197,963 research outputs found
Elas também matam: o romance policial de Maria Alice Barroso, Ana Callado e Kátia Rebello
No full textDissertação (mestrado) - Universidade Federal de Santa Catarina, Centro de Comunicação e Expressão, Programa de Pós-Graduação em Literatura, Florianópolis, 2009.Disponibilizado em PDF apenas o capítulo 1.A presente dissertação aborda o processo de transposição e adaptação dos modelos do romance policial norte-americano e europeu à narrativa policial brasileira de autoria feminina, categorizada em três momentos. O primeiro momento enfoca a construção do romance policial de enigma através do romance de Maria Alice Barroso, Quem matou Pacífico?. O segundo momento privilegia o texto de Ana Arruda Callado, Uma aula de matar, expressando a ruptura com o modelo clássico e a consolidação do novo cânone, agora com o romance noir. O terceiro momento é configurado pelo romance policial brasileiro contemporâneo que mescla os elementos canônicos do enigma e do noir, no romance de Kátia Rebello, Olhos de vidro. Encerram o trabalho considerações, questionamentos e reflexões sobre o romance policial brasileiro de autoria feminina.Disponibilizado em PDF apenas o capítulo 1
Seducida para matar
No full textCompany, JM. (2001). Seducida para matar. La madriguera. (39):57-58. https://riunet.upv.es/handle/10251/41975.Importación Masiva57583
Les fouilles de M. J. Perrot à Bir Abou Matar et Bir Es Safadi (1954)
No full textSchaeffer Claude F.-A. Les fouilles de M. J. Perrot à Bir Abou Matar et Bir Es Safadi (1954). In: Comptes rendus des séances de l'Académie des Inscriptions et Belles-Lettres, 99ᵉ année, N. 3, 1955. pp. 348-352
Nouvelles des fouilles de M. Jean Perrot à Bir Abou Matar (Palestine)
No full textDussaud René. Nouvelles des fouilles de M. Jean Perrot à Bir Abou Matar (Palestine). In: Comptes rendus des séances de l'Académie des Inscriptions et Belles-Lettres, 98ᵉ année, N. 1, 1954. p. 19
Supplemental Material - Adoption of electronic cards using Wi-Fi platform services by clients of banking sector during COVID-19 pandemic
No full textSupplementary Material for Adoption of electronic cards using Wi-Fi platform services by clients of banking sector during COVID-19 pandemic by Ali Matar and Abdelbaset M Alkhawaldeh in International Journal of Engineering Business Management.</p
Prevalence of the genes encoding extended-spectrum β-lactamases, in Escherichia coli resistant to β-lactam and non-β-lactam antibiotics
No full textThe prevalences of extended-spectrum β-lactamases (ESBL) and their encoding bla genes, TEM, SHV and CTX_M, were investigated in isolates of Escherichia coli that were resistant to β-lactam and-or non-β-lactam antibiotics. Of the 250 E. coli isolates investigated, all of which came from patients in a major hospital in southern Lebanon, 61 (13.3percent) were found to have ESBL, their production of β-lactamase being confirmed by the ceftazidime and ceftazidime-clavulanic-acid disc methods. All 61 ESBL isolates were resistant to β-lactams and sensitive to imipenem, piperacillin-tazobactam and cefoxitime. Only 40percent were resistant to fluoroquinolones, 33percent were resistant to aminoglycosides, and 18percent were considered to have multi-drug resistance. The results of the PCR-based amplification of the bla gene in DNA samples from the 61 ESBL isolates indicated that 11 (18percent) of the isolates carried both the TEM and SHV genes, 37 (61percent) carried the TEM gene but not the SHV, and 13 (21percent) had the SHV gene but not the TEM. None of the isolates carried the CTX_M gene. Of the 37 TEM-positive-SHV-negative isolates, 43percent were resistant to fluoroquinolones and 37percent to aminoglycosides. Increased resistance to non-β-lactam antibiotics was observed in the isolates harbouring both the TEM and SHV genes, of which 54percent were resistant to all of the tested antibiotics except imipenem, 36percent were only resistant to fluoroquinolones, and 9.1percent only resistant to aminoglycosides. The possibility that the concomitant presence of TEM- and SHV-type β-lactamases is associated with resistance to non-β-lactam antibiotics requires further research. The prevalences of ESBL and their encoding genes in Gram-negative bacteria collected from various regions in Lebanon will now be investigated. © 2005 The Liverpool