12,967 research outputs found
Author Correction: Evaluation of skin cancer resection guide using hyper‑realistic in‑vitro phantom fabricated by 3D printing
No full textThe original version of this Article contained an error in the spelling of the author Taehun Kim which was incorrectly given as Teahun Kim. The original Article has been corrected
DBLP-derived labeled data for author name disambiguation
No full textThis is a DBLP-derived labeled data originally created by Dr. C. Lee Giles at Penn State University and filtered for duplicate removal and error correction by Dr. Jinseok Kim at University of Michigan. For more details, see references below.1. Kim, Jinseok (2018). Evaluating author name disambiguation for digital libraries: a case of DBLP. Scientometrics. doi:10.1007/s11192-018-2824-5 2. Kim, Jinseok & Kim, Jenna (2018). The impact of imbalanced training data on machine learning for author name disambiguation. Scientometrics. doi: 10.1007/s11192-018-2865-9Each row refers to an author name instance with following feature information separated by tab.author name: full name string extracted from DBLPunique author id: labels assigned manually by Dr. C. Lee Giles's teampaper id: assigned by Dr. Jinseok Kimauthor list: names of authors in the byline of the paperyear: publication yearvenue: conference or journal namestitle: stopwords removed and stemmed by the Porter's stemmerIf you want to use this dataset, please consider to cite papers below.For the original dataset: Han, H., Giles, L., Zha, H., Li, C., & Tsioutsiouliklis, K. (2004). Two Supervised Learning Approaches for Name Disambiguation in Author Citations. JCDL 2004: Proceedings of the Fourth ACM/IEEE Joint Conference on Digital Libraries, 296-305. doi:10.1145/996350.996419For the filtered dataset: 1. Kim, Jinseok (2018). Evaluating author name disambiguation for digital libraries: a case of DBLP. Scientometrics. doi:10.1007/s11192-018-2824-5 or2. Kim, Jinseok & Kim, Jenna (2018). The impact of imbalanced training data on machine learning for author name disambiguation. Scientometrics. doi: 10.1007/s11192-018-2865-9</div
Khoo Kay Kim, professor of Malaysian history : a biobibliometric study
No full textPresents an analysis of the publication productivity, authorship pattern, channels of communication, journal preference and language preference of Professor Dato' Khoo Kay Kim, Professor of Malaysian History in the University of Malaya, Kuala Lumpur. The results of this biobibliometric study indicate that he can be a role model for future Malaysian historians to emulate his various achievements especially in the field of history education
Anxious, Dismal, Giddy, Aggressive: Seth Kim-Cohen interviewed by Mark Peter Wright for Ear Room.
No full textA conversation with author Seth Kim-Cohen
Kim Gordon - no icon
No full textAs cofounder of legendary rock band Sonic Youth, best-selling author, and celebrated artist, Kim Gordon is one of the most singular and influential figures of the modern era. This personally curated scrapbook is an edgy and evocative portrait of Gordon s life, art, and style. Spanning from her childhood on Californian surf beaches in the 60s and 70s to New York s downtown art and music scene in the 80s and 90s where Sonic Youth was born. Through unpublished personal photographs, magazine and newspaper clippings, fashion editorials, and advertising campaigns, interspersed with Gordon s song lyrics, writings, artworks, private objects, and ephemera, this book demonstrates how Kim Gordon has been a role model for generations of women and me
Overview of Recent Progress in Fire Suppression
No full textthis document is published in / Une version de ce document se trouve dans : Invited Keynote Lecture at the 2 nd NRIFD Symposium, Proceedings, Tokyo, Japan, July 17-19, 2002, pp. 1-13 www.nrc.ca/irc/ircpubs NRCC-45690 Title: OVERVIEW OF RECENT PROGRESS IN FIRE SUPPRESSION TECHNOLOGY Author(s): Andrew KIM Corresponding (first) author: Andrew Kim Academic degree: Ph.
