202,810 research outputs found
filip-jezek/Physiolibrary.models: Published version
No full text<p>An additional material to Lumped models of the cardiovascular system of various complexity (DOI 10.1016/j.bbe.2017.08.001)</p>
filip-jezek/full-blood-acidbase: Submission to journal
No full text<p>This version contains three types of physicochemical description of plasma to be combined with behavioral models of erythrocytes. In addition, we provide an implementation of a full blood component (that is an indepentdent component with BEox, tO2 and tCO2 state properties) and implementation of Wolf model in versions 3.49 and 3.51, verified by the original VisSim implementation.</p>
FILIP dimerizes via its SMC_prok_B domain.
No full textFLAG-tagged deletion mutants of FILIP and EYFP-tagged FILIP expression vectors were co-transfected into COS-7 cells. Immunoprecipitation was performed using an anti-FLAG antibody. FILIP d248-685 did not co-immunoprecipitate with EYFP-tagged FILIP. IgG, normal mouse IgG as a negative control.</p
Jan Filip (24.12.1900-30.4.1981)
No full textKruta Venceslas. Jan Filip (24.12.1900-30.4.1981). In: Etudes Celtiques, vol. 19, 1982. p. 325
Hsc70 is a binding partner of FILIP.
No full text(A) An SDS-PAGE gel image of the co-purified proteins with a partial fragment of FILIP including the Herpes_BLLF1 domain fused to glutathione S-transferase. (B) FILIP binds to Hsc70 in vivo. Protein lysates were obtained from the adult mouse heart. Immunoprecipitation was performed using an anti-FILIP antibody, and blots were probed with an anti-Hsc70 antibody. (C) The Herpes_BLLF1 domain of FILIP is responsible for binding to Hsc70. The indicated vectors were transfected into COS-7 cells. Immunoprecipitation was performed using an anti-FLAG antibody, and blots were probed using an anti-Hsc70 antibody. IgG, normal mouse IgG as a negative control.</p
Filip Buonaccorsi Kallimach „De legitimo amore”
No full textThe article is an analysis of three works by Filip Kallimach (Filippo Buonaccorsi) regarding marriage (elegy XXII, Epithalamium Fanniae ac Musaei, carmen Ad se ipsum). In the collections of Roman elegies from the Augustian era, there are no wedding pieces. The source of Callimachus’ idea is found by the author of the article in Epithalamium in Stellam et Violentillam by P. Papinius Statius and, indirectly, through the epithalamium of Statius – in the unpreserved elegies of L. Arruntius Stella, a poet of Domitian times.The article is an analysis of three works by Filip Kallimach (Filippo Buonaccorsi) regarding marriage (elegy XXII, Epithalamium Fanniae ac Musaei, carmen Ad se ipsum). In the collections of Roman elegies from the Augustian era, there are no wedding pieces. The source of Callimachus’ idea is found by the author of the article in Epithalamium in Stellam et Violentillam by P. Papinius Statius and, indirectly, through the epithalamium of Statius – in the unpreserved elegies of L. Arruntius Stella, a poet of Domitian times
filip-jezek/Physiolibrary.models: A simulation study of left ventricular decompression using a double lumen arterial cannula prototype during a veno-arterial ECMO
No full text<p>Second major revision includes simplification of the model for the demonstrative application.</p>
Pite'-maffian
No full textI dett arbete skriver jag om arbetet och processen kring mitt examensarbete som var en konsert på skolan till vilken jag skrivit all musik. Ämnen som behandlas är b.la hur det är att spela med syskon och hur det är att leda ett band med två mycket erfarna musiker. Stort fokus ligger på självkritik då jag tycker det är någoting som oftare borde tas upp. Kompositioner:The Pilot (Filip Olofsson) Pledge drive (Filip Olofsson) The keys (Filip Olofsson) Pitch (Filip Olofsson) The stakeout (Filip Olofsson) The Opposite (Filip Olofsson) Extra: Giant steps (John Coltrane) Medverkande:Robert Nordmark - saxofon, Petter Olofsson - bas, Filip Olofsson - trummor</p
The SMC_prok_B domain of FILIP is responsible for the modification of actin stress fibers.
No full text(A) We observed no apparent actin stress fibers in FILIP-expressing NIH3T3 cells. In contrast, actin stress fibers were clearly visible in FILIP d248-685-expressing NIH3T3 cells. Actin fibers were visualized (red) using Alexa 568-conjugated phalloidin. Green, bicistronic GFP expression vector of FILIP. Scale bar = 10 μm. (B) The graph shows the ratio of cells containing actin stress fibers to total cells examined. The ratios were 345/409 for control cells, 233/413 for FILIP-expressing cells, 405/487 for FILIP d248-685-expressing cells, and 208/350 for FILIP d872-1111-expressing cells. *p < 0.01 (Fisher’s exact test).</p
The regulation of spine length by FILIP in primary neurons requires Hsc70.
No full text(A) Treatment with clofibric acid resulted in shorter spines in piriform neurons. Scale bar = 2 μm. The graph shows summary data from 554 spines from 6 neurons treated with vehicle and 571 spines from 4 neurons treated with clofibric acid. *p < 0.01 (B) Expression of FILIP d872-1111 in hippocampal neurons did not result in elongated spines. Scale bar = 1 μm. The graph shows summary data from 882 spines from 5 neurons (control), 851 spines from 5 neurons (FILIP d872-1111), and 696 spines from 5 neurons (FILIP). *p < 0.01 (C) Inhibition of the function of Hsc70 reverses the effects of FILIP on spine length. Scale bar = 1 μm. The graph shows summary data from 863 spines from 6 neurons (control/vehicle), 821 spines from 6 neurons (control/VER-155008), 603 spines from 6 neurons (FILIP/vehicle), and 741 spines from 6 neurons (FILIP/VER-155008). **p < 0.01, *p < 0.05.</p
- …
