1,720,976 research outputs found

    Ascorbic acid induces apoptosis in adult T-cell leukemia

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    Background: Adult T-cell leukemia (ATL) is an acute malignancy of activated T-cells caused by the human T-cell lymphotrophic virus type-1 (HTLV-1). Materials and Methods: The effects of non-cytotoxic concentrations of ascorbic acid (AA) were evaluated against HTLV-1 positive and negative cells. The effect of AA on apoptosis and proliferation was evaluated by cell cycle analysis. The role of p53, p21 Bax and Bcl-2α on cell cycle modulation and apoptosis was also assessed. The anti-proliferative effects were tested by determining the changes in the expression of transforming growth factors (TGF-α, TGF-β1 and TGF-β2). Results: Ascorbic acid was found to reduce the proliferation of cells and induce apoptosis by the modulation of p53, p21, Bcl-2 and Bax. Conclusion: The results of this study show the anti-proliferative effects of AA against leukemic cells.ALEXANDROW MG, 1995, CANCER RES, V55, P1452; Bazarbachi A, 1996, J ACQ IMMUN DEF SYND, V13, pS186, DOI 10.1097-00042560-199600001-00028; BISSELL MJ, 1980, P NATL ACAD SCI-BIOL, V77, P2711, DOI 10.1073-pnas.77.5.2711; BLAKESLEE JR, 1985, CANCER RES, V45, P3471; Cameron E., 1979, CANC VITAMIN C, P132; CAMERON E, 1973, ONCOLOGY-BASEL, V27, P181; Carlisle DL, 2000, TOXICOL SCI, V55, P60, DOI 10.1093-toxsci-55.1.60; Carr A, 1999, FASEB J, V13, P1007; Casciari JJ, 2001, BRIT J CANCER, V84, P1544, DOI 10.1054-bjoc.2001.1814; Douglas RM, 1998, COCHRANE DB SYST REV, DOI [10.1002-14651858.CD000980, DOI 10.1002-14651858.CD000980]; Dunker N, 2002, GASTROENTEROLOGY, V122, P1364, DOI 10.1053-gast.2002.32991; El-Sabban ME, 2000, BLOOD, V96, P2849; FOLEY GE, 1965, CANCER, V18, P522, DOI 10.1002-1097-0142(196504)18:4522::AID-CNCR28201804183.0.CO;2-J; Gackowski D, 2002, INT J CANCER, V101, P395, DOI 10.1002-ijc.10610; Groninger E, 2002, INT J ONCOL, V21, P1339; Harakeh S, 2004, CHEM-BIOL INTERACT, V148, P101, DOI 10.1016-j.cbi.2004.05.002; Harakeh S, 1995, NUTRITION, V11, P684; HARAKEH S, 1990, P NATL ACAD SCI USA, V87, P7245, DOI 10.1073-pnas.87.18.7245; Harakeh S, 1997, AIDS RES HUM RETROV, V13, P235, DOI 10.1089-aid.1997.13.235; HARAKEH S, 1994, CHEM-BIOL INTERACT, V91, P207, DOI 10.1016-0009-2797(94)90041-8; HARAKEH S, 1991, AM J CLIN NUTR, V54, pS1231; Harakeh Steve, 1994, Journal of Nutritional Medicine, V4, P393, DOI 10.3109-13590849409003588; Jamison JM, 2004, BIOCHEM PHARMACOL, V67, P337, DOI 10.1016-j.bcp.2003.08.040; Kanai M, 2001, GASTROENTEROLOGY, V121, P56, DOI 10.1053-gast.2001.25544; KAZAKOV SA, 1988, NATURE, V335, P186, DOI 10.1038-335186a0; LIEHR JG, 1991, AM J CLIN NUTR, V54, pS1256; Martin A, 1997, ARTERIOSCL THROM VAS, V17, P1583; MEDINA MA, 1994, BIOCHEM MOL BIOL INT, V34, P871; MORTENSEON MM, 1986, J SURG RES, V114, P302; MURATA A, 1976, J NUTR SCI VITAMINOL, V22, P347; MURATA A, 1973, AGR BIOL CHEM TOKYO, V37, P1145; NAIDU A.K., 2003, NUTR J, V2, P7; PAGANELLI GM, 1992, J NATL CANCER I, V84, P47, DOI 10.1093-jnci-84.1.47; PAVELIC K, 1985, BRAIN RES, V342, P369, DOI 10.1016-0006-8993(85)91139-4; Reddy VG, 2001, BIOCHEM BIOPH RES CO, V282, P409, DOI 10.1006-bbrc.2001.4593; SCHWARZ RI, 1991, AM J CLIN NUTR, V54, pS1247; Van Orden K, 1999, J BIOL CHEM, V274, P26321, DOI 10.1074-jbc.274.37.26321; WAHL SM, 1992, J CLIN IMMUNOL, V12, P61, DOI 10.1007-BF00918135; Wang DG, 2002, ONCOGENE, V21, P2785, DOI 10.1038-sj-onc-120537515141

