364 research outputs found
Cicinnus magnapuncta Kaye 1901
No full text<i>Cicinnus magnapuncta</i> (Kaye, 1901) <p>(Figs. 6, 9, 10, 31)</p> <p> <b>Type locality.</b> Trinidad, Tabaquite [NHMUK, syntype examined, designated here as lectotype] <i>Perophora magnapuncta</i> Kaye, 1901: Kaye (1901); Kaye & Lamont (1927).</p> <p> <i>Cicinnus magnapuncta</i> was described and illustrated implicitly from one (but possibly more) specimens collected at Tabaquite, central Trinidad, in June 1898 by W.J. Kaye (Kaye 1901, Kaye & Lamont 1927). Kaye (1901) does not indicate the sex of the type material but his illustration and the single specimen recognized as a type in the NHMUK is female (Fig. 31). The syntype in the NHMUK lacks a collecting data label, though it does bear a label reading “Trinidad, Kaye” and the accession number 1901-72. On the reverse of the accession label, <i>Perophora magnapuncta</i> Kaye is handwritten, in a style similar to that seen for other Kaye types from 1901 in NHMUK, although different from Kaye’s characteristic writing on later types. A red edged type label is also present on the specimen, along with a genitalia preparation label (the genitalia are apparently missing because the slide only contains the terminal two abdominal segments). We therefore believe that this specimen is a syntype, and here designate it as the lectotype with the following labels: C, magnapuncta Keyes [<i>recte</i> Kaye] Type genit.pr. No 6 Mimallonidae / BMNH(E) #805414/ NHMUK010588329/ Type [red edged circular label]/ Trinidad Kaye 1901 -72 [number after 1901 unclear, 72 or 92, written on upper surface of label]; <i>Perophora magnapuncta</i> Kaye [written on lower surface of label]/ LECTOTYPE ♀ <i>Perophora magnapuncta</i> Kaye designated by St Laurent and Cock, 2017 [red handwritten label].</p> <p> <i>Cicinnus magnapuncta</i> was the only mimallonid species described from Trinidad until <i>C. trini</i> described above. Although <i>C. magnapuncta</i> seemed to be endemic to the island, a single male specimen from French Guiana in the MNHN (Fig. 9) may be this species considering the similarities in external appearance to the females and the close affinity of Trinidad Mimallonidae with those of French Guiana. However, due to the lack of males from Trinidad, it is not possible to definitively state at this time that the two populations are conspecific. Interestingly, so far only females of <i>C. magnapuncta</i> have been collected or photographed in Trinidad, thus males seem to either not be strongly attracted to light or are potentially diurnal or crepuscular whereas the females arrive late (23.51 h and 0 0.44 h) at light (K. Sookdeo pers. comm.).</p> <p> Several similar <i>Cicinnus</i> species are known from mainland South America, namely: <i>C. bactriana</i> (Butler, 1878), <i>C. callipius</i> Schaus, 1928, <i>C. candacus</i> Schaus, 1928, <i>C. gaujoni</i> (Dognin, 1922), and <i>C. marona</i> Schaus, 1905. Primary types of all species have been examined by the first author. <i>Cicinnus magnapuncta</i> is unique in having weak maculation, particularly submarginally, such that there is a complete absence of dark petiolate scales. The relatively faint postmedial lines and discal spots, as well as light brown to fawn ground coloration, also can be used to distinguish <i>C. magnapuncta</i> from other species listed previously, which are darker brown or nearly orange in the case of <i>C. marona</i>, and nearly always have stronger maculation.</p> <p> Prior to this work, <i>C. magnapuncta</i> was only known from a single location in Trinidad, therefore we report several new locations for this species, and figure actual specimens (not a painted illustration) for the first time. This species is restricted to forested areas of Trinidad, though the previously mentioned specimen from French Guiana may be this species. In addition to the lectotype collected from Tabaquite in the Central Range, <i>C. magnapuncta</i> has been found on the slopes of the Northern Range.</p> <p> <b>Material examined.</b> (1 ♂ *, 6 ♀ total) <b>TRINIDAD</b>: 2 ♀, Brasso Seco: 14.III.2015 (K. Sookdeo photograph, not collected). 1 ♀, Cumaca Road 0.5 mi: 27.X.1980, M.J.W. Cock [<i>leg.</i>], at MV Light (UWIZM CABI.2457). 3 ♀, Cumaca Road, 4.6 mi: 21.X.1982, M.J.W. Cock [<i>leg.</i>], at MV light (2 ♀ MWJC, 1 ♀ to be deposited USNM). 1 ♀, [Tabaquite]: [VI.1898], Kaye 1901, [lecto] type, BMNH (E)# 805414, NHMUK 010588329 (NHMUK). <b>FRENCH GUIANA:</b> 1 ♂, St. Jean du Maroni: 2.I.1978, T. Porion <i>leg.</i> [*provisionally identified as this species] (MNHN).</p>Published as part of <i>St Laurent, Ryan A. & Cock, Matthew J. W., 2017, Annotated list of Mimallonidae (Lepidoptera, Mimallonoidea) from Trinidad and Tobago, with the description of a new species of Cicinnus Blanchard, 1852 and taxonomic notes, pp. 53-70 in Zootaxa 4268 (1)</i> on pages 60-62, DOI: 10.11646/zootaxa.4268.1.3, <a href="http://zenodo.org/record/579898">http://zenodo.org/record/579898</a>
