the cell radius on the performance of the 5G link in a 3D scenario, considering both the 28 and 73 GHz millimeter-wave channels
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