    Effects of nutrients on matrix metalloproteinases in human T-lymphotropic virus type 1 positive and negative malignant T-lymphocytes

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    Experimental and clinical studies have revealed the effectiveness of a specific nutrient synergy (SNS) mixture composed of ascorbic acid (AA), lysine, proline, arginine, epigallocatechin gallate (EGCG) and other micronutrients in targeting crucial physiological mechanisms involved in cancer progression and metastasis. HTLV-1 causes adult T-cell leukemia (ATL). The spread and metastases of ATL as well as other tumors has been associated with matrix metalloproteinases, especially the gelatinases MMP-2 and MMP-9. The objective of this study was to investigate whether SNS, AA and EGCG affects the gelatinolytic activity of MMP-2 and its transcriptional and translational levels in HTLV-1-positive and -negative malignant T-cells. The results indicated that SNS and EGCG caused a dose-dependent decline in the activity, transcription and translation of MMP-2 after treatment with SNS and EGCG, while AA was only able to inhibit the activity at maximum doses tested and to some extent, the protein expression levels of MMP-2, without affecting their transcriptional levels. The highest activity was noted in the case of SNS which is likely to be due to a synergistic effect of the different constituents in the formulation. These results point towards the potential integration of SNS in the anti-invasive treatment of ATL and related diseases.Annabi B, 2002, BBA-MOL CELL RES, V1542, P209, DOI 10.1016-S0167-4889(01)00187-2; Araujo Thessika Hialla Almeida, 2014, Mem. Inst. Oswaldo Cruz, V0, P21, DOI 10.1590-0074-0276130587; Bazarbachi A, 2004, CANCER RES, V64, P2039, DOI 10.1158-0008-5472.CAN-03-2390; CAMERON E, 1979, CANCER RES, V39, P663; Cha J, 2013, INT J ONCOL, V42, P55, DOI 10.3892-ijo.2012.1712; Chen PN, 2011, J AGR FOOD CHEM, V59, P836; Cook LB, 2014, BLOOD, V123, P3925, DOI 10.1182-blood-2014-02-553602; Demeule M, 2000, BBA-PROTEIN STRUCT M, V1478, P51, DOI 10.1016-S0167-4838(00)00009-1; Deng YT, 2011, J AGR FOOD CHEM, V59, P13318, DOI 10.1021-jf204149c; Devy J, 2011, INVEST NEW DRUG, V29, P730, DOI 10.1007-s10637-010-9410-x; Du JA, 2010, CLIN CANCER RES, V16, P509, DOI 10.1158-1078-0432.CCR-09-1713; Farabegoli F, 2011, BIOSCIENCE REP, V31, P99, DOI 10.1042-BSR20090143; Fromberg A, 2011, CANCER CHEMOTH PHARM, V67, P1157, DOI 10.1007-s00280-010-1418-6; Fu XY, 2003, J BIOL CHEM, V278, P28403, DOI 10.1074-jbc.M304739200; Gialeli C, 2011, FEBS J, V278, P16, DOI 10.1111-j.1742-4658.2010.07919.x; Harakeh S, 2014, ASIAN PAC J CANCER P, V15, P1219, DOI 10.7314-APJCP.2014.15.3.1219; Harakeh S, 1995, NUTRITION, V11, P684; Harakeh S, 2007, ANTICANCER RES, V27, P289; Harakeh S, 2006, LEUKEMIA RES, V30, P869, DOI 10.1016-j.leukres.2005.11.027; HARAKEH S, 1990, P NATL ACAD SCI USA, V87, P7245, DOI 10.1073-pnas.87.18.7245; Harakeh S, 1997, AIDS RES HUM RETROV, V13, P235, DOI 10.1089-aid.1997.13.235; Harakeh S, 2008, MED ONCOL, V25, P30, DOI 10.1007-s12032-007-0036-6; HARAKEH S, 1994, CHEM-BIOL INTERACT, V91, P207, DOI 10.1016-0009-2797(94)90041-8; Harakeh S, 2010, NANOTOXICOLOGY, V4, P177, DOI 10.3109-17435390903569621; HARAKEH S, 1991, AM J CLIN NUTR, V54, pS1231; Jariwalla R J, 1996, Subcell Biochem, V25, P213; Jiang C, 2000, MOL CARCINOGEN, V29, P236, DOI 10.1002-1098-2744(200012)29:42360

