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A Comparative Study on the Yaobian Theory of Cheong Yakyong and Wu Cheng
No full text북허남오(北許南吳)로 대표되는 원대의 대학자 오징(吳澄, 1249-1333)은 조선조의 사상에 많은 영향을 끼쳤고, 조선 역학사(易學史)에 있어서도 그 영향이 적다고 할 수 없다. 현전하는 우리나라 最古의 주역 관련 저작으로 평가받고 있는 조선 초의 학자 권근이 지은 을 보면, 정이천의 , 주희의 와 함께 오징의 의 영향을 쉽게 찾아볼 수 있다. 조선 후기의 대학자 다산 정약용(丁若鏞, 1762-1836)도 역학 관련 저서에서 오징의 견해를 많이 채택하고 있다. 정약용의 대표적인 역학 저작인 을 살펴보더라도 때략 9곳에서 오징의 견해를 인용하고 있어, 오징의 해석에 대한 다산의 평가가 어떠한지 미루어 짐작할 수 있다.The purpose of this paper is to elucidate the origin of Yaobian(爻變) theory of Cheong Yakyong(丁若鏞). for this purpose, this paper tried to make a comparative study of Cheong Yakyong and Wu Cheng(吳澄). Wu Cheng(吳澄) is a great Confucian Scholar in the Yuan Dynasty. He wrote Yizuanyan(易纂言), which is a his representative work about Yi Studies. In Yisuanyan(易纂言), Wu Cheng classified methods of interpretation of Zhou yi into 12 methods as follows. 1. Guatong(卦統), 2. Guadui(卦對), 3. Guabian(卦變), 4. Guazhu(卦主), 5. Biangua(變卦), 6. Hugua(互卦), 7. Xiangli(象例), 8. Zhanli(占例), 9. Cili(辭例), 10. Bianli(變例), 11. Yiyuan(易原), 12. Yiliu(易流). In these 12 methods, Biangua(變卦) is a theory of Yaobian. According to this viewpoint, we know that Wu Cheng use Yaobian theory in the interpretation of Zhou Yi
Dao de jing zhu: [4 juan].
No full text吳澄撰 ; [伍崇曜輯]Date from preface.框13.1 x 9.1 cm., 9行21字, 黑口, 左右雙邊, 無魚尾, 版心中鐫分冊書名, 下鐫叢書名.Wu Cheng zhuan ; [Wu Chongyao ji]Kuang 13.1 x 9.1 cm., 9 xing 21 zi, hei kou, zuo you shuang bian, wu yu wei, ban xin zhong juan fen ce shu ming, xia juan cong shu ming
Induction of Non-Targeted Stress Responses in Mammary Tissues by Heavy Ions
No full textPurpose: Side effects related to radiation exposures are based primarily on the assumption that the detrimental effects of radiation occur in directly irradiated cells. However, several studies have reported over the years of radiation-induced non-targeted/ abscopal effects in vivo that challenge this paradigm. There is evidence that Cyclooxygenase-2 (COX2) plays an important role in modulating non-targeted effects, including DNA damages in vitro and mutagenesis in vivo. While most reports on radiation-induced non-targeted response utilize x-rays, there is little information available for heavy ions.
Methods and Materials: Adult female transgenic gpt delta mice were exposed to an equitoxic dose of either carbon or argon particles using the Heavy Ion Medical Accelerator in Chiba (HIMAC) at the National Institute of Radiological Sciences (NIRS) in Japan. The mice were stratified into 4 groups of 5 animals each: Control; animals irradiated under full shielding (Sham-irradiated); animals receiving whole body irradiation (WBIR); and animals receiving partial body irradiation (PBIR) to the lower abdomen with a 1 x 1 cm2 field. The doses used in the carbon ion group (4.5 Gy) and in argon particle group (1.5 Gy) have a relative biological effectiveness equivalent to a 5 Gy dose of x-rays. 24 hours after irradiation, breast tissues in and out of the irradiated field were harvested for analysis. Induction of COX2, 8-hydroxydeoxyguanosine (8-OHdG), phosphorylated histone H2AX (γ-H2AX), and apoptosis-related cysteine protease-3 (Caspase-3) antibodies were examined in the four categories of breast tissues using immunohistochemical techniques. Analysis was performed by measuring the intensity of more than 20 individual microscopic fields and comparing the relative fold difference.
Results: In the carbon ion group, the relative fold increase in COX2 expression was 1.01 in sham-irradiated group (p > 0.05), 3.07 in PBIR (p 0.05), 11.31 in PBIR (p 0.05), 8.41 in PBIR (p < 0.05) and 10.59 in WBIR (p < 0.05). Results for the argon particle therapy group showed a similar magnitude of changes in the various biological endpoints examined.
