161,790 research outputs found

    Variations on the Author

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    “Variations on the Author” discusses two of Eduardo Coutinho’s recent films (Um Dia na Vida, from 2010, and Últimas Conversas, posthumously released in 2015) and their contribution to the general question of documentary authorship. The director’s filmography is characterized by a consistent yet self-effacing form of authorial self-inscription: Coutinho often features as an interviewer that rather than express opinions propels discourses; an interviewer that is good at listening. This mode of self-inscription characterizes him as an author who is not expressive but who is nonetheless markedly present on the screen. In Um Dia na Vida, however, Coutinho is completely absent form the image, while Últimas Conversas, on the contrary, includes a confessional prologue that moves the director from the margins to the center of his films. This article examines the ways in which these works stand out in the filmography of a director who offers new insights into the notion of cinematic authorship

    Going Beyond Counting First Authors in Author Co-citation Analysis

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    The present study examines one of the fundamental aspects of author co-citation analysis (ACA) - the way co-citation counts are defined. Co-citation counting provides the data on which all subsequent statistical analyses and mappings are based, and we compare ACA results based on two different types of co-citation counting - the traditional type that only counts the first one among a cited work's authors on the one hand and a non-traditional type that takes into account the first 5 authors of a cited work on the other hand. Results indicate that the picture produced through this non-traditional author co-citation counting contains more coherent author groups and is therefore considerably clearer. However, this picture represents fewer specialties in the research field being studied than that produced through the traditional first-author co-citation counting when the same number of top-ranked authors is selected and analyzed. Reasons for these effects are discussed

    Neun Kirchen-Chor-Gesänge mit Orgel-Begleitung

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    NEUN KIRCHEN-CHOR-GESÄNGE MIT ORGEL-BEGLEITUNG Neun Kirchen-Chor-Gesänge mit Orgel-Begleitung ( - ) Cover ( - ) Inhalt (handschriftlich) ( - ) Titelseite ( - ) Widmung (1) Inhalt (2) I Pater Noster (1) II Ave Maria (4) III O salutaris hostia (7) IV Tantum ergo (13) IV (Bis) Tantum ergo (16) V Ave Verum (20) VI Mihi autem adhaerere (22) VII Ave Maris stella (26) VII (Bis) Ave Maris stella (32) VIII O salutaris hostia (37) IX Libera me (39

    Larry O. Spencer, Conference Author Presentation

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    Gen. Larry O. Spencer, USAF (Ret.), author of Dark Horse: A Journey from the Horseshoe to the Pentago

    Dispelling the Myths Behind First-author Citation Counts

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    We conducted a full-scale evaluative citation analysis study of scholars in the XML research field to explore just how different from each other author rankings resulting from different citation counting methods actually are, and to demonstrate the capability of emerging data and tools on the Web in supporting more realistic citation counting methods. Our results contest some common arguments for the continued use of first-author citation counts in the evaluation of scholars, such as high correlations between author rankings by first-author citation counts and other citation counting methods, and high costs of using more realistic citation counting methods that are not well-supported by the ISI databases. It is argued that increasingly available digital full text research papers make it possible for citation analysis studies to go beyond what the ISI databases have directly supported and to employ more sophisticated methods

    NeuN: a useful neuronal marker for diagnostic histopathology.

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    The monoclonal antibody A60 specifically recognizes the DNA-binding, neuron-specific protein NeuN, which is present in most neuronal cell types of vertebrates. In this study we demonstrate the potential use of NeuN as a diagnostic neuronal marker using a wide range of formalin-fixed, paraffin-embedded human surgical and autopsy specimens from the central and peripheral nervous system. After microwave antigen retrieval, almost all neuronal populations revealed strong immunoreactivity for NeuN in nuclei, perikarya, and some proximal neuronal processes, whereas more distal axon cylinders and dendritic ramifications were not stained. The stain greatly enhanced the gray matter architecture. NeuN immunoreactivity was not detected in Purkinje cells, most neurons of the internal nuclear layer of the retina, and in sympathetic chain ganglia. We examined nine gangliogliomas and 14 dysembryoplastic neuroepithelial tumors, one ganglioneuroma, and one dysplastic cerebellar gangliocytoma. The neuronal component of all of these lesions showed marked immunoreactivity for NeuN. In addition, NeuN immunoreactivity was focally seen in one of seven medulloblastomas with prominent neuronal differentiation. There was no staining of non-neuronal structures. The results indicate that NeuN immunoreactivity is a sensitive and specific neuronal marker in formalin-fixed, paraffin-embedded tissues, and may be useful in diagnostic histopathology. </jats:p

