478 research outputs found
Samir Parikh, Jared Bartie, Jennifer Johnson, and Ben Lauritsen
No full textLeft to right: Samir Parikh, Jared Bartie, Jennifer Johnson, Ben Lauritsen at the 2023 Martin Luther King, Jr. lecture featuring Jared Bartiehttps://lawcommons.lclark.edu/mlk_2023_photos/1026/thumbnail.jp
Samir Parikh
No full textPencil and ink drawing on paper of Lewis & Clark Law School Professor Samir Parikh.https://lawcommons.lclark.edu/lansing/1077/thumbnail.jp
Samir Parikh, Robin Holmes-Sullivan, Jennifer Johnson, Jared Bartie, and Ben Lauritsen
No full textLeft to right: Samir Parikh, Robin Holmes-Sullivan, Jennifer Johnson, Jared Bartie, Ben Lauritsen at the 2023 Martin Luther King, Jr. lecture featuring Jared Bartiehttps://lawcommons.lclark.edu/mlk_2023_photos/1027/thumbnail.jp
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Samir Parikh
No full textPencil and ink drawing on paper of Lewis & Clark Law School Professor Samir Parikh.https://lawcommons.lclark.edu/lansing/1077/thumbnail.jp
Recommended from our members
Bankruptcy and debtor/creditor ::examples & explanations /
No full text"In this eighth edition, the coauthors, Brian Blum and Samir Parikh, combine their expertise to enhance the book's treatment of all salient areas of bankruptcy and debtor-creditor law. Although there are many changes in the eighth edition, it maintains the format and approach of previous editions. The textual discussion of the principles, goals, policies, and legal rules of Bankruptcy and Debtor-Creditor law is clear and accessible. The Examples & Explanations pedagogy gives the reader practice interpreting the Bankruptcy Code and applying the rules and principles to factual situations"-
Mitochondrial dysfunction in inherited renal disease and acute kidney injury
No full textMitochondria are increasingly recognized as key players in genetic and acquired renal diseases. Most mitochondrial cytopathies that cause renal symptoms are characterized by tubular defects, but glomerular, tubulointerstitial and cystic diseases have also been described. For example, defects in coenzyme Q10 (CoQ10) biosynthesis and the mitochondrial DNA 3243 A>G mutation are important causes of focal segmental glomerulosclerosis in children and in adults, respectively. Although they sometimes present with isolated renal findings, mitochondrial diseases are frequently associated with symptoms related to central nervous system and neuromuscular involvement. They can result from mutations in nuclear genes that are inherited according to classic Mendelian rules or from mutations in mitochondrial DNA, which are transmitted according to more complex rules of mitochondrial genetics. Diagnosis of mitochondrial disorders involves clinical characterization of patients in combination with biochemical and genetic analyses. In particular, prompt diagnosis of CoQ10 biosynthesis defects is imperative because of their potentially reversible nature. In acute kidney injury (AKI), mitochondrial dysfunction contributes to the physiopathology of tissue injury, whereas mitochondrial biogenesis has an important role in the recovery of renal function. Potential therapies that target mitochondrial dysfunction or promote mitochondrial regeneration are being developed to limit renal damage during AKI and promote repair of injured tissue
Ovarian steroid cell tumor in pregnancy-a rare occurrence: Report of a case and review of the literature
No full textBackground: Steroid-cell tumors of the ovary are very rare, especially during pregnancy, and they must be distinguished from luteoma of pregnancy.
Case: An 18-year-old female, gravida 3, para 1-0-1-1, at 38 weeks' of gestation, had an adnexal mass that was discovered incidentally during a Caesarean section. The tumor was excised and her male infant was normal.
Results: Histologic workup revealed the tumor to be a steroid-cell tumor, which is exceedingly rare in pregnancy.
Conclusions: Ovarian steroid-cell tumors, which are malignant one-third of the time, are difficult to distinguish from luteoma of pregnancy.Peer reviewe
sj-pdf-1-jcb-10.1177_0271678X231156500 - Supplemental material for NAD<sup><b>+</b></sup> precursor nutritional supplements sensitize the brain to future ischemic events
No full textSupplemental material, sj-pdf-1-jcb-10.1177_0271678X231156500 for NAD+ precursor nutritional supplements sensitize the brain to future ischemic events by Wensheng Qu, Kenneth M Ralto, Tao Qin, Yinhong Cheng, Weifeng Zong, Xiang Luo, Miguel Perez-Pinzon, Samir M Parikh and Cenk Ayata in Journal of Cerebral Blood Flow & Metabolism</p
Gender and climate change framework for analysis, policy & action
No full text"This paper by Jyoti Parikh provides a framework to anlayse gender and climate change concerns keeping in view the strengths and vulnerability of poor ‐ women in particular. The author also provides policy recommendations for policies and actions.
sj-pdf-2-jcb-10.1177_0271678X231156500 - Supplemental material for NAD<sup><b>+</b></sup> precursor nutritional supplements sensitize the brain to future ischemic events
No full textSupplemental material, sj-pdf-2-jcb-10.1177_0271678X231156500 for NAD+ precursor nutritional supplements sensitize the brain to future ischemic events by Wensheng Qu, Kenneth M Ralto, Tao Qin, Yinhong Cheng, Weifeng Zong, Xiang Luo, Miguel Perez-Pinzon, Samir M Parikh and Cenk Ayata in Journal of Cerebral Blood Flow & Metabolism</p
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