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    Laing, C W, 14626

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    This record was harvested from a previous catalogue system and will be withdrawn in 2025. Information in this record may be superseded or incomplete. Visit this record in UMA's new catalogue at: https://archives.library.unimelb.edu.au/nodes/view/397999Surname: LAING. Given Name(s) or Initials: C W. Military Service Number or Last Known Location: 14626. Missing, Wounded and Prisoner of War Enquiry Card Index Number: 56603.236836 Item: [2016.0049.30292] "Laing, C W, 14626

    Laing, C E A, SX3779

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    This record was harvested from a previous catalogue system and will be withdrawn in 2025. Information in this record may be superseded or incomplete. Visit this record in UMA's new catalogue at: https://archives.library.unimelb.edu.au/nodes/view/398016Surname: LAING. Given Name(s) or Initials: C E A. Military Service Number or Last Known Location: SX3779. Missing, Wounded and Prisoner of War Enquiry Card Index Number: C8077.236869 Item: [2016.0049.30309] "Laing, C E A, SX3779

    Laing, C.

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    Northern Spirit: 300 Years of Art on Tyneside

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    Permanent display at the Laing Art Gallery, Newcastle co-curated with Tyne and Wear Archives and Museums

    Mutations in the slow skeletal muscle fiber myosin heavy chain gene (MYH7) cause laing early-onset distal myopathy (MPD1)

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    We previously linked Laing-type early-onset autosomal dominant distal myopathy (MPD1) to a 22-cM region of chromosome 14. One candidate gene in the region, MYH7, which is mutated in cardiomyopathy and myosin storage myopathy, codes for the myosin heavy chain of type I skeletal muscle fibers and cardiac ventricles. We have identified five novel heterozygous mutationsArg1500Pro, Lys1617del, Ala1663Pro, Leu1706Pro, and Lys1729del in exons 32, 34, 35, and 36 of MYH7in six families with early-onset distal myopathy. All five mutations are predicted, by in silico analysis, to locally disrupt the ability of the myosin tail to form the coiled coil, which is its normal structure. These findings demonstrate that heterozygous mutations toward the 3 end of MYH7 cause Laing-type early-onset distal myopathy. MYH7 is the fourth distal-myopathy gene to have been identified

    Prediksi Kinerja Simpang Lima Laing Kota Solok Tahun 2024

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    Simpang Lima Laing merupakan salah satu simpang tak bersinyal di Kota Solok. Simpang Lima Laing berada di Jalan Lingkar Utara yang nantinya akan dioperasikan sebagai jalan nasional.. Simpang Lima Laing menghubungkan Jalan Lingkar Utara Barat – Jalan Muchtar – Jalan Kapten Bahar – Jalan Lingkar Utara Timur – Jalan Tembok Raya. Simpang Lima Laing mempunyai dimensi yang berbeda dengan simpang lainnya karena daerah simpang yang tidak simetris, menyebabkan manuver kendaraan terganggu dan adanya potensi kecelakaan lalu-lintas. Maka diperlukan perencanaan desain simpang atau pengaturan simpang dengan mengetahui kinerja Simpang Lima Laing Kota Solok saat ini dan tahun yang akan datang. Penelitian ini berpedoman pada MKJI 1997. Pengumpulan data dilakukan dengan cara survey di lapangan, untuk mendapatkan data geometrik simpang, volume lalu-lintas, dan kondisi simpang. Survey dilakukan pada hari Selasa dan Rabu tanggal 18-19 November 2018 selama periode 12 jam (06.15-18.15 WIB). Berdasarkan hasil perhitungan arus lalu-lintas pada jam puncak, volume kendaraan tertinggi didapatkan pada hari Rabu sebesar 575 smp/jam, kapasitas (C) sebesar 3838 smp/jam, derajat kejenuhan (DS) sebesar 0,15, tundaan sebesar 6,22 det/smp, dan peluang antrian sebesar 0,92-6,78%. Dari nilai derajat kejenuhan yang didapat (DS≤0,75), disimpulkan bahwa kinerja Simpang Lima Laing Kota Solok tahun 2018 tidak jenuh dan belum membutuhkan pengendalian lalu-lintas. Tingkat pelayanan (LOS) Simpang Lima Laing tahun 2018 termasuk pada tingkat layanan A dengan karakteristik arus bebas, dimana volume lalu-lintas rendah, kecepatan tinggi (pengemudi bebas memilih kecepatan yang diinginkan). Prediksi kinerja Simpang Lima Laing Kota Solok pada tahun 2024 jika Jalan Lingkar Utara dioperasionalkan, menggunakan data volume lalu-lintas dari hari survey, arus lalu-lintas pada jam puncak sebesar 2488 smp/jam. Kapasitas simpang sebesar 2732 smp/jam, diperoleh nilai derajat kejenuhan (DS) sebesar 0,91 (DS≥0,75) dimana prediksi kinerja Simpang Lima Laing Kota Solok pada tahun 2024 jika Jalan Lingkar Utara dioperasionalkan mendekati jenuh. Tingkat pelayanan (LOS) Simpang Lima Laing Kota Solok pada tahun 2024 jika Jalan Lingkar Utara dioperasionalkan, termasuk pada tingkat layanan E dengan karakteristik arus tidak stabil, terkadang berhenti. Kata kunci: Kapasitas, derajat kejenuhan, tundaan, peluang antrian, prediksi kinerja simpang tahun 202

