3,864 research outputs found
Revisiting the Composition of Ezra-Nehemiah: In Conversation with Jacob Wright’s Rebuilding Identity: The Nehemiah Memoir and its Earliest Readers (BZAW, 348; Berlin: de Gruyter, 2004)
No full text This conversation with Jacob L. Wright, Rebuilding
Identity: The Nehemiah Memoir and its Earliest Readers (BZAW, 348; Berlin: de
Gruyter, 2004) began in a special session of the Chronicles-Ezra-Nehemiah
section that was held at the national meeting of the Society of Biblical
Literature in November 2006 (Washington, DC). It includes an introduction by the
editor and contributions by Deirdre N. Fulton, David M. Carr, Ralph W. Klein and
a response by Jacob L. Wright. </jats:p
Schooling and education.
No full textSchooling and education by Giles R. Wright with Howard L. Green and Lee R. Parks. Number 4 in the New Jersey Ethnic Life Series. Published by New Jersey Historical Commission
Making a Name for Oneself: Procreation, Martial Prowess and Heroic Death
No full textFor the 2010-11 Annual Lecture in “Milieux Bibliques” (chair occupied by Prof. Thomas Römer), Jacob L. Wright from Emory University treated the subject of “name-making” as represented in ancient Near Eastern sources and the Hebrew Bible. Wright began by citing a letter that the Assyrian king Shamshi-Adad (18th cent. BCE) sent to his son Yasmah-Adad in Mari, petitioning him to “be a man” and to “make a name” for himself by conquering the city of Qatna: Here your brother won a victory, but the..
Jacob L. Wright, David, King of Israel, and Caleb in Biblical Memory, New York, Cambridge University Press, 2014
No full textHunziker-Rodewald Regine. Jacob L. Wright, David, King of Israel, and Caleb in Biblical Memory, New York, Cambridge University Press, 2014. In: Revue d'histoire et de philosophie religieuses, 96e année n°4, Octobre-Décembre 2016. pp. 446-447
Se faire un nom : Procréation, prouesses martiales et mort héroïque dans l’ancien Israël
No full textJacob L. Wright a introduit son sujet par la citation d’une lettre du roi assyrien Shamshi-Adad (xviiie s. av. n. è.) envoyée à son fils Yasmah-Adad à Mari, afin de l’inviter à « être un homme » et à « se faire un nom » en conquérant la ville de Qatna : Ici ton frère a remporté une victoire, mais de ton côté tu demeures parmi les femmes ! Maintenant, alors que tu marches avec ton armée en direction de Qatna, sois un homme ! À la manière de ton frère qui s’est établi « une grand renommée » [š..
Archaelogy and history of eighth-century Judah
No full textResumen: Ensayos de un grupo internacional de expertos sobre el antiguo Cercano Oriente y la Biblia hebrea que honran el trabajo pionero de Oded Borowski en la arqueología y la historia del antiguo Israel y Judá. Los colaboradores abordan la cuestión de lo que sabemos del Judá del siglo VIII desde múltiples ángulos, incluyendo un estudio de los vecinos de Judá, la tierra de Judá y sus ciudades, la vida diaria y la cultura material, las creencias y prácticas religiosas, y las primeras formas de lo que ahora son los textos bíblicos. Entre los colaboradores se encuentran Rami Arav, Shawn Zelig Aster, Assaf Avraham, Jeffrey A. Blakely, Sandra Blakely, Elizabeth Bloch-Smith, Efrat Bocher, Erin Darby, Jennie Ebeling, Zev I. Farber, Avraham Faust, Daniel E. Fleming, Yuval Gadot, Kristine Garroway, Seymour Gitin, James W. Hardin, Gilad Itach, Hayah Katz, Reinhard G. Kratz, Joel M. LeMon, Shani Libi, Oded Lipschits, Donald Redford, Christopher Rollston, Bruce Routledge, Yair Sapir, Konrad Schmid, Cynthia Shafer-Elliott, Brent A. Strawn, Andy Vaughn, Jacob L. Wright, y K. Lawson Younger Jr. Características: - Énfasis en la influencia de Asiria en las culturas políticas, religiosas y materiales de Judá; Múltiples modelos para las primeras etapas de la escritura y composición bíblica; Información actualizada sobre tipologías de cerámica
Harvard meets the crisis: U.S. fiscal policy in the 1930s and the political economy of Lauchlin B. Currie, Jacob Viner, John H. Williams and Harry D. White
No full textThe paper aims to describe the contribution of four Harvard economists to the interpretation of the Great Depression and the policy decision making from 1933 to 1938. Lauchlin B. Currie, Jacob Viner, John H. Williams, Harry D. White, eminent scholars in the field of monetary and international economics, were deeply involved in policy decisions during the New Deal. In our synoptic analysis we will benefit from extensive scholarly work that has been provided in the last few years. We shall examine the extensive biographical connection between Currie, Viner, White and Williams with special regard to their common training at Harvard. Then we shall compare their interpretations of the causes of crisis and their proposals in fiscal, monetary and banking policy. Finally, we shall describe their advisory activity in the Roosevelt administration and try to assess their influence.Great Depression; Monetary Theory; Monetary Policy; Fiscal Policy, Keynesism
Scribes Before and After 587 BCE: A Conversation
No full text The conversation represented here originated in a special
session at the 2006 Annual Meeting of the Association for Jewish Studies that
dealt with the issue of the shifting role of scribes leading up to and following
the Babylonian destruction of Jerusalem in 587 BCE. The essays situate all
scribal groups in relation to the nation's priestly tradition and circles,
address diverse matters of socio-political agenda, and identify trends in the
development of literary methodology. This conversation includes an introduction
by the editor, and contributions by M. Leuchter, Jacob L. Wright, Jeffrey C.
