1,164 research outputs found
Sur la condition de Carleson dans la boule unité C^m
Sia B la sfera unitaria di C^m. Si danno condizioni equivalenti affinchè una misura μ sia di Carleson. Si generalizza il risultato a domini di classe C^∞ limitati in R^n
Equivalence of two series of spherical representations of a free group
The spherical principal series of a non-commutative free group may be analytically continued to yield a series of uniformly bounded representations, much as the spherical
representations π_{(1/2)+ it} of SL(2, R) may be analytically continued in the strip 0 < Re z < 1. This series of uniformly bounded representations was constructed and studied by A. M. MANTERO and A. ZAPPA. Independently T. PYTLIK and R. SZWARC introduced and studied representations of the free group which contain a series of subrepresentations indexed by spherical functions. Both series consist of irreducible representations and include the spherical complementary series. The aim of this paper is to prove that the non-unitary uniformly bounded representations of the two series are also equivalent
Boundary behavior of generalized Poisson integrals on buildings of type A~2
Let Î be an affine building of type A~2and Ω its maximal boundary. We prove that, for every function fâ L1(Ω) , restricted convergence of the normalized generalized Poisson transform PÏf/ PÏ1 to f holds almost everywhere
Impact of exacerbations in the natural course of COPD
Exacerbations represent an important event in the natural history of patients with chronic obstructive pulmonary disease (COPD). They are associated with considerable physiological deterioration and increased airway inflammatory changes, and may enhance disease progression by accelerating the decline in lung function. Some patients are prone to frequent exacerbations, which are an important cause of hospital admission and readmission, and these frequent episodes may have considerable impact on quality of life, activities of daily living and mortality. Although exacerbations become more frequent and more severe as COPD progresses, the rate at which they occur appears to reflect an independent susceptibility phenotype: the “frequent exacerbator
Acute exacerbations of COPD: risk factors for failure and relapse
Marco Mantero,1,2 Paola Rogliani,3 Marta Di Pasquale,1,2 Eva Polverino,4 Ernesto Crisafulli,5 Monica Guerrero,6 Andrea Gramegna,1,2 Mario Cazzola,3 Francesco Blasi1,2 1Department of Pathophysiology and Transplantation, Università degli Studi di Milano, 2Internal Medicine Department, Respiratory Unit and Regional Adult Cystic Fibrosis Center, IRCCS Fondazione Cà Granda Ospedale Maggiore Policlinico, Milan, 3Respiratory Unit, Department of Systems Medicine, University of Rome Tor Vergata, Rome, Italy; 4Respiratory Disease Department, Servei de Pneumologia, Hospital Universitari Vall d’Hebron (HUVH), Institut de Recerca Vall d’Hebron (VHIR), Barcelona, Spain; 5Department of Medicine and Surgery, Respiratory Disease and Lung Function Unit, University of Parma, Parma, Italy; 6Hospital d’Igualada, Consorci Socisanitari de l’Anoia, Barcelona, Spain Abstract: Acute exacerbations are a leading cause of worsening COPD in terms of lung ­function decline, quality of life, and survival. They also have a relevant economic burden on the health care system. Determining the risk factors for acute exacerbation and early relapse could be a crucial element for a better management of COPD patients. This review analyzes the current knowledge and underlines the main risk factors for recurrent acute exacerbations. Comprehensive evaluation of COPD patients during stable phase and exacerbation could contribute to prevent treatment failure and relapses. Keywords: infections, prevention, treatment, COPD, exacerbations 
The reduced group C*-algebra of a triangle building
Let A be an affine building of type Ã2 and let Γ be a discrete group of type-rotating automorphisms acting simply transitively on the vertices of Δ. We prove that the reduced group C*-algebra C*_r(T) is simple. To prove this result we use the sufficient condition for the simplicity of C*_r(T) given in a recent paper by M. Bekka, M. Cowling and P. de la Harpe
Hypertension in Cushing's Syndrome: from Pathogenesis to treatment
Hypertension is one of the most distinguishing features of endogenous Cushing’s syndrome (CS), as it is present in about 80% of adult patients whereas in children its prevalence is about 47%. Hypertension in CS is significantly correlated with the duration of hypercortisolism and results from the interplay between several pathophysiological mechanisms regulating plasma volume, peripheral vascular resistance and cardiac output, all of which are increased in this state. Glucocorticoids cause hypertension through several mechanisms: their intrinsic mineralocorticoid activity; through activation of the renin-angiotensin system; by enhancement of vasoactive substances, and by causing suppression of the vasodilatory systems. In addition, glucocorticoids may exert some hypertensive effects on cardiovascular regulation through the CNS via both glucocorticoid and mineralocorticoid receptors. Hypertension in CS usually resolves with surgical removal of the tumor, but some patients require pharmacological antihypertensive treatment both pre- and postoperatively. Thiazides and furosemide should be avoided, while adrenergic blockade and calcium channel antagonists are usually ineffective. Mineralocorticoid receptor antagonists, Ang II blockers and ACE inhibitors are good anti-hypertensive options; PPAR-γ agonists may help in many aspects of the insulin resistance syndrome. The relatively selective glucocorticoid receptor antagonist Mifepristone (RU 486) could reduce blood pressure in patients with CS. Neuromodulatory agents such as the serotonin inhibitors cyproheptadine and ritanserin, valproid acid, dopamine agonists, somatostatin analogs may occasionally be effective, as well as drugs acting directly at the adrenal levels, such as Ketoconazole and aminoglutetimide or even opDDD. Treating hypertension in CS remains a difficult task and a big challenge, in order to decrease the morbidity and mortality associated with the disease.</jats:p
Update on the management of pediatric acute osteomyelitis and septic arthritis
Acute osteomyelitis and septic arthritis are two infections whose frequencies are increasing in pediatric patients. Acute osteomyelitis and septic arthritis need to be carefully assessed, diagnosed, and treated to avoid devastating sequelae. Traditionally, the treatment of acute osteoarticular infection in pediatrics was based on prolonged intravenous anti-infective therapy. However, results from clinical trials have suggested that in uncomplicated cases, a short course of a few days of parenteral antibiotics followed by oral therapy is safe and effective. The aim of this review is to provide clinicians an update on recent controversies and advances regarding the management of acute osteomyelitis and septic arthritis in children. In recent years, the emergence of bacterial species resistant to commonly used antibiotics that are particularly aggressive highlights the necessity for further research to optimize treatment approaches and to develop new molecules able to fight the war against acute osteoarticular infection in pediatric patients
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