132 research outputs found
Development of a model for drying of solids in a continuous fluidized bed dryer
No full text477-482A mathematical model to describe the
drying characteristics of solids in a continuous fluidized bed dryer has been
developed. The RTD model proposed by Pydi Setty et al.1
is used to obtain the required drying
model. Experimental data in a continuous fluidized bed dryer are satisfactorily
matched with the model developed. Using the model, one can predict the average
moisture content relative to the initial moisture content in a continuous
fluidized bed dryer for the given operating conditions
Awareness and attitude of patients′ parents toward pulp therapy of the primary teeth: A clinical survey
No full textAims and Objective: The present study was carried out to find out the awareness and attitude of parents of patients, toward the pulpal treatment of primary teeth, visiting a dental hospital in a Bangalore suburban area. Materials and Methods: A total of 685 parents of the child patients requiring pulp treatment procedures visiting the dental OPD over a period of 18 months were personally interviewed with a questionnaire and their responses were immediately computed. Results: Urban populations seeking dental treatment are more in number as compared to the rural population. Pain and associated feature was the most common reason among both urban (71.92%) and rural (93.3%) patients visiting a dental office. Conclusions: It is important to create more awareness among the populace of our country about the significance of maintaining a healthy primary dentition and attendant sequalae if not done so
Nocturnal haemoglobin oxygen saturation variability is associated with vitamin C deficiency in Tanzanian children with sickle cell anaemia.
No full textAIM: To compare pulse oximetry in children with sickle cell anaemia (SCA) and controls and test the hypothesis that vitamin C deficiency (VCD; 0.4, compatible with obstructive sleep apnoea (OSA). Eleven of twenty-three with SCA had VCD; logged vitamin C concentrations showed a 66% decrease per 0.1 unit increase in delta12 s ([95% CI -86%, -15%]; p=0.023) and delta12 s >0.4 was associated with VCD (odds ratio 8.75 [1.24-61.7], p=0.029). Daytime and mean nocturnal SpO(2) were lower in SCA but there was no association with vitamin C. CONCLUSION: Obstructive sleep apnoea (OSA), detected from nocturnal haemoglobin oxygen saturation variability, is common in Tanzanian children and associated with vitamin C Deficiency in SCA. The direction of causality could be determined by comparing OSA treatment with vitamin C supplementation
Preferential Judicial Activism
No full textThe Author examines the Supreme Court’s use of “preferential judicial activism”—whereby justices decide whether to formalistically dismiss cases or instead choose to engage judicial activism based on their policy preferences—through contrasting the Court’s reasoning in Shelby v. Holder with its decisions in cases concerning national security. In Shelby, the majority characterized the Court’s review of the Voting Rights Act of 2006 as necessary given the fundamental rights at stake and the unusually broad reach of the Act in mandating federal jurisdiction over voting matters. However, in national security-related cases in which plaintiffs have alleged violations of fundamental rights, the Court’s response has been a rigid and formalistic refusal to address the plaintiffs’ claims, usually based on the acceptance of government invocations of procedural barriers to litigation. This judicial formalism has consistently led to the dismissal of cases alleging serious government abuse in the post-September 11 context. The Author argues that these instances of judicial formalism illustrate the judiciary’s internal struggle to determine its appropriate role when confronted with questions of constitutional rights during times of war or perceived emergency.
Ultimately, taking this dynamic together with Shelby\u27s preferential judicial activism and its undermining of voter protections for racial minorities, it becomes clear that multiple reforms must be undertaken to protect individual rights. The Author concludes that Congress should better assert its role to protect the civil rights of vulnerable populations through passing additional legislation and that courts must acknowledge their own ongoing preferential judicial activism
Leveling the Playing Field: Reforming the Office for Civil Rights to Achieve Better Title IX Enforcement
No full textIn this Article the Author discusses improving Title IX compliance in athletic programs by reforming the Office for Civil Rights ( OCR ), the agency within the Department of Education responsible for Title IX enforcement. The Author addresses several problem areas within OCR\u27s procedures, including OCR\u27s approach toward student grievances, its standards for assessing alleged Title IX violations, and its inadequate monitoring and enforcement of institutions in violation of Title IX.
