749 research outputs found
A reservation based parking lot system to maximize occupancy and revenue
No full textNumerous parking issues are faced by people on a day to day basis. Congestion is caused by customers circling the lot in search of vacant parking spots while the lot may be full. At peak periods the parking lot may not be filled to maximum capacity because there are only primitive ways to indicate to customers the availability of spots in the lot. Thus parking garages tend to lose profit as usage of parking lots is not maximized. The Parking Lot System proposed aims to reduce these parking hassles faced by people and the occupancy issues faced by parking lot owners by providing customers with a facility of reserving parking spots before arrival at the lot. This reduces the parking search traffic as well as the parking search time. Additionally, this system proposes to automate occupancy tracking in the lot which ensures that customers are aware of the availability of parking spaces from remote locations. In case of unavailability of parking spaces, customers are notified at the entrance thereby reducing congestion levels in the lot. The designed system is simulated in real world scenarios to test the occupancy levels achieved by the lot. The practices and strategies used in well developed reservation systems (hotels and aviation industries) like overbooking are modified and applied in this domain to improve occupancy levels. An increase of about 5.19% in the occupancy level is observed. A comparative study of the effect of various business strategies is carried out to determine the best mix of user controllable parameters. The parameters controllable by the parking lot owners are the length of the grace period provided to customers to arrive and claim their reservation, the number of overbooked reservations made by the system and the proportion of spots in the lot available for reservation. A further increase of up to 5.97% in the occupancy level is observed by using different combinations and values of these parameters. For the purpose of simulation, the parking garage is assumed to be located in a city downtown area having customers using it for both corporate as well as leisure purpose.M.S.Includes bibliographical referencesby Sonia Preeti Pint
Preeti Yadav et al. / International Journal on Computer Science and Engineering (IJCSE) Detection of Copy-Move Forgery of Images Using Discrete Wavelet Transform
No full textAbstract-Digital images are used everywhere and it is easy to manipulate and edit because of availability of various image processing and editing software. In a copy-move image forgery, a part of an image is copied and then pasted on a different location within the same image. A copy-move image forgery is done either for hiding some image object, or adding more details resulting in at least some part being cloned. In both the case, image reliability is lost. In this paper an improved algorithm based on Discrete Wavelet Transform (DWT) is used to detect such cloning forgery. In this technique at first DWT (Discrete Wavelet Transform) is applied to the input image for a reduced dimensional representation. Then the compressed image is divided into overlapping blocks. After that Lexicographic sorting is performed, and duplicated blocks are identified. Due to DWT usage, detection is first carried out on lowest level image representation. This approach increases accuracy of detection process and reduces the time needed for the detection process. Keywords- Copy-Move forgery, Digital Tempering, DWT, Cloning. I
Adult Education: Exploring the inequalities in the world’s most populated country
No full textThis month on Humanities Matter, we discuss the variation in literacy rates among India’s social strata, the importance of considering a gendered perspective in adult education policies within the country, and how such policies can empower marginalized communities and bring about social change.
All this and more with Dr. Preeti Dagar, author of “Subaltern Perspectives in Adult Education”, a chapter in the book Adult Education in India, Volume 35 in the series International Issues in Adult Education, published last year by Brill
“LIFE IS WHAT YOU MAKE IT” - CRITICAL ANALYSIS OF PREETI SHENOY’S
No full textAbstract
‘Life is what you make it‘ is a fiction written by Preeti Shenoy a blooming author. She has also written for different publications like Reader’s Digest and The Times of India, as well as taught English and Math to children in India. Her first book was ‘34 Buffer guns and Candies‘ a creative notification that made it to the national-best seller list. The book ‘Life is what you make it’ is the story of Ankita Sharma a girl in her twenties. It focuses attention on a little known disease, bipolar disease.
