1,721,149 research outputs found
Neuroendocrine regulation of human growth hormone secretion. Diagnostic and clinical applications.
No full textINVECCHIAMENTO E SISTEMA ENDOCRINO.LA SOMATOPAUSA, LA ADRENOPAUSA, LE MALATTIE DELLA TIROIDE
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Calcium ion and pituitary hormones: effect of calcium channel blockers on stimulated secretion of pituitary hormones.
No full textThe effects of two calcium channel blockers nifedipine (20 mg sublingual), and verapamil (10 mg i.v.) on growth hormone (GH), thyrotropin (TSH), prolactin (PRL) and gonadotropin (LH and FSH) secretion induced by growth hormone releasing hormone (GHRH), hypoglycemia, thyrotropin releasing hormone (TRH), metoclopramide and gonadotropin releasing hormone (GnRH), were studied in a group of normal volunteers (27 men and 8 women). Neither nifedipine nor verapamil had any effect on PRL, TSH or gonadotropin release. Verapamil did not cause variations in GH secretion following GHRH and insulin-induced hypoglycemia, whereas nifedipine significantly reduced the elevation in GH induced by GHRH; however the GHRH-mediated GH rise still remained within the normal range in all subjects. Our results suggest that neither nifedipine nor verapamil have important effects on stimulated pituitary hormone secretion, at least under conditions of acute administration. Z9
Variations numeriques et anomalies de position des dents des animaux domestiques de la préhistoire
No full textObservations paléodontologiques sur une mandibule de porc d'un village de l'Age du Cuivre
No full textIndagine sull’insegnamento della Medicina del territorio e di famiglia nei CLM in Medicina e Chirurgia. Riflessioni e proposte.
No full textNell'articolo vengono proposte riflessioni e considerazioni sull'insegnamento della Medicina del territorio e di famiglia nei CLM in Medicina e Chirurgia
Interaction of glucose and pyridostigmine on the secretion of growth hormone (GH) induced by GH-releasing hormone (GHRH).
No full textIn order to investigate the mechanisms by which hyperglycaemia induces an inhibition of GHRH-induced GH release, we gave the following treatments to seven normal men: a) GHRH 100 micrograms iv; b) pyridostigmine (PD) 120 mg po 60 min before GHRH; c) glucose 250 mg/kg iv as a bolus (10 min before GHRH) plus 10 mg/kg/min until the end of the test; d) glucose pyridostigmine and GHRH as above. Glucose significantly reduced GHRH-stimulated GH levels, whereas PD significantly enhanced them. When PD and glucose were given together, the effect on GHRH-stimulated GH secretion was not different from the algebraic sum of the single effects of the two substances. Thus glucose seems to be able to exert its inhibition, at least partially, also when pyridostigmine is coadministered. Z9 1
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