Television food advertising: counterproductive to children's health? A content analysis using the Australian Guide to Healthy Eating
No full textOBJECTIVE: To undertake a content analysis of the types of foods advertised during programs, which have particular appeal to children and in timeslots where children are likely to be watching television and to assess conformity with the recommendations of the Australian Guide to Healthy Eating (AGHE). SETTING: Adelaide, South Australia METHODS: Sixty-three hours of programs classified as 'C' (programs specifically produced for children six to 13 years of age) or 'G' (programs for general viewing, suitable for children to view without adult supervision) were videotaped. Advertisements with the intent of selling were included in the analysis. Food advertisements were coded for type of food (using the AGHE), network stations, program classification and viewing time. STATISTICS: A chi-squared test was used to see if any difference existed between the network stations, viewing times and program classification for certain types of food advertising. RESULTS: There were 544 food advertisements: 21% percent for core foods; and 79% for non-core foods of the AGHE. Fast foods, chocolate and confectionery made up almost 50% of food advertisements shown on television. CONCLUSIONS: Television food advertising did not support the recommendations for healthy eating contained in the AGHE. Child viewers were exposed to a television environment that predominantly promotes foods high in fat, sugar and salt. These nutrients are associated with obesity, dental caries and chronic diseases such as cardiovascular disease and diabetes. Changes are required to create a media environment more supportive of good nutritional health and one which enables Australian children to make healthier food choices.Julie A. Zuppa, Heather Morton and Kaye P. Meht
Food advertising and broadcasting legislation - a case of system failure?
No full textThis study analysed a sample of food advertisements shown during 63 hours of children's programming to investigate compliance and non-compliance with one of the Australian Children's Television Standards (CTS): CTS 20.2a. This standard regulates the way premium offers may, and may not, be used to sell products to children. Of the 1721 advertisements contained in the sample, 544 (32%) were for food. A significantly higher number of food advertisements (41%) were shown during ‘C’ programs (which are specifically regulated and produced for children six to 13 years of age and suitable for viewing without adult supervision), compared with 30% during the less regulated ‘G’ programs (P= < 0.001) (suitable for children to view without adult supervision but not produced specifically for a child audience). Over one-third of food advertisements (36%) in ‘C’ time contained a premium offer compared with 17% in ‘G’ time (P= < 0.0001). Using a precisely defined interpretation of CTS 20.2a, this study found 30 (31%) of food advertisements breached the standard during ‘C’ programs. This was a significantly higher proportion than the 54 (12%) of breaches in ‘G’ time (P= < 0.0001). From this study, the current regulatory system has not resulted in more responsible food advertising during ‘C’ programs, and the widespread breaches of CTS 20.2a indicate this standard is ineffective as a means of regulating food advertising. The Australian Broadcasting Authority has recognised that children need protection from unfair marketing practices and the improper use of premium offers to promote a food product, therefore CTS 20.2a needs urgent review to make it more effective.Heather Morton, Rosemary Stanton, Julie Zuppa and Kaye Meht
MMWR. Recommendations and reports : Morbidity and Mortality Weekly Report. Recommendations and reports