    Antimicrobial resistance of Listeria monocytogenes isolated from dairy-based food products

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    In this study Listeria monocytogenes (L. monocytogenes) was isolated from three traditionally consumed Lebanese dairy-based food products. One hundred and sixty four samples (45 samples of Baladi cheese, 36 samples of Shankleesh and 83 of Kishk) were collected from the Bekaa Valley in the Northeast region of Lebanon. Suspected Listeria colonies were selected and initially identified by using standard biochemical tests. Initial identification of the positive L. monocytogenes colonies was confirmed at the molecular level by Polymerase Chain Reaction (n = 30) and the confirmed isolates were evaluated for their susceptibility to 10 commonly used antimicrobials. All of the 30 isolates were confirmed to be L. monocytogenes yielding a PCR product of ∼ 660 base pairs (bp). L. monocytogenes was detected in 26.67percent, 13.89percent and 7.23percent of the Baladi cheese, Shankleesh and Kishk samples, respectively. The highest resistance in L. monocytogenes isolates was noted against oxacillin (93.33percent) followed by penicillin (90percent). The results provide an indication of the contamination levels of dairy-based foods in Lebanon and highlight the emergence of multi-drug resistant Listeria in the environment. © 2009 Elsevier B.V. All rights reserved.ABUIN CMF, 1994, ANTIMICROB AGENTS CH, V38, P1655; Aureli P, 2003, INT J FOOD MICROBIOL, V83, P325, DOI 10.1016-S0168-1605(02)00381-1; AYGUN P, 2006, FOOD CONTROL, V17, P599; Bottarelli A., 1999, Annali della Facoltà di Medicina Veterinaria, Università di Parma, V19, P293; Bower CK, 1999, INT J FOOD MICROBIOL, V50, P33, DOI 10.1016-S0168-1605(99)00075-6; Bubert A, 1999, APPL ENVIRON MICROB, V65, P4688; Charpentier E, 1999, J BACTERIOL, V181, P3368; CHARPENTIER E, 1995, J INFECT DIS, V172, P277; Cocolin L, 2002, APPL ENVIRON MICROB, V68, P6273, DOI 10.1128-AEM.68.12.6273-6282.2002; Conter M, 2009, INT J FOOD MICROBIOL, V128, P497, DOI 10.1016-j.ijfoodmicro.2008.10.018; Danielsson-Tham M-L, 2004, Foodborne Pathog Dis, V1, P153, DOI 10.1089-fpd.2004.1.153; Downes F. P., 2001, COMPENDIUM METHODS M; FACINELLI B, 1993, APPL ENVIRON MICROB, V59, P614; Geornaras I, 2001, INT J FOOD MICROBIOL, V70, P29, DOI 10.1016-S0168-1605(01)00517-7; Gilbert R J, 2000, Commun Dis Public Health, V3, P163; Gillespie I, 2000, J APPL MICROBIOL, V88, P467, DOI 10.1046-j.1365-2672.2000.00981.x; Gilot P, 2002, J CLIN MICROBIOL, V40, P698, DOI 10.1128-JCM.40.2.698-703.2002; GULMEZ M, 2003, J APPL