Landspítali University Hospital Research Archive
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Why should women age 40-49 years not participate in breast cancer screening?
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Syndrome of transient headache and neurologic deficits with cerebrospinal fluid lymphocytosis – two cases and review
No full textTo access publisher's full text version of this article, please click on the hyperlink in Additional Links field or click on the hyperlink at the top of the page marked DownloadHér er lýst tveimur tilfellum af HaNDL (Headache with Neurological
Deficits and cerebrospinal fluid Lymphocytosis) eða heilkenni skammvinns höfuðverkjar með brottfallseinkennum og eitilfrumuhækkun í
mænuvökva. Fyrra tilfellið var þrítugur maður sem fékk endurtekin
köst með höfuðverk, helftareinkennum og mikilli óáttun. Hið síðara
var 41 árs maður sem fékk höfuðverk, skyndilegt málstol og hægri
helftareinkenni. Í báðum tilfellum var töluverð hækkun á eitilfrumum í
mænuvökva. Leit að sýkingarvöldum var neikvæð og segulómskoðanir
af höfði sýndu engar meinsemdir. Einkenni gengu að fullu til baka hjá
báðum sjúklingum.
Orsök HaNDL er óþekkt en sumir telja ástandið orsakast af bólguviðbrögðum í kjölfar veirusýkingar. Horfur eru góðar og sjúklingar
verða einkennalausir á einni til þremur vikum. Mikilvægt er að útiloka
alvarlegri orsakir eins og heilaslag, innanskúmsblæðingu eða sýkingar
í miðtaugakerfi.Here we describe two cases of HaNDL (Headache with Neurological Deficits
and cerebrospinal fluid Lymphocytosis). A thirty year old man with episodes of
headache with lateralizing symptoms and confusion and a 41 year old man with
headache, aphasia and right hemiparesis. Symptoms resolved completely in both
patients. Considerable cerebrospinal fluid lymphocytosis was present but no signs
of CNS infection and MRIs of the brain were normal. Although the cause of HaNDL
is unknown, it is thought to be triggered by a viral infection by some. The prognosis
is excellent and symptoms normally resolve within 1-3 weeks. It is important to
rule out more serious etiologies like stroke, subarachnoid hemorrhage or central
nervous system infections
A prospective study on the role of smoking, environmental tobacco smoke, indoor painting and living in old or new buildings on asthma, rhinitis and respiratory symptoms.
Get PDFTo access publisher's full text version of this article, please click on the hyperlink in Additional Links field or click on the hyperlink at the top of the page marked DownloadWe studied associations between tobacco smoke, home environment and respiratory health in a 10 year follow up of a cohort of 11,506 adults in Northern Europe. Multilevel logistic regression models were applied to estimate onset and remission of symptoms. Current smokers at baseline developed more respiratory symptoms (OR = 1.39-4.43) and rhinitis symptoms (OR = 1.35). Starting smoking during follow up increased the risk of new respiratory symptoms (OR = 1.54-1.97) and quitting smoking decreased the risk (OR = 0.34-0.60). ETS at baseline increased the risk of wheeze (OR = 1.26). Combined ETS at baseline or follow up increased the risk of wheeze (OR = 1.27) and nocturnal cough (OR = 1.22). Wood painting at baseline reduced remission of asthma (OR 95%CI: 0.61, 0.38-0.99). Floor painting at home increased productive cough (OR 95%CI: 1.64, 1.15-2.34) and decreased remission of wheeze (OR 95%CI: 0.63, 0.40-0.996). Indoor painting (OR 95%CI: 1.43, 1.16-1.75) and floor painting (OR 95%CI: 1.77, 1.11-2.82) increased remission of allergic rhinitis. Living in the oldest buildings (constructed before 1960) was associated with higher onset of nocturnal cough and doctor diagnosed asthma. Living in the newest buildings (constructed 1986-2001) was associated with higher onset of nocturnal breathlessness (OR = 1.39) and rhinitis (OR = 1.34). In conclusion, smoking, ETS and painting indoor can be risk factors for respiratory symptoms. Wood painting and floor painting can reduce remission of respiratory symptoms. Smoking can increase rhinitis. Living in older buildings can be a risk factor for nocturnal cough and doctor diagnosed asthma. Living in new buildings can increase nocturnal dyspnoea and rhinitis.
