7,873 research outputs found
Sans-Coma, V., Mas-Coma, S. & Gosalbez, J. — Mamiferos y Helmintos. Ketres Editora, Barcelona, 1987
No full textJourdane J, Fons Roger. Sans-Coma, V., Mas-Coma, S. & Gosalbez, J. — Mamiferos y Helmintos. Ketres Editora, Barcelona, 1987. In: Revue d'Écologie (La Terre et La Vie), tome 44, n°3, 1989. p. 296
Chill-coma and cold death temperature of Apis mellifera
No full textWorker bees have a lower chill‐coma temperature than drones or queens, which is influenced by their acclimatisation temperature as is their food consumption at a given temperature. Most bees die after 50 hrs. in chill coma, and survive longer at 5° C than at 0 or 10° C. Cold death occurs between −2 and −6° C and is unaffected by acclimatisation. Winter bees have lower chill‐coma temperatures than summer bees but after acclimatisation to 35° C there is no difference. The chill‐coma temperature of summer bees decreases with age irrespective of acclimatisation.
RESP-436
Charopa coma
No full textCharopa coma (Gray, 1843) Helix coma Gray, 1843; Zonites coma; Partula coma globosa Suter, 1892; Charopa pseudocoma Suter, 1984; Endodonta (Charopa) coma var. multicostata Murdoch, 1879; Charopa (Charopa) coma Powell, 1979. Description. Powell (1979). Listed in: Climo (1977a); Powell (1979); Spenser and Willan (1995). Locations. Chatham Is., [New Zealand].Published as part of Pugh, Philip J. A. & Scott, Bronwen, 2002, Biodiversity and biogeography of non-marine Mollusca on the islands of the Southern Ocean, pp. 927-952 in Journal of Natural History 36 (8) on page 933, DOI: 10.1080/00222930110034562, http://zenodo.org/record/530097
Variáveis associadas ao desempenho cognitivo tardio de pacientes com traumatismo crânio-encefálico grave
No full textTese (doutorado) - Universidade Federal de Santa Catarina, Centro de Ciências da Saúde. Programa de Pós-Graduação em Ciências MédicasObjetivos: O trauma cranioencefalico (TCE) e uma das principais causas de mortalidade e morbidade. Ha raros estudos prospectivos que investigam a associacao de variaveis clinicas e laboratoriais da fase aguda do TCE e o prognostico cognitivo tardio dos pacientes vitimas de TCE. Este estudo tem como objetivo identificar variaveis clinicas, laboratoriais e biomarcadores de lesao tecidual associados ao prognostico cognitivo em pacientes vitimas de TCE. Metodos: Foram coletadas prospectivamente as variaveis da internacao hospitalar de 234 pacientes consecutivos com TCE grave (GCS admissao . 8). Dos 172 sobreviventes, uma amostra representada de 46 pacientes realizaram avaliacao cognitiva (composta de 15 testes neuropsicologicos) em media 3 (+ - 1,8) anos apos a hospitalizacao. Um sub-grupo de 22 pacientes que foram avaliados cognitivamente realizaram analise dos niveis plasmaticos de TBARS (indicativo de dano por estresse oxidativo a lipideos) e Carbonil (indicador de dano por estresse oxidativo a proteinas) em amostras de sangue coletadas na fase aguda de TCE (mediana de 10, 30 e 70 horas apos o impacto do TCE). Um grupo controle (n=23) pareado por sexo, idade e nivel socio-educacional foi avaliado cognitivamente para comparacao com os pacientes. Resultados: A media de idade dos pacientes foi 34 (+ - 13) anos sendo 85% do sexo masculino, com escolaridade media de 9 (+ - 4,7) anos. Os pacientes apresentaram um desempenho inferior em todos os testes neuropsicologicos. A analise por regressao linear evidenciou uma forte associacao independente (R coeficiente = 0,6 a 0,8) entre maior escolaridade e menor idade e o desempenho cognitivo em 14 dos 15 testes neuropsicologicos avaliados. O desempenho nos testes cognitivos nao esteve associado ao genero, escore de admissao na Escala de Coma de Glasgow (ECG), exame das pupilas, presenca de trauma em outros orgaos, e classificacao da escala de Marshall na tomografia computadorizada na admissao (TC). Niveis elevados de glicose e presenca de hemorragia sub-aracnoide na TC mostraram-se independentemente