747 research outputs found
Retention data
No full textRetention data used in publications in or submitted to Journal of Chromatography A with A. R. Horner as first author.
Retention data for ~ 100 compounds on a Waters BEH C-18 column in acidic acetonitrile/water mobile phases at phase ratio and temperatures giving a range of k for each compound about 1 - 100. These are in the CSV file "RetentionData". The compounds/solutes are identified by number. The correspondence between the number and the compound name is in "Compound List and Conditions". Enthalpies for the compounds and functional group counts are in FGEnthalpyData.xls
2009 economic analysis responding to recession, preparing for recovery
No full textTitle from PDF cover (viewed on Feb. 2, 2010).; At head of title: Ohio Department of Job and Family Services, to strengthen Ohio's families with solutions to temporary challenges.; "Workforce Research Chief Lewis Horner prepared this report with the assistance of Labor Market Analyst Jonathan Calig and under the direction of Bureau [of Labor Market Information] Chief Keith Ewald and Assistant Bureau Chief Rudy Wilkinson"--P. 4 of cover.; "7/2009"--P. 4 of cover.; Includes bibliographical references (p. 25).; Harvested from the web on 2/2/1
Causes of Horner Syndrome: A Study of 318 Patients
No full textBackground: Some reports have indicated that when a cause for Horner syndrome can be determined, it is most often chest and neck tumors and stroke. Others have suggested that Horner syndrome is more frequently caused by surgical procedures in the neck and chest. These differences may be explained by disparate accrual methods. Therefore, we decided to compare the cause of Horner syndrome in cases where the diagnosis was confirmed by apraclonidine testing conducted by ophthalmologists to cases in which the diagnosis was made entirely on clinical grounds mostly by nonophthalmologists. Methods: We applied a new search engine to the inpatient and outpatient electronic medical records text at the University of Michigan Medical Center from 1996 to 2018 for Horner syndrome with and without pharmacologic confirmation through ocular instillation of apraclonidine 0.5%. Among apraclonidine-confirmed cases, 159 met inclusion criteria. Among apraclonidine-unconfirmed cases, more than 2,000 cases were identified, so that we included only the first 159 cases that met inclusion criteria. In these 318 cases, we documented patient demographics, ophthalmologic features, imaging, underlying cause, and whether the cause was discovered before or after the diagnosis of Horner syndrome. Results: In the cohort of 159 apraclonidine-confirmed cases of Horner syndrome, a cause was identified in 97 (61%). Procedures in the neck, chest, skull base, and paraspinal region accounted for most of the identified causes, with cervical carotid dissection the next most common cause. In a cohort of 159 cases of Horner syndrome not tested with apraclonidine because the clinical diagnosis appeared firm, procedures again accounted for the largest percentage, but tumor was the next most common cause. In both groups, when a cause for Horner syndrome could be identified, that cause was nearly always known before Horner syndrome was identified. However, in an important minority of cases, mostly involving carotid dissection or tumor, the identification of Horner syndrome was critical to the discovery of those conditions. Conclusions: The prevalence of causes of Horner syndrome depends on the accrual method. Among pharmacologically-confirmed cases, the cause was often undetermined or due to a preceding neck or chest procedure. Among pharmacologically-unconfirmed cases, a substantial proportion had also been caused by neck and chest procedures, but tumors in that region were also common. When a cause of Horner syndrome was found in both cohorts, it was usually known before Horner syndrome was discovered, making Horner syndrome an afterthought. However, in an important minority of cases where the cause was not yet known, the identification of Horner syndrome was valuable in leading to important diagnoses such as carotid dissection and tumor
Postoperative Horner Syndrome After Lung Transplantation
No full textHorner syndrome arises from a disruption along the oculosympathetic efferent chain and can be caused by a variety of pathological and iatrogenic etiologies. We present 3 cases of postoperative Horner syndrome after bilateral lung transplantation
Horner Syndrome from a Pediatric Otolaryngology Perspective.
No full text© The Author(s) 2019. Introduction: Horner syndrome is described as the clinical triad of miosis, ptosis, and anhidrosis. In pediatric patients the condition may be congenital or acquired from neoplastic, infectious or traumatic conditions, including birth trauma. Most cases of pediatric Horner syndrome present first to a pediatric ophthalmologist however since the neural pathways involve the cervical sympathetic chain otolaryngologists should understand the pathophysiology to avoid delay in management of potentially malignant cases. Objectives: To aid otolaryngologists in recognizing and managing pediatric Horner syndrome by describing 3 unique cases from malignant, traumatic and/or congenital causes. Methods: Case report of 3 pediatric patients with Horner syndrome presenting to our pediatric otolaryngology department. Results: Case #1 is 5-month-old female with ptosis and a left level II 1.5 cm neck mass. Magnetic resonance imaging showed the mass displacing the common carotid artery and excisional biopsy revealed a poorly differentiated neuroblastoma. Case #2 is a 9-year-old female with anisocoria appearing after suffering a severe playground injury. Case #3 is a 3-year-old-male who developed ptosis and anisocoria following re-excision of a recurrent cervical lymphatic malformation. Conclusion: Pediatric Horner syndrome may be a benign finding that is easily overlooked but may reflect a serious underlying condition. Otolaryngologists should be aware of the pathophysiology and differential diagnosis, including malignant causes, to appropriately manage patients
Letter from Jonathan Williams to Alden Partridge, 13 December 1813
No full textStates status of various instruments and equipment at West Point; reiterates request for bookTranscription by Cameron Myette and Alison Horner. Transcriptions are subject to human error
Letter from Jonathan Williams to Alden Partridge, 16 May 1813.
No full textPrivate Porter had borrowed shelvings from a local farmer and now the farmer wants them, or their value, returned to him.Transcription by Cameron Myette and Alison Horner. Transcriptions are subject to human error
Letter from Jonathan Williams to Alden Partridge, 23 September 1813
No full textAsks that papers relevant to the U.S. Military Philosophical Society that might be at West Point be delivered to his address at New York City.Transcription by Cameron Myette and Alison Horner. Transcriptions are subject to human error
Retention data
No full textRetention data used in publications in or submitted to Journal of Chromatography A with A. R. Horner as first author.
Retention data for ~ 100 compounds on a Waters BEH C-18 column in acidic acetonitrile/water mobile phases at phase ratio and temperatures giving a range of k for each compound about 1 - 100
Letter from Jonathan Williams to Alden Partridge, 8 December 1813
No full textRequests a book belonging to the U.S. Military Philosophical Society in the library at West Point be sent him; asks that Partridge inventory and take possession of all belongings of the Society and send them to New York.Transcription by Cameron Myette and Alison Horner. Transcriptions are subject to human error
- …
