276 research outputs found
sj-pdf-1-epx-10.1177_08959048211049428 – Supplemental material for The Signaling Power of Unexcused Absence from School
No full textSupplemental material, sj-pdf-1-epx-10.1177_08959048211049428 for The Signaling Power of Unexcused Absence from School by Jaymes Pyne, Eric Grodsky, Elizabeth Vaade, Bo McCready, Eric Camburn and Dominique Bradley in Educational Policy</p
When Those in Judea Flee to the Mountains: Mobility, Property, and Loss in Mark's Gospel
No full textThe synoptic gospels contain teachings that promote a lifestyle of mobility and a rejection of home and family. Employing recent developments in humanistic geography, place theory and mobility studies, this thesis considers why the author of Mark chooses to present texts that enshrine the surrendering of all one’s property and possessions in order to gain a share in the kingdom of God. It is proposed that Mark's audience is experiencing a turbulent period in history, which will culminate in an imminent loss of home, homeland, Temple, and – emanating from all of those – orientation in the world. It is further argued that the shift in identity instigated by these events plays a formative role in how Mark depicts the life and teachings of Jesus. Mark’s enshrinement of mobility in the gospel could have served to provide adherents of the Jesus Movement with a model for detachment and innovation that facilitated the creation of new notions of homeland in the wake of the catastrophic events leading up to, during, and after 70 CE
A qualitative exploration of internet forum discussions surrounding female sexual function for individuals with Sj\uf6gren\u27s syndrome
No full textCopyright: \ua9 2023 McCready et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Sexual dysfunction is a common experience for women with the autoimmune rheumatic disease, Sj\uf6gren\u27s syndrome (SS); however, the lived experience of how the disease affects sexual functioning and the sexual environment remains unexplored. This qualitative study explores the conversations pertaining to female sexual function and the sexual environment that individuals with SS have on an internet forum. Qualitative data posted on one publicly accessible, worldwide, internet forum was extracted using an automated web scraping tool. A total of 247,694 posts across 23,382 threads were scraped from the forum in July 2019 and June 2022 (from the United Kingdom). A predetermined and theoretically informed keyword search strategy was used to screen the captured data for content relevant to the study aim. The dataset was cleaned to remove duplication and identifying information and screened for topic relevance. The Computer-Assisted Qualitative Data Analysis software tool, ATLAS.ti, was used to facilitate the data analysis process. Thematic analysis was conducted on 1443 female-oriented posts, and four key themes were identified: the symptoms of SS and their impact on the sexual environment; the emotional responses that are commonly evoked in response to sexual difficulties; the strategies that users have implemented to manage sexual problems; and the impact that a partner\u27s behavior may have on the sexual environment. Together these themes provide an insight into the nature of sexual difficulties for females with SS. Our findings provide novel insights to inform clinical discussions between practitioners and patients whilst further outlining the importance of undertaking qualitative research with this population
The limits of performance in the theater of Alfred de Musset
No full textThis dissertation examines the function of performance in the theatrical oeuvre of Alfred de Musset (1810-1857). By analyzing the many ways in which Musset\u27s characters play roles within the plays, I have attempted to chart the possible trajectories of performance. The crux of the argument throughout the dissertation involves the complex relationship between the concrete space of the stage and abstract values--those announced in the theatrical text and those espoused by the audience. I argue that the stage is a privileged site for the negotiation of values, a negotiation mediated by performance. Following Elaine Scarry\u27s analysis of the structure of belief in The Body in Pain, I have shown how in some cases performance serves to verify or substantiate an abstract value held by the character and, moving outward, by the audience as a whole. In other cases, relying on the work of Georges Bataille, I have shown that performance can instead lead to physical destruction, as the individual turns to violence in revolt against a system of values from which he feels excluded. In addition to the work of Scarry and Bataille, the work of Rene Girard aids my examination of the relationship between theatrical performance and the performance of the ritual sacrifice. In Musset\u27s oeuvre, several plays evoke the ritual sacrifice, but always stage its failure and the failure of performance to substantiate meaning. The specific concerns of Musset\u27s theater and of French romantic theater can be contextualize historically in the anxieties populating the Zeitgeist of the Restoration and July Monarchy; however, the dissertation does not attempt to define or develop an argument about their historical specificity. It focuses, on the contrary, on elements of Musset\u27s theater which are far from unique to it, in an attempt to develop an argument about performance and its function as an element of the structure of belief in our culture
Molecular investigation into brachydactyly type A1