Part I introduces the history of Title IX. Part II describes the legislation and regulations that mandate gender equity in educational institutions. Part III summarizes the case law that has affected the scope of Title IX\u27s application. Part IV suggests specific OCR reforms that, in conjunction with local institutional efforts, would improve compliance with Title IX. Part V outlines approaches previously offered to remedy current Title IX enforcement challenges, and discusses why they do not adequately deal with the noncompliance situation. Part VI asserts that, despite challenges, reforming OCR is currently the most effective option to achieve improved Title IX compliance in our educational institutions
Therapeutically reeducating macrophages to treat GBM
No full textGlioblastoma multiforme (GBM) is the most common type of aggressive malignant brain cancer. The current lack of successful therapeutics means that this disease has a dismal prognosis. However, a new study in mice offers hope for patients with GBM by demonstrating the efficacy of a novel drug that targets GBM-associated macrophages.
Comment on : Pyonteck SM, Akkari L, Schuhmacher AJ, Bowman RL, Sevenich L, Quail DF, Olson OC, Quick ML, Huse JT, Teijeiro V, Setty M, Leslie CS, Oei Y, Pedraza A, Zhang J, Brennan CW, Sutton JC, Holland EC, Daniel D, Joyce JA. CSF-1R inhibition alters macrophage polarization and blocks glioma progression. Nat Med. 2013 Oct;19(10):1264-72. doi: 10.1038/nm.3337. Epub 2013 Sep 22. PMID: 24056773; PMCID: PMC3840724.</p
How Different are Pre-trained Transformers for Text Ranking?
No full textIn recent years, large pre-trained transformers have led to substantial gains in performance over traditional retrieval models and feedback approaches. However, these results are primarily based on the MS Marco/TREC Deep Learning Track setup, with its very particular setup, and our understanding of why and how these models work better is fragmented at best. We analyze effective BERT-based cross-encoders versus traditional BM25 ranking for the passage retrieval task where the largest gains have been observed, and investigate two main questions. On the one hand, what is similar? To what extent does the neural ranker already encompass the capacity of traditional rankers? Is the gain in performance due to a better ranking of the same documents (prioritizing precision)? On the other hand, what is different? Can it retrieve effectively documents missed by traditional systems (prioritizing recall)? We discover substantial differences in the notion of relevance identifying strengths and weaknesses of BERT that may inspire research for future improvement. Our results contribute to our understanding of (black-box) neural rankers relative to (well-understood) traditional rankers, help understand the particular experimental setting of MS-Marco-based test collections.</p
Preliminary phytochemical screening and anti diabetic activity of Zingiber officinale rhizomes
No full textAbstract Herbs are plants which are used in a number of ways including cooking, religious, rituals and medicines. In botany herbs are defined as seed producing plant with non woody stems which weather and die back to the ground after season growth. Diabetes mellitus is a chronic metabolic disorder of impaired carbohydrates, fat and protein metabolism. It is characterized by hyperglycaemia expressed as abnormal glucose value, which is due to insulin deficiency and/or insulin resistance which results in decrease utilization of carbohydrate and excessive glycogenolysis and gluconeogenesis from amino acid by fatty acids. The literature servey exposed Zingiber officinale possesing anti-arthritic, anti-migrane, anti-thrombotic, anti-inflamatory , hypolipidaemic , hypocholesterolaemic , anti-nausea properties. Thus in the present study ,an attempt was made to investigate the various phytochemicals present in the petroleum ether and ethanol extract of the rhizomes of zinigiber officinalis