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AKRASIA IN MEDIATION AND IN THE ADHESION TO ARBITRATION PROCESSES
No full textIn this article we propose, still in an embryonic way, to analyze the Akrasia theme in the mediation pacts and the adhesion to arbitration processes.For this purpose, we consider some critical subjects for ADRs and, the Right: the asymmetrical relations, the contexts of imbalance of powers (in this article, labor and, gender); the high degree of conflict and violence (as domestic), bringing particular questions that seem relevant to us.In our point of view the Akrasia theme in ADRs is a significant field to research to a) understand how Akrasia can occur in the adhesion to arbitration and the pacts (settlements or agreements) in the mediation process; b) know how Akrasia can happen in asymmetrical relations and high degree of conflict and violence; c) identify instruments for overcoming Akrasia in ADRs and; d) understand if Akrasia would be able to invalidate the adhesion to ADRs and decision-making acts.It is relevant to think and talk about Akrasia in ADRs that is currently encouraged by the Brazilian Judicial Justice Policies.At these days, in Brazil, there is practically an epidemic phenomenon of ADRs chambers creation, projects in the Judiciary, and where we have quickly trained mediators and arbitrators, who start their work without a due reflection time, in a supposed new glamorous business market.So, We are talking about Akrasia in ADRs, thinking about autonomy in the decision-making process, especially in delicate situations of asymmetry, bringing any particular questions that seem relevant to us, in this context and to this context.Are we accepting Akrasia in arbitration adhesions and the mediation decision-making process, against the autonomy of the will? Is this an issue, a theme that interests just Brazil?Article信州大学経法論集 5(ブラジル・日本国際セミナー特集号) : 179-205(2019)departmental bulletin pape
Diversity of respiratory impedance based on quantitative computed tomography in patients with COPD（定量CTに基づくCOPD患者における呼吸インピーダンスの多様性）
No full text信州大学(Shinshu university)博士（医学）雑誌に発表。INTERNATIONAL JOURNAL OF CHRONIC OBSTRUCTIVE PULMONARY DISEASE. 13:1841-1849 (2018); doi:10.2147/COPD.S163129.Thesis和田 洋典. Diversity of respiratory impedance based on quantitative computed tomography in patients with COPD（定量CTに基づくCOPD患者における呼吸インピーダンスの多様性）. 信州大学, 2019, 博士論文.doctoral thesi
Transcranial motor-evoked potentials of laryngeal muscles for intraoperative neuromonitoring of the vagus nerve during thyroid surgery（甲状腺手術中の迷走神経機能モニタリングとしての喉頭筋経頭蓋運動誘発電位）
No full text信州大学(Shinshu university)博士（医学）雑誌に発表。JOURNAL OF ANESTHESIA. 33(2):221-229 (2019); doi:10.1007/s00540-018-2601-x.Thesis市野 隆. Transcranial motor-evoked potentials of laryngeal muscles for intraoperative neuromonitoring of the vagus nerve during thyroid surgery（甲状腺手術中の迷走神経機能モニタリングとしての喉頭筋経頭蓋運動誘発電位）. 信州大学, 2019, 博士論文.doctoral thesi
Investigation into the relationship between human feeling and characteristics of wood by using subjective and objective evaluations （主観的客観的評価法を用いた木材感性評価に関する研究）
No full text信州大学(Shinshu university)博士（工学）Thesis設楽 稔那子. Investigation into the relationship between human feeling and characteristics of wood by using subjective and objective evaluations （主観的客観的評価法を用いた木材感性評価に関する研究）. 信州大学, 2019, 博士論文. 博士（工学）, 甲第698号, 平成31年03月20日授与.doctoral thesi
カーボン薄膜のイオン透過に関する物理化学的研究
No full text信州大学(Shinshu university)博士（工学）Thesis谷垣 直人. カーボン薄膜のイオン透過に関する物理化学的研究. 信州大学, 2019, 博士論文. 博士（工学）, 甲第708号, 平成31年03月20日授与.doctoral thesi
消費電力量を可視化するロボット競技用消費電力計測教材の開発と評価
No full text中学校技術・家庭科技術分野でのエネルギー変換に関する技術におけるロボット学習向けに，消費電力を可視化できる消費電力計測教材を開発した。対戦する２台の手動制御型ロボットにこの教材から電源を供給することにより，各ロボットの消費電力をリアルタイムにグラフ化してPC画面（ないしプロジェクタ）上に提示できるようにした。さらにあらかじめ設定した消費電力量に至り次第，当該ロボットへの電力供給を停止できる機能を持たせることで，消費電力を強く意識したロボット競技ができるようにした。実際のロボット競技で試用実践し，所定の機能を確認した。Article日本産業技術教育学会誌.54(2):49-57(2012)journal articl
信州大学志賀自然教育園周辺における自動撮影カメラによる中・大型哺乳類相調査：2017年調査の結果
No full textThe large and medium-sized mammal fauna around the Institute of Nature Education in Shiga Heights, Shinshu University, in the northern part of Nagano Prefecture, Japan was surveyed using camera traps from April to December 2017. Seventeen cameras were set up within two plots and a total of 2111 days were spent camera-trapping. Twenty species of wild mammals were recorded by the cameras :Chiroptera sp.,Muridae sp.,Macaca fuscata, Sciurus lis, Lepus brachyurus, Vulpes vulpes, Martes melampus, Meles anakuma, Ursus thibetanus, Cervus nippon, Sus scrofa and Capricornis crispus.Article志賀自然教育研究施設研究業績(56) : 7-14(2019)departmental bulletin pape
Deficiency of Angulin-2/ILDR1, a Tricellular Tight Junction-Associated Membrane Protein, Causes Deafness with Cochlear Hair Cell Degeneration in Mice
