1,721,036 research outputs found
Prediction of fraction of inspired oxygen to achieve a desired arterial partial pressure of oxygen
No full text[No abstract available]ABIZANDA R, 1981, INTENS CARE MED, V7, P247, DOI 10.1007-BF01702629; Hardman JG, 2010, AM J RESP CRIT CARE, V182, P435; OGAWA S, 1989, CRIT CARE MED, V17, P1191, DOI 10.1097-00003246-198911000-000180
Cerebral Oximetry During Deep Hypothermic Circulatory Arrest
No full text[No abstract available]Baraka A, 2006, J CARDIOTHOR VASC AN, V20, P819, DOI 10.1053-j.jvca.2005.04.015; Laussen PC, 2002, PAEDIATR ANAESTH, V12, P199, DOI 10.1046-j.1460-9592.2002.00779.x; Leyvi G, 2006, J CARDIOTHOR VASC AN, V20, P826, DOI 10.1053-j.jvca.2006.01.001; NAWFAL M, 2006, EUR J ANAESTH, V7, P122
Correlation Between Cerebral and Mixed Venous Oxygen Saturation During Moderate Versus Tepid Hypothermic Hemodiluted Cardiopulmonary Bypass
No full textObjective: This study was undertaken to compare cerebral oxygen saturation (RsO2) and mixed venous oxygen saturation (SvO2) in patients undergoing moderate and tepid hypothermic hemodiluted cardiopulmonary bypass (CPB). Design: Prospective study. Settings: University hospital operating room. Participants: Fourteen patients undergoing elective coronary artery bypass graft surgery using hypothermic hemodiluted CPB. Interventions: During moderate (28°-30°C) and tepid hypothermic (33°-34°C) hemodiluted CPB, RsO2 and SvO2 were continuously monitored with a cerebral oximeter via a surface electrode placed on the patient's forehead and with the mixed venous oximeter integrated in the CPB machine, respectively. Measurements and Main Results: Mean ± standard deviation of RsO2, SvO2, PaCO2, and hematocrit were determined prebypass and during moderate and tepid hypothermic phases of CPB while maintaining pump flow at 2.4 L-min-m2 and mean arterial pressure in the 60- to 70-mmHg range. Compared with a prebypass value of 76.0percent ± 9.6percent, RsO2 was significantly decreased during moderate hypothermia to 58.9percent ± 6.4percent and increased to 66.4percent ± 6.7percent after slow rewarming to tepid hypothermia. In contrast, compared with a prebypass value of 78.6percent ± 3.3percent, SvO2 significantly increased to 84.9percent ± 3.6percent during moderate hypothermia and decreased to 74.1percent ± 5.6percent during tepid hypothermia. During moderate hypothermia, there was poor agreement between RsO2 and SvO2 with a gradient of 26percent; however, during tepid hypothermia, there was a strong agreement between RsO2 and SvO2 with a gradient of 6percent. The temperature-uncorrected PaCO2 was maintained at the normocapnic level throughout the study, whereas the temperature-corrected PaCO2 was significantly lower during the moderate hypothermic phase (26.8 ± 3.1 mmHg) compared with the tepid hypothermic phase (38.9 ± 3.7 mmHg) of CPB. There was a significant and positive correlation between RsO2 and temperature-corrected PaCO2 during hypothermia. Conclusions: During moderate hypothermic hemodiluted CPB, there was a significant increase of SvO2 associated with a paradoxic decrease of RsO2 that was attributed to the low temperature-corrected PaCO2 values. During tepid CPB after slow rewarming, regional cerebral oxygen saturation was increased in association with an increase with the temperature-corrected PaCO2 values. The results show that during hypothermic hemodiluted CPB using the alpha-stat strategy for carbon dioxide homeostasis, cerebral