162,900 research outputs found
Dr W Compston performing moon-rock analysis
Research School of Earth Sciences, Physical Sciences - Moon Rocks Analysis - Dr. B. W. Chappell, Mr. M. I. Vernon, Dr. W. Compston, Mrs. A. Brown, Prof. A. E. Ringwood, Dr. J. E. Lovering, Dr. S. R. Taylor, Dr. W. Compston, Mr. J. Penningto
Mr Michael J Vernon
Research School of Earth Sciences, Physical Sciences - Moon Rocks Analysis - Dr. B. W. Chappell, Mr. M. I. Vernon, Dr. W. Compston, Mrs. A. Brown, Prof. A. E. Ringwood, Dr. J. E. Lovering, Dr. S. R. Taylor, Dr. W. Compston, Mr. J. Penningto
[Report to Chief J. E. Curry, by an unknown author #1]
Report to Chief J. E. Curry, by an unknown author. The report contains a list of officers who gave depositions to the United States Attorney
[Report to Chief J. E. Curry, by an unknown author #2]
Report to Chief J. E. Curry, by an unknown author. The report contains a list of officers who gave depositions to the United States Attorney
Dr BW Chappell and Mr J Pennington
Research School of Earth Sciences, Physical Sciences - Moon Rocks Analysis - Dr. B. W. Chappell, Mr. M. I. Vernon, Dr. W. Compston, Mrs. A. Brown, Prof. A. E. Ringwood, Dr. J. E. Lovering, Dr. S. R. Taylor, Dr. W. Compston, Mr. J. Penningto
Dr Ian McDougall, Wang Sung Shan, Dr Bill Compston and Chang Fu Teo
Research School of Earth Sciences - Dr. Ian MacDougall, Wang Sung Shan, Dr. Bill Compston, Chung Fu Tao, Dr. S. W. J. Clements, W. Sinclair, A. E. Ringwood, Prof. Lew Gustafson, Leigh Schmidt, Dr. John Cleary, Dr. Jack Ryan, Dr. I. D. Ryabchikov, Dr. David Green, Mr. Bill Hibberson, Dr. Mike McElhinny, Mr. Charles Barton, Mr. Kurt Kloska, Mr. Dieter Burmann, Prof. Hales & other
Murder on the mountain: author talk with Peter J. Wosh
Author talk by Peter J. Wosh on May 5th, 2022, on his book, "Murder on the Mountain: crime, passion, and punishment in gilded age New Jersey.
Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis
Multiple sclerosis is a common disease of the central nervous system in which the interplay between inflammatory and neurodegenerative processes typically results in intermittent neurological disturbance followed by progressive accumulation of disability. Epidemiological studies have shown that genetic factors are primarily responsible for the substantially increased frequency of the disease seen in the relatives of affected individuals, and systematic attempts to identify linkage in multiplex families have confirmed that variation within the major histocompatibility complex (MHC) exerts the greatest individual effect on risk. Modestly powered genome-wide association studies (GWAS) have enabled more than 20 additional risk loci to be identified and have shown that multiple variants exerting modest individual effects have a key role in disease susceptibility. Most of the genetic architecture underlying susceptibility to the disease remains to be defined and is anticipated to require the analysis of sample sizes that are beyond the numbers currently available to individual research groups. In a collaborative GWAS involving 9,772 cases of European descent collected by 23 research groups working in 15 different countries, we have replicated almost all of the previously suggested associations and identified at least a further 29 novel susceptibility loci. Within the MHC we have refined the identity of the HLA-DRB1 risk alleles and confirmed that variation in the HLA-A gene underlies the independent protective effect attributable to the class I region. Immunologically relevant genes are significantly overrepresented among those mapping close to the identified loci and particularly implicate T-helper-cell differentiation in the pathogenesis of multiple sclerosis
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