and also the study has been under taken to corroborate the anti-diabetic property of ethonolic extract of Zingiber officinale in alloxan induced diabetic rats. The results thus obtained were comparable with the standard drug, glibenclamide. Key-Words: Herbs, Diabetes, Zingiber officinale, Alloxan, Glibenclamide, Anti diabetic activity. Introduction Herbs are plants which are used in a number of ways including cooking, religious, rituals and medicines. In botany herbs are defined as seed producing plant with non woody stems which weather and die back to the ground after season growth. Regarding the health use herbs are source of pharmacologically active substance which affect the living organism. There are some herbal products which can both food and drug 1, 2 . Diabetes mellitus is a chronic metabolic disorder of impaired carbohydrates, fat and protein metabolism. It is characterized by hyperglycaemia expressed as abnormal glucose value, which is due to insulin deficiency and/or insulin resistance which results in decrease utilization of carbohydrate and excessive glycogenolysis and gluconeogenesis from amino acid by fatty acids. It has been defined by the world health organization (WHO) on the basis of laboratory findings as a fasting venous plasma glucose concentration greater than7.3 mmol/L (140mg/dl) or greater than 11.1 mmol/L (200mg/dl) two hours after carbohydrate meal or two hours after oral ingestion of the equivalent of 7.5g of glucose 3, 4 . * Corresponding Author: Ginger is said to be native of South East Asia, but is cultivated in Jamaica, Taiwan and India .More than 35% of the world's Zinger production is from India 5 . Oleo resin (5.3 -8.6%) comprising of non-volatile pungent principles (gingerols-mainly [6]-gingerol). Non pungent substance(fats and waxes) and volatile oil.Volatile oil (1.5 -2.2 %) containing sesquiterpene hydrocarbon viz; α -zingiberen , β -sesquiphellandrene and α -curcumene as major constituents.The literature servey exposed Zingiber officinale possesing antiarthritic, anti-migrane , anti-thrombotic , antiinflamatory , hypolipidaemic , hypocholesterolaemic , anti-nausea properties Material and methods Collection of Plant Material The fresh rhizomes of Zingiber officinale was collected in the Chittoor of Andhra Pradesh, botanically Research Article [Setty et al., 2(12) Preparation of Extract About 500gms of completely dried material was powdered in a mechanical grinder and finely sifted using Sieve-30 and subjected to continuous hot percolation process using soxhlet apparatus, by using solvents petroleum ether (60-80 o C), ethanol. The extracts thus obtained were concentrated to a thick brownish yellow semi-solid mass using Rotary vacuum evaporator. The petroleum ether, ethanol of Zingiber officinale Rose thus obtained extracts were denoted as PEZO and EEZO for petroleum and ethanolic extracts respectively and were used for Phytochemical and pharmacological evaluations Preliminaryphytochemical screening Test for carbohydrates A small quantity of various extracts were dissolved separately in 4 ml of distilled water and filtered. The filtrate was subjected to the following tests to detect the presence of carbohydrates and glycosides. Molisch's test: The filtrate was treated with 2-3 drops of 1% alcoholic ∝-napthol and 2ml of concentrated sulphuric acid was added along the sides of the test tube. Appearance of brown ring at the junction of two liquids shows the presence of carbohydrates. Iodine Test: To the filtrate add 2ml of Iodine solution. Appearance of blue colour shows the presence of starch. Fehling's test: The filtrate was treated with 1ml of Fehling's solution A and B and heated on a water bath. A reddish precipitate was obtained shows the presence of carbohydrates. Another portion of extracts were hydrolyzed with dilute hydrochloric acid for few hours on a water bath and hydrolysate was subjected to the following tests to detect the presence of glycosides. Legal's test: To the