2,945 research outputs found
Carnet de J.D. CHAUPIN
No full textThis 199-page handbook is a monograph on Notre-Dame de Vaulx, a French village located in the Isère département of the Rhône-Alpes region, authored by J.D. CHAUPIN. It was completed in May 1958 and dedicated by the author to his grand-daughter Danielle Marthe CHAUPIN. The first fifty pages contain a glossary of more than 1500 words and the most popular expressions in matheysin patois.The version available here is a second-generation xerox copy of the manuscript collected by Jacqueline DUC, a specialist of matheysin patois.Ce carnet de 199 pages est une monographie sur le village de Notre-Dame de Vaulx, une commune française située dans le département de l'Isère et la région Rhône-Alpes, rédigée par M. J.D. CHAUPIN. Achevé en mai 1958, il est dédicacé à sa petite fille Danielle Marthe CHAUPIN. Il contient sur une cinquantaine de pages un glossaire de plus de 1500 mots ainsi que les expressions les plus courantes en patois matheysin.La version déposée ici est une photocopie de seconde génération du manuscrit réalisée par Jacqueline DUC, spécialiste des patois matheysins
Élévation du niveau de la mer et subsidence des espaces côtiers : Milliman J.D. et Haq B.U., edit, Sea level rise and coastal subsidence, causes, consequences and strategies
No full textPaskoff Roland. Élévation du niveau de la mer et subsidence des espaces côtiers : Milliman J.D. et Haq B.U., edit, Sea level rise and coastal subsidence, causes, consequences and strategies. In: Annales de Géographie, t. 107, n°600, 1998. p. 250
Élévation du niveau de la mer et subsidence des espaces côtiers : Milliman J.D. et Haq B.U., edit, Sea level rise and coastal subsidence, causes, consequences and strategies
No full textPaskoff Roland. Élévation du niveau de la mer et subsidence des espaces côtiers : Milliman J.D. et Haq B.U., edit, Sea level rise and coastal subsidence, causes, consequences and strategies. In: Annales de Géographie, t. 107, n°600, 1998. p. 250
Near-synchronous and delayed initiation of long run-out submarine sediment flows from a record breaking river-flood, offshore Taiwan
No full textSubsea fiber-optic telecommunication cables can break under fast sediment flows that travel 100s of kilometers through the deep ocean in response to earthquakes and submarine landslides. Similar flows are inferred to form from major river floods whose sediment-laden waters plunge and travel along the seabed. However, the complex initiation of flood-related flows and their hazard potential have not been observed until now. Here we use cable fault data from the Gaoping Canyon/Manila Trench off Taiwan to show that a major river flood, formed during Typhoon Morakot (2009), generated two, long run-out, destructive sediment flows; one during peak flood and the other 3 days later. The latter flow was more damaging with speeds and run-out similar to that of landslide-triggered turbidity currents formed in the same catchment. If the second flow was due to remobilized canyon sediment, it occurred during low earthquake (>Mw 2.0) activity, suggesting other triggering mechanisms
Short-term changes in seafloor character due to flood-derived hyperpycnal discharge: Typhoon Mindulle, Taiwan, July 2004
No full textShort-term changes in seafloor character due to flood-derived hyperpycnal discharge: Typhoon Mindulle, Taiwan, July 2004
No full textGeology of Graham Island, British Columbia
Full text linkby J.D. Mackenzie.Series ; Bulletin (Geological Survey of Canada : 1921). Geological series ; no. 72. Memoir (Geological Survey of Canada) ; 88. Accompanies Southern portion of Graham Island, Queen Charlotte Islands, British Columbia [cartographic material] / geology, J.D. Mackenzie ; geography, British Admiralty and Department of the Naval Service of Canada, Department of Lands, British Columbia, J.D. MacKenzie ; C.O. Senecal, geographer and chief draughtsman. Two folded maps in pocket
J.D. Manning: The Life of a WWII Draftee