MICROBIAL, V95, P632; Harakeh S, 2005, SCI TOTAL ENVIRON, V341, P33, DOI 10.1016-j.scitotenv.2004.09.025; Harakeh S, 2006, ENVIRON POLLUT, V143, P269, DOI 10.1016-j.envpol.2005.11.027; HERMAN LMF, 1995, APPL ENVIRON MICROB, V61, P817; HINTON M, 1986, J APPL BACTERIAL S, V15, P775; Hitchins A.D., 1995, FOOD DRUG ADM BACTER; Horwitz W, 2000, OFFICIAL METHODS ANA; Kells J, 2004, INT J FOOD MICROBIOL, V91, P167, DOI 10.1016-S0168-1605(03)00378-7; Klein G, 1998, APPL ENVIRON MICROB, V64, P1825; MacDonald PDM, 2005, CLIN INFECT DIS, V40, P677, DOI 10.1086-427803; Makino SI, 2005, INT J FOOD MICROBIOL, V104, P189, DOI 10.1016-j.ijfoodmicro.2005.02.009; Manfreda G, 2005, INT J FOOD MICROBIOL, V102, P287, DOI 10.1016-j.ijfoodmicro.2004.11.045; MARCO F, 2000, DIAGN MICROBIAL INFE, V38, P29; MARCO L, 1998, ANTIMICROBIAL AGENTS, V42, P2055; MATTHEW A, 2008, M2A9 CLSI, P72; Mayrhofer S, 2004, INT J FOOD MICROBIOL, V97, P23, DOI 10.1016-j.ijfoodmicro.2004.04.006; McLauchlin J, 2004, INT J FOOD MICROBIOL, V92, P15, DOI 10.1016-S0168-1605(03)00326-X; Meyer-Broseta S, 2003, INT J FOOD MICROBIOL, V80, P1, DOI 10.1016-S0168-1605(02)00117-4; Millet L, 2006, INT J FOOD MICROBIOL, V108, P105, DOI 10.1016-j.ijfoodmicro.2005.11.004; Okwumabua O, 2005, FEMS MICROBIOL LETT, V249, P275, DOI 10.1016-j.femsle.2005.06.018; Oliver SP, 2005, FOODBORNE PATHOG DIS, V2, P115, DOI 10.1089-fpd.2005.2.115; Peng H, 2000, J MICROBIOL METH, V41, P113, DOI 10.1016-S0167-7012(00)00134-2; Poros-Gluchowska Joanna, 2003, Acta Microbiologica Polonica, V52, P113; POYARTSALMERON C, 1990, LANCET, V335, P1422, DOI 10.1016-0140-6736(90)91447-I; Prazak AM, 2002, J FOOD PROTECT, V65, P1796; Saleh I, 2009, ANN TROP MED PARASIT, V103, P39, DOI 10.1179-136485909X384965; SANAA M, 1993, J DAIRY SCI, V76, P2891; Schlegelova J, 2002, J VET MED B, V49, P216, DOI 10.1046-j.1439-0450.2002.00520.x; Schwartz T, 2003, FEMS MICROBIOL ECOL, V43, P325, DOI 10.1111-j.1574-6941.2003.tb01073.x; Siegman-Igra Y, 2002, EMERG INFECT DIS, V8, P305; Silva IMM, 2003, INT J FOOD MICROBIOL, V81, P241, DOI 10.1016-S0168-1605(02)00223-4; Tamime AY, 1999, LAIT, V79, P449, DOI 10.1051-lait:1999437; Teuber M, 1999, CELL MOL LIFE SCI, V56, P755, DOI 10.1007-s000180050022; TOUFEILI I, 1995, INT DAIRY J, V5, P179, DOI 10.1016-0958-6946(95)92209-M; Van Kessel JS, 2004, J DAIRY SCI, V87, P2822; Vela AI, 2001, INT J ANTIMICROB AG, V17, P215, DOI 10.1016-S0924-8579(00)00318-6; Walsh D, 2001, J APPL MICROBIOL, V90, P517, DOI 10.1046-j.1365-2672.2001.01273.x; Wayne P. A., 2006, M2A9 CLSI; Zhou XH, 2005, FOOD CONTROL, V16, P125, DOI 10.1016-j.foodcont.2004.01.00124211

