1,720,975 research outputs found
Assessment of individual sensitivity to occupational benzene exposure: biomarkers of genotoxic effect.
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Bone and soft tissue concentrations of aztreonam after intravenous administration.
No full textvedasi allegat
Aztreonam penetration of bone and soft tissue, alter iv infusion and bolus injection.
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Going Beyond Counting First Authors in Author Co-citation Analysis
Full text linkThe present study examines one of the fundamental aspects of author co-citation analysis (ACA) - the way co-citation
counts are defined. Co-citation counting provides the data on which all subsequent statistical analyses and mappings
are based, and we compare ACA results based on two different types of co-citation counting - the traditional type that
only counts the first one among a cited work's authors on the one hand and a non-traditional type that takes into
account the first 5 authors of a cited work on the other hand. Results indicate that the picture produced through this non-traditional author co-citation counting contains more coherent author groups and is therefore considerably clearer. However, this picture represents fewer specialties in the research field being studied than that produced through the traditional first-author co-citation counting when the same number of top-ranked authors is selected and analyzed. Reasons for these effects are discussed
Effects on C57/BL mice of subchronic treatment with benzene [Effetto del trattamento subcronico con benzene in topi C57/Bl]
No full textBenzene, a chemical widely used in industry, is capable of causing aplastic anemia and acute myelogenous leukemia in humans and multiple forms of cancer in rodents. Hepatic cytochrome P450 2E1 catalyzes the formation of phenolic metabolites of benzene. Mielopeoxidase (MPO), present in bone marrow, may further convert the phenolic metabolites to free radicals, which are responsible for in situ bioactivation of benezene. The aim of this work was to study the effects of low and high doses of benzene (5 and 100 mg/Kg) in subchronically treated mice. Treatment with 100 mg/Kg led to a significative increase in MPO and in NAD(P)H:quinone-oxidoreductase (NADHQ). Furthermore "comet assay" in myelocytes revealed DNA damage for both types of benzene treatment
Environmental and biological monitoring of benzene exposure in gasoline stations.
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Benzene toxicity in C57Bl mice after gastic subchronic administration.
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Correlation between environmental and biological monitoring in petrochemical industry operators.
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