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    UMNH:Mamm:966

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    UMNH:Mamm:966 Voucher Specimen Study Ski

    Inscriptions 966 à 977

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    Inscriptions 966 à 977. In: Revue épigraphique du Midi de la France, tome 3, N°71, 1893. pp. 239-248

    RAAPRAPPORT 966

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    On the size of solutions of the inequality &#966; (ax+b)< &#966; (ax)

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    An estimate is given of the size of a positive integer solution n &#8712; N of the inequality &#966;(an+b)< &#966;(an), gcd(a,b)=1. Experiments indicate that this gives a useful indication of the size of the minimal solution

    On the size of solutions of the inequality &#966; (ax+b)< &#966; (ax)

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    An estimate is given of the size of a positive integer solution n &#8712; N of the inequality &#966;(an+b)< &#966;(an), gcd(a,b)=1. Experiments indicate that this gives a useful indication of the size of the minimal solution

    RAAPNOTITIE 966

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    onderzoeksrappor

    Going Beyond Counting First Authors in Author Co-citation Analysis

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    The present study examines one of the fundamental aspects of author co-citation analysis (ACA) - the way co-citation counts are defined. Co-citation counting provides the data on which all subsequent statistical analyses and mappings are based, and we compare ACA results based on two different types of co-citation counting - the traditional type that only counts the first one among a cited work's authors on the one hand and a non-traditional type that takes into account the first 5 authors of a cited work on the other hand. Results indicate that the picture produced through this non-traditional author co-citation counting contains more coherent author groups and is therefore considerably clearer. However, this picture represents fewer specialties in the research field being studied than that produced through the traditional first-author co-citation counting when the same number of top-ranked authors is selected and analyzed. Reasons for these effects are discussed

    MHY 966 functioned as a PPAR α/γ dual agonist.

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    <p>(A) and (B), Docking simulation was performed to identify interaction between LBD and MHY 966. Docking modes of MHY 966 on the LBD of human PPAR α and γ. MHY 966 has similar binding sites compared with known PPAR α and γ agonist, fenofibrate and rosiglitazone, respectively. For luciferase assay, AC2F cells overexpressing PPAR α (C) and PPAR γ (D) were treated with 1 or 5 μM fenofibrate, rosiglitazone, or MHY 966 for 6 h. Bars represents means ± SEMs of triplicates results. *** P < 0.001, ** <i>P</i> < 0.01 and * <i>P</i> < 0.05 versus cell treated with PC DNA group. C and D, .</p
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