Geoghegan, and Lauren A. S. Monroe. </jats:p
Mammalian drug resistant mutants with multiple gene amplifications: Genes encoding the M1 component of ribonucleotide reductase, the M2 component of ribonucleotide reductase, ornithine decarboxylase, p5-8, the H-subunit of ferritin and the L-subunit of ferritin
No full textPT: J; CR: ASHIHARA J, 1979, METHOD ENZYMOL, V58, P259 AZIZ N, 1986, NUCLEIC ACIDS RES, V14, P915 BEAUMONT C, 1987, J BIOL CHEM, V262, P10619 BEAUMONT C, 1989, J BIOL CHEM, V264, P7498 BIN N, 1976, NUCLEIC ACIDS RES, V3, P2303 BLAKE MS, 1984, ANAL BIOCHEM, V136, P175 BOLIN RW, 1982, CANCER, V50, P1683 BOMFORD AB, 1985, HEPATOLOGY, V5, P870 CASEY JL, 1988, P NATL ACAD SCI USA, V85, P1787 CHIRGWIN JM, 1979, BIOCHEMISTRY-US, V18, P5294 CHOY BK, 1988, CANCER RES, V48, P2029 CHOY BK, 1989, BIOCHEM BIOPH RES CO, V162, P1417 COCKING JM, 1987, SOMAT CELL MOLEC GEN, V13, P221 DEBATISSE M, 1986, MOL CELL BIOL, V6, P1776 DONOVAN PB, 1984, AM J HEMATOL, V17, P329 ENGSTROM PF, 1984, AM J CLIN ONCOL-CANC, V7, P313 ENGSTROM Y, 1984, EMBO J, V3, P863 ERIKSSON S, 1981, J BIOL CHEM, V256, P9436 FEINBERG AP, 1983, ANAL BIOCHEM, V132, P6 GRASLUND A, 1982, J BIOL CHEM, V257, P5711 HARDS RG, 1981, J CELL PHYSIOL, V106, P309 HOPPER S, 1972, J BIOL CHEM, V247, P3336 HURTA RAR, 1990, BIOCHEM BIOPH RES CO, V167, P258 KUHN LC, 1984, CELL, V39, P267 LAEMMLI UK, 1970, NATURE, V227, P680 LEIBOLD EA, 1984, J BIOL CHEM, V259, P4327 MADHUBALA R, 1987, FED PROC, V46, P2254 MCCLARTY GA, 1986, SOMAT CELL MOLEC GEN, V12, P121 MCCLARTY GA, 1987, BIOCHEMISTRY-US, V26, P8004 MCCLARTY GA, 1988, BIOCHEM BIOPH RES CO, V154, P975 MCCLARTY GA, 1988, BIOCHEMISTRY-US, V27, P7524 MCCLARTY GA, 1990, J BIOL CHEM, V265, P7539 MCDONALD CJ, 1981, PHARMACOL THERAPEUT, V14, P1 MURRAY MT, 1987, P NATL ACAD SCI USA, V84, P7438 PEGG AE, 1988, CANCER RES, V48, P759 PIVER MS, 1983, AM J OBSTET GYNECOL, V147, P803 POHJANPELTO P, 1985, MOL CELL BIOL, V5, P1385 ROUAULT TA, 1987, P NATL ACAD SCI USA, V84, P6335 SCHIMKE RT, 1984, CANCER RES, V44, P1735 SCHIMKE RT, 1988, J BIOL CHEM, V263, P5989 SRINIVASAN PR, 1987, J BIOL CHEM, V262, P12871 STARK GR, 1986, CANCER SURV, V5, P1 STARK GR, 1989, CELL, V57, P901 STEEPER JR, 1970, ANAL BIOCHEM, V34, P123 TAGGER AY, 1987, BIOCH CELL BIOL, V65, P925 TAGGER AY, 1988, INT J CANCER, V42, P760 THEIL EC, 1987, ANNU REV BIOCHEM, V56, P289 THELANDER L, 1980, J BIOL CHEM, V255, P7426 THELANDER L, 1986, MOL CELL BIOL, V6, P3433 THELANDER M, 1985, J BIOL CHEM, V260, P2737 THOMAS CE, 1986, J BIOL CHEM, V261, P13064 TOWBIN H, 1979, P NATL ACAD SCI USA, V76, P4350 WRIGHT JA, 1987, SOMAT CELL MOLEC GEN, V13, P155 WRIGHT JA, 1989, DRUG RESISTANCE MAMM, V1, P15 WRIGHT JA, 1989, INT ENCY PHARM THERA, V128, P89; NR: 55; TC: 25; J9: BIOCHIM BIOPHYS ACTA; PG: 8; GA: EH920Source type: Electronic(1
Correlation between levels of ferritin and the iron-containing component of ribonucleotide reductase in hydroxyurea-sensitive,hydroxyurea-resistant, and hydroxyurea-revertant cell lines
