1,721,581 research outputs found
Going Beyond Counting First Authors in Author Co-citation Analysis
Get PDFThe present study examines one of the fundamental aspects of author co-citation analysis (ACA) - the way co-citation
counts are defined. Co-citation counting provides the data on which all subsequent statistical analyses and mappings
are based, and we compare ACA results based on two different types of co-citation counting - the traditional type that
only counts the first one among a cited work's authors on the one hand and a non-traditional type that takes into
account the first 5 authors of a cited work on the other hand. Results indicate that the picture produced through this non-traditional author co-citation counting contains more coherent author groups and is therefore considerably clearer. However, this picture represents fewer specialties in the research field being studied than that produced through the traditional first-author co-citation counting when the same number of top-ranked authors is selected and analyzed. Reasons for these effects are discussed
Variations on the Author
Get PDF“Variations on the Author” discusses two of Eduardo Coutinho’s recent films (Um Dia na Vida, from 2010, and Últimas Conversas, posthumously released in 2015) and their contribution to the general question of documentary authorship. The director’s filmography is characterized by a consistent yet self-effacing form of authorial self-inscription: Coutinho often features as an interviewer that rather than express opinions propels discourses; an interviewer that is good at listening. This mode of self-inscription characterizes him as an author who is not expressive but who is nonetheless markedly present on the screen. In Um Dia na Vida, however, Coutinho is completely absent form the image, while Últimas Conversas, on the contrary, includes a confessional prologue that moves the director from the margins to the center of his films. This article examines the ways in which these works stand out in the filmography of a director who offers new insights into the notion of cinematic authorship
Appropriate Similarity Measures for Author Cocitation Analysis
Get PDFWe provide a number of new insights into the methodological discussion about author cocitation analysis. We first argue that the use of the Pearson correlation for measuring the similarity between authors’ cocitation profiles is not very satisfactory. We then discuss what kind of similarity measures may be used as an alternative to the Pearson correlation. We consider three similarity measures in particular. One is the well-known cosine. The other two similarity measures have not been used before in the bibliometric literature. Finally, we show by means of an example that our findings have a high practical relevance.information science;Pearson correlation;cosine;similarity measure;author cocitation analysis
Dispelling the Myths Behind First-author Citation Counts
Get PDFWe conducted a full-scale evaluative citation analysis study of scholars in the XML research field to explore just how different from each other author rankings resulting from different citation counting methods actually are, and to demonstrate the capability of emerging data and tools on the Web in supporting more realistic citation counting methods. Our results contest some common arguments for the continued
use of first-author citation counts in the evaluation of scholars, such as high correlations between author rankings by first-author citation counts and other citation
counting methods, and high costs of using more realistic citation counting methods that are not well-supported by the ISI databases. It is argued that increasingly available digital full text research papers make it possible for citation analysis studies to go beyond what the ISI databases have directly supported and to employ more
sophisticated methods
Generation and Analysis of Gene-Targeted Mouse Models for Oppenheim's Early-Onset Dyt1 Dystonia
No full textA trinucleotide deletion of GAG in the DYT1 gene that encodes torsinA protein is implicated in the neurological movement disorder of Oppenheim's early-onset dystonia. The function of torsinA and the role of the mutation in causing dystonia are unknown. To gain insight into these unknowns, we made two gene-targeted mouse models: a knockin Dyt1 DeltaGAG (KI) to mimic the mutation found in DYT1 dystonic patients and a knockdown with reduced expression of Dyt1 (KD). KI mice exhibited deficient performance on the beam-walking test, a measure of fine motor coordination and balance, and increased locomotive activity. Ubiquitin- and torsinA-containing aggregates were found in neurons of the pontine nuclei of these mice. KI mice also showed dopaminergic system alterations including a significant decrease in striatal dopamine metabolite 4-hydroxy, 3-methoxyphenacetic acid and a reduction of dopamine receptor types 1 (D1) and 2 (D2). The reduction in the level of D2 receptors may be the cause of the elimination of corticostriatal long-term depression (LTD) also detected in KI mice. Further