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Type II diabetes mellitus and hyperhomocysteinemia: a complex interaction

Marc Uemura

Faisal Fa’ak

Cara Haymaker

Natalie McQuail

Elizabeth Sirmans

Courtney W. Hudgens

Lydia Barbara

Chantale Bernatchez

Jonathan L. Curry

Patrick Hwu

Michael T. Tetzlaff

Adi Diab

Uemura et al. Journal for ImmunoTherapy of Cancer  (2017) 5:7 
DOI 10.1186/s40425-017-0208-7ERRATUM Open AccessErratum to: A case report of Grover’s
disease from immunotherapy-a skin
toxicity induced by inhibition of CTLA-4
but not PD-1
Marc Uemura1†, Faisal Fa’ak1†, Cara Haymaker1, Natalie McQuail1, Elizabeth Sirmans1, Courtney W. Hudgens2,
Lydia Barbara1, Chantale Bernatchez1, Jonathan L. Curry2, Patrick Hwu1, Michael T. Tetzlaff2 and Adi Diab1*Erratum
Unfortunately, after publication of this article [1], it was
noticed that the name of the second author was incor-
rectly displayed as Faa’k Faisal. The correct name is Faisal
Fa’ak and can be seen in the corrected author list above.
The original article has also been updated to correct this
error.
Author details
1Department of Melanoma Medical Oncology, University of Texas-MD
Anderson Cancer Center, Houston, TX, USA. 2Department of Pathology,
University of Texas-MD Anderson Cancer Center, Houston, TX, USA.
Reference
1. Uemura M, Fa’ak F, Haymaker C, McQuail N, Sirmans E, Hudgens CW,
Barbara L, Bernatchez C, Curry JL, Hwu P, Tetzlaff MT, Diab A. A case
report of Grover’s disease from immunotherapy-a skin toxicity induced
by inhibition of CTLA-4 but not PD-1. J Immunother Cancer. 2016;4:55.
http://doi.org/10.1186/s40425-016-0157-6.* Correspondence: adiab@mdanderson.org
†Equal contributors
1Department of Melanoma Medical Oncology, University of Texas-MD
Anderson Cancer Center, Houston, TX, USA
© The Author(s). 2017 Open Access This artic
International License (http://creativecommons
reproduction in any medium, provided you g
the Creative Commons license, and indicate if
(http://creativecommons.org/publicdomain/zele is distributed under the terms of the Creative Commons Attribution 4.0
.org/licenses/by/4.0/), which permits unrestricted use, distribution, and
ive appropriate credit to the original author(s) and the source, provide a link to
changes were made. The Creative Commons Public Domain Dedication waiver
ro/1.0/) applies to the data made available in this article, unless otherwise stated.


Erratum to: A case report of Grover’s disease from immunotherapy-a skin toxicity induced by inhibition of CTLA-4 but not PD-1

Visual mismatch negativity to masked stimuli presented at very brief presentation rates

João Batista Borges

Tomas Hansen

Anders Larsson

Göran Hedenstierna

Intensive Care Med
DOI 10.1007/s00134-017-4708-1
IMAGING IN INTENSIVE CARE MEDICINE
The “normal” ventilated airspaces suffer 
the most damaging effects of mechanical 
ventilation
João Batista Borges1,2*, Tomas Hansen3, Anders Larsson1 and Göran Hedenstierna4
© 2017 The Author(s) . This article is published with open access at Springerlink.com
A patient was treated because of acute respiratory dis-
tress syndrome (ARDS) with multiple fractures, lung 
contusion and bilateral pneumothorax. Figure  1a shows 
the first CT scan when the patient was ventilated in 
supine position in control mode with at that time (1996) 
standard high tidal volume of 12 ml/PBW, positive end-
expiratory pressure of 5–10 cmH2O and end-inspiratory 
pressures of 35–40 cmH2O. It is from her early phase 
of ARDS and displays dependent collapse and “normal-
appearing” parenchyma ventrally. A follow-up CT after 
13 days, now during spontaneous breathing without any 
mechanical support, demonstrates the opposite pattern 
with opacities in the ventral portion and clearance of 
the lower half of the lung (Fig. 1b). This shift of densities 
may be explained by the ventilated, “apparently-spared” 
units suffering more than collapsed, dependent units and 
hyperinflated non-ventilated units. This resulted in ven-
tilator-induced lung injury (VILI). More recently we got 
support for this assumption. We studied the distribution 
of early pulmonary inflammation in a porcine VILI model 
by measuring regional pulmonary uptake of [18F]fluoro-
2-deoxy-d-glucose with positron emission tomography 
combined with CT. Ventilated, normally and poorly aer-
ated units in the mid zone of the lung were the primary 
targets of the inflammation and not hyperinflated or col-
lapsed units (Fig. 1c). Decreased tidal volume/distending 
pressure and increased ventilated lung volume should 
thus minimize VILI.
*Correspondence:  joao.batista_borges@surgsci.uu.se 
1 Hedenstierna Laboratory, Department of Surgical Sciences,  
Section of Anaesthesiology & Critical Care, Uppsala University,  
751 85 Uppsala, Sweden
Full author information is available at the end of the article
Author details
1 Hedenstierna Laboratory, Department of Surgical Sciences, Section  
of Anaesthesiology & Critical Care, Uppsala University, 751 85 Uppsala, 
Sweden. 2 Pulmonary Division, Heart Institute (InCor), Hospital das Clinicas, 
University of São Paulo, São Paulo, Brazil. 3 Department of Radiology, Uppsala 
University Hospital, Uppsala, Sweden. 4 Hedenstierna Laboratory, Department 
of Medical Sciences, Clinical Physiology, Uppsala University, Uppsala, Sweden. 
Open Access This article is distributed under the terms of the Creative 
Commons Attribution-NonCommercial 4.0 International License (http://crea-
tivecommons.org/licenses/by-nc/4.0/), which permits any noncommercial 
Fig. 1 a, b Patient with ARDS. c Animal model of ARDS
use, distribution, and reproduction in any medium, provided you give appro-
priate credit to the original author(s) and the source, provide a link to the Crea-
tive Commons license, and indicate if changes were made.  
Received: 13 January 2017   Accepted: 30 January 2017


The “normal” ventilated airspaces suffer the most damaging effects of mechanical ventilation

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