No full textProblem/Condition: Amyotrophic lateral sclerosis (ALS), commonly known as Lou Gehrig\u2019s disease, is a progressive and fatal neuromuscular disease for which no cure or viable treatment has been identified. ALS, like most noncommunicable Diseases, is not a nationally notifiable disease in the United States The prevalence of ALS in the United States during 2010\u20132011 was estimated to be 3.9 cases per 100,000 persons in the general population. Updated prevalence estimates are needed to help monitor disease status, better understand etiology, and identify risk factors for ALS.Period Covered: 2012\u20132013.Description of System: The National ALS Registry, established in 2009, collects data on ALS patients in the United States to better describe the incidence and prevalence of ALS, examine risk factors such as environmental and occupational exposures, and characterize the demographics of those living with ALS. To identify prevalent cases of ALS, data are compiled from four national administrative databases (maintained by the Centers for Medicare and Medicaid Services, the Veterans Health Administration, and the Veterans Benefits Administration). To identify cases not included in these databases and to better understand risk-factors associated with ALS and disease progression, the Registry also includes data that are collected from patients who voluntarily enroll and complete online surveys.Results: During 2012 and 2013, the Registry identified 14,713 and 15,908 persons, respectively, who met the Surveillance case definition of ALS. The estimated ALS prevalence rate was 4.7 cases per 100,000 U.S. population for 2012 and 5.0 per 100,000 for 2013. Due to revisions to the algorithm and use of death data from the National Death Index, an updated prevalence estimate has been calculated retrospectively for October 19, 2010\u2013December 31, 2011. This updated estimate showed a prevalence rate of 4.3 per 100,000 population and a total of 13,282 cases. Since the inception of the Registry, the pattern of characteristics (e.g., age, sex, and race/ethnicity) among persons with ALS have remained unchanged. Overall, ALS was more common among whites, males, and persons aged 60\u201369 years. The age groups with the lowest number of ALS cases were persons aged 18\u201339 years and those aged 6580 years. Males had a higher prevalence rate of ALS than females overall and across all data sources. These findings remained consistent during October 2010\u2013December 2013.Interpretation: The Registry is the only available data source that can be used to estimate the national prevalence for ALS in the United States Use of both administrative national databases and self-report from patients enables a comprehensive approach to estimate ALS prevalence. The overall increase in the prevalence rate from 4.3 per 100,000 persons (revised) during 2010\u20132011 to 4.7 and 5.0 per 100,000 persons, respectively, during 2012\u20132013 likely is not an actual increase in the number of ALS cases. Rather, this increase might be attributed to improved case ascertainment due to the refinement of the algorithm used to identify definite ALS cases, along with an increased public awareness of the Registry. Registry estimates of ALS prevalence are consistent with findings from long-established ALS registries in Europe and from smaller-scale epidemiologic studies previously conducted in the United StatesPublic Health Actions: Data collected by the National ALS Registry are being used to better describe the Epidemiology of ALS in the United States and to help facilitate research. The combined approach of using national administrative databases and a self-enrollment web portal to collect data is novel and potentially could be used for other non-notifiable Diseases such as Parkinson\u2019s disease or multiple sclerosis.Increased public awareness of the Registry might lead to more ALS cases being identified from the secure web portal (https://www.cdc.gov/als), which can ascertain cases apart from the national administrative databases. For example, in 2014, the ALS Ice Bucket Challenge, a social media\u2013centered campaign, received extensive public visibility and created increased awareness of ALS. The Agency for Toxic Substances and Disease Registry (ATSDR) works closely with ALS advocacy and support groups, researchers, health care professionals, and others to promote the National ALS Registry and to identify all cases of ALS in the United States In addition to estimating the prevalence of ALS, the Registry is being used to collect specimens from patient enrollees through a new biorepository, connect patient enrollees with new clinical trials and epidemiologic studies, and fund studies to help learn more about the etiology of ALS. Additional information about the National ALS Registry is available at http://www.cdc.gov/als or by calling toll-free at 1-877-442-9719.Suggested citation for this article: Mehta, MD1 P, Kaye, PhD2 W, Bryan, MPH3 L, et al. Prevalence of Amyotrophic Lateral Sclerosis \u2014 United States, 2012\u20132013. MMWR Surveill Summ 2016;65(No. SS-8):1\u201312. DOI: http://dx.doi.org/10.15585/mmwr.ss6508a1
