1,532 research outputs found

    Taking the Court Seriously: A Proposed Approach to Senate Confirmation of Supreme Court Nominees.

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    Simson, Gary J.. (1990). Taking the Court Seriously: A Proposed Approach to Senate Confirmation of Supreme Court Nominees.. Retrieved from the University Digital Conservancy, https://hdl.handle.net/11299/165912

    Giving the Establishment Clause Its Due

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    Gary J. Simson, Senior Vice Provost for Scholarship for Mercer University, holds the Macon Chair in Law at the law school. Simson received a B.A. summa cum laude in 1971 from Yale College, where he majored in Spanish Literature and was elected to Phi Beta Kappa, and a J.D. in 1974 from Yale Law School, where he was an editor of The Yale Law Journal. After clerking for Judge J. Joseph Smith of the U.S. Court of Appeals for the Second Circuit, Simson began his teaching career at the University of Texas School of Law in 1975 and was promoted to full professor in 1977. Simson joined the Cornell Law School faculty as professor of law in 1980 and remained at Cornell until 2006, serving as Associate Dean for Faculty Development from 1997-2000 and as Associate Dean for Academic Affairs from 2000-2004. In 2006 he became Dean and Joseph C. Hostetler-Baker & Hostetler Professor of Law at Case Western Reserve University School of Law. He left Case in 2010 to become Dean of the School of Law and Macon Chair in Law at Mercer. He served as Dean until moving to his current leadership position in the central university in 2014. Over the years, Simson has taught Constitutional Law, Conflict of Laws, Religious Liberty Clinic, and seminars on freedom of religion and other constitutional law topics. His constitutional law scholarship has addressed such issues as school vouchers, Supreme Court appointments, the death penalty and religion, and single-sex schools. He is also the author of a leading conflict of laws casebook and various articles in the field

    Panel 1

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    Gary Simson, First Amendment Realism Seth Oranburg, Social Media and Democracy after the Capital Riot Commentator: Sue Paint-Thorn

    Michael Rodriguez interviews author Gary Gildner

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    Author Gary Gildner explains why he left his tenured teaching position to move to Idaho to became a full-time writer of poetry. Gildner talks about donating his personal papers to Michigan State University Libraries' Special Collections, his writing style and how he approaches writing. Gildner is interviewed by MSU Librarian Michael Rodriguez for the MSU Libraries' Michigan Writer Series. Held at the MSU Main Library

    Gary J. Simson

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    View the Mercer Law Library catalog record for more information.https://digitalcommons.law.mercer.edu/portraits/1016/thumbnail.jp

    Zoonotic potential of Giardia duodenalis and Cryptosporidium spp. and prevalence of intestinal parasites in young dogs from different populations on Prince Edward Island, Canada

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    The prevalence of Giardia duodenalis, Cryptosporidium spp. and other intestinal parasites was determined in dogs <1 year old from Prince Edward Island, Canada. Fecal samples were collected from the local animal shelter (n=62), private veterinary clinics (n=78) and a pet store (n=69). Intestinal parasites isolated included G. duodenalis, Cryptosporidium spp., Toxocara canis, Isospora spp. and Uncinaria stenocephala. To estimate the zoonotic risk associated with these infections, genotypes of G. duodenalis and Cryptosporidium spp. were determined using 16S rRNA and Hsp70 gene sequencing, respectively. Dogs from the pet store had the highest prevalence of intestinal parasites (78%, 95% CI: 68-88%), followed by the private veterinary clinics (49%, 95% CI: 37-60%), and the local animal shelter (34%, 95% CI: 22-46%). The majority G. duodenalis belonged to host-adapted assemblages D (47%, 95% CI: 31-64%) and C (26%, 95% CI: 13-43%), respectively. Zoonotic assemblages A and B were isolated alone or in mixed infections from 16% (95% CI: 6-31%) of G. duodenalis-positive dogs. All Cryptosporidium spp. were the host-adapted C. canis. While host-adapted, non-zoonotic G. duodenalis genotypes were more common, the presence of G. duodenalis assemblages A and B, T. canis, and U. stenocephala suggests that these dogs may present a zoonotic risk. The zoonotic risk from Cryptosporidium-infected dogs was minimal.Fabienne D. Uehlinger, Spencer J. Greenwood, J. Trenton McClure, Gary Conboy, Ryan O’Handley, Herman W. Barkem

    Mood disorders in familial epilepsy: A test of shared etiology

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    Objective: Mood disorders are the most common comorbid conditions in epilepsy but the cause remains unclear. One possible explanation is a shared genetic susceptibility to epilepsy and mood disorders. We tested this hypothesis by evaluating lifetime prevalence of mood disorders in relatives with and without epilepsy in families containing multiple individuals with epilepsy, and comparing the findings with rates from a general population sample. Methods: The Composite International Diagnostic Interview was administered to 192 individuals from 60 families, including 110 participants with epilepsy of unknown cause (50 focal epilepsy [FE], 42 generalized epilepsy [GE], 6 FE and GE, 12 unclassifiable) and 82 relatives without epilepsy (RWOE). Odds ratios (ORs) for lifetime prevalence of mood disorders in participants with versus without epilepsy were computed through logistic regression, using generalized estimation equations to account for familial clustering. Standardized prevalence ratios (SPRs) were used to compare prevalence in family members with general population rates. Results: Compared with RWOE, ORs for mood disorders were significantly increased in participants with FE (OR = 2.4, 95% confidence interval [CI] = 1.1 - 5.2) but not in those with GE (OR = 1.0, 95% CI = 0.4 - 2.2). In addition, prevalence of mood disorders was increased in individuals with epilepsy who had ≥1 relative with FE. Compared with general population rates, mood disorders were significantly increased in individuals with FE but not in those with GE. Rates were also increased in RWOE, but not significantly so (SPR = 1.4, P = 0.14). Significance: These findings are consistent with the hypothesis of shared genetic susceptibility to epilepsy and mood disorders, but suggest (1) the effect may be restricted to FE, and (2) the shared genetic effect on risk of mood disorders and epilepsy may be restricted to individuals with epilepsy, that is, to those in whom the genetic risk for epilepsy is “penetrant.”Peer reviewedThis is the accepted version of the following article: Insel BJ, Ottman R, Heiman GA. Mood disorders in familial epilepsy: A test of shared etiology. Epilepsia. 2018;00:1–9. https://doi.org/10.1111/epi.13985, which has been published in the Early View form at http://onlinelibrary.wiley.com/doi/10.1111/epi.13985/full. This article may be used for non-commercial purposes in accordance with the Wiley Self-Archiving Policy [https://authorservices.wiley.com/author-resources/Journal-Authors/licensing-open-access/open-access/self-archiving.html]

    Book Review: Alcoholism, Drug Addiction, and the Road to Recovery; Life on the Edge

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    Author: Barry Stimmel, M.D. Reviewer: J. Gary Linn, Ph.D. Publisher: Binghamton, NY: Haworth, 2002 ISBN: 0-7890-0552-2, 414 pages Cost: $39.9
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