1,739,282 research outputs found
A study of the legal aspect of labour relations in Hong Kong: research report.
by Ying Wang-bun.Thesis (M.B.A.)--Chinese University of Hong Kong, 1981.Bibliography: leaf 59
Chinesische Impressionen : Huan Mou, Ying Wang ; 15. Oktober - 27. November 1999 ; eine Ausstellung in der Universitäts- und Landesbibliothek Düsseldorf
Motiv: zwei Bilder: eine lesende und eine laufende Figur in Fluren ("Der Zeitungsleser", 1998 von Huan Mou) - eine Figur mit Kamm und eine sitzende Figur mit langen Haaren (von Ying Wang
Two new genera of fossil palaeontinids from the Middle Jurassic in Daohugou, Inner Mongolia, China (Hemiptera, Palaeontinidae)
Ying, Wang, Dong, Ren (2007): Two new genera of fossil palaeontinids from the Middle Jurassic in Daohugou, Inner Mongolia, China (Hemiptera, Palaeontinidae). Zootaxa 1390: 41-49, DOI: 10.5281/zenodo.17523
FIGURE 1 D–F in Two new genera of fossil palaeontinids from the Middle Jurassic in Daohugou, Inner Mongolia, China (Hemiptera, Palaeontinidae)
FIGURE 1 D–F. Quadraticossus fangi gen. & sp. nov., holotype: D, body with wings; E, forewing; F, hind wing.Published as part of Ying, Wang & Dong, Ren, 2007, Two new genera of fossil palaeontinids from the Middle Jurassic in Daohugou, Inner Mongolia, China (Hemiptera, Palaeontinidae), pp. 41-49 in Zootaxa 1390 on page 45, DOI: 10.5281/zenodo.17523
FIGURE 2. A, B in Two new genera of fossil palaeontinids from the Middle Jurassic in Daohugou, Inner Mongolia, China (Hemiptera, Palaeontinidae)
FIGURE 2. A, B, Quadraticossus longicaulis gen. & sp. nov., holotype: A, photograph of part CNUHNN2006094; B, line drawing.Published as part of Ying, Wang & Dong, Ren, 2007, Two new genera of fossil palaeontinids from the Middle Jurassic in Daohugou, Inner Mongolia, China (Hemiptera, Palaeontinidae), pp. 41-49 in Zootaxa 1390 on page 46, DOI: 10.5281/zenodo.17523
Time-lapse helical X-ray computed tomography (CT) data of tensile fatigue damage in GFRP
The X-ray CT data here is published with the paper -
Wang, Y.; Mikkelsen, L.P.; Pyka, G.; Withers, P.J. Time-Lapse Helical X-ray Computed Tomography (CT) Study of Tensile Fatigue Damage Formation in Composites for Wind Turbine Blades. Materials 2018, 11, 2340.
https://doi.org/10.3390/ma11112340
More information about the data and the material can be found in the paper above.
If using the data here, please cite the above paper.
Contact details for author: Ying Wang, [email protected]</p
Erratum: High performance platinum single atom electrocatalyst for oxygen reduction reaction
Nature Communications 8: Article number: 15938 (2017); Published: 24 July 2017; Updated: 27 September 2017. The affiliation details for one of the corresponding authors, Ying Wang, are incorrect in this Article. This author is incorrectly affiliated with ‘University of Chinese Academy of Sciences, Beijing 100049, China.</jats:p
Neuroinflammation and its resolution in Alzheimer's disease
Alzheimer's disease (AD) is the most common dementia with high prevalence among an increasing aged population. Despite the existence of symptom-reliving drugs for AD, the clinical trials performed until now have failed to find drugs that cure or stop the progression of AD. New perspectives and strategies for treatments are therefore direly needed. Chronic inflammation as indicated by persistent activation of microglia and increased proinflammatory mediators is one of the major characteristics for AD, together with pathological accumulation of β-amyloid (Aβ), hyperphosphorylated tau proteins and neuronal loss. In normal physiological conditions, inflammation is ended by resolution, an active process associated with restoration and regeneration mediated by specialised pro-resolving lipid mediators (SPMs). Previous studies have shown that there are alterations in the resolution of inflammation in AD that can cause neurodegeneration by impairment in neuroprotective signalling, control of inflammation, and in the removal of the pathogenic Aβ peptide. The current studies focus on the impairment of pro-resolving mechanisms in the context of AD. The prospect of reducing harmful