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Phedimusゾク リョクカ ショクブツ ニ オケル キセツテキ ケイタイ ヘンカ ノ マルチ スペクトル プロファイリング ト イデンガクテキ カイセキ
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イヌ ノ コウクウビクウロウ ノ エキガクテキ トクチョウ ナラビニ コウハツケンシュ ミニチュアダックスフンド ノ ジョウガク ケンシ シシュウビョウ ノ シンコウ パターン
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キチンナノファイバー ノ シツカンセツナイ チュウシャ ニオケル ヘンケイセイ シツカンセツショウ ノ ヨクセイコウカ : ヘンケイセイ カンセツショウ モデル ラット オ モチイタ ケンキュウ
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ＡＭＩＧＯ２ ハ フクマク チュウヒ サイボウ ヘノ セッチャク ソクシン オ カイシテ 、 ランソウ ショウエキセイ ガン ニオケル フクマク ハシュ ノ ハンイ カクダイ ニ カンヨスル
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Thyroid hormone may predict treatment failure in Kawasaki disease
No full textBackground, In systemic inflammatory conditions, inflammatory cytokines can cause low thyroid hormone levels. There are no reports discussing the relation between thyroid hormone levels and response to treatment for Kawasaki disease. Methods, We investigated 67 patients who underwent treatment in the acute phase of Kawasaki disease. We divided patients into two groups based on their response to initial intravenous immunoglobulin (IVIG) treatment: the responder group (n=40), and the non-responder group (n=27). The serum levels of the thyroid hormones free triiodothyronine (FT3), free thyroxine (FT4), and thyroid-stimulating hormone (TSH) were compared before and after treatment in all patients, and between responder and non-responder groups. Results, FT3, FT4, and TSH levels were low before the initial treatment and increased significantly after treatment (p<0.05). FT3, FT4, and TSH levels before treatment were significantly lower in the non-responder group than in the responder group (p<0.05). Logistic regression analysis implied that adding FT4 to the Gunma score improved the accuracy of treatment failure. Conclusions, Thyroid hormone and TSH levels were lower in the non-responder group than in the responder group in the initial IVIG treatment for Kawasaki disease. This study suggests that Kawasaki disease in the acute phase is associated with low thyroid hormone levels and TSH. It is possible that these hormone levels predict response to the initial IVIG
Paleo-health of Neolithic wet-rice farmers in the Yangtze River Delta : a comparison with early millet farmers in northern China
No full textSeveral cases that do not fit the agricultural adaptation model, in which people’s health deteriorated with the shift from hunting-gathering to farming, have been reported, such as the introduction of rice agriculture during the Yayoi Period in Japan and the Iron Age in Southeast Asia, where health was maintained or improved. However, the health of rice farmers in the homeland areas has rarely been reported. This study aims to clarify the frequency and degree of stress markers inscribed on Neolithic human bones in the Yangtze River Delta, one of the origins of rice agriculture, and to elucidate the state of adaptation when humans first engaged in rice agriculture. The materials of this study are the Early Neolithic site of Majiabang, and the Late Neolithic sites of Guangfulin and Jiangzhuang. Several millet farming groups in northern China were used as comparative materials. The results show that the Neolithic rice farming groups in the Yangtze River Delta generally had a higher frequency of stress markers than the millet farming groups in northern China. In particular, the Late Neolithic Guangfulin assemblage had higher frequencies, while the Early Neolithic Majiabang assemblage tended to have relatively low frequencies of porotic hyperostosis and cribra orbitalia. These results suggest that people’s health deteriorated in the Late Neolithic period, when the scale of paddy rice cultivation expanded, as a result of the new subsistence activities and rice-oriented diet