hydrolysate 1ml of pyridine and few drops of sodium nitroprusside solution were added and then it was made alkaline with sodium hydroxide solution. Appearance of pink to red colour shows the presence of glycosides. Borntrager's test: Hydrolysate was treated with chloroform and the chloroform layer was separated. To this equal volume of dilute ammonia solution was added. Ammonia layer acquires pink colour shows the presence of glycosides. Detection of fixed oils and fats Filter paper test: Small quantities of various extracts were pressed separately between the filter papers. Appearance of oil stain on the paper indicates the presence of fixed oils. Saponification test: Few drops of 0.5N alcoholic potassium hydroxide were added to small quantities of various extracts along with a drop of phenolphthalein. The mixture was heated on a water bath for 1-2 hours. Formation of soap indicates the presence of fixed oils and fats. Detection of protiens and free aminoacids Small quantities of various extracts were dissolved in few ml of water and then they were subjected to the following tests. Million's test: The above-prepared extracts were treated with Millon's reagent. Red colour formed shows the presence of proteins and free amino acids. Biuret test: To the above prepared extracts equal volume of 5% sodium hydroxide and 1% copper sulphate solution were added. Violet colour produced shows the presence of proteins and free amino acids. Ninhydrine test: The extracts were treated with Ninhydrine reagent. Purple colour produced shows the presence of proteins and free amino acids. Detection of saponins The extracts were diluted with 20ml of distilled water and it was agitated in a measuring cylinder for 15 minutes. The formation of 1cm layer of foam shows the presence of saponins. Detection of tannins and phenolic compounds Small quantities of the various extracts were taken separately in water and test for the presence of phenolic compounds and tannins was carried out with the following reagents. 1) 5% Ferrric chloride solution -violet colour 2) 1% solution of gelatin containing10% NaClwhite precipitate 3) 10% lead acetate solution -white precipitate Detection of phytosterols Small quantities of various extracts were dissolved in 5ml of chloroform separately. Then this chloroform Research Article [Setty et al., 2(12) Libermann Burchard test: The above prepared chloroform solution was treated with a few drops of concentrated sulphuric acid followed by few drops of diluted acetic acid, 3ml of acetic anhydride. A bluish green colour appeared indicates the presence of phytosterols. Detection of alkaloids Small quantities of various extracts were separately treated with few drops of dilute hydrochloric acid and filtered. The filtrates were used for the following tests. 1) Mayer's reagent cream precipitate 2) Dragendorff's reagent orange brown precipitate 3) Hager's reagent yellow precipitate 4) Wagner's reagent reddish brown precipitate Detection of gums and mucilages A small quantity of various extracts were added separately to 25ml of absolute alcohol with constant stirring and filtered. The precipitate was dried in air and examined for its swelling properties. No swelling was observed indicates the absence of gums and mucilages. Detectionof flavonoids 1. Small quantities of various extracts were dissolved separately in aqueous sodium hydroxide. Appearance of yellow colour indicates the presence of Flavonoids. 2. To the small portion of each extract, concentrated sulphuric acid was added. Yellow orange colour was obtained shows the presence of Flavonoids. 