MMWR. Morbidity and Mortality Weekly Report, Vol. 67, No. 46, November 23, 2018
No full textPrevalence of Amyotrophic Lateral Sclerosis \u2014 United States, 2015 / Paul Mehta, MD; Wendy Kaye, PhD; Jaime Raymond, MPH; et al.-- Lead in Spices, Herbal Remedies, and Ceremonial Powders Sampled from Home Investigations for Children with Elevated Blood Lead Levels \u2014 North Carolina, 2011\u20132018 / Kim A. Angelon-Gaetz, PhD; Christen Klaus; Ezan A. Chaudhry; et al.Self-Directed Walk With Ease Workplace Wellness Program \u2014 Montana, 2015\u20132017 / Robin P. Silverstein, MS, MPH; Melissa VanderVos, MPH; Heather Welch; et al.Notes from the Field: Multiple Modes of Transmission During a Thanksgiving Day NoroVirus Outbreak \u2014 Tennessee, 2017 / Julia Brennan, MS, MPH; Steffany J. Cavallo, MPH; Katie Garman, MPH; et al.Correction and Republication: Abortion Surveillance \u2014 United States, 2014 --QuickStats: Percentage of All Emergency Department (ED) Visits Made by Patients with Diagnosed Depression, by Sex and Age Group \u2014 National Hospital Ambulatory Medical Care Survey, United States, 201
RoMEO Studies 2: How academics wish to protect their open-access research paper
No full textThis paper is the second in a series of studies (see Gadd, E., C. Oppenheim, and S. Probets. RoMEO Studies 1: The impact of copyright ownership on author-self-archiving. Journal of Documentation. 59(3) 243-277) emanating from the UK JISC-funded RoMEO Project (Rights Metadata for Open-archiving). It considers the protection for research papers afforded by UK copyright law, and by e-journal licences. It compares this with the protection required by academic authors for open-access research papers as discovered by the RoMEO academic author survey. The survey used the Open Digital Rights Language (ODRL) as a framework for collecting views from 542 academics as to the permissions, restrictions, and conditions they wanted to assert over their works. Responses from self-archivers and non-archivers are compared. Concludes that most academic authors are primarily interested in preserving their moral rights, and that the protection offered research papers by copyright law is way in excess of that required by most academics. It also raises concerns about the level of protection enforced by e-journal licence agreement
Roy et al. 2023 Supplemental Information for "Sediment-encased pressure–temperature maturation experiments elucidate the impact of diagenesis on melanin-based fossil color and its paleobiological implications."
No full text<p><em>Supplemental Information for</em></p>
<p>Sediment-encased pressure–temperature maturation experiments elucidate the impact of diagenesis on melanin-based fossil color and its paleobiological implications.</p>
<p>Arindam Roy<em><sup>*</sup></em> (<a href="https://orcid.org/0000-0002-4890-6851">https://orcid.org/0000-0002-4890-6851</a>)</p>
<p>Michael Pittman<em><sup>*</sup></em> (<a href="https://orcid.org/0000-0002-6149-3078">https://orcid.org/0000-0002-6149-3078</a>)</p>
<p>Thomas G. Kaye (<a href="https://orcid.org/0000-0001-7996-618X">https://orcid.org/0000-0001-7996-618X</a>)</p>
<p>Evan T. Saitta (<a href="https://orcid.org/0000-0002-9306-9060">https://orcid.org/0000-0002-9306-9060</a>)</p>
<p>*Corresponding author(s)</p>
<p><strong>Email: </strong><a href="mailto:[email protected]">[email protected]</a> ; <a href="http://palaeopittman.com/2022/03/27/lab-members/[email protected]">[email protected]</a></p>
<p>The Dataset contains two files, (1) Supporting Information and (2) Supporting Data PCA worksheet. </p>
<p>The first contains Supplementary tables and figures (.docx file) while the structural organisation of the second (.xlsx file) is provided below:</p>
<p>Authors: </p>
<p>Arindam Roy* (https://orcid.org/0000-0002-4890-6851)<br>
Michael Pittman* (https://orcid.org/0000-0002-6149-3078)<br>
Thomas G. Kaye (https://orcid.org/0000-0001-7996-618X)<br>
Evan T. Saitta (https://orcid.org/0000-0002-9306-9060)<br>
*Corresponding author(s)</p>
<p>README: </p>
<p>We received ToF-SIMS data (Samples 1–30, 34–49) pertaining to purified melanosome extracts of modern bird feathers (both fresh and capsule-matured) from Caitlin Colleary (Associate Curator of Vertebrate Paleontology, Cleveland Museum of Natural History), based on their previous work (Colleary et al. 2015). Citation below: </p>