The plot is set in the 80s and moves across two cities. Being a bright student she got admitted into one of South India’s best colleges. What follows is a series of events, love affairs, pranks and studies that are instrumental for every college going student. But Ankita’s life soon turn-turvy when she is diagnosed with a mental illness. She develops Bipolar disorder
To study the linear and nonlinear optical properties of Se-Te-Bi-Sn/PVP (polyvinylpyrrolidone) nanocomposites
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Living Arrangements of Rural Elderly Women in Haryana, India
Get PDFIn this study we have investigating the living arrangements, decision making pattern, and position of elderly women in Haryana state. In spite of it we are also analyzing the dependence and dependents of elderly women after getting old age pension which is known as Old Age Samman Allowance (OASA) in Haryana and Indira Gandhi National Old Age Pension Scheme (IGNOAPS) at national level. The study was done in Haryana state of India and data was collected from 200 elderly women of age 62 years or above from four different village (50 women from each village) of Haryana. In results majority of elderly women were living with their family members and only 2 percent elderly women were living alone and dominance of male members either the spouse or son as head of the family was observed. Closest family member were their children either son or daughter. Though the elderly women were not head of the family, but majority of them had involvement in decision making related to household matters. Economic dependency of more than 35. 0 percent of the selected elderly women in spite of getting financial assistance of Rs. 2250 per month as OASA assistance raises the question of utilization of this assistance by the actual beneficiaries. Around half of the financially independent elderly women were supporting at least one person in their family. The elderly women either supported their son or grandson but not the spouse. In total old age samman allowance is very beneficial and much needed to the elderly as they become self-dependent and could live their life with full of dignity and according to their wishes
Substrate dependent linear and nonlinear optical properties of chalcogenide Se_82Te_15Bi_3.0 thin film
No full textDie Analyse des neuronalen Zytoskeletts und synaptischer Defekte im Mausmodel der Amyotrophen Lateralsklerose und der Spinalen Muskelatrophie
No full textAmyotrophic lateral sclerosis and spinal muscular atrophy are the two most common motoneuron diseases. Both are characterized by destabilization of axon terminals, axon degeneration and alterations in neuronal cytoskeleton. Accumulation of neurofilaments has been observed in several neurodegenerative diseases but the mechanisms how elevated neurofilament levels destabilize axons are unknown so far. Here, I show that increased neurofilament expression in motor nerves of pmn mutant mice causes disturbed microtubule dynamics. Depletion of neurofilament by Nefl knockout increases the number and regrowth of microtubules in pmn mutant motoneurons and restores axon elongation. This effect is mediated by interaction of neurofilament with the stathmin complex. Depletion of neurofilament increases stathmin-Stat3 interaction and stabilizes the microtubules. Consequently, the axonal maintenance is improved and the pmn mutant mice survive longer. We propose that this mechanism could also be relevant for other neurodegenerative diseases in which neurofilament accumulation is a prominent feature.
Next, using Smn-/-;SMN2 mouse as a model, the molecular mechanism behind synapse loss in SMA is studied. SMA is characterized by degeneration of lower α-motoneurons in spinal cord; however, how reduction of ubiquitously expressed SMN leads to MN-specific degeneration remains unclear. SMN is involved in pre-mRNA splicing (Pellizzoni, Kataoka et al. 1998) and its deficiency in SMA affects the splicing machinery. Neuromuscular junction denervation precedes neurodegeneration in SMA. However, there is no evidence of a link between aberrant splicing of transcripts downstream of Smn and reduced presynaptic axon excitability observed in SMA. In this study, we observed that expression and splicing of Nrxn2, that encodes a presynaptic protein is affected in the SMA mouse and that Nrxn2 could be a candidate that relates aberrant splicing to synaptic motoneuron defects in SMA.Die Amyotrophe Lateralsklerose und die spinale Muskelatrophie sind die beiden häufigsten Formen der Motoneuronerkrankungen. Sie sind charakterisiert durch eine Destabilisierung der Axonendigungen, durch Axondegeneration und durch Änderungen im neuronalen Zytoskelett. Eine Anhäufung von Neurofilamenten konnte in einigen neurodegenerativen Erkrankungen beobachtet werden. Der genaue Mechanismus, welcher zu einer Destabilisierung des Axons führt, ist bis heute jedoch unklar. Hiermit zeige ich, dass eine gesteigerte Expression von Neurofilamenten in motorischen Nerven von pmn mutierten Mäusen zu einer Störung der Mikrotubuli – Dynamik führt. Ein Neurofilamentabbau durch Nefl knockout steigert die Anzahl an neu wachsenden Mikrotubuli in pmn mutierten Motoneuronen und führt zu erneutem Axonwachstum. Dieser Effekt wird durch eine Interaktion zwischen dem Neurofilament und dem Stathmin Komplex vermittelt. Ein Abbau des Neurofilaments führt zu einer Erhöhung der Stathmin-Stat3 Interaktion und zu einer Stabilisierung der Mikrotubuli. Demzufolge ist die Versorgung der Axone verbessert und die pmn mutierten Mäuse überleben länger. Wir vermuten, dass dieser Mechanismus auch für andere neurodegenerative Erkrankungen, bei denen Neurofilamentanhäufung ein dominantes Merkmal ist, relevant sein könnte.
Des Weiteren studierte ich mit Hilfe des Smn-/-;SMN2 Mausmodels, den molekularen Mechanismus der sich hinter dem Synapsenverlust bei SMA verbirgt. SMA ist charakterisiert durch eine Degeneration der unteren -Motoneuronen im Rückenmark. Es ist jedoch unklar, wie ein Verlust des ubiquitär exprimierten SMN Proteins zu einer MN-spezifischen Degeneration führt. Smn ist involviert in den Prozess des pre-mRNA Splicing (Pellizzoni, Kataoka et al. 1998) und ein Verlust des Proteins führt zu einer Störung des Splicing. Eine Denervierung der motorischen Endplatte führt zu einer Neurodegeneration in SMA. Es gibt jedoch keinen Hinweis auf eine kausale Verbindung zwischen anomalem Splicen von stromabwärts gelegenen Transkripten des Smn und einer Reduktion präsynaptischer Axone, wie man es bei SMA beobachten kann. In dieser Studie konnten wir beobachten, dass Expression und Splicing von Nrxn2, welches für ein präsynaptisches Protein kodiert, in SMA Mäusen betroffen ist und dass Nrxn2 ein Kandidat sein könnte, der eine Verbindung zwischen Störungen im Splice Prozess und synaptischen Motoneuron-Defekten in der SMA herstellen könnte
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