associados a um menor desempenho no teste de Retencao de Aprendizagem de Rey e de Memoria Logica respectivamente. Embora os niveis plasmaticos de TBARS e Carbonil tenham sido significativamente elevados na fase aguda do TCE, estes biomarcadores nao se mostraram associados ao desempenho cognitivo dos pacientes. Conclusoes: Baixa escolaridade e idade mais avancada sao preditores independentes de pior desempenho cognitivo tardio apos o TCE grave. O exame de TC e glicemia mostraram limitada capacidade de predicao do desempenho cognitivo enquanto que o exame das pupilas, ECG na admissao, presenca de trauma associado nao foram preditores do desempenho em nenhum dos testes neuropsicologicos avaliados. A medida dos niveis plasmaticos de TBARS e Carbonil tambem nao se mostrou associada com o desempenho cognitivo dos pacientes. A identificacao de variaveis clinicas e laboratoriais associadas ao prognostico cognitivo apos o TCE grave permanece um desafio para a area de neuropsicologia clinica
The Curing Coma Campaign: Framing Initial Scientific Challenges-Proceedings of the First Curing Coma Campaign Scientific Advisory Council Meeting
No full textComa and disordered consciousness are common manifestations of acute neurological conditions and are among the most pervasive and challenging aspects of treatment in neurocritical care. Gaps exist in patient assessment, outcome prognostication, and treatment directed specifically at improving consciousness and cognitive recovery. In 2019, the Neurocritical Care Society (NCS) launched the Curing Coma Campaign in order to address the grand challenge of improving the management of patients with coma and decreased consciousness. One of the first steps was to bring together a Scientific Advisory Council including coma scientists, neurointensivists, neurorehabilitationists, and implementation experts in order to address the current scientific landscape and begin to develop a framework on how to move forward. This manuscript describes the proceedings of the first Curing Coma Campaign Scientific Advisory Council meeting which occurred in conjunction with the NCS Annual Meeting in October 2019 in Vancouver. Specifically, three major pillars were identified which should be considered: endotyping of coma and disorders of consciousness, biomarkers, and proof-of-concept clinical trials. Each is summarized with regard to current approach, benefits to the patient, family, and clinicians, and next steps. Integration of these three pillars will be essential to the success of the Curing Coma Campaign as will expanding the curing coma community to ensure broad participation of clinicians, scientists, and patient advocates with the goal of identifying and implementing treatments to fundamentally improve the outcome of patients
Diagnosing Level of Consciousness: Limits of the Glasgow Coma Scale Total Score
No full textpeer reviewedIn nearly all clinical and research contexts, the initial severity of a traumatic brain injury (TBI) is measured
using the Glasgow Coma Scale (GCS) total score. The GCS total score however, may not accurately reflect
level of consciousness, a critical indicator of injury severity. We investigated the relationship between GCS
total scores and level of consciousness in a consecutive sample of 2455 adult subjects assessed with the
GCS 69,487 times as part of the multi-center Transforming Research and Clinical Knowledge in TBI (TRACKTBI) study. We assigned each GCS subscale score combination a level of consciousness rating based on published criteria for the following disorders of consciousness (DoC) diagnoses: coma, vegetative state/
unresponsive wakefulness syndrome, minimally conscious state, and post-traumatic confusional state, and present our findings using summary statistics and four illustrative cases. Participants had the following characteristics: mean (standard deviation) age 41.9 (17.6) years, 69% male, initial GCS 3–8 = 13%; 9–12 = 5%; 13–15 = 82%.
