106,495 research outputs found
Letter From T. T. Pollard to Eartha White
No full textCorrespondence: Letter from T. T. Pollard, Community Chest Association Executive Committee Secretary, to Miss E. M. M. White requesting her monthly report. Date: February 10, 193
Pollard, John T, TX5249
No full textThis record was harvested from a previous catalogue system and will be withdrawn in 2025. Information in this record may be superseded or incomplete. Visit this record in UMA's new catalogue at: https://archives.library.unimelb.edu.au/nodes/view/411141Surname: POLLARD. Given Name(s) or Initials: JOHN T. Military Service Number or Last Known Location: TX5249. Missing, Wounded and Prisoner of War Enquiry Card Index Number: 21738.226854
Item: [2016.0049.43407] "Pollard, John T, TX5249
Handwritten biographical information on Paulina T. McClung Merritt
No full textA handwritten biography of Paulina T. McClung Merritt by an unknown author, 1892.
Vascular endothelial growth factor restores delayed tumor progression in tumors depleted of macrophages
No full textGenetic depletion of macrophages in Polyoma Middle T oncoprotein (PyMT)-induced mammary tumors in mice delayed the angiogenic switch and the progression to malignancy. To determine whether vascular endothelial growth factor A (VEGF-A) produced by tumor-associated macrophages regulated the onset of the angiogenic switch, a genetic approach was used to restore expression of VEGF-A into tumors at the benign stages. This stimulated formation of a high-density vessel network and in macrophage-depleted mice, was followed by accelerated tumor progression. The expression of VEGF-A led to a massive infiltration into the tumor of leukocytes that were mostly macrophages. This study suggests that macrophage-produced VEGF regulates malignant progression through stimulating tumor angiogenesis, leukocytic infiltration and tumor cell invasion
The drowned and the saved: archaeological perspectives on the sea as grave
No full textNo abstract available
Letter, [Author unclear] to Paulina T. Merritt
No full textHandwritten letter to Paulina Merritt from an unknown author, October 1, 1876.
Identity and dislocation in Caribbean women's literature: a study of the writings of Velma Pollard
No full textJamaican-born Velma Pollard has been publishing poetry and short stories for nearly
thirty years. Her first poems appeared in the 1970s, her first volume of short stories in
1989, and her first novel in 1994. Despite this considerable literary output, in the evergrowing
critical literature on Caribbean women's writing Pollard's work has not attracted
any of the scholarly treatment accorded to other writers. Given this lack of critical
attention to Pollard's considerable body of work, this thesis aims to provide the first
detailed and contextualised study of her writings (excluding the majority of her poetry
and of her writings on linguistics), and to accord Pollard the recognition her work
deserves.
Chapter 1 of this thesis situates Pollard's writings in the context of Caribbean
(women's) literature, and writings on identity, dislocations and (Caribbean) migration. I
argue that Pollard's principal contribution to Caribbean literature is found in her
engagement with two main subjects, return migration and relationships (male-female and
female-female), within a wider context of debates on identity and dislocation.
Chapter 2 introduces Pollard's work by way of a general discussion of her novella
Karl, which won the Casa de las Americas literary award in 1992. I consider Karl to be
central to Pollard's work, not least because it features many of the themes explored by
her later writings, including her novel, Homestretch, which is the subject of Chapter 3.
Pollard's first novel, Homestretch, which was published in 1994, explores the themes
of identity and dislocation through the experiences of 'return migrants' and 'repeat
migrants' and their comparison of life in England, the United States and Jamaica. The
novel chronicles how these migrants come to reconnect with and accept their cultural
heritage.
In chapters 4 and 5 I discuss selected stories taken from Pollard's two collections
of short stories, Considering Woman ('Cages', 'My Sisters', 'My Mother', and 'Gran') and
from Karl and Other Stories ('A Night's Tale', 'Miss Chandra', 'Betsy Hyde', and 'Altamont
Jones'). In these stories Pollard explores male-female relationships and the lives of
several generations and a wide range of Caribbean women and men. Pollard utilises the
West Indian setting, speech, situations and conflicts in these stories to graphically
describe familiar Caribbean role models and to provide a narrative and literary
examination of the frustrations and conflicting desires of women in the region.
In my conclusion, I address the ethnographic quality and significance of her work,
and its contribution to an understanding of the Caribbean
Letter from Stephen T. Mather to Carl Hayden
No full textLetter from Stephen T. Mather to Carl T. Hayden thanking Hayden for his advice about visiting Arizon
Heterogeneous and tissue-specific regulation of effector T cell responses by IFN-gamma during Plasmodium berghei ANKA infection.
No full textIFN-γ and T cells are both required for the development of experimental cerebral malaria during Plasmodium berghei ANKA infection. Surprisingly, however, the role of IFN-γ in shaping the effector CD4(+) and CD8(+) T cell response during this infection has not been examined in detail. To address this, we have compared the effector T cell responses in wild-type and IFN-γ(-/-) mice during P. berghei ANKA infection. The expansion of splenic CD4(+) and CD8(+) T cells during P. berghei ANKA infection was unaffected by the absence of IFN-γ, but the contraction phase of the T cell response was significantly attenuated. Splenic T cell activation and effector function were essentially normal in IFN-γ(-/-) mice; however, the migration to, and accumulation of, effector CD4(+) and CD8(+) T cells in the lung, liver, and brain was altered in IFN-γ(-/-) mice. Interestingly, activation and accumulation of T cells in various nonlymphoid organs was differently affected by lack of IFN-γ, suggesting that IFN-γ influences T cell effector function to varying levels in different anatomical locations. Importantly, control of splenic T cell numbers during P. berghei ANKA infection depended on active IFN-γ-dependent environmental signals--leading to T cell apoptosis--rather than upon intrinsic alterations in T cell programming. To our knowledge, this is the first study to fully investigate the role of IFN-γ in modulating T cell function during P. berghei ANKA infection and reveals that IFN-γ is required for efficient contraction of the pool of activated T cells
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