220,311 research outputs found
Wu Rongfu and Wu Decai talking about Temples in Mianning
No full textWu Decai and Wu Rongfu talk about local Mianning temples (Lingshansi), recall visiting temples in their youth, and about temples nowadaysEquipment: Fostex Fr-2 AKG C 480 B+CK61-ULS (cardiod
Wu Rongfu and Wu Decai talking about Watermills in Mianning
No full textWu Decai talks about different types of mills (for different types of grain)Equipment: Fostex Fr-2 AKG C 480 B+CK61-ULS (cardiod
Wu Rongfu's life story
No full textWu Rongfu briefly tells about his childhood and life.Equipment: Fostex Fr-2 AKG C 480 B+CK61-ULS (cardiod
K.C. Wu Collection
No full textK.C. Wu (1903-1984) served as mayor of several Chinese cities during the 1930s and 1940s and then in December 1949 was appointed Governor of Taiwan by Chiang Kai-shek. Wu emigrated to the United States in 1954. Wu joined the faculty of Armstrong from 1966 to 1973. The collection consists of biographical information, manuscripts mainly of The Chinese Heritage (1982) and early novel Flags and Cross, lecture notes, clippings and information on his lawsuit with the National Endowment for the Humanities in 1972. The Wu family genealogy and writings of K.C. Wu’s father Wu Jing Ming b. 1875, see series four.https://digitalcommons.georgiasouthern.edu/finding-aids-lane/1030/thumbnail.jp
Personal narrative by Wu Decai
No full textWu Decai speaks about her life, since her childhood, through marriage until presentEquipment: Tascam DR-100 AKG C 480 B+CK62-ULS (omnidirectional) + AKG C 480 B Preamplifier (stereo
Control and Filtering for Discrete Linear Repetitive Processes with H infty and ell 2--ell infty Performance
No full textRepetitive processes are characterized by a series of sweeps, termed passes, through a set of dynamics defined over a finite duration known as the pass length. On each pass an output, termed the pass profile, is produced which acts as a forcing function on, and hence contributes to, the dynamics of the next pass profile. This can lead to oscillations which increase in amplitude in the pass to pass direction and cannot be controlled by standard control laws. Here we give new results on the design of physically based control laws for the sub-class of so-called discrete linear repetitive processes which arise in applications areas such as iterative learning control. The main contribution is to show how control law design can be undertaken within the framework of a general robust filtering problem with guaranteed levels of performance. In particular, we develop algorithms for the design of an H? and dynamic output feedback controller and filter which guarantees that the resulting controlled (filtering error) process, respectively, is stable along the pass and has prescribed disturbance attenuation performance as measured by and – norms
Wu, Chieh-ping -- 1981-82 -- Correspondence, Individual -- letter, 1980-12-29
No full textLetter from Graber, C. D. to Wu, Chieh-Ping dated 1980-12-29.Sabin Collection Fair Use Policy</a
Participation of c-FLIP in NLRP3 and AIM2 inflammasome activation
No full textCellular FLICE-inhibitory protein (c-FLIP) is an inhibitor of caspase-8 and is required for macrophage survival. Recent studies have revealed a selective role of caspase-8 in noncanonical IL-1 beta production that is independent of caspase-1 or inflammasome. Here we demonstrated that c-FLIPL is an unexpected contributor to canonical inflammasome activation for the generation of caspase-1 and active IL-1 beta. Hemizygotic deletion of c-FLIP impaired ATP-and monosodium uric acid (MSU)-induced IL-1 beta production in macrophages primed through Toll-like receptors (TLRs). Decreased IL-1 beta expression was attributed to a reduced activation of caspase-1 in c-FLIP hemizygotic cells. In contrast, the production of TNF-alpha was not affected by downregulation in c-FLIP. c-FLIPL interacted with NLRP3 or procaspase-1. c-FLIP is required for the full NLRP3 inflammasome assembly and NLRP3 mitochondrial localization, and c-FLIP is associated with NLRP3 inflammasome. c-FLIP downregulation also reduced AIM2 inflammasome activation. In contrast, c-FLIP inhibited SMAC mimetic-, FasL-, or Dectin-1-induced IL-1 beta generation that is caspase-8-mediated. Our results demonstrate a prominent role of c-FLIPL in the optimal activation of the NLRP3 and AIM2 inflammasomes, and suggest that c-FLIP could be a valid target for treatment of inflammatory diseases caused by over-activation of inflammasomes
Wu-exclusive up-regulated DEGs.
No full text(A) Overlap between all human groups for human-exclusive up-regulated DEGs (up-regulated in any human group, but no mouse group). 1298 genes were upregulated only in Wu and no other human or mouse groups. (B) Although only up-regulated in Wu these 1298 DEGs, nevertheless, return very similar IPA Cytokine USR pathways as those shown in Fig 4A and 4B. (C) The 3%, 18% and 21% of DEGs in the IL6R, TNF and IFNg networks (Fig 4D IL6R, S5 Fig TNF, and S6 Fig IFNG) that were up-regulated only in human (green) comprised 194 genes. Of these DEGs, 73% were found up-regulated exclusively in the Wu dataset. (D) When these 143 DEGs were analysed by IPA Diseases or Functions the highest and lowest annotation by z-score suggest more cell survival and less cell death, consistent with Fig 3C. Thus IL6R, TNF, and IFNG networks contain genes that are also associated with cell survival. The presence of DEGs in these later networks that are only up-regulated in humans (Fig 4D IL6R, S5 Fig TNF, and S6 Fig IFNG, green) is largely due to the Wu dataset. The RNA-Seq data suggests that the tissues used to generate the Wu dataset had less virus (Fig 2A) and less cell death (as also seen in Fig 3C), with pathways somewhat distinct (Fig 3A), perhaps because these samples were collected at a later time point when recovery was well underway and/or because a series of medication were used by the patients. The 3%, 18% and 21% of network genes up-regulated in humans might suggest humans up-regulate these network genes in response to SARS-CoV2 infection, whereas mice do not. However, this may largely be due to the fact that no comparable mouse data set was available (e.g. medicated in the same way). (PDF)</p
Jing wu ti yu hui fu xing di ba jie li jian shi jiu zhi dian li ji chun jie lian huan da hui te kan /c[bian ji zhu ren Luo Junchou ; bian ji wei yuan Liang Shengchi ... [deng]].
No full text書名據封面.內文刊載黃漢勛所著之"精武國術部之的演變"(p. 26).黃漢勛為第一至第三屆"香港精武體育會"之國術主任, 自第五屆起為體育主任.Shu ming ju feng mian.Nei wen kan zai Huang Hanxun suo zhu zhi "Jing wu guo shu bu zhi de yan bian" (p. 26).Huang Hanxun wei di yi zhi di san jie "Xianggang jing wu ti yu hui" zhi guo shu zhu ren, zi di wu jie qi wei ti yu zhu ren
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