    Resistance of Brucella abortus isolated from Lebanese dairy-based food products against commonly used antimicrobials

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    Considering the marked importance of Brucella organisms as food-borne pathogens and the lack of published literature on the evaluation of the microbiological quality of dairy-based food products in the Middle East, this study was performed to address this gap. The main aim of the present study was to assess the antimicrobial resistance patterns of Brucella isolates recovered from a total of 164 cultured samples of Lebanese dairy-based food products (Baladi cheese, Shankleesh and Kishk). Standard polymerase chain reaction (PCR) was used to stereotype colonies previously confirmed by biochemical tests to be Brucella strains and to distinguish between the RB51 vaccine and field Brucella strains. Real-time PCR was applied to differentiate among the various Brucella species. Confirmed PCR field Brucella abortus isolates were evaluated for their susceptibility to eight commonly used antimicrobials. The highest number of resistant B. abortus isolates (n = 4 out of 6) was shown against Streptomycin and Ciprofloxacin, whereas 3 out of 6 isolates tested were resistant to Gentamicin. A lower number of resistant isolates were noted against Rifampicin, Tetracycline and Trimethoprim-sulfamethoxazole (n = 2 out of 6) and the lowest number for Doxycycline and Ceftriaxone (n = 1 out of 6). Such results are alarming and reflect the significance and importance of implementing more strict hygiene standards and regulations to reduce food-borne illnesses and control the excessive use of antimicrobials in this region. © INRA, EDP Sciences, 2010.Al Dahouk Sascha, 2003, Clin Lab, V49, P487; ANON JB, 1995, COMMUN DIS REP CDR W, V5, P151; Bricker BJ, 2002, VET MICROBIOL, V90, P435, DOI 10.1016-S0378-1135(02)00228-6; BRICKER BJ, 1994, J CLIN MICROBIOL, V32, P2660; Centers for Disease Control and Prevention, 2001, BAS LAB PROT PRES ID; DEBUYSER M, 2004, INT J FOOD MICROBIOL, V67, P1; DIANA MP, 2007, INFECT IMMUN, V75, P379; Downes F. P., 2001, COMPENDIUM METHODS M; FERNANDO AM, 2009, J BACTERIOL, V191, P2530; Gilbert R J, 2000, Commun Dis Public Health, V3, P163; HARAKEH S, 2009, SCI TOTAL ENVIRON, V408, P4022; Harakeh S, 2006, ENVIRON POLLUT, V143, P269, DOI 10.1016-j.envpol.2005.11.027; Hayes MC, 2001, J DAIRY SCI, V84, P292; Horwitz W, 2000, OFFICIAL METHODS ANA; LEALKLEVEZAS DS, 1995, J CLIN MICROBIOL, V33, P3087; *NAT COMM CLIN LAB, 2004, M2A6 NCCLS; OuahraniBettache S, 1996, J APPL BACTERIOL, V81, P154, DOI 10.1111-j.1365-2672.1996.tb04493.x; Redkar R, 2001, MOL CELL PROBE, V15, P43, DOI 10.1006-mcpr.2000.0338; Saleh I, 2009, ANN TROP MED PARASIT, V103, P39, DOI 10.1179-136485909X384965; Schelling E, 2003, PREV VET MED, V61, P279, DOI 10.1016-j.prevetmed.2003.08.004; Tantillo G, 2001, J FOOD PROTECT, V64, P164; Thakur S. D., 2002, Journal of Communicable Diseases, V34, P287; Vemulapalli R, 1999, CLIN DIAGN LAB IMMUN, V6, P760; WALLACH JC, 1998, FEMS IMMUNOL MED MIC, V19, P31511