No full textabstract: The biography of J.D. Manning tells the story of the first man drafted in the United States at the dawn of World War II. Growing up, he lived an ordinary, small-town life in Washburn, Wisconsin. However, due to a clerical error, by the time he was inducted into the military, J.D. had assumed a second identity. While listed under a different name throughout his military service, J.D. decided to turn the military into a career. He extended his service and went on to Officer Candidate School before serving in the war. Ultimately, J.D. died in the Battle of Cherbourg. His story outlines the importance of humanizing war at a time when statistics and numbers tend to impersonalize such a large, historical event. J.D.'s biography provides an understanding of how even the most ordinary, typical life of a drafted solider during WWII can produce an extraordinary story. J.D. was not special. He was but one death in a body count of over 400,000 American soldiers during the war. Yet, his story teaches us that one does not have to be special to be important. Every American soldier has made a contribution to our country, yet only a select few have ever had their stories told. This biography of J.D. will add one more story to the limited collection existing today
Depolarization and decreased surface expression of K+ channels contribute to NSAID-inhibition of intestinal restitution
No full textNon-steroidal anti-inflammatory drugs (NSAIDs) contribute to gastrointestinal ulcer formation by inhibiting epithelial cell migration and mucosal restitution; however, the drug-affected signaling pathways are poorly defined. We investigated whether NSAID inhibition of intestinal epithelial migration is associated with depletion of intracellular polyamines, depolarization of membrane potential (Em) and altered surface expression of K+ channels. Epithelial cell migration in response to the wounding of confluent IEC-6 and IEC-Cdx2 monolayers was reduced by indomethacin (100μM), phenylbutazone (100μM) and NS-398 (100μM) but not by SC-560 (1μM). NSAID-inhibition of intestinal cell migration was not associated with depletion of intracellular polyamines. Treatment of IEC-6 and IEC-Cdx2 cells with indomethacin, phenylbutazone and NS-398 induced significant depolarization of Em, whereas treatment with SC-560 had no effect on Em. The Em of IEC-Cdx2 cells was: −38.5±1.8mV under control conditions; −35.9±1.6mV after treatment with SC-560; −18.8±1.2mV after treatment with indomethacin; and −23.7±1.4mV after treatment with NS-398. Whereas SC-560 had no significant effects on the total cellular expression of Kv1.4 channel protein, indomethacin and NS-398 decreased not only the total cellular expression of Kv1.4, but also the cell surface expression of both Kv1.4 and Kv1.6 channel subunits in IEC-Cdx2. Both Kv1.4 and Kv1.6 channel proteins were immunoprecipitated by Kv1.4 antibody from IEC-Cdx2 lysates, indicating that these subunits co-assemble to form heteromeric Kv channels. These results suggest that NSAID inhibition of epithelial cell migration is independent of polyamine-depletion, and is associated with depolarization of Em and decreased surface expression of heteromeric Kv1 channels.ID: S0006295207001931; M3: Article; Accession Number: S0006295207001931; Author: L.C. Freeman (b); Author: D.F. Narvaez (a); Author: A. McCoy (a); Author: F.B. von Stein (c); Author: S. Young (b); Author: K. Silver (a); Author: S. Ganta (b); Author: D. Koch (b); Author: R. Hunter (b); Author: R.F. Gilmour (c); Author: J.D. Lillich (a, ⁎); Affiliation: Department of Clinical Sciences, Kansas State University, Manhattan, KS 66506, United States; Affiliation: Department of Anatomy and Physiology, Kansas State University, Manhattan, KS 66506, United States; Affiliation: Department of Biomedical Sciences, Cornell University, Ithaca, NY 14853, United States; Keyword: Non-steroidal anti-inflammatory drugs; Keyword: Intestinal epithelial cells; Keyword: Membrane potential; Keyword: Potassium channels; Number of Pages: 12; Language: English;Source type: Electronic(1)http://search.ebscohost.com/login.aspx?direct=true&db=edselp&AN=S0006295207001931&site=eds-live&scope=sit
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