    Bibliographie Leo Perutz : Stand: 26. April 2010

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    In den gut eineinhalb Jahrzehnten, die seit der Bibliographie von Hans-Harald Müller und Wilhelm Schernus (Nr. 140) vergangen sind, hat sich die Perutz-Forschung etabliert. Dies zeigt schon ein flüchtiger quantitativer Vergleich. Der Abschnitt "Wissenschaftliche Untersuchungen" bestand damals aus einer Handvoll Magisterarbeiten und Dissertationen; in der vorliegenden Bibliographie nimmt die Sekundärliteratur mehr als die Hälfte des Raums ein. Die Rechtfertigung einer neuen Perutz-Bibliographie liegt denn auch vor allem darin, ein aktuelles, möglichst vollständiges Verzeichnis der wissenschaftlichen Literatur zu Leo Perutz (unter Einschluß der vor 1990 erschienenen Titel) vorzulegen. Der Abschnitt "Primärliteratur" schließt dagegen chronologisch an Müller und Schernus an

    Changes in the expression of NeuN.

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    <p>Micrographs show immunofluorescence staining for NeuN in the nucleus of neurons in the motor cortex labeled with anti-NeuN. A, E, I and M: vehicle; B, F, J and N: LPS; C, G, K and O: Stx2; D, H, L and P: Stx2+LPS; at two, four, seven and twenty days after the respective treatment. Arrows show nuclei of degenerating neurons (H); asterisk shows normal nuclei of neurons (A). Quantification of phenotypic nuclear abnormalities in neurons at different days and for different treatments (Q). *: significant differences among all treated and control groups, ** significant differences between Stx2 and Stx2+LPS treatments (p <0.001).</p

    Simvastatin reduces caspase-3/NeuN positive cells after SAH.

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    <p>Representative images shown are from cerebral cortex. Vehicle treated animals show numerous caspase-3/NeuN positive cells (<b>F</b> and <b>L</b>, arrow heads). In comparison, there are fewer positive cells in post-SAH simvastatin treated animals (<b>O</b>), and almost no positive caspase-3/NeuN positive cells in the pre-treatment mice (<b>I</b>), none in naïve mice (<b>C</b>). Scale bars are 100 µm in <b>A</b> to <b>C</b>, 50 µm in <b>D</b> to <b>O</b>.</p

    GDNF neuroprotective effect on NMDA-induced cell death revealed by NeuN immunohistochemistry.

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    <p>A-E: NeuN immunoreactivity in the CA1 pyramidal layer of control OHSC (A); NMDA-exposed HOSC for 24 h in absence (B) or in presence of GDNF (C); and of NMDA-exposed HOSC for 48 h in absence (D) or in presence of GDNF (E). F-J: NeuN immunoreactivity in the CA3 pyramidal layer of control OHSC (F); NMDA-exposed HOSC for 24 h in absence (G) or in presence of GDNF (H); and of NMDA-exposed HOSC for 48 h in absence (I) or in presence of GDNF (J). K-O: NeuN immunoreactivity in the granule cell layer of dentate gyrus (DG) of control OHSC (K); NMDA-exposed HOSC for 24 h in absence (L) or in presence of GDNF (M); and of NMDA-exposed HOSC for 48 h in absence (N) or in presence of GDNF (O). Scale bar: 50 µm in A-O. Quantification of NeuN-positive cells in the CA1, CA3 and DG is reported. In each experimental group cells were counted in 9 non-overlapping microscope fields subfields from three different slices. Data are expressed as percentage of NeuN-positive cells counted in control slices. *p<0.05 versus NMDA-exposed HOSCs.</p
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