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X-ray diffractograms for titanium dioxide, graphitic carbon nitride, and their composites
No full textX-ray diffractometry (XRD) spectra for titanium dioxide (TiO2), graphitic carbon nitride (g-C3N4), and their composites annealed at 200 and 300 oC. Bulk graphitic carbon nitride was prepared by polymerizing pure urea at 600 oC. Sol of TiO2 nanoparticles was annealed at 200 and 300 oC. Composites of TiO2 with g-C3N4 were prepared by adding urea to the TiO2 sol, lyophilizing the resulting mixture, and then annealing it at 200 and 300 oC
Spatial and temporal modulation in time-domain diffuse optical tomography: Dataset and MATLAB Code for Visualization
No full textThis dataset contains Hadamard pattern illumination and time-domain diffuse optical tomography (TD-DOT) measurements, including both simulated time domain data and frequency-domain converted data. MATLAB scripts are provided for visualizing Hadamard patterns, data and reconstructing 3D maps of optical properties. The dataset supports the forthcoming publication “Spatial and temporal modulation in time-domain diffuse optical tomography"
Effects of parental characteristics and different intensive feeding methods on the growth and survival of perch (Perca fluavitilis) larvae.
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Mechanobiochemical finite element model to analyze impact‑loading‑induced cell damage, subsequent proteoglycan loss, and anti‑oxidative treatment effects in articular cartilage
No full textJoint trauma often leads to articular cartilage degeneration and post-traumatic osteoarthritis (PTOA). Pivotal determinants include trauma-induced excessive tissue strains that damage cartilage cells. As a downstream effect, these damaged cells can trigger cartilage degeneration via oxidative stress, cell death, and proteolytic tissue degeneration. N-acetylcysteine (NAC) has emerged as an antioxidant capable of inhibiting oxidative stress, cell death, and cartilage degeneration post-impact. However, the temporal effects of NAC are not fully understood and remain difficult to assess solely by physical experiments. Thus, we developed a computational finite element analysis framework to simulate a drop tower impact of cartilage in Abaqus, and subsequent oxidative stress-related cell damage, and NAC treatment upon cartilage proteoglycan content in Comsol Multiphysics, based on prior ex vivo experiments. Model results provide evidence that immediate NAC treatment can reduce proteoglycan loss by mitigating oxidative stress, cell death (improved proteoglycan biosynthesis) and enzymatic proteoglycan depletion. Our simulations also indicate that delayed NAC treatment may not inhibit cartilage proteoglycan loss despite reduced cell death after impact. These results enhance understanding of the temporal effects of impact-related cell damage and treatment that are critical for the development of effective treatments for PTOA. In the future, our modeling framework could increase understanding of time-dependent mechanisms of oxidative stress and downstream effects in injured cartilage and aid in developing better treatments to mitigate PTOA progression.
This dataset includes the files needed to run the computational models