motor learning behavioral testing revealed a severe deficiency of motor skill adaptation in KI mice that may be correlated with the absence of LTD. KD mice that expressed close to 60% of normal torsinA level showed that a reduced level of torsinA even in the absence of mutant protein is adequate to alter the motor behavioral development of mice. These mice, like the KI mice, had deficient performance on the beam-walking test and had increased locomotive activity. They also had a significantly reduced level of 4-dihydroxyphenylacetic acid, another dopamine metabolite. Our results show that the DeltaGAG mutation in Dyt1 causes abnormalities in fine motor coordination and balance, spontaneous locomotive activity level, and a decline in motor skill adaptation. A reduction in D1 and D2 receptor levels that appears to affect D2-receptor dependent corticostriatal LTD ablation may be the mechanism responsible for the motor skill transfer deficit. In addition, the similarity in behavioral phenotype between the two mutant mouse lines indicates that the DeltaGAG mutation is most likely either a loss-of-function or dominant negative mutation, and not a gain-of-function mutation.Made available in DSpace on 2015-09-28T15:50:11Z (GMT). No. of bitstreams: 2
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Previous issue date: 2006Embargo set by: Seth Robbins for item 88523
Lift date: Forever
Reason: Restricted to the U of I community idenfinitely during batch ingest of legacy ETDsRestricted to the U of I community idenfinitely during batch ingest of legacy ETDsU of I Only104 p.Thesis (Ph.D.)--University of Illinois at Urbana-Champaign, 2006
koamabayili/VECTRON-author-checklist: VECTRON author checklist
No full textWe have done our best to complete the author checklist relating to the use of animals in the hut study. Note that the objective for the hut study was to evaluate the IRS treatment applications for residual efficacy against Anopheles mosquitoes, including the local An. coluzzii mosquito population. Cows were only used to attract mosquitoes into the huts and no tests were carried out directly on the cows. The author checklist is intended for use with studies where experiments are carried out on animals, which is why we have had such difficulty in completing this for the hut study, as many of the questions do not relate to how the cows were used
Author-wise bibliometric analysis based on entropy.
No full textAuthor-wise bibliometric analysis based on entropy.</p
Generation and Analysis of Gene-Targeted Mouse Models for Oppenheim's Early-Onset Dyt1 Dystonia
No full text104 p.Thesis (Ph.D.)--University of Illinois at Urbana-Champaign, 2006.A trinucleotide deletion of GAG in the DYT1 gene that encodes torsinA protein is implicated in the neurological movement disorder of Oppenheim's early-onset dystonia. The function of torsinA and the role of the mutation in causing dystonia are unknown. To gain insight into these unknowns, we made two gene-targeted mouse models: a knockin Dyt1 DeltaGAG (KI) to mimic the mutation found in DYT1 dystonic patients and a knockdown with reduced expression of Dyt1 (KD). KI mice exhibited deficient performance on the beam-walking test, a measure of fine motor coordination and balance, and increased locomotive activity. Ubiquitin- and torsinA-containing aggregates were found in neurons of the pontine nuclei of these mice. KI mice also showed dopaminergic system alterations including a significant decrease in striatal dopamine metabolite 4-hydroxy, 3-methoxyphenacetic acid and a reduction of dopamine receptor types 1 (D1) and 2 (D2). The reduction in the level of D2 receptors may be the cause of the elimination of corticostriatal long-term depression (LTD) also detected in KI mice. Further motor learning behavioral testing revealed a severe deficiency of motor skill adaptation in KI mice that may be correlated with the absence of LTD. KD mice that expressed close to 60% of normal torsinA level showed that a reduced level of torsinA even in the absence of mutant protein is adequate to alter the motor behavioral development of mice. These mice, like the KI mice, had deficient performance on the beam-walking test and had increased locomotive activity. They also had a significantly reduced level of 4-dihydroxyphenylacetic acid, another dopamine metabolite. Our results show that the DeltaGAG mutation in Dyt1 causes abnormalities in fine motor coordination and balance, spontaneous locomotive activity level, and a decline in motor skill adaptation. A reduction in D1 and D2 receptor levels that appears to affect D2-receptor dependent corticostriatal LTD ablation may be the mechanism responsible for the motor skill transfer deficit. In addition, the similarity in behavioral phenotype between the two mutant mouse lines indicates that the DeltaGAG mutation is most likely either a loss-of-function or dominant negative mutation, and not a gain-of-function mutation.U of I OnlyRestricted to the U of I community idenfinitely during batch ingest of legacy ETD
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