262 research outputs found
LLR-based attention-weighted projection decoding for sparse vector coded short-packet transmissions
Receiver Metric Design for Short-Block Channels : A Perspective for Reliable 6G Signaling Scenarios
Optimization strategies for low-latency 5G NR LDPC decoding on general purpose processor
International audienceIn recent years, with the progression of the computational abilities of General-Purpose Processors (GPPs), there has been a heightened interest in the implementation of software Low-Density Parity-Check (LDPC) decoders. This investigation provides a comprehensive analysis of the most effective strategies for optimizing the decoding latency of 5G LDPC on GPPs. Our proposed optimization mechanisms consist of the implementation of Advanced Vector Extensions 512 (AVX-512) instructions in computationally intensive routines and the application of code transformation techniques, specifically optimization through unrolling to tackle the primary computational challenges and minimize the overall latency of the decoder. To assess the efficiency of our proposed techniques, thorough simulations were carried out to determine the decoding time. Our findings indicate that the implementation of the aforementioned optimization techniques on computational routines causing time bottlenecks can lead to a significant reduction of at least 30% in computational delay, even under unfavorable conditions. This discovery demonstrates the feasibility of developing a low-latency software 5G NR LDPC decoder on an x86 architecture
Genetic and clinical characteristics of Korean maturity-onset diabetes of the young (MODY) patients.
Maturity-onset diabetes of the young (MODY) is mostly caused by mutations of the hepatocyte nuclear factor (HNF)-1alpha (MODY3) and glucokinase (MODY2) genes in Caucasians. But most Japanese and Chinese MODY patients are not linked to known MODY genes. In this study, we examined the genetic and clinical characteristics of Korean subjects with MODY and early onset type 2 diabetes who had been diagnosed before 15 years of age. The study included 23 unrelated subjects fulfilling the criteria for MODY (three consecutive generations of type 2 diabetes with at least one member diagnosed under the age of 25 year) and 17 unrelated subjects diagnosed with early onset type 2 DM under the age of 15 years. The HNF-4alpha (MODY1), glucokinase (MODY2) and HNF-1alpha (MODY3) genes were analysed by direct sequencing. Mutations in the HNF-1alpha gene were found in two patients (5%). One of these, P393fsdelC, was novel, and was found in a patient classified in the MODY group. The GCK gene mutation, R191W, was identified in one patient classified as early-onset type 2 DM (2.5%). No mutations were found in the HNF-4alpha gene, except the T130I variant, which is a known rare polymorphism. In conclusion, the mutations in the HNF-1alpha gene and GCK account for a small proportion, about 5% and 2.5%, respectively, in Korean MODY and early onset type 2 patients. The majority of MODY cases in the Korean population are due to defects in unknown genes
Test of the German Resilience
From its early post-war catch-up phase, Germany’s formidable export engine has been its consistent driver of growth. But Germany has almost equally consistently run current account surpluses. Exports have powered the dynamic phases and helped emerge from stagnation. Volatile external demand, in turn, has elevated German GDP growth volatility by advanced countries’ standards, keeping domestic consumption growth at surprisingly low levels. As a consequence, despite the size of its economy and important labor market reforms, Germany’s ability to act as global locomotive has been limited. With increasing competition in its traditional areas of manufacturing, a more domestically-driven growth dynamic, especially in the production and delivery of services, will be good for Germany and for the global economy. Absent such an effort, German growth will remain constrained, and Germany will play only a modest role in spurring growth elsewhere
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