173,892 research outputs found
Oral History Interview with C. W. Horner
No full textThe National Museum of the Pacific War presents an interview with C W Horner. Horner joined the Navy in 1938. He served in the Fire Control Division aboard the USS Helena (CL-50). The Helena was moored at 1010 Dock Navy Yard on the base (southeast) side of Pearl Harbor when the Japanese attacked. Horner recalls his experiences through that fateful event
Der Hochloblichen Streitbarn und Mannhaften Ehren-Frey-Compagnie zu Basel, gewiedmet den 18. Merz 1791 : An diesem Tag und solchem Jahr, Da jedes Thor verschlossen war
No full textC. Horner = Jakob Christoph Horner (1725-1805)?Anfang des Lieds: "Versprechen ist und bleibt ein Schuld, ..."Gedich
Oral History Interview with C. W. Horner
No full textThe National Museum of the Pacific War presents an interview with C W Horner. Horner joined the Navy in 1938. He served in the Fire Control Division aboard the USS Helena (CL-50). The Helena was moored at 1010 Dock Navy Yard on the base (southeast) side of Pearl Harbor when the Japanese attacked. Horner recalls his experiences through that fateful event
Retention data
No full textRetention data used in publications in or submitted to Journal of Chromatography A with A. R. Horner as first author.
Retention data for ~ 100 compounds on a Waters BEH C-18 column in acidic acetonitrile/water mobile phases at phase ratio and temperatures giving a range of k for each compound about 1 - 100. These are in the CSV file "RetentionData". The compounds/solutes are identified by number. The correspondence between the number and the compound name is in "Compound List and Conditions". Enthalpies for the compounds and functional group counts are in FGEnthalpyData.xls
Trauer-Ode über den tödtlichen Hinscheid Herrn M. Andreas Fäschen, SS. M. C. gewesenen Waisenvaters, auch Organisten zu St. Theodor und St. Clara im Mindern Basel : Den 1sten May 1792
No full text[C. Horner]C. Horner = Jakob Christoph Horner (1725-1805)?Gelegenheitsschrift: TodLebensdaten: XX.02.1735-30.04./01.05.1792Bestattungsdatum: 03.05.179
Horner, Rachael C.
No full textJames M. Horner - husbandhttps://stars.library.ucf.edu/cfm-ch-memoranda-1940/1066/thumbnail.jp
Retention data
No full textRetention data used in publications in or submitted to Journal of Chromatography A with A. R. Horner as first author.
Retention data for ~ 100 compounds on a Waters BEH C-18 column in acidic acetonitrile/water mobile phases at phase ratio and temperatures giving a range of k for each compound about 1 - 100
Likelihood of Diagnosing Neuroblastoma in Isolated Horner Syndrome
No full textBackground: The need for an extensive evaluation for neuroblastoma in children with Horner syndrome is controversial. Methods: A retrospective study design was used. The cohort included 47 children with anisocoria who were diagnosed with Horner syndrome and 135 children with neuroblastoma evaluated at a pediatric medical center between 2007 and 2015. To detect neuroblastoma, patients with Horner syndrome underwent brain and cervical MRI, abdominal ultrasound, and/or measurement of urinary vanillylmandelic acid (VMA). The neuroblastoma group was evaluated for signs/symptoms of Horner syndrome at the time of diagnosis. Results: Seven patients with Horner syndrome were lost to follow-up, and the findings of the remaining 40 were categorized according to the age of the patient. Horner syndrome most frequently was idiopathic (58%), and in only 1 patient did the discovery of neuroblastoma precede the appearance of Horner syndrome. In the 21 patients aged 1-18 years, Horner syndrome was acquired in 15 patients and congenital in 6. The most common etiology was trauma (62%). Imaging was performed in 14 patients and VMA testing in 13. Neuroblastoma was diagnosed in 5 patients; in none was it related to Horner syndrome. In the 135 patients with neuroblastoma, most of the tumors were diagnosed at Stage 4 (60%) or Stage 3 (30%) with 53% originating in the abdomen. In one patient (0.74%) with signs/symptoms of Horner syndrome at diagnosis of neuroblastoma, the tumor had been identified prenatally and the diagnosis confirmed by imaging postnatally. Conclusions: The absence of occult neuroblastoma in children with Horner syndrome and of signs/symptoms of Horner syndrome in the children diagnosed with neuroblastoma suggests that Horner syndrome might not be as frequent a cause of neuroblastoma as previously thought. We recommend that full investigation for neuroblastoma be reserved for suspicious cases associated with additional systemic signs or symptoms
To C. E. Horner, Manager, Water Cooler Division re: sending parts for VA evaluation.--Correspondence
No full textTo C. E. Horner, Manager, Water Cooler Division re: sending parts for VA evaluation
Recurrent horner syndrome and persistent trigeminal artery
No full textIntroduction: A persistent primitive trigeminal artery (PTA) is the most common type of fetal carotid-basilar anastomosis in adulthood; however, its prevalence is low and estimated between 0.1% and 0.7% in angiographic studies. PTA is usually diagnosed incidentally during an angiographic examination, though some symptomatic cases associated with cranial nerve dysfunction have been described. We describe the unusual association between PTA and ipsilateral Horner syndrome triggered by hypertensive crisis.
Case Report: A 40-year-old man presented to medical attention for recurrent episodes, over the last 3 years, characterized by hypertensive crisis (up to 220/150 mm Hg), dizziness, tachycardia, flushing of the forehead on the left side, ptosis, miosis, and conjunctival injection of the left eye without pain. Imaging revealed a left PTA originating from the carotid siphon with moderate ectasia of the proximal tract. We speculate that the recurrent Horner syndrome observed in our patient could be due to the disruption of the postganglionic peripheral fibers in the pericarotid plexus. Adequate antihypertensive therapy led to resolution of signs and symptoms.
Conclusions: To our knowledge, we present the first case of persistent PTA and ipsilateral Horner syndrome during hypertensive crisis. Adequate antihypertensive therapy can be useful in patients bearing trigeminal artery variants
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