oxygen saturation is significantly higher during tepid than moderate hypothermia. © 2006 Elsevier Inc. All rights reserved.BARAKA A, 1992, J CARDIOTHOR VASC AN, V1, P35; COOK DJ, 1996, J THORACIC CARDIOVAS, V111, P672; CZINN EA, 1995, ANESTH ANALG, V80, P492, DOI 10.1097-00000539-199503000-00010; Daubeney PEF, 1996, ANN THORAC SURG, V61, P930, DOI 10.1016-0003-4975(95)01186-2; Duebener LF, 2004, J CARDIOTHOR VASC AN, V18, P423, DOI 10.1053-j.jvca.2004.05.018; Edmonds HL, 1996, J CARDIOTHOR VASC AN, V10, P15, DOI 10.1016-S1053-0770(96)80174-1; Holzschuh M, 1997, NEUROL RES, V19, P246; JOBSISVANDERVLIET FF, 1985, ADV EXP MED BIOL, V191, P833; LARACH DR, 1988, ANESTHESIOLOGY, V69, P185, DOI 10.1097-00000542-198808000-00006; Lozano S, 2004, J CARDIOTHOR VASC AN, V18, P645, DOI 10.1053-j.jvca.2004.07.027; BLAND JM, 1986, LANCET, V1, P307; McLean RF, 1996, J CARDIOTHOR VASC AN, V10, P45, DOI 10.1016-S1053-0770(96)80178-9; MURKIN JM, 1987, ANESTH ANALG, V66, P825; Newman MF, 2001, NEW ENGL J MED, V344, P395, DOI 10.1056-NEJM200102083440601; RICHARD FM, 1996, J CARDIOTHOR VASC AN, V10, P45; Roach GW, 1996, NEW ENGL J MED, V335, P1857, DOI 10.1056-NEJM199612193352501; Samra SK, 1999, J NEUROSURG ANESTH, V11, P1, DOI 10.1097-00008506-199901000-00001; SHAABAN AM, 2004, ACTA ANAESTHESIOLOGI, V48, P837; Yao FSF, 2004, J CARDIOTHOR VASC AN, V18, P552, DOI 10.1053-j.jvca.2004.07.007; Yeh T, 2001, J THORAC CARDIOV SUR, V122, P192, DOI 10.1067-mtc.2001.11316714121
Going Beyond Counting First Authors in Author Co-citation Analysis
Full text linkThe present study examines one of the fundamental aspects of author co-citation analysis (ACA) - the way co-citation
counts are defined. Co-citation counting provides the data on which all subsequent statistical analyses and mappings
are based, and we compare ACA results based on two different types of co-citation counting - the traditional type that
only counts the first one among a cited work's authors on the one hand and a non-traditional type that takes into
account the first 5 authors of a cited work on the other hand. Results indicate that the picture produced through this non-traditional author co-citation counting contains more coherent author groups and is therefore considerably clearer. However, this picture represents fewer specialties in the research field being studied than that produced through the traditional first-author co-citation counting when the same number of top-ranked authors is selected and analyzed. Reasons for these effects are discussed
Persistant dysphonia following endotracheal intubation
No full textVoice production is a complex process that involves more than one system, yet most causes of dysphonia are attributed to disturbances in the laryngeal structures and little attention is paid to extralaryngeal factors. Persistent dysphonia after general anesthesia is a challenge to both anesthesiologists and otolaryngologists. The etiology is often multivariable and necessitates a team approach for proper diagnosis. Laryngeal symptoms are subdivided into phonatory disturbances and airway related complaints. When they become persistent for more than 72 hours or are coupled with airway symptoms such as hemoptysis, stridor, dyspnea or aspiration, the anesthesiologist should suspect injury to the vocal folds or cricoarytenoid joints. Here-below, the laryngeal manifestations of endotracheal intubation and the pathophysiology of vocal fold scarring are discussed.