    Going Beyond Counting First Authors in Author Co-citation Analysis

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    The present study examines one of the fundamental aspects of author co-citation analysis (ACA) - the way co-citation counts are defined. Co-citation counting provides the data on which all subsequent statistical analyses and mappings are based, and we compare ACA results based on two different types of co-citation counting - the traditional type that only counts the first one among a cited work's authors on the one hand and a non-traditional type that takes into account the first 5 authors of a cited work on the other hand. Results indicate that the picture produced through this non-traditional author co-citation counting contains more coherent author groups and is therefore considerably clearer. However, this picture represents fewer specialties in the research field being studied than that produced through the traditional first-author co-citation counting when the same number of top-ranked authors is selected and analyzed. Reasons for these effects are discussed

    Variations on the Author

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    “Variations on the Author” discusses two of Eduardo Coutinho’s recent films (Um Dia na Vida, from 2010, and Últimas Conversas, posthumously released in 2015) and their contribution to the general question of documentary authorship. The director’s filmography is characterized by a consistent yet self-effacing form of authorial self-inscription: Coutinho often features as an interviewer that rather than express opinions propels discourses; an interviewer that is good at listening. This mode of self-inscription characterizes him as an author who is not expressive but who is nonetheless markedly present on the screen. In Um Dia na Vida, however, Coutinho is completely absent form the image, while Últimas Conversas, on the contrary, includes a confessional prologue that moves the director from the margins to the center of his films. This article examines the ways in which these works stand out in the filmography of a director who offers new insights into the notion of cinematic authorship

    Appropriate Similarity Measures for Author Cocitation Analysis

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    We provide a number of new insights into the methodological discussion about author cocitation analysis. We first argue that the use of the Pearson correlation for measuring the similarity between authors’ cocitation profiles is not very satisfactory. We then discuss what kind of similarity measures may be used as an alternative to the Pearson correlation. We consider three similarity measures in particular. One is the well-known cosine. The other two similarity measures have not been used before in the bibliometric literature. Finally, we show by means of an example that our findings have a high practical relevance.information science;Pearson correlation;cosine;similarity measure;author cocitation analysis

    Dispelling the Myths Behind First-author Citation Counts

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    We conducted a full-scale evaluative citation analysis study of scholars in the XML research field to explore just how different from each other author rankings resulting from different citation counting methods actually are, and to demonstrate the capability of emerging data and tools on the Web in supporting more realistic citation counting methods. Our results contest some common arguments for the continued use of first-author citation counts in the evaluation of scholars, such as high correlations between author rankings by first-author citation counts and other citation counting methods, and high costs of using more realistic citation counting methods that are not well-supported by the ISI databases. It is argued that increasingly available digital full text research papers make it possible for citation analysis studies to go beyond what the ISI databases have directly supported and to employ more sophisticated methods

    Antimicrobial-resistant patterns of Escherichia coli and Salmonella strains in the aquatic Lebanese environments

