1,721,033 research outputs found
Going Beyond Counting First Authors in Author Co-citation Analysis
Get PDFThe present study examines one of the fundamental aspects of author co-citation analysis (ACA) - the way co-citation
counts are defined. Co-citation counting provides the data on which all subsequent statistical analyses and mappings
are based, and we compare ACA results based on two different types of co-citation counting - the traditional type that
only counts the first one among a cited work's authors on the one hand and a non-traditional type that takes into
account the first 5 authors of a cited work on the other hand. Results indicate that the picture produced through this non-traditional author co-citation counting contains more coherent author groups and is therefore considerably clearer. However, this picture represents fewer specialties in the research field being studied than that produced through the traditional first-author co-citation counting when the same number of top-ranked authors is selected and analyzed. Reasons for these effects are discussed
Variations on the Author
Get PDF“Variations on the Author” discusses two of Eduardo Coutinho’s recent films (Um Dia na Vida, from 2010, and Últimas Conversas, posthumously released in 2015) and their contribution to the general question of documentary authorship. The director’s filmography is characterized by a consistent yet self-effacing form of authorial self-inscription: Coutinho often features as an interviewer that rather than express opinions propels discourses; an interviewer that is good at listening. This mode of self-inscription characterizes him as an author who is not expressive but who is nonetheless markedly present on the screen. In Um Dia na Vida, however, Coutinho is completely absent form the image, while Últimas Conversas, on the contrary, includes a confessional prologue that moves the director from the margins to the center of his films. This article examines the ways in which these works stand out in the filmography of a director who offers new insights into the notion of cinematic authorship
Appropriate Similarity Measures for Author Cocitation Analysis
Get PDFWe provide a number of new insights into the methodological discussion about author cocitation analysis. We first argue that the use of the Pearson correlation for measuring the similarity between authors’ cocitation profiles is not very satisfactory. We then discuss what kind of similarity measures may be used as an alternative to the Pearson correlation. We consider three similarity measures in particular. One is the well-known cosine. The other two similarity measures have not been used before in the bibliometric literature. Finally, we show by means of an example that our findings have a high practical relevance.information science;Pearson correlation;cosine;similarity measure;author cocitation analysis
Dispelling the Myths Behind First-author Citation Counts
Get PDFWe conducted a full-scale evaluative citation analysis study of scholars in the XML research field to explore just how different from each other author rankings resulting from different citation counting methods actually are, and to demonstrate the capability of emerging data and tools on the Web in supporting more realistic citation counting methods. Our results contest some common arguments for the continued
use of first-author citation counts in the evaluation of scholars, such as high correlations between author rankings by first-author citation counts and other citation
counting methods, and high costs of using more realistic citation counting methods that are not well-supported by the ISI databases. It is argued that increasingly available digital full text research papers make it possible for citation analysis studies to go beyond what the ISI databases have directly supported and to employ more
sophisticated methods
Gastrointestinalis stromatumorok 1-es típusú neurofibromatosisban = Gastrointestinal stromal tumors in neurofibromatosis type 1
No full textAz 1-es típusú neurofibromatosis, más néven Recklinghausen-kór a leggyakoribb autoszomális dominánsan öröklődő betegségek egyike. A neurofibromatosis génje a 17-es kromoszómán található NF1 tumorszuppresszor gén. A gén mutációja a tumorszuppresszor funkció kiesése következtében benignus és malignus tumorok kialakulásához vezet. Gastrointestinalis manifesztáció az esetek 25%-ában észlelhető, a leggyakrabban GIST. Az irodalomban jól ismert a két betegség szoros asszociációja, jelenleg már több mint 160 esetről van tudomásunk. Neurofibromatosisos betegek 7%-ában alakul ki GIST, és a GIST-betegek között az NF1 előfordulása 150-180-szoros az átlagpopulációhoz képest. A neurofibromatosishoz társuló GIST külön entitás, a sporadikus GIST-től eltérően általában multiplex, és szinte mindig a vékonybélben fordul elő. Enyhe női túlsúllyal, általában fiatalabb korban jelenik meg. Szövettani jellemzői közül az orsósejtes típus, a skeinoid testek jelenléte és a gyakori S100-pozitivitás emelhető ki. Mitotikus aktivitása alacsony, általában a prognózisa is kedvezőbb. A sporadikus GIST-re jellemző c-KIT- és PDGFRA-mutáció igen ritkán fordul elő, azzal a hipotézissel összhangban, hogy a neurofibromatosishoz társuló GIST patogenezise nem c-KIT-függő. Feltételezik, hogy neurofibromatosisban a GIST patomechanizmusa különbözik a sporadikustól, a GIST-tumor megjelenése a neurofibromatosis klinikai spektrumának része. C-KIT- és PDGFRA-mutáció az ismert néhány esetben feltehetőleg a tumorgenezis késői lépéseként alakul ki. A GIST terápiáját forradalmasító imatinib ebben a betegcsoportban nem hatékony, de kellő mennyiségű adat még nem áll rendelkezésre.
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Neurofibromatosis type 1 or Recklinghausen disease is one of the most common hereditary autosomal dominant diseases. The disease-causing gene can be found on chromosome 17 as an NF1 tumor suppressor gene. The mutation of this gene leads to the loss of tumorsuppressor function, which in turn causes the development of benign and malignant tumors. In 25% of the cases gastrointestinal manifestations are found, most often GIST. The close correlation of the two diseases are well known in the literature, there are more than 160 published cases. GIST develops in 7% of patients with neurofibromatosis, and among these patients the occurrence of NF1 is 150-180 times more frequent than in the general population. Neurofibromatosis associated with GIST is a different entity and, unlike sporadic GIST, it is usually multiplex and almost always develops in the small bowel. There is a slightly higher incidence among women than in men, and the disease develops at young age. Histological characteristics include spindle cell type, skeinoid fibers and frequent S100 positivity. Low mitotic activity usually suggests better prognosis. c-KIT and PDGFRA mutation is very rare, in agreement with the hypothesis that the pathogenesis of NF1-GIST is not c-KIT dependent. It is presumed that neurofibromatosis associated and sporadic GIST have different pathogenesis, and that the development of GIST tumor in neurofibromatosis is part of the hereditary disease. c-KIT and PDGFRA mutations – as shown in a few known cases – probably develop at a later step of tumor genesis. Imatinib, which has revolutionized the therapy of GIST, cannot be used in this patient group, however, as of today not enough information is available
koamabayili/VECTRON-author-checklist: VECTRON author checklist
No full textWe have done our best to complete the author checklist relating to the use of animals in the hut study. Note that the objective for the hut study was to evaluate the IRS treatment applications for residual efficacy against Anopheles mosquitoes, including the local An. coluzzii mosquito population. Cows were only used to attract mosquitoes into the huts and no tests were carried out directly on the cows. The author checklist is intended for use with studies where experiments are carried out on animals, which is why we have had such difficulty in completing this for the hut study, as many of the questions do not relate to how the cows were used
Author-wise bibliometric analysis based on entropy.
No full textAuthor-wise bibliometric analysis based on entropy.</p
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