3. Shinoda's test: Small quantities of the extracts were dissolved in alcohol. To those pieces of magnesium followed by concentrated hydrochloric acid was added drop wise and heated. Appearance of magenta colour shows the presence of Flavonoids. Pharmacological screening of anti diabetic activity Animals: Wistar albino rats weighing about 150-200gms of either sex were used for the pharmacological studies. The animals were kept under standard conditions(day/night rhythm)8.00am to 8.00pm, 22 ± 2°c room temperature, 50-60% relative humidity standard pellet diet(Hindustan lever, Bangalore)and water ad libitum. The animals were housed for one week in poly propylene cages prior to the experiments to acclimatize to laboratory conditions. It is randomly distributed into three different groups with six animals in each group under identical conditions throughout the experiments. The experimental protocol was approved by The Institutional Animal Ethics Committee (IAEC) of SLN College of pharmacy, pallur. As the ethanolic extract was showing presence of more number of phytoconstituents, and comparatively more extractive yield, it was selected for the pharmacological screening at dose of 200mg/kg BW. Animals were divided into three groups into six rats each. Group -I (Control) -Alloxan injected (150mg/kg of body weight) intra peritonially and kept without any treatment to study the diabetic nature, Group -II (Standard) -Alloxan injected (150mg/kg of body weight) intra peritonially and Glibenclamide (10 mg/kg of body weight), and Group -III (Ethanolic Extract) -Alloxan injected (150mg/kg of body weight) intra peritonially and EEZO (200mg/kg of body weight). Method The acclimatized animals were kept fasting for 24 hrs with water ad libitum and injected intraperitonially at a dose of 150mg/kg of body weight of alloxan monohydrate freshly prepared in normal saline (0.9 % w/v) solution. Before starting the experiment animals were separated according to their body weight. After one hour of Alloxan administration, animals were given feed ad libitum and 1 ml of (100ml/dL) glucose I.P to combat encountering severe hypo glycaemia. After 72 hours of alloxan injection, the animals were tested for evidence of diabetes by estimating their blood glucose level by using glucometer. The blood glucose levels were more than 140 mg/dl was criteria for diabetes according to world health organization. To the animals the EEZO (200 mg/kg of body weight, intra peritonially) and standard drug glibenclamide (10 mg/kg body weight) were administered by dissolving in 2% Tween 80, water and normal saline respectively. The blood glucose levels were monitored at interval of initial (zero hour), 1 st .3 rd , 5 th and 7 th day of administration of single dose for acute and prolong action was studied respectively. The body weight of the animals from all the groups was recorded and all the parameters were tabulated Statistical analysis The data represents mean ± SEM results were analysed statistically by ANOVA followed by Dunnett's't' test using computer fitted Graph pad prism software student's version 5.0. The difference was considered significant when P< 0.05. Research Article [Setty et al., 2(12): Dec., 2011
Immediate Text Search on Streams Using Apoptosic Indexes
No full textApplications that involve streams of documents require a mechanism for search over the newest arrivals. In this paper we explore provision of immediate indexing and fast search of recent documents only, in contrast to focus on dynamic construction of an index of all observed material. Our contribution is a new structure, an apoptosic index, that operates in a fixed volume of memory and in which expired index entries vanish without significant overhead; there is neither explicit removal of old data nor explicit memory management. We demonstrate the practicality of apoptosic indexes with a straightforward implementation and experiments on microblog and newswire data, showing dramatically faster performance than observed with alternatives
Hematological manifestations of celiac disease
