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Male-Centred Norms and Intimate Partner Femicides: A Case Study in German Homicide Law by David Linkerhaegner
Get PDFThis paper examines male-centred norms and femicide from a feminist criminology perspective, conducting a case study of the German criminal law\u27s response to intimate partner femicide. Focusing on the legal framework of homicide in the German Criminal Code (StGB), it critiques the doctrinal understanding of the statutory wording ‘despicable reasons’ in the context of femicide as perpetuating gendered biases rooted in male-centred norms. By analysing case law and feminist criminological scholarship, the paper highlights how the case law’s concept of ‘ordinary psychological motives’ fails to adequately address gendered violence. It calls for a different perception of ‘the ordinary’ to better reflect the gendered dynamics of intimate partner femicide on a case-by-case approach
Book review: Data Protection, Privacy and Artificial Intelligence: To Govern or to Be Governed, That Is the Question
Get PDFWilliam George Black, “Neomagus,” and Folk-Medicine (1876)
Get PDFThis article examines William George Black’s coinage of the term ‘folk medicine’, under the pseudonym ‘Neomagus’, in the Journal of the British Archaeological Association of 1878. This was prior to the publication of his seminar Folk Medicine in 1882, almost forty years after Thom’s coinage of the term ‘folk lore’. As Secretary of South Scotland for the Folk-Lore Society of London, Black – who published his seminal Folk Medicine in 1882 – played a key role in the development of the discipline. This article explores the wide-ranging influences, and probable German origins of this central term, of his ground-breaking work in defining folk medicine
Contributors\u27 Biographies
No full textEthnology, Folklore, Scottish culture, Scottish history, Scotland, Narratology, Material Culture, Archaeology, Fieldwork, Diaspora, local studies, regional studie
The Lost Paradise: The Role of the Witness in Shaping the Historiography of Jerusalem
Get PDFJerusalem is not merely occupied; it is actively rewritten. This piece examines how Palestinian witness testimony defies the gradual violence of erasure, transforming the city from a site of dispossession into an assertion of historical continuity. Drawing from decolonial theory, phenomenology, and political philosophy, I argue that testimony is not merely an act of remembrance but an epistemic and ontological intervention that challenges both material oppression and the state’s monopolization of historical truth. I interrogate the material conditions of occupation, positioning them within a broader philosophy of spatial domination: the destruction of Palestinian homes, the calculated deprivation of infrastructure, and the juridical apparatus that renders Palestinian existence precarious. Drawing on Nur-eldeen Masalha’s concept of memoricide, I examine how settler-colonial power operates not just through territorial expansion but through the systematic erasure and reconstruction of historical narratives, turning Jerusalem into a battleground of meaning as much as land. Testimony emerges here not as passive recollection but as an act of existential resistance. The act of witnessing asserts both the primacy of lived experience and the refusal of epistemic erasure, positioning Palestinian memory as a challenge to the structures that seek to render it illegible. Judith Butler’s work on ‘grievability’ is mobilized to interrogate how international frameworks of recognition operate to exclude Palestinian suffering from the category of the politically visible. Further on, I situates testimony within digital landscapes, analyzing the El-Kurd family’s plight in Sheikh Jarrah as an extension of what James C. Scott terms ‘hidden transcripts,’ a counter-history that bypasses the gatekeepers of official discourse. ‘Digital sumud’ is introduced as a contemporary iteration of Palestinian endurance, where social media functions not just as documentation but as a subversion of hegemonic narratives. Ultimately, this essay argues that Palestinian testimony does more than document loss. It disrupts, resists, and reclaims. The struggle over Jerusalem is not just territorial; it is a war over meaning itself, and through the voices of those who refuse to be erased, the city remains a living archive of defiance
5-Hydroxytryptamine receptors in GtoPdb v.2025.1
