2,486 research outputs found
Leucocytozoon ibisi Adlard & Peirce & Lederer 2002, n. sp.
No full textLeucocytozoon ibisi n. sp. (gures 5, 6) Type host. Threskiornis molucca Cuvier (white ibis). Type locality. Currumbin, Queensland, Australia. Basis of description Macrogametocyte. A parasite with small round morphs only, not exceeding about 13 m m in diameter (table 1), exhibiting the usual staining characteristics of the genus. Nucleus slightly ovoid occupying a central to subcentral position with a small round karyosome visible; cytoplasm granular with a few hyaline vacuoles sometimes present. Host–parasite complex indicates a maximum diameter of about 15 m m; host cell nucleus occupies a peripheral position around a quarter to onethird of the parasite circumference (gure 5). Microgametocyte. Similar to the macrogametocyte but more variable in shape and slightly smaller in overall dimensions (table 1); exhibiting the usual diOEerentiating characteristics of the genus. The host cell nucleus covers slightly more of the parasite circumference (gure 6). No distinctive karyosome visible in dispersed parasite nucleus. Ratio of macrogametocyte s to microgametocyte s is 3:2. Hapantotype. IRCAH: G46312 5 from Threskiornis molucca, coll. Lederer, 20 November 1998, Currumbin, Gold Coast, Queensland, Australia.Published as part of Adlard, R. D., Peirce, M. A. & Lederer, R., 2002, New species of Leucocytozoon from the avian families Otidae, Podargidae and Threskiornithidae, pp. 1261-1267 in Journal of Natural History 36 (11) on page 1265, DOI: 10.1080/00222930110049962, http://zenodo.org/record/529850
Aedophron Lederer 1857
No full textGenus <i>Aedophron</i> Lederer, 1857 <p> <b>Type species:</b> <i>Heliothis rhodites</i> Eversmann, 1851</p>Published as part of <i>Matov, Alexej, Zahiri, Reza & Holloway, Jeremy D., 2008, The Heliothinae of Iran (Lepidoptera: Noctuidae), pp. 1-37 in Zootaxa 1763</i> on page 15, DOI: <a href="http://zenodo.org/record/181966">10.5281/zenodo.181966</a>
P<sub>2</sub> purinoceptor stimulation attenuates PTH inhibition of phosphate uptake by a G protein-dependent mechanism
No full text Pages F309'F316: E. D. Lederer and K. R. McLeish. “P2 purinoceptor stimulation attenuates PTH inhibition of phosphate uptake by a G protein-dependent mechanism.” The American Heart Association of Kentucky should have been acknowledged for their support of E. D. Lederer. </jats:p
P<sub>2</sub> purinoceptor stimulation attenuates PTH inhibition of phosphate uptake by a G protein-dependent mechanism
No full text Pages F309–F316: E. D. Lederer and K. R. McLeish. ”P2 purinoceptor stimulation attenuates PTH inhibition of phosphate uptake by a G protein-dependent mechanism.“ The American Heart Association of Kentucky should have been acknowledged for their support of E. D. Lederer. </jats:p
Personal Papers (MS 80-0002)
No full textLetter from Harris L. Kempner to Lederer discussing the arrival of a C. O. D. parcel to his house and Mrs. Kempner paying the remaining balance for articles, requesting a refund and enclosing a receipt
Leucocytozoon podargii Adlard & Peirce & Lederer 2002, n. sp.
