125,368 research outputs found
Joshua Davis: Author of Spare Parts
Citation: K-State First (2016). Joshua Davis: Author of Spare Parts [Flier]. Manhattan, Kansas: K-State First.Flyer advertising Joshua Davis's author talk at Kansas State University
Steven Johnson Author Talk Poster
K-State Book NetworkA poster advertising an author talk by Steven Johnson at Kansas State University on September 3, 2014. Steven Johnson's book "The Ghost Map" was the 2014-2015 common book
Personal Papers (MS 80-0002)
Letter from Daniel W. Kempner to Arnold K. Mueller sending a check regarding the silver covered glass basket
ATP-independent anoxic activation of ATP-sensitive K+ channels in dorsal vagal neurons of juvenile mice in situ
The role of ATP in anoxic activation of ATP-sensitive K+ (K-ATP) channels was studied in dorsal vagal neurons of mouse brainstem slices. In the whole-cell configuration, cyanide-induced chemical anoxia evoked within 10 s a 300-pA outward current that gave rise to a hyperpolarization of 24 mV. These responses were mimicked by nitrogen-aerated saline, rotenone or diazoxide and abolished by tolbutamide. The cyanide-induced hyperpolarization was due to activation of 70 pS K-ATP channels that were half-maximally blocked by 5 muM internal ATP, Dialyzing the cells with either 1, 20 or 0 mM ATP did not, however, affect the time to onset, the kinetics or the magnitude of the cyanide-induced hyperpolarization. Impairment of ATP consumption by ouabain, vanadate or reduced temperature had no effect either. Thus, anoxia-induced activation of these K-ATP channels cannot be explained by a fall of cellular ATP or a concomitant rise of ADP. Anoxia-related changes of the actin cytoskeleton or the composition of the plasma membrane are also not likely to be involved, as cytochalasin D did not affect the cyanide-evoked hyperpolarization and phosphatidylinositol 4,5-bisphosphate failed to decrease the ATP sensitivity of single K-ATP channels. Finally, because of a lack of effects of reduced/oxidized glutathione and the oxidase blocker diphenyliodonium on the cyanide-induced hyperpolarization, cellular redox state does not appear to be involved. Our results indicate that despite a high sensitivity to ATP in excised patches, anoxic activation of K-ATP channels is independent of cellular ATP. Rather the ATP block seems to be removed as a consequence of impaired mitochondrial function. (C) 2002 IBRO. Published by Elsevier Science Ltd. All rights reserved
K. Mueller, Settlements of the Ptolemies, Louvain (2006)
Ruffini Giovanni. K. Mueller, Settlements of the Ptolemies, Louvain (2006). In: Topoi, volume 15/2, 2007. pp. 673-686
K. Mueller, Settlements of the Ptolemies, Louvain (2006)
Ruffini Giovanni. K. Mueller, Settlements of the Ptolemies, Louvain (2006). In: Topoi, volume 15/2, 2007. pp. 673-686
Mueller, Catherine Mueller (Birth, 1889-11-17)
Address: 76 Glass7318/Pg.176/1889/W F/Germ./Germ./Mrs. K. Drantzgurger, Mid.Original record filed in drawer labeled 'MUELLER-MULLER'
Supplement to Schlögl H, Villringer A, Miehle K, Fasshauer M, Stumvoll M, Mueller K: Metreleptin robustly increases resting-state brain connectivity in treatment-naïve female patients with lipodystrophy. Journal of the Endocrine Society, 2023
Supplement to Schlögl H, Villringer A, Miehle K, Fasshauer M, Stumvoll M, Mueller K: Metreleptin robustly increases resting-state brain connectivity in treatment-naïve female patients with lipodystrophy. Journal of the Endocrine Society, 2023. DOI: bvad07
De novo sulfur SAD phasing of the lysosomal 66.3 kDa protein from mouse
Lakomek K, Dickmanns A, Mueller U, et al. De novo sulfur SAD phasing of the lysosomal 66.3 kDa protein from mouse. Acta Crystallographica Section D. 2009;65(3):220-228
Normal stresses at the gelation transition
A simple Rouse-type model, generalized to incorporate the effects of chemical cross-links, is used to obtain a theoretical prediction for the critical behavior of the normal-stress coefficients Psi(1) and Psi(2) in polymeric liquids when approaching the gelation transition from the sol side. While the exact calculation shows Psi(2)=0, a typical result for these types of models, an additional scaling ansatz is used to demonstrate that Psi(1) diverges with a critical exponent l=k+z. Here, k denotes the critical exponent of the shear viscosity and z the exponent governing the divergence of the time scale in the Kohlrausch decay of the shear-stress relaxation function. For cross-links distributed according to mean-field percolation, this scaling relation yields l=3, in accordance with an exact expression for the first normal-stress coefficient based on a replica calculation. Alternatively, using three-dimensional percolation for the cross-link ensemble we find the value lapproximate to4.9. Results on time-dependent normal-stress response are also presented
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