All GCS total scores between 4–14 were associated with more than one DoC diagnosis; the greatest variability
was observed for scores of 7–11. Further, a wide range of total scores was associated with identical DoC diagnoses. Importantly, a diagnosis of coma was only possible with GCS total scores of 3–6. The GCS total score does
not accurately reflect level of consciousness based on published DoC diagnostic criteria. To improve the classification of patients with TBI and to inform the design of future clinical trials, clinicians and investigators should
consider individual subscale behaviors and more comprehensive assessments when evaluating TBI severityTRACK-TB
Coma and brain death
No full textComa must persist for at least 1 hour to distinguish it from transient unconsciousness. Traumatic and nontraumatic coma are common problems in pediatric practice with high mortality and morbidity. Emergency neuroimaging is worthwhile even when etiology is known, as treatable complications, such as venous sinus thrombosis, as well as extradural and intracerebral hemorrhage, are commonly diagnosed. There is a wide range of possible etiologies in the previously well child, most of which may be diagnosed from neuroimaging and laboratory testing available as an emergency, or can be treated presumptively, e.g., with antimicrobials for infections. The modified Child's Glasgow Coma Scale (CGCS) for recording depth of consciousness in children is widely used and should be supplemented by examination for the signs of reversible central and uncal brainstem herniation due to acute intracranial hypertension. An evidence-based guideline for the investigation and management of decreased level of consciousness in children, written by an expert panel using the DELPHI principles, is available. Monitoring and rehabilitation should also be part of the management plan. Etiology, depth and duration of coma, and serial neurophysiology and imaging are predictors of outcome in survivors but must be interpreted cautiously. There are no reports of children meeting adult brain death criteria making good neurological recovery.</p
HIV Prevention and Social Desirability: Husband–Wife Discrepancies in Reports of Condom Use
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•Resumen:
Greater use of condoms within marriage would help limit the spread of HIV in sub-Saharan Africa. Using data from the Malawi Diffusion and Ideational Change Project (MDICP), the authors examined the influence that the fidelity norm and the traditional association between marriage and reproduction have on condom use with a spouse. The sample included 749 married couples. The authors used latent class analysis to estimate a "true," or latent measure of condom use by couples based on the individual reports of husbands and wives and to explore the reasons why individuals tend to misreport their use of condoms. They found that married couples with more children were more likely to use condoms and that having been informed by experts about AIDS prevention at home induced men and women to overreport condom use within marriage in a survey but may not necessarily increase the extent to which condoms are used.Depto. de Sociología AplicadaFac. de EducaciónTRUEpu
Relação da escala de coma de Glasgow com a introdução de dieta via oral em pacientes com traumatismo crânioencefálico
No full textTCC(graduação) - Universidade Federal de Santa Catarina. Centro de Ciências da Saúde. Fonoaudiologia.Introdução: Conceitua-se o Traumatismo Crânioencefálico (TCE) como qualquer agressão de ordem traumática que acarrete comprometimento anatômico ou funcional do couro cabeludo, crânio, meninges, encéfalo ou de seus vasos. O grau de alteração é comumente determinado utilizando-se a Escala de Coma de Glasgow (ECG) para avaliação do prognóstico funcional após o TCE. Objetivo: Analisar a progressão da dieta dos pacientes com diagnóstico de TCE. Metodologia: Estudo prospectivo de corte transversal que analisou os prontuários de pacientes com TCE. A população pesquisada foi formada por todos os pacientes que possuíram diagnóstico médico de TCE, de ambos os sexos e atendidos no período de maio a setembro de 2013 no Hospital Governador Celso Ramos da cidade de Florianópolis, SC. Resultados: Participaram do estudo 25 pacientes, a maioria do gênero masculino e com média de idade de 46 anos. Com relação à gravidade prevaleceram os TCEs classificados como graves, tendo como primeiro impacto a lesão frontal. O momento de introdução de dieta por via oral segura somente ocorreu quando os pacientes alcançaram níveis altos da EGC (entre 11 e 12). Na admissão os pacientes estavam com dieta zero por via oral (nível 1 da FOIS) e durante a internação e consequente melhora do nível de consciência e alerta evoluíram para dieta por via oral de pelo menos uma consistência (nível 4 da FOIS) e na alta hospitalar alguns evoluíram para mais de uma consistência, porém com necessidade de preparo especial (nível 5 da FOIS). Não houve relação do tempo de internação com a gravidade