No full textLeucocytozoon podargii n. sp. (gures 1–4) Type host. Podargus strigoides (Latham) (tawny frogmouth). Type locality. Currumbin, Queensland, Australia. Basis of description Macrogametocyte. Only round morphs occur, exhibiting the usual characteristics of the genus. Maximum diameter is about 15 m m (table 1). Cytoplasm is granular with occasional hyaline vacuoles present; nucleus generally oval in shape occupying a central to subcentral position but lacking a distinct karyosome. Maximum length of the host–parasite complex is 17 m m. The host cell nucleus forms a cap around the periphery covering about one-third (gure 1), but in most infections about half the parasites were observed without any remnant of the host cell nucleus (gure 2). Microgametocyte. Similar in size to the macrogametocyt e with the usual diOEerentiating characteristics. The nucleus is dispersed but has a distinctive karyosome which stains a deep red (gure 3). The prevalence of microgametocytes is very low with the ratio to macrogametocyte s being 1:50. Schizonts. In histopathology sections stained with H and E, the presence of schizonts was observed in skeletal muscle, lung and spleen. The schizonts in muscle were small compared to those in lung and spleen (gure 4). Megaloschizonts in lung and spleen tissue measured up to 163 m m in diameter and were characterized by cytomere formation with each section packed with merozoites. Hapantotype. IRCAH: G46311 2 (gametocytes) from Podargus strigoides, coll. Reid, 14 October 1999, Currumbin, Gold Coast, Queensland, Australia; IRCAH: G46311 3 (schizonts) from P. strigoides, coll. Lederer, 14 November 1998, Currumbin, Gold Coast, Queensland, Australia. Parahapantotype. IRCAH: G463114, G463115 and G463116 (gametoctyes) from P. strigoides, coll. Reid, 16 August 1999, 19 September 1999 and 10 October 1999; IRCAH: G46311 7 (schizonts) from P. strigoides, coll. Lederer, 14 November 1998, Currumbin, Gold Coast, Queensland, Australia. Other hosts. Probably occurs throughout the range of the Podargidae. Vectors. Unknown, but assumed to be simuliids. Etymology. Named after the genus of the type host. Comments. Leucocytozoon podargii is a parasite with only round morphs occurring in a small family of birds with a distribution restricted to the Australasian region. The inclusion of schizont stages is a bonus particularly as the presence of megaloschizonts occurs in the absence of second generation elongate morphs. In this respect L. podargii appears to have a life cycle similar to that of Leucocytozoon marchouxi Mathis and Leger (see Peirce et al., 1997). Nothing is currently known regarding the potential pathogenicity of L. podargii; the reason for euthanasia in this particular bird was related to causes other than the Leucocytozoon infection. The low prevalence of microgametocyte s in all the positive birds examined seems to be a characteristic of L. podargii infections. The earliest published record of leucocytozoids from the Podargidae was by Cleland (1915) who recognized Leucocytozoon sp. from P. strigoides from Eidsvold in southern Queensland. Later, Mackerras and Mackerras (1960) assigned leucocytozoids from the same host and locality to Leucocytozoon caprimulgi Kerandel originally described from the nightjar Caprimulgus fossii Hartlaub, a species with Afro-Tropical distribution. Their contention was that host relationships dictated conspecicity of the leucocytozoid. However, C. fossii is placed within the family Caprimulgidae (see Sibley and Monroe, 1990, 1993) while it has long been recognized that P. strigoides is a member of the Australasian-endemic bird family, Podargidae. Taking into account the established host-specicity at family level of Leucocytozoon spp. (see discussion in Bennett et al., 1991) and in the absence of any data to the contrary, we describe this species as L. podargii.Published as part of Adlard, R. D., Peirce, M. A. & Lederer, R., 2002, New species of Leucocytozoon from the avian families Otidae, Podargidae and Threskiornithidae, pp. 1261-1267 in Journal of Natural History 36 (11) on pages 1262-1263, DOI: 10.1080/00222930110049962, http://zenodo.org/record/529850
Aedophron phlebophora Lederer 1858