inflammation while at the same time increasing protective and pro-homeostatic activities present a promising strategy for treating AD.In Paper I and II, we focused on answering the fundamental question, whether and how the neuroinflammation (Paper I) and its resolution (Paper II) are altered in AD patients. We aimed to identify dissimilar inflammation-related protein mediators (Paper I) and SPMs (Paper II) profiles in the cerebrospinal fluid (CSF) of patients diagnosed with subjective cognitive impairment (SCI), mild cognitive impairment (MCI) or AD. We found an inflammatory pattern in the CSF that could differentiate SCI and AD. Comorbidities have an influence on the inflammatory pattern. SPMs were decreased in the CSF of AD patients and were associated with AD pathologies and cognition, suggesting that SPMs have potential to be novel biomarkers for AD. In Paper III and IV, the aim of the studies was to explore the pro-resolving role of maresin 1 (MaR1) in the context of Aβ42-induced inflammation in human microglial cell models. In Paper III, AD-like neuroinflammation was induced exposure to Ab42 monomers in both human monocyte-derived microglia (MdM) and a differentiated human monocyte cell line (THP-1 cells). We showed that one of the SPMs MaR1 reduced Aβ42-induced elevation in pro-inflammatory activation and stimulated the Aβ42 uptake. In Paper IV, RNA-Sequencing (RNA-Seq) was used to study the effects of MaR1 on the transcriptome of Aβ42-treated MdM to obtain a broader view regarding the pro-resolving roles of MaR1. We found that Aβ42 up-regulated inflammatory pathways and that co-incubation with MaR1 down-regulated some of these pathways. Proteomics confirmed the finding.In conclusion, the inflammation-related protein mediator profile and SPMs in CSF have a potential to contribute to the diagnosis of AD and are correlated to AD pathologies and cognition. SPM MaR1 attenuates AD-like neuroinflammation and supports the hypothesis that stimulating the resolution of inflammation could be a new therapeutic strategy in AD.List of scientific papersI. Ying Wang, Ceren Emre, Helena Gyllenhammar-Schill, Karin Fjellman, Helga Eyjolfsdottir, Maria Eriksdotter, Marianne Schultzberg, Erik Hjorth. Cerebrospinal fluid inflammatory markers in Alzheimer’s disease: influence of comorbidities. Curr Alzheimer Res. 2021. https://doi.org/10.2174/1567205018666210330162207 II. Khanh V. Do, Erik Hjorth, Ying Wang, Bokkyoo Jun, Marie-Audrey I. Kautzmann, Maria Eriksdotter, Marianne Schultzberg, Nicolas G. Bazan CSF profile of lipid mediators in Alzheimer’s disease. [Manuscript]III. Ying Wang, Axel Leppert, Shuai Tan, Bram van der Gaag, Nailin Li, Marianne Schultzberg, Erik Hjorth. Maresin 1 attenuates pro-inflammatory activation induced by b-amyloid and stimulates its uptake. J Cell Mol Med. 2021;25(1):434-447. https://doi.org/10.1111/jcmm.16098 IV. Ying Wang, Xiang Zhang, Henrik Biverstål, Nicolas G. Bazan, Shuai Tan, Xiaofei Li, Nailin Li, Marianne Schultzberg, Erik Hjorth. Pro-resolving lipid mediator reduces Ab42-induced gene expression in monocyte-derived microglia. [Manuscript]</p
Appendix_JTR_Revision_2 – Supplemental material for Do Food Image and Food Neophobia Affect Tourist Intention to Visit a Destination? The Case of Australia
Supplemental material, Appendix_JTR_Revision_2 for Do Food Image and Food Neophobia Affect Tourist Intention to Visit a Destination? The Case of Australia by Mun Yee Lai, Ying Wang and Catheryn Khoo-Lattimore in Journal of Travel Research</p
sj-doc-1-smo-10.1177_20503121221107126 – Supplemental material for Evidence and implication of interventions across various socioecological levels to address HIV testing uptake among men who have sex with men in the United States: A systematic review
Supplemental material, sj-doc-1-smo-10.1177_20503121221107126 for Evidence and implication of interventions across various socioecological levels to address HIV testing uptake among men who have sex with men in the United States: A systematic review by Ying Wang, Jason Mitchell and Yu Liu in SAGE Open Medicine</p
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