Weaning from mechanical ventilation
No full textPURPOSE OF REVIEW: Liberation from mechanical ventilation is a defining moment for intubated patients, and thus a critical clinical decision. Extubating the patient too early exposes the patient to extubation failure and reintubation. Waiting too long increases the complications of prolonged intubation. Tools to help the physician with this critical decision and to test readiness have been available for decades, and are continuously being improved. New methods to improve extubation outcomes are also being developed. This review covers the latest studies in order to help physicians take advantage of the latest developments in a rapidly evolving field. RECENT FINDINGS: This review highlights the recent advances in assessing and testing for readiness of weaning and liberation from mechanical ventilation, the cause of weaning failure, the value of weaning protocols, and the role of noninvasive positive pressure ventilation in liberating patients from invasive mechanical ventilation. SUMMARY: Recent findings are shedding more light on this topic, and transforming 'the artistic' aspect of weaning and liberation from mechanical ventilation into a more 'scientific' approach that will expedite liberation from mechanical ventilation yet without encountering high failure rates, and without exposing patients to unnecessary risks. © 2012 Wolters Kluwer Health | Lippincott Williams and Wilkins.Alvisi R, 2000, EUR RESPIR J, V15, P656, DOI 10.1034-j.1399-3003.2000.15d06.x; BANNER MJ, 1993, CRIT CARE MED, V21, P1333, DOI 10.1097-00003246-199309000-00017; Bickenbach J, 2011, MINERVA ANESTESIOL; Bickenbach J, 2011, MINERVA ANESTESIOL, V77, P427; Blackwood B, 2010, COCHRANE DB SYST REV, DOI 10.1002-14651858.CD006904.pub2; Boutou AK, 2011, HEART LUNG, V40, P105, DOI 10.1016-j.hrtlng.2010.02.002; BROCHARD L, 1994, AM J RESP CRIT CARE, V150, P896; BROCHARD L, 1991, ANESTHESIOLOGY, V75, P739, DOI 10.1097-00000542-199111000-00004; Burns KEA, 2010, COCHRANE DB SYST REV, DOI 10.1002-14651858.CD004127.pub2; Cader SA, 2010, J PHYSIOTHER, V56, P171, DOI 10.1016-S1836-9553(10)70022-9; Chen CW, 2011, RESP CARE, V56, P976, DOI 10.4187-respcare.00966; Cohen J, 2009, CRIT CARE, V13, DOI 10.1186-cc7724; Delisle S, 2011, RESP CARE, V111, P1211; Del Rosario N, 1997, EUR RESPIR J, V10, P2560, DOI 10.1183-09031936.97.10112560; El-Khatib M, 2001, INTENS CARE MED, V27, P52, DOI 10.1007-s001340000758; El-Khatib MF, 2002, CHEST, V121, P475, DOI 10.1378-chest.121.2.475; El-Khatib MF, 2008, INTENS CARE MED, V34, P505, DOI 10.1007-s00134-007-0939-x; Engoren M, 1998, CRIT CARE MED, V26, P1817; EPSTEIN SK, 1995, AM J RESP CRIT CARE, V152, P545; ESTEBAN A, 1995, NEW ENGL J MED, V332, P345, DOI 10.1056-NEJM199502093320601; Esteban A, 2004, NEW ENGL J MED, V350, P2452, DOI 10.1056-NEJMoa032736; Esteban A, 1999, AM J RESP CRIT CARE, V159, P512; Ezingeard E, 2006, INTENS CARE MED, V32, P165, DOI 10.1007-s00134-005-2852-5; Ferrer M, 2003, AM J RESP CRIT CARE, V168, P70, DOI 10.1164-rccm.200209-1074OC; Figueroa-Casas JB, 2010, RESP CARE, V55, P549; Girault C, 2011, AM J RESP CRIT CARE; Goodman Sheila, 2006, Nurs Crit Care, V11, P23; Grasso S, 2007, CRIT CARE MED, V35, P96, DOI 10.1097-01.CCM.0000250391.89780.64; HALL JB, 1987, JAMA-J AM MED ASSOC, V257, P1621, DOI 10.1001-jama.257.12.1621; Hunter BC, 2010, J MUSIC THER, V47, P198; Kim WY, 2011, CRIT CARE MED; Krieger BP, 1997, CHEST, V112, P1029, DOI 10.1378-chest.112.4.1029; Kuhlen R, 2003, EUR J ANAESTH, V20, P10; LEE KH, 1994, CHEST, V105, P540, DOI 10.1378-chest.105.2.540; MacIntyre NR, 2001, CHEST, V120, p375S, DOI 