No full textBrachydactyly type A1 (BDA1) is a rare, autosomal-dominant genetic trait that affects normal limb and bone development, resulting in short or absent middle halan es. Despite being the first human trait described in terms of autosomal dominant Mendelian inheritance, the underlying molecular cause had not been examined until relatively recently. In 2001, mutations of the Indian Hedgehog (IHH) gene were identified in three Chinese families with BDA1. Preliminary studies in our laboratory further demonstrated linkage of BDA1 to chromosome 5p in a Canadian kindred with a mild variant of the trait. The goal of this thesis was to characterize the BDA1 locus on chromosome 5 by reducing the critical region and examining the transcribed portions of candidate genes for mutations. To reduce the chromosome 5p critical region in the Canadian kindred, 23 microsatellite markers were examined and the number of family members analyzed was increased to include 22 affected and 16 unaffected individuals. By doing so, we defined a 7 cM (3.03 Mb) critical region flanked by the recombinant markers D5S1986 and D5S426. Fifteen candidate genes within the region were examined for mutations, however none were identified. Further characterization of the chromosome 5p critical region was made by recruiting twelve additional families into the study. Of the twelve families, five had a G → A transition at codon 100 of the IHH gene. The mutation causes an aspartate to asparagine amino acid substitution and supports a role for IHH in normal bone development. Linkage analysis to chromosome 5 in two other pedigrees indicated that the trait was not linked to this locus in these families, and provided further support for genetic heterogeneity underlying the aetiology of BDA1. IHH mutations were not found in the remaining five families, thus increasing the likelihood that the chromosome 5 locus is involved in these pedigrees. Linkage could not be established in these families, however, since the trait was sporadic, and not familial. Identification of additional BDA1 families in which the trait is linked to chromosome 5, and further mutational analysis will be required to identify the gene from chromosome 5p that causes BDA1
Which Students to Serve?: Universal or Targeted Eligibility for Postsecondary Opportunity Programs
No full textDramatic changes in the higher education landscape and the recent recession have intensified the challenges students face in postsecondary enrollment and completion. In response, some states, communities, and institutions have developed postsecondary opportunity programs (POPs) -- comprehensive college access and success programs offering a combination of funding and support services.
POPs have the potential to transform students' educational careers as well as enhance their future success; therefore, the decision of who will be eligible to access these programs is extremely important and often highly contentious. The debate over eligibility has influenced education policies and programs for pre-K, school choice, financial aid, and college admissions. But while public policy literature has much to say about the theoretical implications of universal and targeted eligibility, less research exists on how these classic debates play out in policymaking and practice.
This brief examines how the designers of POPs determine eligibility for their programs, focusing on their decision either to employ universal eligibility or target students who are underrepresented in postsecondary education. The authors explore the factors that influence the eligibility decision for 10 programs, including how closely the identified aspects align with those cited in the existing literature. To conclude, the brief offers lessons for researchers, practitioners, and policymakers and recommendations for POPs stakeholders moving forward
Useful, Used, and Peer Approved: The Importance of Rigor and Accessibility in Postsecondary Research and Evaluation
No full textTraditionally, researchers, policymakers, and practitioners have perceived a tension between rigor and accessibility in quantitative research and evaluation in postsecondary education. However, this study indicates that both producers and consumers of these studies value high-quality work and clear findings that can reach multiple audiences. The authors discuss the perceived importance of rigor and accessibility, barriers to accomplishing both in practice, and suggestions for achieving an appropriate balance between the two
Molecular investigation into brachydactyly type A1
No full textBrachydactyly type A1 (BDA1) is a rare, autosomal-dominant genetic trait that affects normal limb and bone development, resulting in short or absent middle halan es. Despite being the first human trait described in terms of autosomal dominant Mendelian inheritance, the underlying molecular cause had not been examined until relatively recently. In 2001, mutations of the Indian Hedgehog (IHH) gene were identified in three Chinese families with BDA1. Preliminary studies in our laboratory further demonstrated linkage of BDA1 to chromosome 5p in a Canadian kindred with a mild variant of the trait. The goal of this thesis was to characterize the BDA1 locus on chromosome 5 by reducing the critical region and examining the transcribed portions of candidate genes for mutations. To reduce the chromosome 5p critical region in the Canadian kindred, 23 microsatellite markers were examined and the number of family members analyzed was increased to include 22 affected and 16 unaffected individuals. By doing so, we defined a 7 cM (3.03 Mb) critical region flanked by the recombinant markers D5S1986 and D5S426. Fifteen candidate genes within the region were examined for mutations, however none were identified. Further characterization of the chromosome 5p critical region was made by recruiting twelve additional families into the study. Of the twelve families, five had a G → A transition at codon 100 of the IHH gene. The mutation causes an aspartate to asparagine amino acid substitution and supports a role for IHH in normal bone development. Linkage analysis to chromosome 5 in two other pedigrees indicated that the trait was not linked to this locus in these families, and provided further support for genetic heterogeneity underlying the aetiology of BDA1. IHH mutations were not found in the remaining five families, thus increasing the likelihood that the chromosome 5 locus is involved in these pedigrees. Linkage could not be established in these families, however, since the trait was sporadic, and not familial. Identification of additional BDA1 families in which the trait is linked to chromosome 5, and further mutational analysis will be required to identify the gene from chromosome 5p that causes BDA1
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