There was no statistical significance observed in Caspase-3 expression among the 4 groups.
Conclusions: Our data show that both carbon and argon ions induced non-targeted, out of field induction of COX2 and DNA damages in breast tissues. These effects may pose new challenges to evaluate the risks associated with radiation exposure and understanding radiation-induced side effects
Flexible Resource Allocation in Complex Processing Networks with Reliability Considerations
No full textInduction of Non-Targeted Stress Responses in Mammary Tissues by Heavy Ions
No full textPurpose: Side effects related to radiation exposures are based primarily on the assumption that the detrimental effects of radiation occur in directly irradiated cells. However, several studies have reported over the years of radiation-induced non-targeted/ abscopal effects in vivo that challenge this paradigm. There is evidence that Cyclooxygenase-2 (COX2) plays an important role in modulating non-targeted effects, including DNA damages in vitro and mutagenesis in vivo. While most reports on radiation-induced non-targeted response utilize x-rays, there is little information available for heavy ions.
Methods and Materials: Adult female transgenic gpt delta mice were exposed to an equitoxic dose of either carbon or argon particles using the Heavy Ion Medical Accelerator in Chiba (HIMAC) at the National Institute of Radiological Sciences (NIRS) in Japan. The mice were stratified into 4 groups of 5 animals each: Control; animals irradiated under full shielding (Sham-irradiated); animals receiving whole body irradiation (WBIR); and animals receiving partial body irradiation (PBIR) to the lower abdomen with a 1 x 1 cm2 field. The doses used in the carbon ion group (4.5 Gy) and in argon particle group (1.5 Gy) have a relative biological effectiveness equivalent to a 5 Gy dose of x-rays. 24 hours after irradiation, breast tissues in and out of the irradiated field were harvested for analysis. Induction of COX2, 8-hydroxydeoxyguanosine (8-OHdG), phosphorylated histone H2AX (γ-H2AX), and apoptosis-related cysteine protease-3 (Caspase-3) antibodies were examined in the four categories of breast tissues using immunohistochemical techniques. Analysis was performed by measuring the intensity of more than 20 individual microscopic fields and comparing the relative fold difference.
Results: In the carbon ion group, the relative fold increase in COX2 expression was 1.01 in sham-irradiated group (p > 0.05), 3.07 in PBIR (p 0.05), 11.31 in PBIR (p 0.05), 8.41 in PBIR (p < 0.05) and 10.59 in WBIR (p < 0.05). Results for the argon particle therapy group showed a similar magnitude of changes in the various biological endpoints examined.
There was no statistical significance observed in Caspase-3 expression among the 4 groups.
Conclusions: Our data show that both carbon and argon ions induced non-targeted, out of field induction of COX2 and DNA damages in breast tissues. These effects may pose new challenges to evaluate the risks associated with radiation exposure and understanding radiation-induced side effects
[Tai shang wu ji da dao zi ran zhen yi wu cheng fu shang jing太 上 無 極 大 道 自 然 眞 一 五 稱 符 上 經] commentaires du Xian gong 仙公.
No full textLing bao zhen yi wu chengjing 靈 寶 眞 一 五 稱 經, cf. Tai shang wu ji da dao zi ran zhen yi wu cheng fu shang jing.Tai shang wu ji da daozi ran zhen yi wu cheng fu shang jing 太 上 無 極 大 道 自 然 眞 一 五 稱 符 上經Numérisation effectuée à partir d'un document original.Déb. manque. Sans division en j.Quelques variantes par rapport à Dao 671, j.shang, fasc. 352, 潔 一, ff. 2 b 1-14 b 1 et j.xia, fasc. 352, 潔 二, ff. 1 a 2-14 b 7. Titrefinal : Ling bao zhen yi wu cheng jing juan 靈 寶 眞一 五 稱 經 卷. Cf. TKDM ,pp. 1-2. Écr. élégante, de style ancien, car. légèrement inclinés àgauche. Encre foncée. 1 car. omis ajouté entre les col. 3 et 4 de la f. 2.Quelques car. effacés et récrits, plusieurs car. minuscules inscrits à droite des car. fautifs. 28 col. par f., 16 ou 17 car. par col., texte interrompu par 5 groupes de car. talismaniques (ff. 1 à 6). Commentaires en petits car. sur col. simples ou dédoublées. Marges sup. 2,7 à 3,5 cm, inf. 3,4 à 4,4 cm.Réglure
Two new species of the genus Gieysztoria (Rhabdocoela: Dalyelliidae) from China
No full textZhang, Hang, Li, Yi-Kui, Wu, Cheng-Chen, Wang, An-Tai (2014): Two new species of the genus Gieysztoria (Rhabdocoela: Dalyelliidae) from China. Zoological Systematics 39 (4): 485-495, DOI: 10.11865/zs.2014040
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