30,812 research outputs found
AN EXPERIMENTAL CLASSIFICATION OF DRAIN AND GATE CURRENT PULSES IN LOW-VOLTAGE POWER MOSFETS DURING RADIATION EXPOSURE
No full textExpression of p53, pRB, p21, p27 cyclin D1, Ki-67 and HPV presence in endophytic schneiderian papillomas
No full textNot availabl
Is the physiological maturity at harvest influencing nectarine flavour after cold storage?
No full textPeach and nectarine are often harvested immature to withstand the postharvest
handling without considering the consequence on fruit quality and therefore
consumer’s satisfaction and their likelihood of repurchase. Indeed, the inaccurate
determination of the optimal harvest time leads to a considerable reduction in fruit
quality at consumption. Despite fruit volatile organic compounds (VOCs) being
positively associated with the consumer acceptance, VOCs are not well-considered in
the optimization of pre- and postharvest fruit management. The aim of this research
was to investigate the effect of fruit physiological maturity at harvest on the
development of VOCs during a simulated postharvest chain. Fruits of ‘Stark Red Gold’
nectarine, harvested at different maturity stages, determined by the “index of
absorbance difference” (IAD), were stored at 1°C (90-95% RH) for 20 days then
maintained 6 days in shelf-life at 20°C. The evolution in aroma production was
monitored in each day of shelf life by proton transfer reaction – time of flight – mass
spectrometry (PTR-ToF-MS). Besides the VOC measurement, fruit were assessed for
the main quality traits: soluble solids content, flesh firmness and titratable acidity.
Results of this research shows that the concentration of the main aroma compounds
such as lactones (mainly γ- and δ-decalactones) and esters (mainly methyl acetate)
increased during the progression of ripening while aldehydes (mainly pentanal) and
C6-aldehydes (predominantly hexanal and 3-hexenal) decreased. However the
concentration of these compounds, irrespective of the harvest maturity stages, was
significantly reduced after cold storage, indicating a negative effect of low
temperature in the development of aroma compounds. In addition fruit senescence
was clearly marked by an enhanced emission of off-flavour compounds, such as
ethanol, acetaldehyde and methanol
Erratum to: Effect of moderate red wine intake on cardiac prognosis after recent acute myocardial infarction of subjects with Type 2 diabetes mellitus (Diabetic Medicine, (2006), 23, 9, (974-981), 10.1111/j.1464-5491.2006.01886.x)
No full textIn an article by Marfella et al, the author name C. Saron is incorrect and should be listed as C. Sardu. Therefore the correct author list is: R. Marfella, F. Cacciapuoti, M. Siniscalchi, F. C. Sasso, F. Marchese, F. Cinone, E. Musacchio, M. A. Marfella, L. Ruggiero, G. Chiorazzo, D. Liberti, G. Chiorazzo, G. F. Nicoletti, C. Sardu, F. D'Andrea, C. Ammendola, M. Verza and L. Coppola.In an article by Marfella et al, the author name C. Saron is incorrect and should be listed as C. Sardu. Therefore the correct author list is: R. Marfella, F. Cacciapuoti, M. Siniscalchi, F. C. Sasso, F. Marchese, F. Cinone, E. Musacchio, M. A. Marfella, L. Ruggiero, G. Chiorazzo, D. Liberti, G. Chiorazzo, G. F. Nicoletti, C. Sardu, F. D'Andrea, C. Ammendola, M. Verza and L. Coppola
Numerical simulation of SEB induced by heavy ions in silicon carbide Schottky diode
No full textA simulation study is presented aimed at describing phenomena involved in heavy
ion induced SEB of SiC Schottky diode. It is demonstrated that the failure is caused by
a strong local increase of the temperature
Distribuzione del novellame di Mugilidi (Pisces, Mugilidae) e di spigola (Pisces, Moronidae) in habitat costieri italiani a differente tipologia
No full textElaboration on Kwapien's theorem: Representing bounded mean zero functions f as coboundary f = g ◦ T − g
No full textIn [8] Kwapien proved that every mean zero function f ∈ L∞[0, 1] we can write as f = g ◦ T − g for some g ∈ L∞[0, 1] and some measure preserving transformation T of [0, 1]. However, as was discovered in [4] there is a gap in the proof for the case that f is not continuous. The aim of this bachelor thesis is filling in that gap in the proof. We first extend Kwapien’s proof for continuous functions to certain other measure spaces. Thereafter, we use the method of proof suggested by Kwapien, to proof the theorem for mean zero function f ∈ L∞[0, 1] for which λ(f−1({x})) = 0 for all x ∈ R. Using this result we then proof that every mean zero function f ∈ L∞[0, 1] can be written as a sum f =(g1 ◦ T1 − g1) + (g2 ◦ T2 − g2) where g1, g2 ∈ L∞[0, 1] and where T1, T2 are measure preserving transformations of [0, 1]. We finish this thesis with an application of Kwapien’s theorem in the study to singular traces Applied Mathematic
MAGFET based current sensing for power integrated circuit
No full textA new on-chip non-invasive integrated current sensing, compatible with standard CMOS technology, has been developed, using a 1.2 um BiCMOS ALCATEL technology. to sense the current in the drain side of a power MOSFET. The circuit is based on a split-drain magnetic sensor. implemented on the same chip of an integrated gate driver for a power MOSFET. A CMOS biasing circuit with a differential current output is also developed. The simulation results of the current sensing show a conversion gain of 1.25 mV/mT. (C) 2003 Elsevier Science Ltd. All rights reserved
Are tryptase and cathepsin D related to Helicobacter pylori infection and mucosal gastrin in peptic ulcer?
No full textThe pathogenesis of peptic ulcer is a complex phenomenon and several factors are thought to be involved in this process. Among others, Helicobacter pylori infection, hypergastrinaemia and some proteases seem to play an essential role in inducing peptic ulceration. We investigated whether tryptase (a serine endoprotease released by mast cells) and cathepsin D (a lysosomal hydrolase which seems able to derange the extracellular matrix) play a part in peptic ulcer disease and whether they are linked to Helicobacter pylori infection and mucosal content of gastrin. We studied 13 controls, 25 patients with gastric ulcer, 47 with duodenal ulcer and 11 with duodenitis. Tryptase and cathepsin D were measured in mucosal biopsy specimens (body and antrum of the stomach and duodenum) using IRMA methods. Gastrin was assayed in the antral mucosa by means of a RIA method. Helicobacter pylori infection was histologically evaluated (Giemsa). Tryptase and cathepsin D levels were higher (25%) in patients with active peptic ulcer, whether gastric or duodenal. The mucosal content of cathepsin D, but not that of tryptase, was associated with Helicobacter pylori infection. Tryptase, on the other hand, was related to gastrin content. No correlation was found between the two enzymes. It is concluded that tryptase and cathepsin D probably reflect different pathophysiological modifications in ulcer disease. Cathepsin D seems to be mainly related to the phlogistic reaction of the mucosa to Helicobacter pylori infection; tryptase may reflect and indirect link between the action of gastrin and the function of mast cells. © 1994 Springer-Verlag
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