A note on Kim-Ma characterization of the Hilbert ball
No full textThis is an open access article under the CC BY license.[No abstract available]Kortney Rose Foundation, KRF, (2002-070-C00005); National Research Foundation of Korea, NRF* Corresponding author. E-mail addresses: [email protected] (K.-T. Kim), [email protected] (D. Ma). 1 Research supported in part by the grant KRF 2002-070-C00005 from The Korea Research Foundation
High-throughput in vitro processing of human primary microRNA by the recombinant microprocessor
No full text© 2021 The Author(s)We describe a protocol to conduct a high-throughput in vitro processing assay, using 1,881 human primary microRNAs (pri-miRNAs) and recombinant Microprocessor complex, followed by deep sequencing library generation. This comprehensive approach allows the mapping of cleavage sites and the measurement of processing efficiency of a large number of substrates simultaneously. Our protocol is readily modifiable to investigate the effects of chemicals and regulatory proteins. Moreover, cis-acting elements can be examined by replacing the wild-type pri-miRNAs with mutant variants. For complete details on the use and execution of this profile, please refer to Kim et al. (2021).11Nscopu
PTTH-induced Metamorphosis in Drosophila melanogaster is Affected by TOR, Retinoic Acid, and Juvenile Hormone Signaling
No full textThe developmental timing of Drosophila melanogaster is mainly controlled by 20-hydroxyecdysone (20E), which initiates moulting and metamorphosis. The synthesis of the 20E precursor, ecdysone (E), in the prothoracic gland (PG) is regulated by the neuropeptide prothoracicotropic hormone (PTTH) synthesized in the prothoracicotropes. The aim of this study is to contribute to the understanding of the regulation of PTTH production and secretion. We investigated the effect of the candidate PTTH regulators juvenile hormone (JH), 20E, retinoic acid (RA), insulin/insulin growth factor signalling (IIS), and target of rapamycin (TOR) on ptth expression. This was achieved through applying the GAL4-UAS system to either knock down or overexpress genes, thought to be involved in the regulation, in the prothoracicotropes. The impact of these alterations on the timing of pupariation was monitored and the expression of ptth was analyzed by using green fluorescent protein (GFP) driven by the ptth promoter. The results suggest that inhibition of RA and JH signalling in the prothoracicotropes accelerate pupariation, which are the first molecular indications of RA and JH being direct regulators of the prothoracicotropes. Additionally, overactivating TOR signalling delays metamorphosis and seems to suppress ptth expression, which implies that nutrition has a negative effect on PTTH production and secretion. This is supported by our observation of an accelerated pupariation when TOR related signalling is suppressed. Further, the results indicate that IIS and 20E are not critical regulators of PTTH secretion.The developmental timing of Drosophila melanogaster is mainly controlled by 20-hydroxyecdysone (20E), which initiates moulting and metamorphosis. The synthesis of the 20E precursor, ecdysone (E), in the prothoracic gland (PG) is regulated by the neuropeptide prothoracicotropic hormone (PTTH) synthesized in the prothoracicotropes. The aim of this study is to contribute to the understanding of the regulation of PTTH production and secretion. We investigated the effect of the candidate PTTH regulators juvenile hormone (JH), 20E, retinoic acid (RA), insulin/insulin growth factor signalling (IIS), and target of rapamycin (TOR) on ptth expression. This was achieved through applying the GAL4-UAS system to either knock down or overexpress genes, thought to be involved in the regulation, in the prothoracicotropes. The impact of these alterations on the timing of pupariation was monitored and the expression of ptth was analyzed by using green fluorescent protein (GFP) driven by the ptth promoter. The results suggest that inhibition of RA and JH signalling in the prothoracicotropes accelerate pupariation, which are the first molecular indications of RA and JH being direct regulators of the prothoracicotropes. Additionally, overactivating TOR signalling delays metamorphosis and seems to suppress ptth expression, which implies that nutrition has a negative effect on PTTH production and secretion. This is supported by our observation of an accelerated pupariation when TOR related signalling is suppressed. Further, the results indicate that IIS and 20E are not critical regulators of PTTH secretion
Taesun Yoo,1 Sun-Gyun Kim,2 Soo Hyun Yang,3 Hyun Kim,3 Eunjoon Kim,1,2 and Soo Young Kimcorresponding author4
No full textBackground DLG2, also known as postsynaptic density protein-93 (PSD-93) or chapsyn-110, is an excitatory postsynaptic scaffolding protein that interacts with synaptic surface receptors and signaling molecules. A recent study has demonstrated that mutations in the DLG2 promoter region are significantly associated with autism spectrum disorder (ASD). Although DLG2 is well known as a schizophrenia-susceptibility gene, the mechanisms that link DLG2 gene disruption with ASD-like behaviors remain unclear. Methods Mice lacking exon 14 of the Dlg2 gene (Dlg2(-/-) mice) were used to investigate whether Dlg2 deletion leads to ASD-like behavioral abnormalities. To this end, we performed a battery of behavioral tests assessing locomotion, anxiety, sociability, and repetitive behaviors. In situ hybridization was performed to determine expression levels of Dlg2 mRNA in different mouse brain regions during embryonic and postnatal brain development. We also measured excitatory and inhibitory synaptic currents to determine the impacts of Dlg2 deletion on synaptic transmission in the dorsolateral striatum. Results Dlg2(-/-) mice showed hypoactivity in a novel environment. They also exhibited decreased social approach, but normal social novelty recognition, compared with wild-type animals. In addition, Dlg2(-/-) mice displayed strong self-grooming, both in home cages and novel environments. Dlg2 mRNA levels in the striatum were heightened until postnatal day 7 in mice, implying potential roles of DLG2 in the development of striatal connectivity. In addition, the frequency of excitatory, but not inhibitory, spontaneous postsynaptic currents in the Dlg2(-/-) dorsolateral striatum was significantly reduced. Conclusion These results suggest that homozygous Dlg2 deletion in mice leads to ASD-like behavioral phenotypes, including social deficits and increased repetitive behaviors, as well as reductions in excitatory synaptic input onto dorsolateral spiny projection neurons, implying that the dorsal striatum is one of the brain regions vulnerable to the developmental dysregulation of DLG2. C. The Author(s) 202011Nsciescopu
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