do trauma e não houve associação entre a gravidade do trauma e o nível de FOIS na alta hospitalar. O uso de via alternativa de alimentação mostrou relação direta com o tempo de internação e o risco para disfagia esteve relacionado ao maior tempo de permanência na UTI e maior uso de TQT. Conclusão: A progressão da dieta dos pacientes com diagnóstico de TCE ocorre com segurança quando ele atinge o nível 12 da ECG. O uso de via alternativa de alimentação mostrou relação direta com o tempo de internação.Introduction: Traumatic Brain Injury (TBI) is defined as any lesion of traumatic origin which results in anatomical or functional damage of the scalp, skull, meninges, brain or its vessels. The degree of impairment is commonly determined using the Glasgow Coma Scale (GCS) to assess the functional outcome after TBI.Objective:To analyze the dietary progression in patients with TBI. Methodology: A prospective cross-sectional study which will examine TBI patients’ medical records. The studied population consisted of all patients who had been medically diagnosed with TBI from, both sexes and undergoing treatment between May and September 2013 in the Governador Celso Ramos Hospital, city of Florianópolis, Brazil. Results: The study enrolled 25 patients, the majority were male, mean age of 46 years. Regarding the severity of TBI, there was a prevalence of severe cases.The introduction of safe oral dietary intake only occurred when patients achieved higher levels in GCS (around 11 and 12). On admission, patients’ oral intake was absent (FOIS level 1); during hospitalization and consequent improvement of consciousness and alertness levels, they were able to progress to oral feeding for at least one consistency (FOIS level 4) and at discharge some individuals presented oral intake for more than one consistency, although requiring special preparation (FOIS level 5). There was no relationship between length of stay and severity of injury, as same as no as sociation between the severity of injury and FOIS level at discharge. The use of alternative feeding route showed direct correlation with the length of stay and risk for dysphagia was related to longer ICU stay and increased use of tracheostomy.Conclusion: The dietary progression in patients with TBI occurs safely when patient reaches level 12 in the Glasgow Coma Scale. The use of alternative feeding routes was directly related to the length of stay
Auditory event-related potentials as indicators of good prognosis in coma of non-anoxic etiology
No full textThe aim of this study is to evaluate whether auditory event-related potentials can predict the prognosis of recovery from coma resulting from different etiologies. The results of this study could then be used as an adjuvant test in helping the clinician evaluate patients in coma. We performed P300 auditory event-related potentials on 21 patients who developed a state of coma at our institution. We compared the results to the Glasgow coma scale at the onset of coma, on day 3, and day 21. We found that patients who developed coma secondary to cardiopulmonary arrest had no P300, and did not develop one, irrespective of their GCS, or their survival. Patients who developed coma from causes other than cardiopulmonary arrest who had a P300 at the onset of their coma, or developed one in the days that followed, ended up surviving their coma. On the other hand, patients in coma from non-cardiac causes who did not have, or developed a P300, did not survive their coma. We concluded that P300 had no prognostic value in coma secondary to anoxic brain injury, while it was an indicator of good prognosis if it was present in patients in coma from nonanoxic causes.Baguley IJ, 1997, ARCH PHYS MED REHAB, V78, P1248, DOI 10.1016-S0003-9993(97)90339-7; DEGIORGIO CM, 1993, ACTA NEUROL SCAND, V87, P423; DOEBRICH HM, 1986, SURG NEUROL, V26, P112; DONCHIN E, 1986, PROG BRAIN RES, V70, P87; GOTT PS, 1991, ARCH NEUROL-CHICAGO, V48, P1267; GREENBERG RP, 1982, J NEUROSURG, V56, P1, DOI 10.3171-jns.1982.56.1.0001; JOHNSON R, 1978, ELECTROEN CLIN NEURO, V44, P424, DOI 10.1016-0013-4694(78)90027-5; KANE NM, 1993, LANCET, V341, P688, DOI 10.1016-0140-6736(93)90453-N; KROPOTOV JD, 1991, ELECTROEN CLIN NEURO, V78, P40, DOI 10.1016-0013-4694(91)90017-X; Lew HL, 1999, AM J PHYS MED REHAB, V78, P367, DOI 10.1097-00002060-199907000-00014; Mazzini L, 2001, ARCH PHYS MED REHAB, V82, P57, DOI 10.1053-apmr.2001.18076; OMAHONY D, 1990, LANCET, V336, P1265, DOI 10.1016-0140-6736(90)92887-N; POLICH J, 1989, ELECTROEN CLIN NEURO, V74, P312, DOI 10.1016-0168-5597(89)90061-0; PRATAPCHAND R, 1988, ACTA NEUROL SCAND, V78, P185; Reuter B. M., 1989, TOPOGRAPHIC BRAIN MA, P192; SIGNORINO M, 1995, LANCET, V345, P255, DOI 10.1016-S0140-6736(95)90252-X; SQUIRES KC, 1976, SCIENCE, V193, P1142, DOI 10.1126-science.959831; YAMAGUCHI S, 1991, ELECTROEN CLIN NEURO, V78, P50, DOI 10.1016-0013-4694(91)90018-Y; YINGLING CD, 1990, LANCET, V336, P873, DOI 10.1016-0140-6736(90)92372-O0
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