No full textThe reduction of ribonucleotides to deoxyribonucleotides, a rate-limiting step in DNA synthesis, is catalyzed by ribonucleotide reductase. This enzyme is composed of two components, M1 and M2. Recent work has shown that inhibition of ribonucleotide reductase by the antitumor drug hydroxyurea leads to a destabilized iron centre in protein M2. We have examined the relationship between the levels of ferritin, the iron storage protein, and the iron-containing M2 component of ribonucleotide reductase. These studies were carried out with hydroxyurea-sensitive, -resistant, and -revertant cell lines. Hydroxyurea-resistant mouse L cells contained M2 gene amplification and elevated levels of enzyme activity, M2 message, and total cellular M2 protein concentration. Hydroxyurea-revertant cells exhibited a wild-type M2 gene copy number, and approximately wild-type levels of enzyme activity, M2 message, and M2 protein concentration. In addition, we observed that the hydroxyurea-resistant cells possessed elevated levels of L-chain ferritin message and total cellular H-chain ferritin protein when compared to wild-type cells. In contrast, the revertant cell population contained approximately wild-type levels of ferritin mRNA and protein. In keeping with these observations, obtained with mouse L cells, was the finding that hydroxyurea-resistant Chinese hamster ovary cells with increased ribonucleotide reductase activity exhibited elevated expression of both ferritin and M2 genes, which declined in drug-sensitive revertant hamster cell lines with decreased levels of ribonucleotide reductase activity. This is the first demonstration that reversion of hydroxyurea resistance and a decline in ribonucleotide reductase activity are accompanied by decreased ferritin expression, and supports the concept that ferritin is important in establishing resistance to hydroxyurea, and may play a role in DNA synthesis, through the regulation of functional iron-containing M2 protein levels required for ribonucleotide reduction.PT: J; CR: BEAUMONT C, 1987, J BIOL CHEM, V262, P10619 BEAUMONT C, 1989, J BIOL CHEM, V264, P7498 CERI H, 1988, BIOCH CELL BIOL, V66, P541 CHIRGWIN JM, 1979, BIOCHEMISTRY-US, V18, P5294 CHOY BK, 1988, CANCER RES, V48, P2029 COCKING JM, 1987, SOMAT CELL MOLEC GEN, V13, P221 DESPHANDE VV, 1985, J BIOL CHEM, V260, P757 EDWARDS DR, 1985, MOL CELL BIOL, V5, P3280 HARDS RG, 1981, J CELL PHYSIOL, V106, P309 HOPPER S, 1972, J BIOL CHEM, V247, P3336 HURTA RAR, 1990, BIOCHEM BIOPH RES CO, V167, P258 HURTA RAR, 1990, BIOCHIM BIOPHYS ACTA, V1087, P165 KOHGO Y, 1980, J BIOL CHEM, V255, P5195 LEIBOLD EA, 1984, J BIOL CHEM, V259, P4327 LEWIS WH, 1979, SOMATIC CELL GENET, V5, P83 MCCLARTY GA, 1987, BIOCHEMISTRY-US, V26, P8004 MCCLARTY GA, 1988, BIOCHEM BIOPH RES CO, V154, P975 MCCLARTY GA, 1990, J BIOL CHEM, V265, P7539 MURRAY MT, 1987, P NATL ACAD SCI USA, V84, P7438 ROUAULT TA, 1987, P NATL ACAD SCI USA, V84, P6335 STEEPER JR, 1970, ANAL BIOCHEM, V34, P123 TAGGER AY, 1988, INT J CANCER, V42, P760 THEIL EC, 1987, ANNU REV BIOCHEM, V56, P289 THELANDER L, 1980, J BIOL CHEM, V255, P7426 THELANDER L, 1986, MOL CELL BIOL, V6, P3433 THELANDER M, 1985, J BIOL CHEM, V260, P2737 THOMAS CE, 1986, J BIOL CHEM, V261, P13064 THOMPSON LH, 1971, J CELL PHYSL, V78, P431 TONIN PN, 1987, CYTOGENET CELL GENET, V45, P102 WRIGHT JA, 1981, ADV ENZYME REGUL, V19, P105 WRIGHT JA, 1987, SOMAT CELL MOLEC GEN, V13, P155 WRIGHT JA, 1989, DRUG RESISTANCE MAMM, V1, P15 WRIGHT JA, 1989, INT ENCY PHARM THERA, V128, P89 WRIGHT JA, 1990, BIOCH CELL BIOL, V68, P1364; NR: 34; TC: 14; J9: BIOCHEM CELL BIOL; PG: 8; GA: GK421Source type: Electronic(1
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