<p>Colleary, C., A. Dolocan, J. Gardner, S. Singh, M. Wuttke, R. Rabenstein, J. Habersetzer, S. Schaal, M. Feseha, M. Clemens, B. F. Jacobs, E. D. Currano, L. L. Jacobs, R. L. Sylvestersen, S. E. Gabbott, and J. Vinther. 2015. Chemical, experimental, and morphological evidence for diagenetically altered melanin in exceptionally preserved fossils. Proceedings of the National Academy of Sciences USA 112(41):12592-7. doi: https://doi.org/10.1073/pnas.1509831112</p>
<p>We further augmented this data set by adding ToF-SIMS spectra from our own samples(31-33, 50-77) pertaining to sediment encased maturation experiments (190ºC to 300ºC) and fossilised feathers of paravian dinosaurs housed at the Shandong Tianyu Museum of Natural History, Linyi Shi, Shandong, China.</p>
<p>We conducted Principal Components Analysis with this Data and this dataset effectively serves as a PCA worksheet. The file can be opened/edited using Microsoft 365 Excel (.xlsx) with the following organisation of sheets.</p>
<p>Sheets: </p>
<p>|----- PCA whole : contains Sample ID, treatment categories, mass by charge (m/Z) ratios of peaks, peak identity and raw intensity counts<br>
|-----PCA Norm : same data as PCA whole but peak raw intensity counts normalised.<br>
|-----PCA Mean Centered: same data as PCA Norm but with peak raw intensity counts normalised.<br>
|-----PCA All Loading Matrix: Loading matrix for PCA using all peaks.<br>
|-----PCA Scores<br>
|-----PCA No Lipids RAW: same data as PCA whole but without peaks suspected to arise from lipids (e.g., CxH- ) <br>
|-----PCA No Lipids Norm: same data as PCA without Lipids RAW but with peak raw intensity counts normalised.<br>
|-----PCA No Lipids MeanCentred: same data as PCA without Lipids Norm but with peak raw intensity counts normalised.<br>
|-----PCA No Lipids Loading Matrix: Loading matrix for PCA using peaks excluding peaks of lipid origin (CxH-).<br>
|-----PCA No Lipids Scores</p>
<p><br>
The dataset can be created in Microsoft Office 365 (Excel: .xlsx file). The file can also be also be opened and edited using the following softwares.<br>
1. Google Sheets<br>
2. Apache Open Office<br>
3. Libre Office<br>
4. PAST 4 (free software for scientific data analysis, with functions for data manipulation, plotting, univariate and multivariate statistics, ecological analysis, time series and spatial analysis, morphometrics and stratigraphy).</p>
Amyotroph Lateral Scler Frontotemporal Degener
No full textObjective:The majority of cases of the fatal neurodegenerative disease amyotrophic lateral sclerosis (ALS) are of unknown etiology. A proportion of these cases are likely to be attributable to contaminant exposures, although the specific environmental etiology of ALS remains largely a mystery. Certain forms of the neurotoxic metal mercury readily cross into the central nervous system. Fish is a dietary source of methymercury, but also of beneficial components, such as omega-3 polyunsaturated fatty acids. Prior work using clinic-based studies of toenails and hair as keratinous biomarkers of exposure suggest elevated mercury levels in ALS patients compared with controls. We sought to validate this relationship in a U.S. case-control comparison of mercury levels in nail clippings.Methods:We performed trace element analysis using inductively coupled plasma mass spectrometry (ICP-MS) on the nail clippings of n=70 female, geographically representative ALS patients from the National ALS Biorepository and compared them to n=210 age-matched controls from a set of n=1216 nationally distributed controls from the Sister and Two Sister Studies.Results:Compared to the lowest quartile of nail mercury, moderate levels were associated with decreased risk of ALS (P=4.18e-6). However, the odds of having nail mercury levels above the 90th percentile were 2.3 fold higher among ALS patients compared with controls (odds ratio (OR)=2.3, 95% confidence interval 1.10 \u2013 4.58, adjusted for age and smoking status).Conclusions:This finding suggests that excessive mercury exposure may be associated with the neurodegenerative health of aging populations.ZIA ES103086/ImNIH/Intramural NIH HHSUnited States/P42 ES007373/ES/NIEHS NIH HHSUnited States/P30 CA023108/CA/NCI NIH HHSUnited States/CC999999/ImCDC/Intramural CDC HHSUnited States/Z01 ES044005/ImNIH/Intramural NIH HHSUnited States/R01 TS000288/TS/ATSDR CDC HHSUnited States
Roy et al. 2023 Supplemental Information for "Sediment-encased pressure–temperature maturation experiments elucidate the impact of diagenesis on melanin-based fossil color and its paleobiological implications."