No full textCeliac disease, an autoimmune disease once thought to be uncommon, is now being increasingly identified. Our improved diagnostic modalities have allowed us to diagnose more and more patients with atypical symptoms who improve on gluten-free diet (GFD). We discuss here the latest findings regarding the various hematological manifestations of celiac disease and their management. Anemia remains the most common hematological manifestation of celiac disease due to many mechanisms, and can be the sole presenting symptom. Other manifestations include thrombocytosis and thrombocythemia, leukopenia, thromboembolism, increased bleeding tendency, IgA deficiency, splenic dysfunction, and lymphoma. The diagnosis of celiac disease should always be kept in mind when a patient presents with unexplained and isolated hematological finding. Once diagnosed, patients should adhere to GFD and be educated about the potential complications of this disease. We herein present an algorithm for adequate management and follow-up. © 2012 Informa Healthcare.Agarwal S, 2009, J ALLERGY CLIN IMMUN, V124, P658, DOI 10.1016-j.jaci.2009.06.018; ANAND BS, 1977, BRIT J HAEMATOL, V37, P409, DOI 10.1111-j.1365-2141.1977.tb01012.x; Annibale B, 2001, AM J GASTROENTEROL, V96, P132, DOI 10.1016-S0002-9270(00)02256-5; Bagdi E, 1999, BLOOD, V94, P260; Bai J, 2012, WORLD GASTROENTEROLO; Bergamaschi G, 2008, HAEMATOL-HEMATOL J, V93, P1785, DOI 10.3324-haematol.13255; Bode S, 1996, SCAND J GASTROENTERO, V31, P54, DOI 10.3109-00365529609031627; Bottaro G, 1999, AM J GASTROENTEROL, V94, P691; Brigden ML, 1999, CRIT CARE MED, V27, P836, DOI 10.1097-00003246-199904000-00050; Broudy VC, 1995, BLOOD, p593a; Carroccio A, 2002, EUR J GASTROEN HEPAT, V14, P897, DOI 10.1097-00042737-200208000-00017; Cataldo F, 2003, PEDIATR ALLERGY IMMU, V14, P320, DOI 10.1034-j.1399-3038.2003.00053.x; Cataldo F, 1998, GUT, V42, P362; Catassi C, 2005, GASTROENTEROLOGY, V128, pS79, DOI 10.1053-j.gastro.2005.02.027; Cavallaro R, 2004, EUR J GASTROEN HEPAT, V16, P219, DOI 10.1097-01.meg.0000085537.79233.10; Cellier C, 2000, LANCET, V356, P203, DOI 10.1016-S0140-6736(00)02481-8; Chen CS, 2007, J GEN INTERN MED, V22, P1608, DOI 10.1007-s11606-007-0297-y; Chulilla JAM, 2009, WORLD J GASTROENTERO, V15, P4627, DOI 10.3748-wjg.15.4627; COLLIN P, 1992, SCAND J GASTROENTERO, V27, P367, DOI 10.3109-00365529209000089; Conley ME, 1999, CLIN IMMUNOL, V93, P190, DOI 10.1006-clim.1999.4799; CORAZZA GR, 1983, GUT, V24, P228, DOI 10.1136-gut.24.3.228; Corazza GR, 1982, GUT, P415; CORAZZA GR, 1990, CLIN LAB HAEMATOL, V12, P269; Croese J, 1979, MED J AUSTRALIA, P335; Cunningham-Rundles C, 2001, J CLIN IMMUNOL, V21, P303, DOI 10.1023-A:1012241117984; Dahele A, 2001, AM J GASTROENTEROL, V96, P745, DOI 10.1016-S0002-9270(00)02409-6; Dameshek W., 1955, JAMA-J AM MED ASSOC, P163; de Serre NPM, 2000, HISTOPATHOLOGY, V37, P70; Dickey W, 1998, BRIT MED J, V317, P1290; Dickey W, 1998, J CLIN GASTROENTEROL, P21; Durand JM, 1993, CARDIOVASC SURG, P273; EICHNER ER, 1979, AM J MED, V66, P311, DOI 10.1016-0002-9343(79)90554-0; ELIAKIM R, 1982, ARCH INTERN MED, V142, P1037, DOI 10.1001-archinte.142.5.1037; Fairley N H, 1937, Br Med J, V1, P375; Fine KD, 1996, NEW ENGL J MED, V334, P1163, DOI 10.1056-NEJM199605023341804; Fisgin T, 2004, ACTA HAEMATOL-BASEL, V111, P211, DOI 10.1159-000077568; Fish JC, 1970, SURGERY, P658; Freeman HJ, 2010, WORLD J GASTROENTERO, V16, P2991, DOI 10.3748-wjg.v16.i24.2991; Geha RS, 2007, J ALLERGY CLIN IMMUN, V120, P776, DOI 10.1016-j.jaci.2007.08.053; GOUGH KR, 1962, GUT, V3, P232, DOI 10.1136-gut.3.3.232; GOYENS P, 1985, J PEDIATR GASTR NUTR, V4, P677, DOI 10.1097-00005176-198508000-00033; Granel B, 2005, QJM-INT J MED, V98, P70, DOI 10.1093-qjmed-hci011; GREEN PA, 1960, GASTROENTEROLOGY, V38, P399; Green PHR, 2003, AM J MED, V115, P191, DOI 10.1016-S0002-9343(03)00302-4; Grigg AP, 1999, AUST NZ J MED, V29, P566, DOI 10.1111-j.1445-5994.1999.tb00764.x; Halfdanarson TR, 2007, BLOOD, V109, P412, DOI 10.1182-blood-2006-07-031104; Harper JW, 2007, AM J HEMATOL, V82, P996, DOI 10.1002-ajh.20996; Hershko C, 2008, HAEMATOL-HEMATOL J, V93, P1761, DOI 10.3324-haematol.2008.000828; Hjelt K., 1991, J PEDIAT GASTROENTER, P27; HOLMES GKT, 1989, GUT, V30, P333, DOI 10.1136-gut.30.3.333; Holmes