828 research outputs found
Reply to comment by K. Heki and Y. Enomoto on “Preseismic ionospheric electron enhancements revisited”
No full textReply to comment by K. Heki and Y. Enomoto on “Preseismic ionospheric electron enhancements revisited”
No full textPreseismic ionospheric electron enhancements revisited
No full text[1] Possible enhancement of ionospheric Total Electron Content (TEC) immediately before the 2011 Tohoku-oki earthquake (Mw9.0) has been reported by Heki (2011). Critical responses to it often come in two stages; they first doubt the enhancement itself and attribute it to an artifact. Second (when they accept the enhancement), they doubt the significance of the enhancement among natural variability of space weather origin. For example, Kamogawa and Kakinami (2013) attributed the enhancement to an artifact falsely detected by the combined effect of the highly variable TEC under active geomagnetic condition and the occurrence of a tsunamigenic ionospheric hole. Here we closely examine the time series of vertical TEC before and after the 2011 Tohoku-oki earthquake. We first demonstrate that the tsunami did not make an ionospheric hole, and next confirm the reality of the enhancement using data of two other sensors, ionosonde and magnetometers. The amplitude of the preseismic TEC enhancement is within the natural variability, and its snapshot resembles to large-scale traveling ionospheric disturbances. However, distinction could be made by examining their propagation properties. Similar TEC anomalies occurred before all the M ≥ 8.5 earthquakes in this century, suggesting their seismic origin
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No full textNeurotransmitter is released at synapses by fusion of synaptic vesicles with the plasma membrane. To sustain synaptic transmission, compensatory retrieval of membranes and vesicular proteins is essential. We combined capacitance measurements and pH-imaging via pH-sensitive vesicular protein marker (anti-synaptotagmin2-cypHer5E), and compared the retrieval kinetics of membranes and vesicular proteins at the calyx of Held synapse. Membrane and Syt2 were retrieved with a similar time course when slow endocytosis was elicited. When fast endocytosis was elicited, Syt2 was still retrieved together with the membrane, but endocytosed organelle re-acidification was slowed down, which provides strong evidence for two distinct endocytotic pathways. Strikingly, CaM inhibitors or the inhibition of the Ca2+-calmodulin-Munc13-1 signaling pathway only impaired the uptake of Syt2 while leaving membrane retrieval intact, indicating different recycling mechanisms for membranes and vesicle proteins. Our data identify a novel mechanism of stimulus-and Ca2+-dependent regulation of coordinated endocytosis of synaptic membranes and vesicle proteins
Special issue on precision technology in Tsukuba Science City (Part2) Fine ceramics in machine industry.
No full textPictorial 1 : A Wooden Print on the Meiji-Sanriku Earthquake Tsunami Showing a Reddish Pillar Burst Out from the Great Wave
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On the Publication of an Issue on the Strategy of the Grobal Standards Related to Fundamental Technology
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