No full textTricellular tight junctions seal the extracellular spaces of tricellular contacts, where the vertices of three epithelial cells meet, and are required for the establishment of a strong barrier function of the epithelial cellular sheet. Angulins and tricellulin are known as specific protein components of tricellular tight junctions, where angulins recruit tricellulin. Mutations in the genes encoding angulin-2/ILDR1 and tricellulin have been reported to cause human hereditary deafness DFNB42 and DFNB49, respectively. To investigate the pathogenesis of DFNB42, we analyzed mice with a targeted disruption of Ildr1, which encodes angulin-2/ILDR1. Ildr1 null mice exhibited profound deafness. Hair cells in the cochlea of Ildr1 null mice develop normally, but begin to degenerate by two weeks after birth. Tricellulin localization at tricellular contacts of the organ of Corti in the cochlea was retained in Ildr1 null mice, but its distribution along the depth of tricellular contacts was affected. Interestingly, compensatory tricellular contact localization of angulin-1/LSR was observed in the organ of Corti in Ildr1 null mice although it was hardly detected in the organ of Corti in wild-type mice. The onset of hair cell degeneration in Ildr1 null mice was earlier than that in the reported Tric mutant mice, which mimic one of the tricellulin mutations in DFNB49 deafness. These results indicate that the angulin-2/ILDR1 deficiency causes the postnatal degenerative loss of hair cells in the cochlea, leading to human deafness DFNB42. Our data also suggest that angulin family proteins have distinct functions in addition to their common roles of tricellulin recruitment and that the function of angulin-2/ILDR1 for hearing cannot be substituted by angulin-1/LSR.ArticlePLOS ONE.10(3):e0120674(2015)journal articl
Influence of Individual Differences on the Calculation Method for FBG-Type Blood Pressure Sensors
No full textIn this paper, we propose a blood pressure calculation and associated measurement method that by using a fiber Bragg grating (FBG) sensor. There are several points at which the pulse can be measured on the surface of the human body, and when a FBG sensor located at any of these points, the pulse wave signal can be measured. The measured waveform is similar to the acceleration pulse wave. The pulse wave signal changes depending on several factors, including whether or not the individual is healthy and/or elderly. The measured pulse wave signal can be used to calculate the blood pressure using a calibration curve, which is constructed by a partial least squares (PLS) regression analysis using a reference blood pressure and the pulse wave signal. In this paper, we focus on the influence of individual differences from calculated blood pressure based on each calibration curve. In our study, the calculated blood pressure from both the individual and overall calibration curves were compared, and our results show that the calculated blood pressure based on the overall calibration curve had a lower measurement accuracy than that based on an individual calibration curve. We also found that the influence of the individual differences on the calculated blood pressure when using the FBG sensor method were very low. Therefore, the FBG sensor method that we developed for measuring the blood pressure was found to be suitable for use by many people.ArticleSENSORS.17(1):48(2016)journal articl
Clinical Characteristics and Predictors of Mortality in Patients with Combined Pulmonary Fibrosis and Emphysema Syndrome and Lung Cancer
No full textRationale: We performed this retrospective study to clarify the clinical characteristics, survival and mortality predictors in patients with combined pulmonary fibrosis and emphysema (CPFE) and lung cancer.Methods: We retrospectively reviewed the medical records of a total of 123 patients with lung cancer, as confirmed according to histological or cytological examinations. Based on the findings of chest CT, the patients were categorized into four groups: LC+normal (n=70); LC+emphysema (n=26); LC+fibrosis (n=10); LC+CPFE (n=17). The clinical characteristics and survival of the LC+CPFE group were compared with those of the other groups. In addition, mortality predictors were evaluated in the LC+CPFE group.Results: The proportion of females was significantly higher in the LC+normal group than in the LC+CPFE and LC+emphysema groups. Significantly more patients were diagnosed with squamous cell carcinoma in the LC+CPFE group than in the LC+normal group. The proportion of patients whose primary mass was located in “nonsubpleural” areas was significantly higher in patients with CPFE who also had lung cancer in the upper lobe than in those with CPFE who also had lung cancer in the other sites. There were significant differences in survival between the LC+normal group and the other groups, whereas there were no significant differences in survival among the LC+emphysema, LC+fibrosis and LC+CPFE groups. In the LC+CPFE group, the patients with a high level of serum KL-6 at diagnosis and upper lobe lung cancer demonstrated a high risk of death. A high level of serum KL-6 at diagnosis was also independently associated with a high risk of death.Conclusions: Patients with CPFE and lung cancer may have distinct clinical characteristics. Strict follow-up is required in patients with CPFE and lung cancer whose serum KL-6 level at diagnosis is higher than the normal range and/or the primary mass of lung cancer is located in the upper lobe.ArticleJournal of Pulmonary and Respiratory Medicine.5(3):263(2015)journal articl