10.1378-chest.120.6_suppl.375S; Mårtensson Irene E, 2002, Intensive Crit Care Nurs, V18, P219, DOI 10.1016-S0964339702000630; Martin AD, 2011, CRIT CARE, V15, DOI 10.1186-cc10081; Molina-Saldarriaga FJ, 2010, MED INTENSIVA, V34, P453, DOI 10.1016-j.medin.2010.03.007; Monaco F, 2010, BRIT J ANAESTH, V105, P326, DOI 10.1093-bja-aeq184; Nava S, 1998, ANN INTERN MED, V128, P721; Nelson JE, 2004, CRIT CARE MED, V32, P1527, DOI 10.1097-01.CCM.0000129485.08835.5A; Nevins ML, 2001, CHEST, V119, P1840, DOI 10.1378-chest.119.6.1840; Ouanes-Besbes L, 2011, J CRIT CARE, V26, P15, DOI 10.1016-j.jcrc.2010.01.002; Papaioannou Vasilios E., 2011, BMC Physiology, V11, P2, DOI 10.1186-1472-6793-11-2; Papaioannou VE, 2010, J CRIT CARE, V26, P262; Patel KN, 2009, RESP CARE, V54, P1462; Penuelas R, 2011, AM J RESP CRIT CARE, V184, P430; Perren A, 2002, INTENS CARE MED, V28, P1058, DOI 10.1007-s00134-002-1353-z; Raurich JM, 2011, RESP CARE, V53, P1012; Raurich JM, 2009, ANAESTH INTENS CARE, V37, P726; Routsi C, 2010, CRIT CARE, V14, DOI 10.1186-cc9326; Segal LN, 2010, INTENS CARE MED, V36, P487, DOI 10.1007-s00134-009-1735-6; Su CL, 2011, RESP CARE; Teixeira C, 2008, J CRIT CARE, V23, P572, DOI 10.1016-j.jcrc.2007.12.011; Thille AW, 2011, CRIT CARE MED; Vallverdu I, 1998, AM J RESP CRIT CARE, V158, P1855; Vianello A, 2011, J CRIT CARE, V26, P517, DOI 10.1016-j.jcrc.2010.12.008; White CE, 2010, J TRAUMA, V68, P1310, DOI 10.1097-TA.0b013e3181da90db; YANG KL, 1991, NEW ENGL J MED, V324, P1445, DOI 10.1056-NEJM199105233242101; Zapata L, 2011, INTENS CARE MED, V37, P477, DOI 10.1007-s00134-010-2101-458
Variations on the Author
Full text link“Variations on the Author” discusses two of Eduardo Coutinho’s recent films (Um Dia na Vida, from 2010, and Últimas Conversas, posthumously released in 2015) and their contribution to the general question of documentary authorship. The director’s filmography is characterized by a consistent yet self-effacing form of authorial self-inscription: Coutinho often features as an interviewer that rather than express opinions propels discourses; an interviewer that is good at listening. This mode of self-inscription characterizes him as an author who is not expressive but who is nonetheless markedly present on the screen. In Um Dia na Vida, however, Coutinho is completely absent form the image, while Últimas Conversas, on the contrary, includes a confessional prologue that moves the director from the margins to the center of his films. This article examines the ways in which these works stand out in the filmography of a director who offers new insights into the notion of cinematic authorship
Correspondence: A reply
No full text[No abstract available]Taha SK, 2010, ANAESTHESIA, V65, P358, DOI 10.1111-j.1365-2044.2010.06243.x0
Appropriate Similarity Measures for Author Cocitation Analysis
Full text linkWe provide a number of new insights into the methodological discussion about author cocitation analysis. We first argue that the use of the Pearson correlation for measuring the similarity between authors’ cocitation profiles is not very satisfactory. We then discuss what kind of similarity measures may be used as an alternative to the Pearson correlation. We consider three similarity measures in particular. One is the well-known cosine. The other two similarity measures have not been used before in the bibliometric literature. Finally, we show by means of an example that our findings have a high practical relevance.information science;Pearson correlation;cosine;similarity measure;author cocitation analysis
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