Get PDF5-HT receptors (nomenclature as agreed by the NC-IUPHAR Subcommittee on 5-HT receptors [200] and subsequently revised [182]) are, with the exception of the ionotropic 5-HT3 class, GPCRs where the endogenous agonist is 5-hydroxytryptamine. The diversity of metabotropic 5-HT receptors is increased by alternative splicing that produces isoforms of the 5-HT2A (non-functional), 5-HT2C (non-functional), 5-HT4, 5-HT6 (non-functional) and 5-HT7 receptors. Unique amongst the GPCRs, RNA editing produces 5-HT2C receptor isoforms that differ in function, such as efficiency and specificity of coupling to Gq/11 and also pharmacology [40, 499]. Most 5-HT receptors (except 5-ht1e and 5-ht5b) play specific roles mediating functional responses in different tissues (reviewed by [478, 394])
Cannabinoid receptors in GtoPdb v.2025.1
Get PDFCannabinoid receptors (nomenclature as agreed by the NC-IUPHAR Subcommittee on Cannabinoid Receptors [127]) are activated by endogenous ligands that include N-arachidonoylethanolamine (anandamide), N-homo-γ-linolenoylethanolamine, N-docosatetra-7,10,13,16-enoylethanolamine and 2-arachidonoylglycerol. Potency determinations of endogenous agonists at these receptors are complicated by the possibility of differential susceptibility of endogenous ligands to enzymatic conversion [5].There are currently three licenced cannabinoid medicines each of which contains a compound that can activate CB1 and CB2 receptors [119]. Two of these medicines were developed to suppress nausea and vomiting produced by chemotherapy. These are nabilone (Cesamet®), a synthetic CB1/CB2 receptor agonist, and synthetic Δ9-tetrahydrocannabinol (Marinol®; dronabinol), which can also be used as an appetite stimulant. The third medicine, Sativex®, contains mainly Δ9-tetrahydrocannabinol and cannabidiol, both extracted from cannabis, and is used to treat multiple sclerosis and cancer pain
Chemokine receptors in GtoPdb v.2025.1
Get PDFChemokine receptors (nomenclature as agreed by the NC-IUPHAR Subcommittee on Chemokine Receptors [459, 458, 32]) comprise a large subfamily of 7TM proteins that bind one or more chemokines, a large family of small cytokines typically possessing chemotactic activity for leukocytes. Additional hematopoietic and non-hematopoietic roles have been identified for many chemokines in the areas of embryonic development, immune cell proliferation, activation and death, viral infection, and as antibacterials, among others. Chemokine receptors can be divided by function into two main groups: G protein-coupled chemokine receptors, which mediate leukocyte trafficking, and "Atypical chemokine receptors", which may signal through non-G protein-coupled mechanisms and act as chemokine scavengers to downregulate inflammation or shape chemokine gradients [32].Chemokines in turn can be divided by structure into four subclasses by the number and arrangement of conserved cysteines. CC (also known as β-chemokines; n= 28), CXC (also known as α-chemokines; n= 17) and CX3C (n= 1) chemokines all have four conserved cysteines, with zero, one and three amino acids separating the first two cysteines respectively. C chemokines (n= 2) have only the second and fourth cysteines found in other chemokines. Chemokines can also be classified by function into homeostatic and inflammatory subgroups. Most chemokine receptors are able to bind multiple high-affinity chemokine ligands, but the ligands for a given receptor are almost always restricted to the same structural subclass. Most chemokines bind to more than one receptor subtype. Receptors for inflammatory chemokines are typically highly promiscuous with regard to ligand specificity, and may lack a selective endogenous ligand. G protein-coupled chemokine receptors are named acccording to the class of chemokines bound, whereas ACKR is the root acronym for atypical chemokine receptors [33]. There can be substantial cross-species differences in the sequences of both chemokines and chemokine receptors, and in the pharmacology and biology of chemokine receptors. Endogenous and microbial non-chemokine ligands have also been identified for chemokine receptors. Many chemokine receptors function as HIV co-receptors, but CCR5 is the only one demonstrated to play an essential role in HIV/AIDS pathogenesis. The tables include both standard chemokine receptor names [717] and aliases
Formylpeptide receptors in GtoPdb v.2025.1
Get PDFThe formylpeptide receptors (nomenclature agreed by the NC-IUPHAR Subcommittee on the formylpeptide receptor family [150]) respond to exogenous ligands such as the bacterial product fMet-Leu-Phe (fMLF) and endogenous ligands such as lipoxin A4 (LXA4), 15-epi-lipoxin A4, annexin I , cathepsin G, amyloid β42, serum amyloid A and spinorphin, derived from β-haemoglobin. FPR1 also serves as a plague receptor for selective destruction of human immune cells by Y. pestis [135]. The FPR1/2 agonists \u27compound 17b\u27 and \u27compound 43\u27 have shown cardiac protective functions [149, 64]