School of Tropical Medicine.Bonnet R, 2000, ANTIMICROB AGENTS CH, V44, P1936, DOI 10.1128-AAC.44.7.1936-1942.2000; Bradford PA, 2001, CLIN MICROBIOL REV, V14, P933, DOI 10.1128-CMR.14.4.933-951.2001; MATAR GM, 1999, BRIT MED J MIDDLE E, V7, P6; MEDEIROS AA, 1984, BRIT MED BULL, V40, P18; *NAT COMM CLIN LAB, 2004, 100S14 M NAT COMM CL; NUESCHINDERBINE.MT, 1997, ANTIMICROBIAL AGENTS, V41, P283; Paterson DL, 2000, CLIN INFECT DIS, V30, P473, DOI 10.1086-313719; Saurina G, 2000, J ANTIMICROB CHEMOTH, V45, P895, DOI 10.1093-jac-45.6.895; Tran JH, 2002, P NATL ACAD SCI USA, V99, P5638, DOI 10.1073-pnas.082092899; Tzouvelekis LS, 2000, INT J ANTIMICROB AG, V14, P137, DOI 10.1016-S0924-8579(99)00165-X78
Polymerase chain reaction identification of coagulase-negative staphylococci and of strain diversity and spread of Staphylococcus epidermidis in a major medical center in Lebanon
No full textA 2-step polymerase chain reaction (PCR) assay and random amplification of polymorphic DNA (RAPD) analysis, respectively, were assessed to identify coagulase-negative staphylococci organisms to the species level and to determine the strain diversity and spread of Staphylococcus epidermidis, the most frequently isolated species, in a medical center in Beirut, Lebanon. Our data indicated that PCR was faster and was more efficient in identifying S. epidermidis isolates than is conventional biochemical testing. RAPD analysis have shown that S. epidermidis strains were scattered across the different clinical services, demonstrating various clusters of infection in the medical center. © 2006 by The Society for Healthcare Epidemiology of America. All rights reserved.ARBEIT RD, 1997, STAPHYLOCOCCI HUMAN, P253; Diekema DJ, 2001, CLIN INFECT DIS S2, V32, P114; Huebner J, 1999, ANNU REV MED, V50, P223; MARTIN MA, 1989, ANN INTERN MED, V110, P9; Martineau F, 2001, J CLIN MICROBIOL, V39, P2541, DOI 10.1128-JCM.39.7.2541-2547.2001; MATAR GM, 1995, LEB SCI B, V8, P63; Matar GM, 2005, BMC MICROBIOL, V5, DOI 10.1186-1471-2180-5-29; Mendoza M, 1998, INT J SYST BACTERIOL, V48, P1049; NCCLS (National Committee for Clinical Laboratory Standards), 2002, PERF STAND ANT SUSC; PATRICK CC, 1990, J PEDIATR-US, V116, P497, DOI 10.1016-S0022-3476(05)81593-8; PETERS G, 1995, CURR OPIN INFECT DIS, V8, pS12, DOI 10.1097-00001432-199503001-00004; PFALLER M A, 1988, Clinical Microbiology Reviews, V1, P281; Ronald A, 2002, AM J MED, V113, p14S52
Unified approach for polymeric patterning via controlling the propagation of frontal photopolymerization waves
No full textDr. Duane M. Jackson, Morehouse College, July 2011
No full textThis video is a conversation with Dr. Duane M. Jackson. Dr. Jackson talks about his paper, "Recall and the Serial Position Effect: The Role of Primacy and Recency on Accounting Students' Performance." Jackie Daniel, AUC Woodruff Library, is the interviewer
First detection of oxacillinase-mediated resistance to carbapenems in Klebsiella pneumoniae from Morocco
No full textThe frequency of carbapenem resistance due to class-D β-lactamases (i.e. oxacillinases) among the world's Enterobacteriaceae is increasing. Recently, in Morocco, two isolates of carbapenem-resistant Klebsiella pneumoniae were recovered from the same patient, one harbouring plasmid-encoded bla- OXA-48 and the other the bla- OXA-1 gene. This represents the first evidence of bla OXA-48-mediated carbapenem-resistance in Enterobacteriaceae in Morocco. © 2010 W. S. Maney andamp; Son Ltd.Aktas Z, 2008, CHEMOTHERAPY, V54, P101, DOI 10.1159-000118661; [Anonymous], 2008, M100S18 CLIN LAB STA; BROWN DFJ, 1991, J ANTIMICROB CHEMOTH, V27, P185, DOI 10.1093-jac-27.2.185; Carrer A, 2008, ANTIMICROB AGENTS CH, V52, P2950, DOI 10.1128-AAC.01672-07; CARRER A, 2010, ANTIMICROBIAL AGENTS, V54, P1312; Cuzon G, 2008, ANTIMICROB AGENTS CH, V52, P3463, DOI 10.1128-AAC.00543-08; Gulmez D, 2008, INT J ANTIMICROB AG, V31, P523, DOI 10.1016-j.ijantimicag.2008.01.017; Leavitt A, 2007, ANTIMICROB AGENTS CH, V51, P3026, DOI 10.1128-AAC.00299-07; Matar GM, 2010, ANN TROP MED PARASIT, V104, P271, DOI 10.1179-136485910X12647085215651; Matar GM, 2008, CLIN MICROBIOL INFEC, V14, P887, DOI 10.1111-j.1469-0691.2008.02059.x; Poirel L, 2007, FUTURE MICROBIOL, V2, P501, DOI 10.2217-17460913.2.5.50126272
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