1,721,045 research outputs found
Methodology matters ⋯ but so does interpretation!
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Acquired perforating disorder reminiscent of elastosis perforans serpiginosa in a patient with Behçet disease
No full text[No abstract available]Abbas O, 2010, INT J DERMATOL, V49, P1244, DOI 10.1111-j.1365-4632.2009.04233.x; Abdullah L, 2010, CLIN EXP DERMATOL, V35, P935, DOI 10.1111-j.1365-2230.2010.03863.x; Alpsoy E, 2003, J Eur Acad Dermatol Venereol, V17, P521, DOI 10.1046-j.1468-3083.2003.00625.x; Saray Y, 2006, J EUR ACAD DERMATOL, V20, P679, DOI 10.1111-j.1468-3083.2006.01571.x; WEISS SJ, 1989, NEW ENGL J MED, V320, P3650
Immunological Differences in Women Compared with Men: Overview and Contributing Factors
No full textCitation: Ghazeeri G, Abdullah L, Abbas O. Immunological differences in women compared with men: overview and contributing factors. Am J Reprod Immunol 2011 Gender differences in the innate and adaptive immune systems have long been observed in humans. These immunological differences in immune function manifest as diverse susceptibilities to different types of infections and varied risks of developing autoimmune disorders and maybe even, cancers. Several factors contribute to the development of this immunological dimorphism including sex hormones, genetic makeup, environmental causes, and more recently microchimerism. Although the aim behind this sexual immune dimorphism is still unclear, it is tempting to believe that the higher risk of developing autoimmune diseases in women somehow serves the higher evolutionary goal of reproduction and creating new life. © 2011 John Wiley and Sons A-S
Recurrent vesicular eruption on the right hand
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Immunological differences in women compared with men: overview and contributing factors.
No full textCitation: Ghazeeri G, Abdullah L, Abbas O. Immunological differences in women compared with men: overview and contributing factors. Am J Reprod Immunol 2011; 66: 163-169 Gender differences in the innate and adaptive immune systems have long been observed in humans. These immunological differences in immune function manifest as diverse susceptibilities to different types of infections and varied risks of developing autoimmune disorders and maybe even, cancers. Several factors contribute to the development of this immunological dimorphism including sex hormones, genetic makeup, environmental causes, and more recently microchimerism. Although the aim behind this sexual immune dimorphism is still unclear, it is tempting to believe that the higher risk of developing autoimmune diseases in women somehow serves the higher evolutionary goal of reproduction and creating new life. © 2011 John Wiley and Sons A-S.Ackerman LS, 2006, ARCH DERMATOL, V142, P371, DOI 10.1001-archderm.142.3.371; Alter MJ, 1999, NEW ENGL J MED, V341, P556, DOI 10.1056-NEJM199908193410802; Asai K, 2001, SHOCK, V16, P340, DOI 10.1097-00024382-200116050-00003; Bouman A, 2004, AM J REPROD IMMUNOL, V52, P19, DOI 10.1111-j.1600-0897.2004.00177.x; Bouman A, 2005, HUM REPROD UPDATE, V11, P411, DOI 10.1093-humupd-dmi008; CACCIARI E, 1981, J IMMUNOGENET, V8, P337; Cutolo M, 2004, J RHEUMATOL, V31, P419; Dal H, 2008, J DERMATOL, V35, P55, DOI 10.1111-j.1346-8138.2008.00416.x; Damian DL, 2008, J INVEST DERMATOL, V128, P447, DOI 10.1038-sj.jid.5701058; Dao H, 2007, GENDER MED, V4, P308; Evans PC, 1999, BLOOD, V93, P2033; Gammill HS, 2010, INT J DEV BIOL, V54, P531, DOI 10.1387-ijdb.082767hg; Giacomoni PU, 2009, J DERMATOL SCI, V55, P144, DOI 10.1016-j.jdermsci.2009.06.001; Giltay EJ, 2000, J CLIN ENDOCR METAB, V85, P1648, DOI 10.1210-jc.85.4.1648; Gleicher N, 2007, J AUTOIMMUN, V28, P1, DOI 10.1016-j.jaut.2006.12.004; Grimaldi CM, 2005, MOL IMMUNOL, V42, P811, DOI 10.1016-j.molimm.2004.05.014; Heldring N, 2007, PHYSIOL REV, V87, P905, DOI 10.1152-physrev.00026.2006; Huber JP, 2010, J IMMUNOL, V185, P813, DOI 10.4049-jimmunol.1000469; IVARSSON SA, 1995, ACTA PAEDIATR, V84, P63, DOI 10.1111-j.1651-2227.1995.tb13485.x; Jacob CO, 2009, P NATL ACAD SCI USA, V106, P6256, DOI 10.1073-pnas.0901181106; Klein SL, 2004, PARASITE IMMUNOL, V26, P247, DOI 10.1111-j.0141-9838.2004.00710.x; Lahita RG, 2000, RHEUM DIS CLIN N AM, V26, P951, DOI 10.1016-S0889-857X(05)70178-2; Lasithiotakis K, 2008, CANCER, V112, P1795, DOI 10.1002-cncr.23359; Lockshin MD, 2010, ANN NY ACAD SCI, V1193, P22, DOI 10.1111-j.1749-6632.2009.05293.x; Medina E, 2001, J INFECT DIS, V184, P846, DOI 10.1086-323292; Naugler WE, 2007, SCIENCE, V317, P121, DOI 10.1126-science.1140485; Oliver JE, 2009, ARTHRITIS RES THER, V11, DOI 10.1186-ar2825; Olsen NJ, 1996, ENDOCR REV, V17, P369, DOI 10.1210-er.17.4.369; Orbach H, 2007, AUTOIMMUN REV, V6, P537, DOI 