No full text<p><em>Supplemental Information for</em></p>
<p>Sediment-encased pressure–temperature maturation experiments elucidate the impact of diagenesis on melanin-based fossil color and its paleobiological implications.</p>
<p>Arindam Roy<em><sup>*</sup></em> (<a href="https://orcid.org/0000-0002-4890-6851">https://orcid.org/0000-0002-4890-6851</a>)</p>
<p>Michael Pittman<em><sup>*</sup></em> (<a href="https://orcid.org/0000-0002-6149-3078">https://orcid.org/0000-0002-6149-3078</a>)</p>
<p>Thomas G. Kaye (<a href="https://orcid.org/0000-0001-7996-618X">https://orcid.org/0000-0001-7996-618X</a>)</p>
<p>Evan T. Saitta (<a href="https://orcid.org/0000-0002-9306-9060">https://orcid.org/0000-0002-9306-9060</a>)</p>
<p>*Corresponding author(s)</p>
<p><strong>Email: </strong><a href="mailto:[email protected]">[email protected]</a> ; <a href="http://palaeopittman.com/2022/03/27/lab-members/[email protected]">[email protected]</a></p>
<p>The Dataset contains two files, (1) Supporting Information and (2) Supporting Data PCA worksheet. </p>
<p>The first contains Supplementary tables and figures (.docx file) while the structural organisation of the second (.xlsx file) is provided below:</p>
<p>Sheets: </p>
<p>|----- PCA whole : contains Sample ID, treatment categories, mass by charge (m/Z) ratios of peaks, peak identity and raw intensity counts.</p>
<p>|-----PCA Norm : same data as PCA whole but peak raw intensity counts normalised.</p>
<p>|-----PCA Mean Centered: same data as PCA Norm but with peak raw intensity counts normalised.</p>
<p>|-----PCA All Loading Matrix: Loading matrix for PCA using all peaks.</p>
<p>|-----PCA without Lipids RAW: same data as PCA whole but without peaks suspected to arise from lipids (e.g., CxH- ) </p>
<p>|-----PCA without Lipids Norm: same data as PCA without Lipids RAW but with peak raw intensity counts normalised.</p>
<p>|-----PCA without Lipids MeanCentred: same data as PCA without Lipids Norm but with peak raw intensity counts normalised.</p>
<p>|-----PCAwithoutLipidsLoading Matrix: Loading matrix for PCA using peaks excluding peaks of lipid origin (CxH-).</p>
<p>The dataset can be created in Microsoft Office 365 (Excel: .xlsx file). The file can also be also be opened and edited using the following softwares.</p>
<ol>
<li>Google Sheets</li>
<li>Apache Open Office</li>
<li>Libre Office</li>
<li>PAST 4 (free software for scientific data analysis, with functions for data manipulation, plotting, univariate and multivariate statistics, ecological analysis, time series and spatial analysis, morphometrics and stratigraphy).</li>
</ol>
Exploring parents' perceptions of television food advertising directed at children: a South Australian study
No full text- …