Keywords: Asthma; Environmental tobacco smoke; Onset and remission; Painting; Rhinitis; Smoking.Icelandic Research Council
Swedish Heart-Lung Foundation
Vardal Foundation for Health Care and Allergic Research
Swedish Association Against Asthma and Allergy
Swedish Council for Work Live and Social Research
Swedish AFA Insurance
Bror Hjerpstedt Foundation
Research Council of Norway
Norwegian Asthma and Allergy Association
Danish Lung Association
Estonian Science Foundatio
Question: Would you consider teaching physiotherapy?
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Hvernig getum við sjúkraþjálfarar skilgreint og mælt forvarnargildi meðferðar? Viðtal við Dr. Elizabeth Dean, sjúkraþjálfara
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Physiotherapy in Mental Health: A Historical Overview
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Physiotherapy Assessments in Mental Health Care - Are we measuring the right things in a societal perspective?
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Clinical practice guideline on spinal stabilisation of adult trauma patients: Endorsement by the Scandinavian Society of Anaesthesiology and Intensive Care Medicine.
Get PDFTo access publisher's full text version of this article, please click on the hyperlink in Additional Links field or click on the hyperlink at the top of the page marked DownloadThe Clinical Practice Committee of the Scandinavian Society of Anaesthesiology and Intensive Care Medicine endorses the clinical practice guideline New clinical guidelines on the spinal stabilisation of adult trauma patients-consensus and evidence based. The guideline can serve as a useful decision aid for clinicians caring for patients with traumatic spinal cord injury. However, it is important to acknowledge that the overall certainty of evidence supporting the guideline recommendations was low, implying that further research is likely to have an important impact on the confidence in the estimate of effect.
Keywords: SSAI; clinical practice guideline; endorsement; spinal stabilisation; trauma.Scandinavian Society of Anaesthesiology and Intensive Care Medicin
October sixth; a historic day in the fight against malaria
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Critical care management of patients with spontaneous subarachnoid haemorrhage – a review
No full textTo access publisher's full text version of this article, please click on the hyperlink in Additional Links field or click on the hyperlink at the top of the page marked DownloadSjálfsprottnar innanskúmsblæðingar eru blæðingar inn í innanskúmshol
heilans sem ekki eru afleiðingar áverka. Algengasta orsökin er brostinn
æðagúll í slagæðakerfi heilans. Þessum blæðingum geta fylgt alvarlegir
fylgikvillar, svo sem endurblæðing, æðasamdráttur og heilablóðþurrð.
Dánartíðni er há og stór hluti þeirra sem lifir af situr uppi með langtíma
afleiðingar blæðingarinnar. Lokun á blæðingarstað er lykilatriði í meðferð
þessara sjúklinga en slíkt inngrip ber að framkvæma sem allra fyrst,
eða innan 72 klukkustunda. Þörf er á nánu eftirliti á gjörgæsludeild
þar sem áhersla er lögð á nákvæma blóðþrýstingsstjórnun, vöktun á
vökvajafnvægi og blóðsöltum og náið eftirlit með meðvitundarstigi. Allir
sjúklingar með sjálfsprottna innanskúmsblæðingu ætti að meðhöndla
með kalsíumhemlinum nímódipíni sem sýnt hefur verið fram á að minnki
hættuna á æðasamdrætti og heilablóðþurrð sem er meðal alvarlegustu
fylgikvillar þessara blæðinga.Spontaneous subarachnoid haemorrhage is characterized by extravasation
of blood into the subarachnoid space without a preceding trauma. The leading
cause is a ruptured intracranial aneurysm. Serious neurologic complications can
occur, such as rebleeding, cerebral vasospasm and delayed cerebral ischemia.
Subarachnoid haemorrhage is a serious condition with a high mortality rate and
those who survive often suffer long-term consequences. Prevention of rebleeding
by aneurysm repair is essential and guidelines recommend this procedure should
be done as soon as possible or within 72 hours. Management requires intensive
care with emphasis on accurate blood pressure control, maintaining normal fluid
and electrolyte balance and monitoring the level of consciousness. All patients
should be treated with the calcium channel blocker nimodipine to reduce the risk
of vasospasm and delayed cerebral ischemia which are among the most serious
complications of subarachnoid haemorrhage