1,750 research outputs found
Dépistage génétique et résurgence de l'eugénisme
No full textDuster Troy, Devaux Laetitia. Dépistage génétique et résurgence de l'eugénisme. In: Quaderni, n°22, Hiver 1994. Exclusion-Intégration : la communication interculturelle. pp. 167-182
Le fantôme de Frantz Fanon
No full textDuster Troy, Wellman David. Le fantôme de Frantz Fanon. In: Actes de la recherche en sciences sociales. Vol. 71-72, mars 1988. Penser la politique-1. pp. 135-136
Sociologist and Author Troy Duster to Address Race and DNA, Diversity and Curricular Reform
No full textNews release announces that Troy Duster kicks off the University of Dayton\u27s 2005-2006 Diversity Lecture Series with a pair of public lecture on Aug. 25-26
Writers Talk featuring authors Troy Hicks and Elaine Wolf
No full textElaine Wolf, author of Camp, talks to OSU students Erin Reilly-Sanders and Allison Fetzer. Author and teacher Troy Hicks talks to OSU employee Kevin Cordi about the impact of technology on the teaching of writing.The media can be accessed here: http://streaming.osu.edu/knowledgebank/WritersTalk-Audio/WT_2013-3-18-Hicks_Wolf.mp3Ohio State University. Center for the Study and Teaching of Writin
Remarks and observations on the plain of Troy, made during an excursion in June, 1799 /
No full textSignatures: pi⁴ A-G⁴.Errata: p. [2] at end; p. [1] and [3] at end blank.ESTC(RLIN)Mode of access: Internet.Library copy bound with: Description of the plain of Troy : with a map of that region, delineated from an actual survey : read in French before the Royal Society of Edinburgh, Feb. 21 and 28 and March 21 1791 / by the author, M. Chevalier. Edinburgh : Printed for T. Cadell, 1791. (90-B15413
Observations upon a treatise, entitled A description of the Plain of Troy, by Monsieur Le Chevalier /
No full textErrata: p. [1] at end.Signatures: [A]1 B-H⁴.Includes bibliographical references.ESTC(RLIN)Mode of access: Internet.Library copy bound with: Description of the plain of Troy : with a map of that region, delineated from an actual survey : read in French before the Royal Society of Edinburgh, Feb. 21 and 28 and March 21 1791 / by the author, M. Chevalier. Edinburgh : Printed for T. Cadell, 1791. (90-B15413
Social Diversity in Humans: Implications and Hidden Consequences for Biological Research
No full textHumans are both similar and diverse in such a vast number of dimensions that for human geneticists and social scientists to decide which of these dimensions is a worthy focus of empirical investigation is a formidable challenge. For geneticists, one vital question, of course, revolves around hypothesizing which kind of social diversity might illuminate genetic variation-and vice versa (i.e., what genetic variation illuminates human social diversity). For example, are there health outcomes that can be best explained by genetic variation-or for social scientists, are health outcomes mainly a function of the social diversity of lifestyles and social circumstances of a given population? Indeed, what is a "population," how is it bounded, and are those boundaries most appropriate or relevant for human genetic research, be they national borders, religious affiliation, ethnic or racial identification, or language group, to name but a few? For social scientists, the matter of what constitutes the relevant borders of a population is equally complex, and the answer is demarcated by the goal of the research project. Although race and caste are categories deployed in both human genetics and social science, the social meaning of race and caste as pathways to employment, health, or education demonstrably overwhelms the analytic and explanatory power of genetic markers of difference between human aggregates
Linoleic acid causes greater weight gain than saturated fat without hypothalamic inflammation in the male mouse
No full textA significant change in the Western diet, concurrent with the obesity epidemic, was a substitution of saturated fatty acids with polyunsaturated, specifically linoleic acid (LA). Despite increasing investigation on type as well as amount of fat, it is unclear which fatty acids are most obesogenic. The objective of this study was to determine the obesogenic potency of LA vs. saturated fatty acids and the involvement of hypothalamic inflammation. Forty-eight mice were divided into four groups: low-fat or three high-fat diets (HFDs, 45% kcals from fat) with LA comprising 1%, 15% and 22.5% of kilocalories, the balance being saturated fatty acids. Over 12 weeks, bodyweight, body composition, food intake, calorimetry, and glycemia assays were performed. Arcuate nucleus and blood were collected for mRNA and protein analysis. All HFD-fed mice were heavier and less glucose tolerant than control. The diet with 22.5% LA caused greater bodyweight gain, decreased activity, and insulin resistance compared to control and 1% LA. All HFDs elevated leptin and decreased ghrelin in plasma. Neuropeptides gene expression was higher in 22.5% HFD. The inflammatory gene Ikk was suppressed in 1% and 22.5% LA. No consistent pattern of inflammatory gene expression was observed, with suppression and augmentation of genes by one or all of the HFDs relative to control. These data indicate that, in male mice, LA induces obesity and insulin resistance and reduces activity more than saturated fat, supporting the hypothesis that increased LA intake may be a contributor to the obesity epidemic.Peer reviewe
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