    Cryptostigma saundersi Laing 1925

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    Cryptostigma saundersi Laing (Fig. 22) Cryptostigma saundersi Laing: Laing 1925: 59; Ben-Dov 1993: 99; Qin & Gullan 1989: 225. Material examined. Lectotype, here designated. Cryptostigma saundersi Laing, adult ♀. BRAZIL: Rio de Janeiro, ex trunk of a tree protected by ants, 9.viii.1923, coll. L.G. Saunders (# 4), 1 (1) (BMNH). Paralectotype. Same data as lectotype, except that no date given, 1 slide with 1 specimen with dorsum and venter under separate cover slips (BMNH). Adult female (Fig. 22) Unmounted material. Alcohol preserved specimens with a pale yellowish brown longitudinal stripe and several intertwining stripes on dorsum; also with semicircular stripes on declivity anterior to anal lobes; remainder of dorsum of a darker brown; a small darker brown to almost black area around anal plates. Ventral surface dark brown with a paler marginal area. Length (unmounted) approximately 7 mm, breadth from 5–6 mm (Laing, 1925). Mounted material. Body outline oval, constricted at stigmatic areas, 6.0–7.0 mm long, 6.5–7.0 mm wide (n=2). Dorsum. Derm membranous, with numerous (68–92) subcircular compound orbicular pores, scattered on dorsum, size variable, those closest to body margin smallest, abundant on body margin and submargin but absent from mid-dorsum; each orbicular pore associated with 4–15 bilocular microducts, and about 6–24 slender setae around pore margins; each seta 13–19 µm long. Dorsal setae absent, except for those on margins of orbicular pores. Simple pores of 2 sizes: larger pores each about 8 µm wide; smaller pores each about 4.5 µm wide, scattered throughout dorsum. Dorsal microducts of 2 types: small microducts each about 2 µm wide and unilocular; larger microducts each about 6.0 µm wide and appearing bilocular, both types scattered on dorsum. Preopercular pores absent. Narrow sclerotic crescent present around anal plates. Anal plates together quadrate, with rounded angles, each plate 243–265 µm long, 108–140 µm wide, anterolateral margin 135–150 µm long, posterolateral margin 173–185 µm long; with 10–21 setae on dorsal surface; plates located about 1/5 of body length from posterior margin. Anal ring with 10 setae. Eyespots absent. Margin. Margin smooth. Marginal setae each 13–17 µm long and conical, with well-developed bases; in 2 or 3 rows; numerous, with 100 or more setae between anterior and posterior stigmatic areas. Stigmatic clefts deep; stigmatic sclerotization enclosing spiracles; with 3–5 stigmatic setae in each cleft, usually broken off. Venter. Derm membranous. Ventral setae slender, each 23–41 µm long, those on abdominal segments usually longest, shortest around body margin. Ventral microducts each about 4.5 µm wide, present in transverse intersegmental groups, and around margin; each microduct surrounded by a slightly sclerotized area. Tubular ducts present around vulva and in a small area on last 3 or 4 abdominal segments. Clypeolabral shield 447–530 µm wide. Multilocular disc-pores each 8–10 µm wide with 5–11 (mostly 8) loculi, present in perivulvar region and medially on all abdominal segments, and also transversely on meso- and metathorax; also with pores extending from mid areas between meso- and metathoracic legs in a narrow line that connects to mesothoracic legs and posterior spiracles; with 1 pore near each antenna. Spiracular disc-pores each 6–7 µm wide with 6–9 (mostly 5) loculi, present in a small group around each spiracle. Antennae reduced, with about 3 segments, total length 102–155 µm. Legs reduced, segments mostly fused, total length 119–237 µm long; tarsal and claw digitules slender, knobbed; claw denticle absent. Spiracles located near margin; anterior peritremes each 205–245 µm wide, posterior peritremes each 248–310 µm wide. Diagnosis. The adult female of C. saundersi can be diagnosed by the following features: (i) sclerotic pores absent, (ii) complex orbicular pores present, totalling 34, (iii) dorsal setae absent, except for those present on orbicular pores, (iv) preopercular pores absent, (v) each anal plate with about 21 setae on dorsal surface, (vi) with about 100 or more marginal setae between anterior and posterior stigmatic areas, (vii) 1 multilocular disc-pore present near each antenna, (viii) ventral microducts present in transverse linear intersegmental rows and around margin, and (ix) ventral tubular ducts present. C. saundersi appears closest to C. chacoensis, but the following features separate C. saundersi from C. chacoensis (character states on C. chacoensis in brackets): (i) 60 or more complex orbicular pores (22–33); a multilocular disc-pore present next to each antenna (absent); and ventral microducts present in rows (scattered). Remarks. The above description is based on a single slide labelled as paratype and agrees well with the original description. According to its collector, L.G. Sanders, the insects were found in the trunk of an undetermined tree, and tended by ants of an undescribed species of the genus Azteca (Laing, 1925). First-instar nymphs were not available during present study. Host plants. Host not determined. Associated Hymenoptera. Dolichoderinae: Azteca sp. (Laing, 1925). Distribution. Neotropical Region: Brazil.Published as part of Kondo, Takumasa, 2010, Taxonomic revision of the myrmecophilous, meliponiphilous and rhizophilous soft scale genus Cryptostigma Ferris (Hemiptera: Coccoidea: Coccidae) 2709, pp. 1-72 in Zootaxa 2709 on pages 54-5
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