No full textAedophron phlebophora Lederer, 1858 Pl. 2, fig. 12; male genitalia Pl. 6, fig. 30; female genitalia Pl. 9, fig. 46. Aedophron phlebophora Lederer, 1858, Wien. Ent. Monatschr. 2: 144 (TL.: Syria: Damaskus). Synonymy: Aedophron monotonia Amsel, 1935, Aedophron phlebophora postnigra Draudt, 1937, Aedophron phlebophora postrosea Draudt, 1937, Aedophron pallida Schwingenschuss, 1938. References: Christoph 1873; Hampson 1903; Schwingenschuss 1938; Wiltshire 1951; Hacker & Kautt 1999; Hacker 2001; Hacker & Meineke 2001; Ebert & Hacker 2002 (Aedophron phlebophora). Bionomics: Univoltine (Hacker 2001; Kravchenko et al. 2005). Moths flying from April to August. A few notes on the larvae and bionomics have been given by Schwingenschuss (1938). The species inhabits dry steppe biotopes, from lowlands (70 m) to moderate altitude (2800 m). Larvae feed on Phlomis spp. Distribution: Ponto-Turkestanian. Europe (European part of Turkey), Near East, Caucasus, Transcaucasia, Central Asia (Turkmenistan). – In Iran (Pl. 11, fig. 60) occurs in provinces West Azerbaijan, East Azerbaijan, Mazandaran, Tehran, Qazvin, Zanjan, Kermanshah, Kordestan, Lorestan, Esfahan, Kohkiluyeh va Boyer-Ahmad, Fars and Khuzestan. Material examined: 100 specimens from provinces West Azerbaijan, East Azerbaijan, Mazandaran, Tehran, Qazvin, Zanjan, Kermanshah, Kordestan, Lorestan, Esfahan, Kohkiluyeh va Boyer-Ahmad, Fars and Khuzestan, collected between 26.IV to 2.VIII on elevations from 70 to 2800 m.Published as part of Matov, Alexej, Zahiri, Reza & Holloway, Jeremy D., 2008, The Heliothinae of Iran (Lepidoptera: Noctuidae), pp. 1-37 in Zootaxa 1763 on page 15, DOI: 10.5281/zenodo.18196
Als der HochWohlgebohrne Herr/ Herr Wolff Erich von Bennigsen ... Hoff-Richter in Wittenberg/ Mit ... Fraeulein Eleonoren Charlotten Vitzthumen von Eckstädt ... Am 7. Iunii des 1698sten Jahres Sein Vermählungs-Fest HochAdelich begienge
No full textWolte mit einen wohlmeinenden Glückwunsch ... dabey auffwärtig seyn ... treuergebenster Diener Christian Hartman Lederer/ D
Jiří Lederer (1922–1983) – dziennikarz, dysydent, wygnaniec polityczny
No full textBohaterem szkicu biograficznego jest Jiří Lederer, czeski dziennikarz, działacz polityczny, dysydent, kilkakrotnie więziony. Autor opisuje jego ideowe korzenie i ewolucję od socjalizmu, przez komunizm, do demokratycznego socjalizmu spod znaku Praskiej Wiosny (socjalizmu z ludzką twarzą), aż po przymusową emigrację.The protagonist of the biographical sketch is Jiří Lederer, a Czech journalist, political activist, and dissident who was imprisoned several times. The author describes Lederer’s ideological roots and ideological evolution from socialism through communism, democratic socialism under the banner of Prague Spring (socialism with a human face) to forced emigration
Surfactant protein A and D polymorphisms and methylprednisolone pharmacogenetics in donor lungs
No full textObjective: Surfactant proteins A and D are important molecules involved in lung allograft innate immunity. Genetic polymorphisms of surfactant proteins A and D are associated with various lung diseases. In this study, surfactant protein A and D expression responses were investigated during pharmacogenetics upon methylprednisolone treatment as observed during lung transplantation. Methods: A human cell line (NCI-H441) and precision-cut lung slices from 16 human donors were incubated with methylprednisolone, and surfactant protein A1, surfactant protein A2, and surfactant protein D messenger RNA and surfactant protein A protein expression were assayed. Surfactant protein A1, A2, and D polymorphisms and surfactant protein A gene and protein expressions were determined. Results: In NCI-H441 cells, methylprednisolone treatment at 10−5 M and 10−6 M reduced surfactant protein A1 and surfactant protein A2 messenger RNA and surfactant protein A protein expression (P <.05). A pharmacogenetic relationship was observed in human donor precision-cut lung slices between the surfactant protein A2 (1Ax) variants: Surfactant protein A1, A2, and D messenger RNA expression were greater for 1A0 versus 1A1 (P <.05); surfactant protein A1/surfactant protein A2 genotype 6A26A2/1A01A0 (n = 5) showed greater surfactant protein A1, A2, and D messenger RNA expression and surfactant protein A protein expression compared with the other surfactant protein A1/surfactant protein A2 genotypes (n = 11) (P <.05). Conclusions: The surfactant protein A genotype and methylprednisolone stimuli influence donor lung surfactant protein A and D expression. Lungs carrying the surfactant protein A2 variant 1A0 have a greater expression of surfactant protein A when treated with methylprednisolone. Surfactant protein A polymorphisms could be used to personalize immunosuppressive regimens
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