1,898,492 research outputs found
C-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIOS
No full textC-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIOSC-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIOSJp
C-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIOS
No full textC-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIOSC-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIOSJp
C-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIOS
No full textC-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIOSC-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIOSJp
C-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIOS
No full textC-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIO
C-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIOS
No full textC-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIO
C-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIOS
No full textC-194 PROMUEVE IPN MODELO INSTITUCIONAL DE ACELERACIÓN DE NEGOCIO
Additional file 2 of Increased expression of miR-194-5p through the circPVRL3/miR-194-5p/SOCS2 axis promotes proliferation and metastasis in pancreatic ductal adenocarcinoma by activating the PI3K/AKT signaling pathway
No full textAdditional file 2: Figure S1. The expression validation of miR-194-5p and KEGG pathway analysis. (A) The expression of miR-194-5p-related genes was observed by RT‒qPCR in the NC group and groups treated with 50 nM miR-194-5p mimic or 200 nM miR-194-5p inhibitor in PANC-1 cells. (B) The image shows the clustering heatmap of DEGs by RNA sequencing in PANC-1 cells with treated miR-194-5p mimic or not. (C) The KEGG analysis showed that genes related to overexpression of miR-194-5p are related to the cell cycle and miRNAs in cancer based on the results of mRNA-seq
Additional file 4 of Increased expression of miR-194-5p through the circPVRL3/miR-194-5p/SOCS2 axis promotes proliferation and metastasis in pancreatic ductal adenocarcinoma by activating the PI3K/AKT signaling pathway
No full textAdditional file 4: Figure S3. MiR-194-5p affects the expression of SOCS2 which reverses the effects of miR-194-5p on cell proliferation and migration. (A) and (B), the number of colonies or cells was counted in the control group, miR-194-5p mimic or miR-194-5p inhibitor group, miR-194-5p mimic or miR-194-5p inhibitor group and SOCS2 overexpression plasmid or si-SOCS2 co-transfected group by colony formation and Transwell assays. (C) HE staining (scale bar = 100 μm) and the expression of SOCS2 by IHC staining (scale bar = 50 μm) of tumors derived from the subcutaneous xenograft model. The results were evaluated by IHC scoring (n = 5)
Going Beyond Counting First Authors in Author Co-citation Analysis
Full text linkThe present study examines one of the fundamental aspects of author co-citation analysis (ACA) - the way co-citation
counts are defined. Co-citation counting provides the data on which all subsequent statistical analyses and mappings
are based, and we compare ACA results based on two different types of co-citation counting - the traditional type that
only counts the first one among a cited work's authors on the one hand and a non-traditional type that takes into
account the first 5 authors of a cited work on the other hand. Results indicate that the picture produced through this non-traditional author co-citation counting contains more coherent author groups and is therefore considerably clearer. However, this picture represents fewer specialties in the research field being studied than that produced through the traditional first-author co-citation counting when the same number of top-ranked authors is selected and analyzed. Reasons for these effects are discussed
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