22,555 research outputs found
The political role of the people's liberation army 1949-1973
This thesis is to study the political role of the People's Liberation Army from the approach of structure and function. The framework of the thesis consists of three major parts, first, the influence of Chinese traditional political culture on, and the formation of, the political role of the PL A; second, the influence of domestic political struggles and external military conflicts on the development of the political role of the PLA; and the third, the analysis of the transition of the PLA's political role from the structure and personnel arrangements of the CCPCC Within the above-mentioned three scopes, this thesis make a thorough discussion on the following: (1) The relationship between the structure of the PRC and the formation of the PLA's political role; (2) How has ideology influenced the army's political role; (3) What is Mao's viewpoint and his influence on the development of the army's political role; (4) What is the link between the army and the party, and how has this developed; (6) What accounts for the expansion of the PLA's political functions; (7) What is the influence of political factional struggles on the PLA's political role; (8) Is it political institution or military institution that controls the recruitment of the military elite; (9) What are the disparities between the military elite in handling international conflicts and what are their political considerations; (10) What is the Party's position in the army; (11) How have the Party’s important meetings and personnel arrangements influenced the rise and fall of the PLA's political role
Mu tian zi zhuan: liu juan.
郭璞註 ; 程榮校.綫裝, 1函.框20.1x14.2公分, 9行20字, 小字雙行同, 白口, 單白魚尾, 左右雙邊. 版心上鐫題名, 中鐫卷次, 下鐫葉次.明刻"漢魏叢書"本?卷末鐫"錢塘郭志學寫"Xian zhuang, 1 han.Kuang 20.1 x 14.2 gong fen, 9 hang 20 zi, xiao zi shuang hang tong, bai kou, dan bai yu wei, zuo you shuang bian. Ban xin shang juan ti ming, zhong juan juan ci, xia juan ye ci.Ming ke "Han Wei cong shu" ben?Juan mo juan "Qiantang Guo Zhixue xie"Guo Pu zhu ; Cheng Rong jiao
Qiu lai ju qin pu
"Qiu lai ju qin pu" wei gu qin da shi Cheng Gongliang xian sheng de gu qin qu ji, shou lu le "Gui qu lai ci", "Yi gu ren", "Xiao xiang shui yun", "Tao yuan chun xiao", "Pao xiu luo lan (ba shou)" deng er shi jiu shou qin qu, wan zheng cheng xian le Cheng Gongliang xian sheng zai ji cheng chuan tong qu mu, da pu, gai bian he chuang zuo fang mian de qin xue cheng j
Graduate recital, piano. Cheng, Y.-J., 1999
Recorded during a live performance at Dalton Center Recital Hall, Western Michigan University, Kalamazoo, Michigan, April 15, 1999, 5:00 p.m., the 484th concert of the School of Music's 1998-1999 season.Yuan-Jen Cheng, piano.In partial fulfillment of the requirements of the Master of Music degree in piano performance, Western Michigan University, 1999.Information from performance program.Partita no. 1 in B-flat major, BWV 825 / Johann Sebastian Bach -- Six bagatelles, op. 126 / Ludwig van Beethoven -- Intermezzo no. 1 in C major. Presto con fuoco ; Intermezzo no. 2 in D-flat major. Assez modéré mais sans lenteur ; Intermezzo in A-flat major. Très allant, tout droit, sans aucun rubata / Francis Poulenc -- Sonata reminiscenza in A minor, op. 38, no. 1: from Forgotten melodies, first cycle / Nicolas Medtner -- Songs without words. Kinderstücke, op. 102, no. 5. Allegro vivace ; Spinnerlied, op. 67, no. 4. Presto ; Duetto, op. 38, no. 6. Andante con moto / Felix Mendelssohn
The granulocytic inducer C/EBPalpha inactivates the myeloid master regulator PU.1
Verschiedene Transkriptionsfaktoren spielen eine Rolle in der Entwicklung myeloischer Zellen. PU.1, ein Transkriptionsfaktor aus der ETS-Familie, ist sowohl für die Entwicklung lymphatischer als auch für die Entwicklung myeloischer Zellen von Bedeutung. Der Transkriptions
faktor C/EBPalpha, ein an den CCAAT-Enhancer bindendes Protein, ist hingegen wesentlich verantwortlich für die Entwicklung von Granulozyten. Wir stellen hier den ersten Nachweis dafür vor, dass C/EBPalpha die Funktion von PU.1 blockiert. PU.1 und C/EBPalpha können einander binden und sind in myeloischen Zellen kolokalisiert. Wenn C/EBPalpha PU.1 bindet, kann PU.1 einen minimalen Promotor mit Bindungsstelle für PU.1 nicht mehr aktivieren. Wir zeigen, dass der Leuzin-Zipper in der DNA-bindenden Domäne von C/EBPalpha mit der beta3/beta4-Region in der DNA-bindenden Domäne von PU.1 interagieren kann. Dadurch wird der Koaktivator von PU.1, c-jun, aus seiner Bindung mit PU.1 verdrängt. C/EBPalpha hemmt PU.1 nicht, indem es Korepressoren rekrutiert. Vielmehr vermindert C/EBPalpha die Expression von PU.1 in U-937-Zellen mit induzierbarem C/EBPalpha, indem es den autoregulatorischen Effekt PU.1 auf den PU.1-Promotor hemmt.
Ausserdem blockiert C/EBPalpha die durch PU.1 bedingte Entwicklung dendritischer Zellen aus CD34+ menschlichen Nabel blutzellen. Diese funktionelle Blockade von PU.1 durch C/EBPalpha könnte einer der Mechanismen sein, mit denen C/EBPalpha den durch PU.1 determinierten Weg der Zelldifferenzierung hemmt und sich Zellen unter dem Einfluss von C/EBPalpha zu Granulozyten entwickeln.Several transcription factors have been shown to play a role in myelopoiesis. PU.1, an ets-family transcription factor, is required for the development of both myeloid and lymphoid lineages while the transcription factor CCAAT/enhancer binding protein family member C/EBPalpha is essential for granulocytic development. We present here the first evidence that C/EBPalpha blocks the function of PU.1. PU.1 and C/EBPalpha interact physically and co-localize in myeloid cells. As a consequence of this interaction C/EBPalpha can inhibit the function of PU.1 to activate a minimal promoter containing only PU.1 DNA binding sites. We further demonstrate that the leucine zipper in the DNA binding domain of C/EBPalpha interacts with the beta3/beta4 region in the DNA binding domain of PU.1, and as a result displaces the PU.1 co-activator c-Jun. Finally, C/EBPalpha blocks PU.1 induced dendritic cell development from CD34+ human cord blood cells. The functional blocking of PU.1 by C/EBPalpha could be the mechanism by which C/EBPalpha inhibits the cell fates specified by PU.1, and directs cell development to the granulocytic lineage
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