10.1016-j.autrev.2006.10.005; Ozbalkan Z, 2005, ARTHRITIS RHEUM, V52, P1564, DOI 10.1002-art.21026; Paust S, 2011, NAT IMMUNOL, V131, P500; Paust S, 2010, IMMUNOL REV, V235, P286, DOI 10.1111-j.0105-2896.2010.00906.x; Poon SC, 2003, J INVEST DERMATOL, V121, P184; Prieto Jesús, 2008, J Hepatol, V48, P380, DOI 10.1016-j.jhep.2007.11.007; Romagnani S, 2006, CLIN EXP ALLERGY, V36, P1357, DOI 10.1111-j.1365-2222.2006.02606.x; Rubtsov AV, 2010, AUTOIMMUN REV, V9, P494, DOI 10.1016-j.autrev.2010.02.008; Sader MA, 2005, CLIN ENDOCRINOL, V62, P56, DOI 10.1111-j.1365-2265.2004.02173.x; Saha S, 2011, CLIN REV ALLERG IMMU, V40, P8, DOI 10.1007-s12016-009-8182-6; SanchezGuerrero J, 1996, ARTHRITIS RHEUM, V39, P657, DOI 10.1002-art.1780390418; Schenten D, 2011, ADV IMMUNOL, V109, P87, DOI 10.1016-B978-0-12-387664-5.00003-0; Scofield RH, 2008, ARTHRITIS RHEUM, V58, P2511, DOI 10.1002-art.23701; Streilein J. Wayne, 1994, Journal of Investigative Dermatology, V103, p107S, DOI 10.1111-1523-1747.ep12399400; Szekeres-Bartho J, 2001, INT IMMUNOPHARMACOL, V1, P1037, DOI 10.1016-S1567-5769(01)00035-2; Tanriverdi F, 2003, J ENDOCRINOL, V176, P293, DOI 10.1677-joe.0.1760293; Verthelyi D, 2001, INT IMMUNOPHARMACOL, V1, P983, DOI 10.1016-S1567-5769(01)00044-3; Waldorf KMA, 2008, IMMUNOL INVEST, V37, P631, DOI 10.1080-08820130802205886; Wang J, 2008, CLIN RHEUMATOL, V27, P1183, DOI 10.1007-s10067-008-0937-6; Whitacre CC, 1999, SCIENCE, V283, P1277, DOI 10.1126-science.283.5406.1277; Yancey AL, 2001, INFECT IMMUN, V69, P2865, DOI 10.1128-IAI.69.5.2865-2871.2001; Yeh SH, 2010, ONCOLOGY-BASEL, V78, P172, DOI 10.1159-000315247; Zandman-Goddard G, 2007, AUTOIMMUN REV, V6, P366, DOI 10.1016-j.autrev.2006.10.00114141
Going Beyond Counting First Authors in Author Co-citation Analysis
Full text linkThe present study examines one of the fundamental aspects of author co-citation analysis (ACA) - the way co-citation
counts are defined. Co-citation counting provides the data on which all subsequent statistical analyses and mappings
are based, and we compare ACA results based on two different types of co-citation counting - the traditional type that
only counts the first one among a cited work's authors on the one hand and a non-traditional type that takes into
account the first 5 authors of a cited work on the other hand. Results indicate that the picture produced through this non-traditional author co-citation counting contains more coherent author groups and is therefore considerably clearer. However, this picture represents fewer specialties in the research field being studied than that produced through the traditional first-author co-citation counting when the same number of top-ranked authors is selected and analyzed. Reasons for these effects are discussed
Can you identify this condition?
No full text[No abstract available]Al-Enezi S, 2001, J PEDIATR, V138, P602, DOI 10.1067-mpd.2001.112173; ALSALEH QA, 1994, INT J DERMATOL, V33, P52, DOI 10.1111-j.1365-4362.1994.tb01497.x; ALTMAN J, 1971, ARCH DERMATOL, V104, P385, DOI 10.1001-archderm.104.4.385; DEREURE O, 1994, ACTA DERM-VENEREOL, V74, P208; Kawaguchi Hiroshi, 1999, Journal of Dermatology (Tokyo), V26, P599; Kennedy D, 1996, PEDIATR DERMATOL, V13, P95; Lee S H, 2001, Australas J Dermatol, V42, P252, DOI 10.1046-j.1440-0960.2001.00530.x; Morag C, 1985, Pediatr Dermatol, V3, P15, DOI 10.1111-j.1525-1470.1985.tb00479.x; Oskay T, 2003, CLIN EXP DERMATOL, V28, P557, DOI 10.1046-j.1365-2230.2003.01337.x; ROGERS M, 1992, PEDIATR DERMATOL, V9, P342, DOI 10.1111-j.1525-1470.1992.tb00623.x0
Question: Can you identify this condition? Acute hemorrhagic edema of infancy.
No full text[No abstract available
Variations on the Author
Full text link“Variations on the Author” discusses two of Eduardo Coutinho’s recent films (Um Dia na Vida, from 2010, and Últimas Conversas, posthumously released in 2015) and their contribution to the general question of documentary authorship. The director’s filmography is characterized by a consistent yet self-effacing form of authorial self-inscription: Coutinho often features as an interviewer that rather than express opinions propels discourses; an interviewer that is good at listening. This mode of self-inscription characterizes him as an author who is not expressive but who is nonetheless markedly present on the screen. In Um Dia na Vida, however, Coutinho is completely absent form the image, while Últimas Conversas, on the contrary, includes a confessional prologue that moves the director from the margins to the center of his films. This article examines the ways in which these works stand out in the filmography of a director who offers new insights into the notion of cinematic authorship
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