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Chronic cardiac disease should be considered when using left ventricular dimensions to assess volume status and fluid responsiveness
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Metabolism and Pathophysiology of Bariatric Surgery: Nutrition, Procedures, Outcomes and Adverse Effects
Pathophysiology of Bariatric Surgery: Metabolism, Nutrition, Procedures, Outcomes and Adverse Effects uses a metabolic and nutritional theme to explain the complex interrelationships between obesity and metabolic profiles before and after bariatric surgery. The book is sectioned into seven distinct areas, Features of Obesity, Surgical Procedures, Nutritional Aspects, Metabolic Aspects, Diabetes, Insulin Resistance and Glucose Control, Cardiovascular and Physiological Effects, and Psychological and Behavioral Effects. Included is coverage on the various types of bariatric surgery, including Roux-en-Y gastric bypass, gastric banding, sleeve gastrectomy, biliopancreatic diversion, and jejunoileal bypass, as well as the variations upon these procedures. Provides information on diet, nutrition, surgical procedures, outcomes, and side effects in relation to bariatric surgery in one comprehensive text Contains a Dictionary of Terms, Key Facts, and Summary Points in each chapter Includes access to a companion website with accompanying videos.</p
Parkinson's disease, nutrition, and surgery in context of critical care
Parkinson’s disease is a common neurodegenerative disorder with a higher risk of hospitalization than the general population. Therefore, there is a high likelihood of encountering a person with Parkinson’s disease in acute or critical care. Most people with Parkinson’s disease are over the age of 60 years and are likely to have other concurrent medical conditions. Parkinson’s disease is more likely to be the secondary diagnosis during hospital admission. The primary diagnosis may be due to other medical conditions or as a result of complications from Parkinson’s disease symptoms. Symptoms include motor symptoms, such as slowness of movement and tremor, and non-motor symptoms, such as depression, dysphagia, and constipation. There is a large degree of variation in the presence and degree of symptoms as well as in the rate of progression. There is a range of medications that can be used to manage the motor or non-motor symptoms, and side effects can occur. Improper administration of medications can result in deterioration of the patient’s condition and potentially a life-threatening condition called neuroleptic malignant-like syndrome. Nutrients and delayed gastric emptying may also interfere with intestinal absorption of levodopa, the primary medication used for motor symptom management. Rates of protein-energy malnutrition can be up to 15 % in people with Parkinson’s disease in the community, and this is likely to be higher in the acute or critical care setting. Nutrition-related care in this setting should utilize the Nutrition Care Process and take into account each individual’s Parkinson’s disease motor and non-motor symptoms, the severity of disease, limitations due to the disease, medical management regimen, and nutritional status when planning nutrition interventions. Special considerations may need to be taken into account in relation to meal and medication times and the administration of enteral feeding. Nutrition screening, assessment, and monitoring should occur during admission to minimize the effects of Parkinson's disease symptoms and to optimise nutrition-related outcomes
Anterior cingulate cortex, pain perception, and pathological neuronal plasticity during chronic pain
Features and Physiology of Spinal Stretch Reflex Pathways in People with Chronic Spinal Cord Injury
In spastic individuals with chronic spinal cord injury (SCI), the stretch reflex gain and reflex stiffness are high at rest and do not decrease with muscle activation. The soleus H-reflex is also large during voluntary muscle contraction, and the soleus stretch and H-reflexes are abnormally modulated during walking. Hyperexcitable stretch reflexes are most problematic in mid-late swing through early stance as they can trigger clonus and negatively affect locomotion. These observations of stretch reflex behaviors in SCI do not appear to match what has been found in hemiparetic stroke or cerebral palsy. Different from pharmacologically or surgically disabling a reflex pathway, changing a reflex behavior through neurobehavioral training such as locomotor training and reflex operant conditioning could provide new means to enhance motor function recovery in people with SCI
Risk stratification to guide management in cardiogenic shock due to acute myocardial infarction. An illustrative case series and review of the literature
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