GKT, 2002, DIGEST LIVER DIS, V34, P229, DOI 10.1016-S1590-8658(02)80198-0; Howard MR, 2002, J CLIN PATHOL, V55, P754, DOI 10.1136-jcp.55.10.754; Howdle PD, 2003, QJM-INT J MED, V96, P345, DOI 10.1093-qjmed-hcg058; Jacobs P, 2004, DM-DIS MON, V50, P46, DOI 10.1016-j.disamonth.2003.02.003; Jaffe Elaine S, 2009, Hematology Am Soc Hematol Educ Program, P523, DOI 10.1182-asheducation-2009.1.523; Johnston SD, 1998, EUR J GASTROEN HEPAT, V10, P353, DOI 10.1097-00042737-199804000-00014; Jones S, 2006, NUTR J, V5, DOI 10.1186-1475-2891-5-24; KRASINSKI SD, 1985, AM J CLIN NUTR, V41, P639; Kumar N, 2006, MAYO CLIN PROC, V81, P1371; Lamia K, 2009, AM J GASTROENTEROL, P256; Le Quellec A, 1990, ANN PATHOL, P194; Leeds JS, 2008, BRIT MED BULL, V88, P157, DOI 10.1093-bmb-ldn044; Leffler DA, 2010, AM J GASTROENTEROL, V105, P2520, DOI 10.1038-ajg.2010.276; Lubel JS, 2005, MED J AUSTRALIA, V183, P321; Ludvigsson JF, 2009, BMC GASTROENTEROL, V9, DOI 10.1186-1471-230X-9-57; Ludvigsson JF, 2007, BRIT J HAEMATOL, V139, P121, DOI 10.1111-j.1365-2141.2007.06766.x; Ludvigsson JF, 2008, GUT, V57, P1074, DOI 10.1136-gut.2007.133868; Mant MJ, 2006, CLIN GASTROENTEROL H, V4, P451, DOI 10.1016-j.cgh.2005.12.010; MARSH GW, 1970, BRIT J HAEMATOL, V19, P445, DOI 10.1111-j.1365-2141.1970.tb06972.x; Maurizio G, 2003, THROMB HAEMOSTASIS, P203; Mawhinney H, 1971, LANCET, P121; MCCARTHY CF, 1966, GUT, V7, P140, DOI 10.1136-gut.7.2.140; MCKINLEY M, 1995, J CLIN GASTROENTEROL, V20, P113, DOI 10.1097-00004836-199503000-00008; McNeill A, 2006, J CLIN PATHOL, V59, P216, DOI 10.1136-jcp.2005.027698; Miehsler W, 2004, GUT, V53, P542, DOI 10.1136-gut.2003.025411; Morello F, 2003, NEUROL SCI, V24, P85, DOI 10.1007-s100720300079; Mulder CJ, 1983, ARCH INTERN MED, P842; Mulder CJJ, 2000, SCAND J GASTROENTERO, V35, P32; Nutritional anaemias, 1968, WHO TECH REP SER, P5; OGRADY JG, 1984, GASTROENTEROLOGY, V87, P1326; OFARRELLY C, 1986, BRIT MED J, V293, P908; Oh RC, 2003, AM FAM PHYSICIAN, V67, P979; Olen O, 2008, SCAND J GASTROENTERO, V43, P416, DOI 10.1080-00365520701814028; Palmer KR, 1983, GUT, P384; Pettit JE, 1972, BRIT MED J, P438; Pittschieler K, 1995, ACTA PAEDIATR, P705; Rakesh K, 2009, EUR J GASTROEN HEPAT, P1092; Romaldini CC, 2002, J PEDIATR GASTR NUTR, V35, P513, DOI 10.1097-00005176-200210000-00010; Roshan B, 2011, AM J GASTROENTEROL, V106, P923, DOI 10.1038-ajg.2011.104; RUBIN CE, 1962, GASTROENTEROLOGY, V43, P621; Rubio-Tapia A, 2009, GASTROENTEROLOGY, V136, P99, DOI 10.1053-j.gastro.2008.10.013; Rubio-Tapia A, 2010, GUT, V59, P547, DOI 10.1136-gut.2009.195131; Saibeni S, 2004, AM J GASTROENTEROL, V99, P1966, DOI 10.1111-j.1572-0241.2004.30203.x; Schilling V, 1924, KLIN WOCHENSCHR, P1960; Schmitz F, 2002, INT J COLORECTAL DIS, V17, P192, DOI 10.1007-s00384-001-0361-8; Setty M, 2008, MOL DIAGN THER, V12, P289; Silano M, 2008, DIGEST DIS SCI, V53, P972, DOI 10.1007-s10620-007-9952-8; Smedby KE, 2005, GUT, V54, P54, DOI 10.1136-gut.2003.032094; Snow CF, 1999, ARCH INTERN MED, V159, P1289, DOI 10.1001-archinte.159.12.1289; Sopirjakova P, 2011, ACTA MED HRADEC KRAL, P25; Soranzo N, 2009, NAT GENET, V41, P1182, DOI 10.1038-ng.467; Spencer RP, 1989, CRIT REV CLIN LAB SC, P299; Stene-Larsen G, 1988, SCAND J GASTROENTERO, P1105; van Tilburg NH, 2000, BLOOD, V95, P2855; Villalta D, 2007, ANN NY ACAD SCI, V1109, P212, DOI 10.1196-annals.1398.025; Voigt W, 2008, J MED CASE REPORTS, P96; Vorechovsky I, 2000, J IMMUNOL, V164, P4408; Wilkins BS, 2002, BRIT J HAEMATOL, V117, P265, DOI 10.1046-j.1365-2141.2002.03425.x; William BM, 2007, HEMATOLOGY, V12, P89, DOI 10.1080-10245330600938463; William BM, 2007, HEMATOLOGY, P1; Woof JM, 2006, J PATHOL, V208, P270, DOI 10.1002-path.1877; Working Party of the British Committee for Standards in Haematology Clinical Haematology Task Force, 1996, BMJ, P430; Yel L, 2010, J CLIN IMMUNOL, V30, P10, DOI 10.1007-s10875-009-9357-x; Zamani F, 2009, J MED CASE REPORTS, P68; Zenjari T, 1995, GASTROEN CLIN BIOL, V19, P95345
- …