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    This study is the first to be conducted in Lebanon on the isolation and molecular characterization and the antimicrobial resistance profile of environmental pathogenic bacterial strains. Fifty-seven samples of seawater, sediment, crab, and fresh water were collected during the spring and summer seasons of 2003. The isolation of Escherichia coli and Salmonella using appropriate selective media revealed that 94.7percent of the tested samples were contaminated with one or both of the tested bacteria. The polymerase chain reaction (PCR) was then used to identify the species of both bacteria using various sets of primers. Many pathogenic E. coli isolates were detected by PCR out of which two were identified as O157:H7 E. coli. Similarly, the species of many of the Salmonella isolates was molecularly identified. The confirmed isolates of Salmonella and E. coli were then tested using the disk diffusion method for their susceptibility to four different antimicrobials revealing high rates of antimicrobial resistance. © 2005 Elsevier Ltd. All rights reserved.Ammon A, 1999, J INFECT DIS, V179, P1274, DOI 10.1086-314715; *AOAC, 1995, OFF METH AN AOAC INT; ARAJ F, 2000, J MED LIBANAIS, V48, P221; BANWART CJ, 1989, BASIC FOOD MICROBIOL; Bielaszewska M, 1998, J CLIN MICROBIOL, V36, P2135; BROOK MG, 1994, EPIDEMIOL INFECT, V112, P63; Duijkeren E. van, 2003, Journal of Clinical Microbiology, V41, P3574, DOI 10.1128-JCM.41.8.3574-3578.2003; El-Fadel M., 2003, Management of Environmental Quality, V14, P353, DOI 10.1108-14777830310479441; GANNON VPJ, 1992, APPL ENVIRON MICROB, V58, P3809; Gillespie BE, 1997, J DAIRY SCI, V80, P471; Gray JT, 2001, BERL MUNCH TIERARZTL, V114, P370; GRIFFIN, 1991, EPIDEMIOLOGY REV, V13, P60; HOLMBERG SD, 1984, SCIENCE, V225, P833, DOI 10.1126-science.6382605; Huovinen P., 1999, ACTA VET SCADINAVI S, V96, P7; IRVINE KN, 1995, MODERN METHODS MODEL, P205; KARMALI MA, 1985, J INFECT DIS, V151, P775; LEE SJ, 1992, J GEN MICROBIOL, V138, P1843; Loynachan AT, 2004, J FOOD PROTECT, V67, P1484; Luk JM, 1997, J CLIN MICROBIOL, V35, P714; LUK JMC, 1993, J CLIN MICROBIOL, V31, P2118; Lukasova J, 2004, J VET MED B, V51, P77, DOI 10.1111-j.1439-0450.2004.00727.x; MCKEON DM, 1995, WATER RES, V29, P1902, DOI 10.1016-0043-1354(95)00013-B; National Committee for Clinical Laboratory Standards, 2000, M7A5 NCCLS, VM100-S10; NIEMI M, 1983, APPL ENVIRON MICROB, V45, P79; Osek J, 2002, VET MED-CZECH, V47, P149; Raveh D, 2003, J HOSP INFECT, V55, P196, DOI 10.1016-S0195-6701(03)00286-X; Reischl U, 2002, J CLIN MICROBIOL, V40, P2555, DOI 10.1128-JCM.40.7.2555-2565.2002; Sayah RS, 2005, APPL ENVIRON MICROB, V71, P1394, DOI 10.1128-AEM.71.3.1394-1404.2005; Schwartz T, 2003, FEMS MICROBIOL ECOL, V43, P325, DOI 10.1111-j.1574-6941.2003.tb01073.x; Senkel IA, 2003, J FOOD PROTECT, V66, P2237; Soto SM, 2003, ANTIMICROB AGENTS CH, V47, P421, DOI 10.1128-AAC.47.1.421-425.2003; Tauxe RV, 1996, HLTH ENV DIGEST, V10, P1; Wait DA, 2001, WATER SCI TECHNOL, V43, P139; WATSTESON Y, 2001, ACTA VET SCANDINAV S, V95, P79; Yoo S, 2004, J KOREAN MED SCI, V19, P15; Zann LP, 2000, STATE MARINE ENV REP16181
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